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1 rves) and a diabetic neuropathy model (i.e., streptozotocin-induced diabetes).
2 co-induced in livers of mice in response to streptozotocin induced diabetes.
3 g were evaluated in C57BL/6J adult mice with streptozotocin-induced diabetes.
4 is markedly reduced soon after the onset of streptozotocin-induced diabetes.
5 pose tissue LPL expression in male rats with streptozotocin-induced diabetes.
6 ed in coronary venules at the early onset of streptozotocin-induced diabetes.
7 yte stasis (leukostasis) in the rat model of streptozotocin-induced diabetes.
8 nal brush border membrane (BBM) of mice with streptozotocin-induced diabetes.
9 ancreatic protein using acini from rats with streptozotocin-induced diabetes.
10 oglycemia for more than 80 days in mice with streptozotocin-induced diabetes.
11 iferative islet cells and with resistance to streptozotocin-induced diabetes.
12 cible human Nox5 transgenic mouse exposed to streptozotocin-induced diabetes.
13 hyperglycemia and ex vivo, in a rat model of streptozotocin-induced diabetes.
14 iminished opioid responsiveness in rats with streptozotocin-induced diabetes.
15 in situ and by glycemic control in rats with streptozotocin-induced diabetes.
16 c pancreata were transplanted into mice with streptozotocin-induced diabetes.
17 lar leakage in both early and late stages of streptozotocin-induced diabetes.
18 ects, and granulation tissues from mice with streptozotocin-induced diabetes.
19 so observed in liver extracts from mice with streptozotocin-induced diabetes.
20 eability (RVP) and hemodynamics in rats with streptozotocin-induced diabetes.
21 ture healing in a model of multiple low-dose streptozotocin-induced diabetes.
22 a more severe hyperglycemia associated with streptozotocin-induced diabetes.
23 duced obesity and insulin resistance or with streptozotocin-induced diabetes.
24 ibitor apocynin in models of endotoxemia and streptozotocin-induced diabetes.
25 he epididymal fat pad of syngeneic mice with streptozotocin-induced diabetes.
26 Moreover, HcB-19 mice are resistant to streptozotocin-induced diabetes.
27 x1) to ameliorate hyperglycemia in mice with streptozotocin-induced diabetes.
28 N loss also protects animals from developing streptozotocin-induced diabetes.
29 in whole retinas after 2, 4, and 8 weeks of streptozotocin-induced diabetes.
30 Pten mutant mice are resistant to developing streptozotocin-induced diabetes.
31 rated in vivo function and were able to cure streptozotocin-induced diabetes.
32 f these NF-kappaB subunits were resistant to streptozotocin-induced diabetes.
33 evel is increased in the livers of rats with streptozotocin-induced diabetes.
34 SCHAD knockout (SCHADKO) mice into mice with streptozotocin-induced diabetes.
35 observed in rats with a 10-week duration of streptozotocin-induced diabetes.
36 creased HbA(1c) levels in the mouse model of streptozotocin-induced diabetes.
37 in transgenic mice, which were resistant to streptozotocin-induced diabetes.
38 etinal microvascular cells after 5 months of streptozotocin-induced diabetes.
39 ponsible for the hyperfiltration observed in streptozotocin-induced diabetes; (2) a combined alterati
40 assessed whether CNTF protects mice against streptozotocin-induced diabetes (a model of type 1 diabe
42 ective of this study is to determine whether streptozotocin-induced diabetes activates cell death pat
44 e of 7 nontreated, nontransplanted mice with streptozotocin-induced diabetes and 7 normal mice), demo
45 vessels isolated from rats with 6 months of streptozotocin-induced diabetes and compared it with tha
47 rsensitive to collagen-induced arthritis and streptozotocin-induced diabetes and develop heightened a
48 tions were prepared from retina of rats with streptozotocin-induced diabetes and from the isolated re
50 diabetes, examining the kidneys of rats with streptozotocin-induced diabetes and kidney cells exposed
52 the pathogenesis of murine multiple low-dose streptozotocin-induced diabetes and support the concept
53 in dorsal root ganglion neurons in rats with streptozotocin-induced diabetes and tactile allodynia, u
54 ively expressed in beta cells prevented both streptozotocin-induced diabetes and the metabolic defici
55 transplanted into immunodeficient mice with streptozotocin-induced diabetes, and glycemia was initia
56 -reperfusion between control rats, rats with streptozotocin-induced diabetes, and rats with hyperglyc
57 leukocyte 12-LO in mice ameliorates low dose streptozotocin-induced diabetes by increasing islet resi
58 In conclusion, CNTF protects mice against streptozotocin-induced diabetes by increasing pancreatic
62 alogue, was markedly attenuated in rats with streptozotocin-induced diabetes, consistent with a profo
63 % (P < 0.01) in apoE knockout (KO) mice with streptozotocin-induced diabetes consuming an atherogenic
64 GR5 dual agonist INT-767 to DBA/2J mice with streptozotocin-induced diabetes, db/db mice with type 2
66 48 only (B48 mice) to examine the effects of streptozotocin-induced diabetes (DM) on apoB-100- and ap
70 e unchanged during chronic food restriction, streptozotocin-induced diabetes, hypoglycemia when food
71 e retinas of rats with 2 months' duration of streptozotocin-induced diabetes, IGF-I mRNA levels were
72 asting, increased by insulin, and reduced by streptozotocin-induced diabetes, implicating FGFR4 as a
73 cose levels in two mouse models of IDDM: (1) streptozotocin-induced diabetes in C57BL/6J mice on a re
74 volved in leukostasis in the early stages of streptozotocin-induced diabetes in mice by direct observ
75 ype I diabetes, we studied multiple low-dose streptozotocin-induced diabetes in mice deficient in eit
80 e ability of 6-month cultured islets to cure streptozotocin-induced diabetes in this mouse model.
81 , we assessed the consequence of 4 months of streptozotocin-induced diabetes in wild type (+/+) and C
83 onsecutive days in an in vivo mouse model of streptozotocin-induced diabetes inhibited the loss of ti
84 ss GFP driven by the mouse insulin promoter, streptozotocin-induced diabetes led to the appearance of
85 poE(-/-)) mice (n = 22), including mice with streptozotocin-induced diabetes mellitus (n = 8), nondia
90 fasted mice, from rats with cancer cachexia, streptozotocin-induced diabetes mellitus, uremia induced
95 e mouse models of insulin action deficiency: streptozotocin-induced diabetes, ob/ob genotype and live
96 lar tissues of normal rats, and in rats with streptozotocin-induced diabetes of 6 and 12 weeks' durat
98 present study was to determine the effect of streptozotocin-induced diabetes on BBM glucose transport
100 the corneal clock, we studied the effects of streptozotocin-induced diabetes on the mitosis of epithe
104 were able to protect recipient mice against streptozotocin-induced diabetes, restoring a physiologic
106 egrees C, degrees C into 42 B6AF/1 mice with streptozotocin-induced diabetes resulted in a return to
108 c BB rats, we have found that 8-10 months of streptozotocin-induced diabetes results in fewer dystrop
109 ning porcine islets for 17 days in rats with streptozotocin-induced diabetes results in normalization
112 p53inp2 exhibited enhanced muscle wasting in streptozotocin-induced diabetes that was dependent on au
116 of six pairs of cynomolgus monkeys (CM) with streptozotocin-induced diabetes was closely matched for
117 traperitoneal rPdx1 injection into mice with streptozotocin-induced diabetes, we assessed its action
119 in D (CypD)-deficient mice (Ppif (-/-)) with streptozotocin-induced diabetes, we observed an increase
120 n neurons and the GABAB)receptor function in streptozotocin-induced diabetes, we performed whole-cell
123 tic RAGE apoE double-knockout (KO) mice with streptozotocin-induced diabetes were treated with the AG
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