ライフサイエンスコーパス: stress

1 e against herbivores, pathogens, and abiotic stress.

2 spected to be also regulatable by mechanical stress.

3 orphological changes as a result of cellular stress.

4 and UTP molecules released following injury/stress.

5 ng MSNs of mice susceptible to social defeat stress.

6 n Mycobacterium smegmatis under hypochlorite stress.

7 the molecular responses of PTPs to oxidative stress.

8 ct and store proteins for immediate use post-stress.

9 ial dysfunction that is induced by oxidative stress.

10 ed during heat stress and recovery from heat stress.

11 y students would improve their resilience to stress.

12 o hydrogen peroxide (H2O2)-induced oxidative stress.

13 gy and lipid metabolism as well as oxidative stress.

14 el significantly repressed in mice under the stress.

15 nd behavioral responses to acute and chronic stress.

16 deployed, particularly in situations of high stress.

17 nesis and defense against biotic and abiotic stress.

18 against weight loss associated with chronic stress.

19 of BHB on rats exposed to acute and chronic stress.

20 apoptosis under genotoxic and hematopoietic stress.

21 undance in Daojiao for a time course of cold stress.

22 ntermittently at stresses above the critical stress.

23 protect cells against hypoxia and oxidative stress.

24 othelial cells suppresses miR-204-induced ER stress.

25 vivo, and protects osteocytes from oxidative stress.

26 of lateral root (LR) formation under osmotic stress.

27 aggregates under the condition of oxidative stress.

28 ironmental impacts known to induce oxidative stress.

29 rectly controls the sensitivity to curvature stress.

30 er protein damage originating from oxidative stress.

31 ctivity and were hypersensitive to oxidative stress.

32 o assist in their maintenance upon injury or stress.

33 osed to cytokines or thapsigargin-induced ER stress.

34 lity and apoptotic cell death with oxidative stress.

35 d expression patterns indicative of cellular stress.

36 ER) homeostasis create a condition termed ER stress.

37 ptation for survival rather than escape from stress.

38 ich appears relatively impervious to drought stress.

39 against hydrogen peroxide-induced oxidative stress.

40 ndividual yeast cells, both before and after stress.

41 miRNA-mediated down-regulation under drought stress.

42 ant-specific responses to abiotic and biotic stresses.

43 tion in response to physiological mechanical stresses.

44 neously and cause damages that exceed single stresses.

45 control wheat plants under drought and salt stresses.

46 0.79); concurrent validity (r with perceived stress=0.55, r with sleep quality=0.39) and predictive v

47 to their molecular resilience to nutritional stress, a characteristic that is relevant to organismal

48 ll a:b ratio with leaf age and physiological stress, a further separation of total plant-based chloro

49 creased stress that appear intermittently at stresses above the critical stress.

50 Many cellular stresses activate senescence, a persistent hyporeplicati

51 of the MAPK pathway results in mitogen- and stress-activated protein kinase 1/2 (MSK1/2)-catalyzed p

52 Since oxidative stress activates protein kinase D1 (PKD1) in tumor cells

53 l functions, including response to oxidative stress, addictive behaviour, and regulatory functions em

54 nslation of select mRNAs that participate in stress alleviation.

55 Our results demonstrate how stress alters CA1 circuit function through the impairmen

56 n IEC lines, we found that induction of cell stress alters the cytoskeleton in IECs via changes in th

57 r quality of life for patients; 2) surrogate stress and anxiety; 3) optimistic health expectations; 4

58 development of tunicamycin-induced renal ER stress and apoptosis.

59 otein (ZIKV-C) was associated with ribosomal stress and apoptosis.

60 volumetric reductions: apoptosis, oxidative stress and autophagy.

61 ationship between endoplasmic reticulum (ER) stress and cGVHD, and aimed to create effective treatmen

62 the cooperative induction of DNA replication stress and damage by ATR inhibition and cytarabine, and

63 rove life-quality is misleading as oxidative stress and exacerbation occur when oxidant foods (e.g. f

64 rehalose pretreatment can sensitise cells to stress and impair survival.

65 4, will minimize the negative impact of heat stress and increase global food productivity, benefiting

66 elation to biomarkers of oxidative/nitrative stress and inflammation and to explore whether changes i

67 allows assessment of mitochondrial oxidative stress and mitophagy in vivo, and were preceded by incre

68 and a substrate for enzymes signaling energy stress and oxidative stress response, nicotinamide adeni

69 c enzyme pyruvate kinase under environmental stress and propose a method to protect and store protein

70 nd quantify proteins synthesized during heat stress and recovery from heat stress.

71 sites are associated with enhanced oxidative stress and reduced endothelial NO production is a furthe

72 nism of transcription regulation on cellular stress and reveal functional similarities to the mammali

73 s a pivotal trait governing root penetration stress and root elongation rate in soils of greater stre

74 tter was associated with increased oxidative stress and significant ultrastructural impairment of mit

75 multiple bend locations under high torsional stress and that the positions of these sharp bends are d

76 ression and the ability to control oxidative stress and the phenotypic severity of SCD.

77 easing frequency in the coming century - can stress and ultimately kill native coldwater fish in lake

78 nal axis responses to both acute and chronic stress and were protected against weight loss associated

79 umor suppressor responds to various cellular stresses and regulates cell fate.

80 that osmotic stress induces transient energy stress, and AMPK activation allows cells to manage this

81 changing conditions and to control oxidative stress, and its dysfunction can lead to hypoxia-dependen

82 titis, mitochondrial function, autophagy, ER stress, and lipid metabolism were measured in pancreatic

83 Fatty liver, oxidative stress, and mitochondrial dysfunction are key pathophysi

84 b/db mice developed hyperglycemia, oxidative stress, and nephropathy at age 20 weeks compared with th

85 ation and timing of drivers of physiological stress, and the extent of novel climates.

86 by constant exposure to exogenous oxidative stress, and this upregulation is CLOCK-dependent.

87 rcade configurations that concentrated shear stresses, and (3) repetitive, localized biting.

88 pulsation-which increases waist longitudinal stresses-and transverse rupture.

89 how plants may respond to, and perhaps sense stresses; and how organisms with a similar sensitive cyt

90 plore whether changes in oxidative/nitrative stress are a function of prenatal exposure to NP/BPA and

91 published and new data, we propose oxidative stress as a common pathological mechanism leading to PVI

92 f the injured nociceptors revealed oxidative stress as a key biological process.

93 g were also observed, suggesting replication stress as a root causative factor in CHKi hypersensitivi

94 tunicamycin-induced vascular/endothelial ER stress, associated impairment of endothelium-dependent v

95 ecular and cellular determinants in mPFC for stress-associated mental disorders.

96 Our data indicate that stress-associated suicide risk is elevated in carriers o

97 The states of stress at the studied wellbore is in the normal faulting

98 etabolite formation in APAP-induced chemical stress, both the hepatotoxicity and localised Nrf2-luc r

99 bserved mitochondrial fission during osmotic stress, but blocking fission did not affect AMPK activat

100 response (UPR) to endoplasmic reticulum (ER) stress by Mvarphis in a longitudinal study of fish-deriv

101 demonstrated higher antifouling and tensile stress (by 31%) when compared to pure PES membranes.

102 Psychosocial stress can also modify pollutant effects.

103 Whereas severe stress is detrimental, mild stress can be beneficial for health and survival, known

104 Ribosome profiling in ER-stressed cells lacking these factors revealed that Ire1-

105 t from nitrogen starvation and other abiotic stresses commonly used to induce oil accumulation in alg

106 qH operates under stress conditions such as cold and high light and is pho

107 g us to evaluate the function of GRIKs under stress conditions.

108 This study reveals that appropriate water stress could increase capsaicinoid yield in some, but no

109 limate projections, we show that future heat stress could reduce the forest edge growth enhancement b

110 chondrial function and endoplasmic reticulum stress, could have contributed to the neutral trial resu

111 Can viewing others experiencing stress create a "contagious" physiological stress respon

112 Overall, our results indicate that stress cross-over occurs in animal populations and may h

113 te how distinct signalling mechanisms direct stress-dependent versus homeostatic regeneration, and hi

114 In these stresses, different kinases phosphorylate eukaryotic ini

115 ssembly and facilitates their premature post-stress disassembly.

116 sponse to traumatic stress on post-traumatic stress disorder (PTSD), this study examined longitudinal

117 (CRFR2) to be associated with post-traumatic stress disorder (PTSD)-like symptoms.

118 IFICANCE STATEMENT Anxiety and posttraumatic stress disorder patients generalize fear to nonfearful f

119 al aetiological mechanism for post-traumatic stress disorder.

120 tral sensory hyperactivity in post-traumatic stress disorder.

121 Here, we describe mGluR5 findings in stress disorders, particularly major depressive disorder

122 Genotoxic stress drives damaged DNA out of the nucleus by forming

123 e, we test for the first time how early-life stress drives developmental programming and transgenerat

124 tein, which promoted cellular survival under stress due to downregulation of the E3 ligase RNF144A.

125 ted intraindividual variability in perceived stress during a 4-month follow-up period.

126 so indicates an association between maternal stress during pregnancy and TL in the offspring.

127 vestigating sweetpotato response to chilling stress during storage.

128 iated beta-galactosidase activity, oxidative stress, early phosphorylation of mitogen-activated prote

129 ting, symptom questionnaires, and dobutamine stress echocardiography.

130 bles cells to cope more effectively with the stress elicited by oncogenic Ras protein.

131 Stress elicits neuroendocrine, autonomic, and behavioral

132 We found that efficient stress erythropoiesis in vivo requires E2F-2, and we als

133 locations in the materials where the thermal stress exceeds the yield stress undergo plastic deformat

134 AX, JNK signaling, and endoplasmic reticulum stress, explaining why SRp55 depletion triggers beta-cel

135 city, induced elongation, and promoted actin stress fiber organization.

136 Incorporation of NM-II into actin stress fiber provides a traction force to promote actin

137 uniaxial cyclic stretch results in an actin stress fiber reinforcement response that stabilizes the

138 We identify zyxin as a regulator of stress fibre mechanics, as stress fibres are fluid-like

139 Furthermore, stress fibres and intermediate filaments modulate the me

140 as a regulator of stress fibre mechanics, as stress fibres are fluid-like without flow reversal in it

141 stable AJs and redistributed to radial actin stress fibres of remodelling focal AJs.

142 y, normal laminar and elevated laminar shear stress for a period of 72 hours.

143 ctivation allows cells to manage this energy stress for proliferation in new osmotic states.

144 the spatiotemporal distribution of cellular stresses from measured traction forces in motile tissues

145 ics, promotes normal induction of protective stress genes, and rescues stress sensitivity of TDP-43-e

146 personal violence, pre- and early postpartum stress, gestational age at birth, infant sex, and postna

147 SFP1 is induced in stressed glucose-grown cells, whereas RTG3 is upregulate

148 show that oxygen vacancies can move under a stress-gradient-induced depolarisation field.

149 pport of critical slowing down along natural stress gradients in tidal marsh ecosystems.

150 Upon DNA damage, p53 mRNA is released from stress granules and associates with polyribosomes to inc

151 toplasmic ribonucleoprotein granules such as stress granules and those seeded by the aggregation of s

152 mRNA accumulation in stress granules correlates with longer coding and UTR re

153 rodegeneration and promotes the formation of stress granules.

154 nelles involved in RNA metabolism, including stress granules.

155 Hypoxic stress has a strong impact on tumor cell biology.

156 commonly used paradigm-chronic social defeat stress-has proven challenging to implement in females.

157 avior otherwise facilitated by developmental stress hormone exposure.

158 ions, addition of methyl jasmonate, a biotic stress hormone, induced expression in all leaf tissues.

159 In the absence of oxidative stress, HPbetaCD addition induces a paradoxical response

160 fication of ventricular volume and function, stress imaging, shunt quantification, and tissue charact

161 However, few studies have investigated how stress impact humans raised in an overprotective manner.

162 combat-related and non-combat related mental stress, impaired sympathetic and cardiovagal baroreflex

163 use it has the ability to withstand numerous stresses imposed by host immunity.

164 tein-RNA interactions within 1 min following stress imposition.

165 hways that induce oxidative, thiol and metal stress in bacteria could be a useful strategy to design

166 which is a documented consequence of chronic stress in chickens.

167 itivity responding to experimentally-induced stress in desiccated mosses, indicating that spectral im

168 diating the vasoconstrictor response to cold stress in hypertension.

169 As predicted, preadolescent stress in mice resulted in a significant blunting of the

170 metabolism, energy production and oxidative stress in others.

171 (c/c) colonocytes showed increased metabolic stress in response to DSS with higher levels of phospho-

172 on, exhibit elevated PLIN2 expression and ER stress in their beta cells.

173 dy of the roles of replication and oxidative stresses in mediating cellular senescence in cancer cell

174 t measurements of spatially resolved surface stresses in the continuous thickening regime, we report

175 Cellular stress, including oxidative stress, results in increased

176 r of repeats varies under different types of stress indicating that natural variation in repeat numbe

177 potential therapeutic approach for treating stress-induced behavioral disorders, and that dynamics a

178 2), an epigenetic regulator, is critical for stress-induced cardiac hypertrophy.

179 ction is required for ETI and containment of stress-induced cell death in Arabidopsis.

180 Stress-induced changes in target cell PAg levels are spe

181 s also been shown to be prophylactic against stress-induced depressive-like behavior in mice.

182 females may be more sensitive than males to stress-induced drug seeking.

183 Our results show that osmotic stress induces transient energy stress, and AMPK activat

184 of the Pap1-dependent genes still relied on stress induction, another subset of stress-responsive ge

185 and protected against UVB-induced oxidative stress, inflammation and papillomagenesis.

186 pipeline for the genetic studies of abiotic stress iron deficiency chlorosis (IDC) of soybean is rep

187 Endoplasmic reticulum stress is an evolutionarily conserved cell stress respon

188 SV-1 reproduction triggered by physiological stress is characteristic of reactivation of lifelong lat

189 Whereas severe stress is detrimental, mild stress can be beneficial for

190 nse to a stressor.Animals' response to acute stress is known to be influenced by sex and genetics.

191 ing correlates elicited by early-life social stress is lacking.

192 n transporter, SLC6A4, coupled with prenatal stress is reported to increase the risk for children to

193 astrocytes, including proteasome inhibition, stress kinase activation, mechanistic target of rapamyci

194 ion of the autophagy substrate p62/SQSTM1 in stressed Kras(G12D) acinar cells is associated with PDAC

195 -grown cells, whereas RTG3 is upregulated in stressed lactate-grown cells.

196 and ALG-2 levels are detected along with ER stress markers and associated caspases in transgenic bra

197 le tissues and show that such traction-based stresses match those calculated from instantaneous cell

198 Such form of cross-over of stress may be beneficial if it enhances adaptive respons

199 It has been suggested that prenatal maternal stress may increase the risk of childhood externalizing

200 During stress, men recruit regions associated with emotion and

201 ic adaptation of the hematopoietic system in stress myelopoiesis.

202 ufficient to increase movement in the TST in stress-naive mice, while stimulating above the carrier f

203 vestigated the causal influence of two major stress neuromodulators, cortisol and noradrenaline, on l

204 subsequently function better than similarly stressed neurons that did not produce exophers.

205 Proteotoxically stressed neurons that generate exophers subsequently fun

206 We report that the environmental stress of carbon dioxide (CO2) anesthesia converts an as

207 for interventions but because of suboptimal stress of the 2/4 protocol in some patients.

208 warming temperatures and increased moisture stress on plants.

209 pact of the epigenetic response to traumatic stress on post-traumatic stress disorder (PTSD), this st

210 the significant effect of hemodynamic shear stress on RBC-induced microvascular injury.

211 l effects of maternal exposure to early-life stress on several phenotypic traits in their offspring i

212 defence against endogenous and environmental stresses on the other hand.

213 dynamically altering gene expression during stress or disease.

214 Response to stress or external threats is a key factor in mood and a

215 Restraint stress or optogenetic C1 neuron (C1) stimulation (10 min

216 antiviral response through activating the ER stress pathway during viral infection.

217 explain with existing models that emphasize stress pathways and accelerated limbic system developmen

218 It also activates ER stress pathways that promote acinar cell death.

219 mortality reduce canopy leaf area during the stress period and for a lagged recovery window thereafte

220 Thermal tensile stress perpendicular to the laser scanning direction is

221 pting chemicals induce endoplasmic reticulum stress, perturb NF-kappaB, and p53 signaling, and dimini

222 and signaling in roots of flooded and water stressed plants of Carrizo citrange revealed that the ho

223 Therefore, ER stress poses a promising target in colorectal cancers, w

224 Furthermore, changes in IL-6 following stress predicted intraindividual variability in perceive

225 Furthermore, we find that acute stress preferentially activates neuronal subpopulations

226 The mechanisms by which chronic stress promote the development of pancreatic ductal aden

227 Finally, in an oxidative stress-prone background, Pml(-/-) animals display a long

228 a FAD-dependent, two-domain multifunctional stress protein acting as a Phase II enzyme, activating c

229 in rather than in the compressional tectonic stress regime as in the periphery of the Tarim Basin, wh

230 e studied wellbore is in the normal faulting stress regime within the Tarim Basin rather than in the

231 recruit regions associated with emotion and stress regulation, self-referential processing and cogni

232 The oxidative stress regulator Spx is ubiquitously found among Gram-po

233 witch of susceptibility versus resilience to stress-related disorders.

234 Investigation of several stress-related parameters in SeCspA and SeCspB transgeni

235 t in human fibroblasts rendered senescent by stress, replicative exhaustion, or oncogene activation,

236 ospho-mimicking Atf1 mutant display enhanced stress resistance, and although expression of the Pap1-d

237 are both selectively enhanced by integrated stress response (ISR) activation.

238 s, whose break-down products are involved in stress response against herbivores, pathogens, and abiot

239 in situ temperatures, possessing a suite of stress response and nutrient cycling genes to fix carbon

240 m stress is an evolutionarily conserved cell stress response associated with numerous diseases, inclu

241 FANCD2 also functions during the replication stress response by mediating the restart of temporarily

242 actor eIF2alpha, enabling the translation of stress response genes; among these is GADD34, the protei

243 g stress create a "contagious" physiological stress response in the observer?

244 mycin complex 1 function, and hyperammonemic stress response including autophagy markers normally fou

245 r inappropriate activation of the integrated stress response may contribute to pathogenesis in a subs

246 all, we identify miR-500a-5p as an oxidative stress response miRNA whose activity may define breast c

247 stabilizes a complex between 14-3-3 and the stress response regulator GCN1, inducing GCN1 turnover a

248 re, we report that the fungal nitrooxidative stress response suppresses host defences to facilitate t

249 IF2alpha kinases function in the integrative stress response to inhibit general protein synthesis coi

250 mature cytoplasmic tRNAs are cleaved during stress response to produce tRNA fragments that function

251 determine the role of gamma2-AMPK in cardiac stress response using bioengineered cell lines and mouse

252 ose involved in the metabolism of sugars and stress response were highlighted.

253 le pathways to control metabolism, oxidative stress response, and cell cycle.

254 ranscription rate during the early stages of stress response, indicating orthogonal transcriptional c

255 nzymes signaling energy stress and oxidative stress response, nicotinamide adenine dinucleotide (NAD(

256 hypothalamic circuit coordinating the global stress response.

257 osphate (G3P) is important for environmental stress responses by eukaryotic microalgae.

258 Thus two distinct proteotoxic stress responses control phospholipid metabolism.

259 n by the MAPK Sty1 is required for oxidative stress responses in fission yeast cells by promoting tra

260 C2 signaling pathways to coordinate cellular stress responses with sterol homeostasis.

261 nd expression of genes involved in oxidative stress responses, tumor progression and chemoresistance.

262 hanism to optimize the balance of growth and stress responses.

263 nt of their involvement in the regulation of stress responses.

264 ine alanine-rich kinase) and OSR1 (oxidative stress responsive kinase), which then phosphorylate and

265 ously known as CIKS or Act1) is an oxidative stress-responsive cytoplasmic adapter molecule that is a

266 elied on stress induction, another subset of stress-responsive genes was constitutively expressed in

267 ion of SeCsp could enhance the expression of stress-responsive genes.

268 reases year-round in the absence of moisture stress resulting in net CO2 uptake increases in the shou

269 Cellular stress, including oxidative stress, results in increased O-GlcNAcylation of numerous

270 vation, but deletion of the unfolded protein stress-sensing luminal domain of the UPR transducers PER

271 with nuclear translocation of the lysosomal stress-sensing transcription factor EB and, eventually,

272 tion of protective stress genes, and rescues stress sensitivity of TDP-43-expressing animals.

273 well with the differential regulation of ER stress sensors, in particular Perk.

274 into dormancy after exposure to inflammatory stress stimuli.

275 dependent mechanism in response to different stresses, subsequently maintaining hydrogen peroxide (H2

276 ations between MACL and markers of oxidative stress such as urinary methionine sulfoxide were observe

277 nals as a means of adapting to environmental stresses such as those caused by hypoxia, ischemia, and

278 Neisseria gonorrhoeae senses and responds to stress; such responses may be modulated by MisRS (NGO017

279 In rodents, many stress susceptibility and resilience studies have focuse

280 The remainder were Stress Test Originated Phantom Perception (STOPP) subjec

281 risk scores with an echocardiogram, exercise stress test, computerized tomographic coronary angiogram

282 nary angiography and revascularization after stress testing were ascertained.

283 ing (exercise electrocardiography or nuclear stress testing) from 2009 to 2015.

284 localized regions of substantially increased stress that appear intermittently at stresses above the

285 d compromised proliferation, and replication stress that could be rescued with an antioxidant.

286 olecular damage may be incurred by oxidative stress that is imparted by high iron status in early lif

287 a cardiac phenotype and response to ischemic stress that is similar to wild-type aged hearts (i.e., i

288 ng test procedures is an important source of stress that may impair reliability of test responses.

289 fidence with chloroplast HSP22E/F under heat stress thus revealing chloroplast processes affected by

290 nt protein deacetylase, senses environmental stress to alter intestinal integrity.

291 rated rats were subjected to water avoidance stress (to induce visceral hypersensitivity), then given

292 t lines indicated that these lines possessed stress tolerance characteristics, including lower malond

293 s where the thermal stress exceeds the yield stress undergo plastic deformation mediated by GND activ

294 armacologically and high-fat diet-induced ER stress using Zip14(-/-) (KO) mice, which exhibit impaire

295 ct with endothelia under physiological shear stress, using recently developed live cell imaging and p

296 Under field conditions, these stresses usually occur simultaneously and cause damages

297 on of endothelial Sirt1 in mice, promotes ER stress via upregulation of miR-204, whereas overexpressi

298 -driven effects can be classified into point stresses, where a temperature event during a sensitive s

299 These include oxidative stresses, which are present throughout the respiratory t

300 Cells adjust to hypoxic stress within the tumor microenvironment by downregulati