ライフサイエンスコーパス: stress-activated protein kinase

1 n oxidative stress independently of the Spc1 stress-activated protein kinase.

2 elta, or on c-Jun N-terminal kinase, another stress-activated protein kinase.

3 nt both on mitochondrial respiration and p38 stress-activated protein kinase.

4 PK), but not c-Jun N-terminal protein kinase/stress-activated protein kinase.

5 2alpha) on serine 51 is effected by specific stress-activated protein kinases.

6 gnaling was mediated by the p38 subfamily of stress-activated protein kinases.

7 feration and phosphorylation of mitogen- and stress-activated protein kinases.

8 8gamma in particular, and also p38alpha, two stress-activated protein kinases.

9 zipper transcription factor and a target for stress-activated protein kinases.

10 rtery cells is mediated by activation of the stress-activated protein kinases.

11 ly, protein kinase A (PKA), the mitogen- and stress-activated protein kinase 1 (MSK1) and the AMP-act

12 this study, we examined whether mitogen- and stress-activated protein kinase 1 (MSK1) is a downstream

13 Mitogen- and stress-activated protein kinase 1 (MSK1) is a growth-fac

14 rminal "kinase-dead" mutants of mitogen- and stress-activated protein kinase 1 (MSK1), a downstream k

15 dy, we investigated the role of mitogen- and stress-activated protein kinase 1 (MSK1), a nuclear kina

16 ATF1 can be phosphorylated by mitogen- and stress-activated protein kinase 1 (MSK1), which is activ

17 Here, mitogen- and stress-activated protein kinase 1 (MSK1), which itself i

18 in kinase (MAPK) activation and mitogen- and stress-activated protein kinase 1 (MSK1)-ranges of intra

19 and activated by UVB-activated mitogen- and stress-activated protein kinase 1 (Msk1).

20 els and also those of activated mitogen- and stress-activated protein kinase 1 (MSK1).

21 osphorylates the nuclear kinase mitogen- and stress-activated protein kinase 1 (MSK1).

22 thways is the activation of the mitogen- and stress-activated protein kinase 1 (MSK1).

23 ells was not due to a defect in mitogen- and stress-activated protein kinase 1 or 90-kDa ribosomal S6

24 N- or C-terminal mutant mitogen- and stress-activated protein kinase 1 or its inhibitor H89 h

25 argets (ribosomal S6 kinase and mitogen- and stress-activated protein kinase 1) within the ventral te

26 0 ribosomal S6 kinase 2 but not mitogen- and stress-activated protein kinase 1.

27 ischemic phosphorylation of c-Jun N-terminal stress-activated protein kinase 1/2 (JNK1/2), p38, mitog

28 of the MAPK pathway results in mitogen- and stress-activated protein kinase 1/2 (MSK1/2)-catalyzed p

29 aB kinase-alpha (IKK-alpha) and Mitogen- and Stress-activated protein Kinases 1 and 2 (MSK1/2) have b

30 her, CREB1 was activated by the mitogen- and stress-activated protein kinases 1 and 2 (MSK1/2).

31 s shown to be mediated by RSK2, mitogen- and stress-activated protein kinase-1 (MSK1), and mitogen-ac

32 ibition of protein kinase A and mitogen- and stress-activated protein kinase-1 activity.

33 kinase, ribosomal S6 kinase-2, mitogen- and stress-activated protein kinase-1, p38 MAPK, phosphatidy

34 , alpha-PDGFR, but not beta-PDGFR, activates stress-activated protein kinase-1/c-Jun NH(2)-terminal k

35 ediated transformation through activation of stress-activated protein kinase-1/c-Jun NH2-terminal kin

36 ges triggered the activation of mitogen- and stress-activated protein kinases-1 and -2 (MSK1, MSK2) a

37 report that the MAPK family member known as stress-activated protein kinase-1c (SAPK1c, also known a

38 e (ERK) and the apoptotic/growth suppressive stress-activated protein kinase 2 (p38(MAPK)), and that

39 on formation and activation of FAK, Src, and stress-activated protein kinase 2, p38, were dysregulate

40 1/2 and p38 mitogen-activated protein kinase/stress-activated protein kinase 2.

41 d, tempo, but not tempol, potently activated stress-activated protein kinase (2 h, >3-fold).

42 protein kinases c-jun N-terminal kinase and stress-activated protein kinase-2.

43 y blocking the CA-4-P-mediated activation of stress-activated protein kinase-2/p38.

44 (71% over control) posttreatment, preceding stress-activated protein kinase activation and apoptosis

45 oxy-2-nonenal, have been reported to mediate stress-activated protein kinase activation and cell toxi

46 ne dithiocarbamate, markedly attenuated both stress-activated protein kinase activation and the induc

47 of the CD40ct is critical for NF-kappaB and stress-activated protein kinase activation, as well as t

48 Signals which upregulate Jun kinase (JNK)/stress-activated protein kinase activity also lead to as

49 oth LMPCD40 and LMP1 are now shown to induce stress-activated protein kinase activity in the absence

50 lysis of tobacco (Nicotiana tabacum) Osmotic Stress-Activated Protein Kinase activity in tobacco Brig

51 Rapid activation of p38, p44/42, stress-activated protein kinase and c-Jun N-terminal kin

52 associated verotoxins (VTs), VT1 and VT2, on stress-activated protein kinase and nuclear factor kappa

53 PK) superfamily, c-Jun amino-terminal kinase/stress-activated protein kinase and p38 MAPK.

54 ptosis are both accompanied by activation of stress-activated protein kinase and p38 mitogen-activate

55 nd activation of the c-Jun N-terminal kinase/stress-activated protein kinase and p38 pathways.

56 pha on the phosphorylation of JNK1/2 and p38 stress-activated protein kinase and their downstream tra

57 nd JNK/SAPK (c-Jun N-terminal protein kinase/stress-activated protein kinase)], and AKT.

58 CD40 ligation was known to activate the stress-activated protein kinase, and both LMPCD40 and LM

59 n of the protein kinases JNK1/2, ERK1/2, p38 stress-activated protein kinase, and mitogen-activated p

60 APKs: extracellular signal-regulated kinase, stress-activated protein kinase, and p38), protein phosp

61 on, or F-actin disruption, activated the JNK stress-activated protein kinase, and that interfering wi

62 -activated protein kinase 2 and mitogen- and stress-activated protein kinase are activated in psoriat

63 that Raf-1 interacts with the proapoptotic, stress-activated protein kinase ASK1 (apoptosis signal-r

64 role for Tpx1 in the activation of the Sty1 stress-activated protein kinase by peroxide, we find tha

65 ion of mitogen-activated protein kinases and stress-activated protein kinases by TNF and IL-1, sugges

66 The stress-activated protein kinase c-Jun N-terminal kinase

67 ligation, R-848 stimulated activation of the stress-activated protein kinases c-Jun kinase and p38 an

68 ts displayed higher levels of phosphorylated stress-activated protein kinase (c-jun N-terminal Kinase

69 used activation of c-Jun NH2-terminal kinase/stress-activated protein kinase, c-jun gene induction, a

70 The stress-activated protein kinase, c-Jun N-terminal kinase

71 Another stress-activated protein kinase, c-Jun N-terminal kinase

72 , was transiently phosphorylated and another stress-activated protein kinase, c-Jun NH(2)-terminal ki

73 In this study the role of the stress-activated protein kinases, c-Jun N-terminal kinas

74 ular signal-regulated kinases (ERK), and the stress-activated protein kinases, c-Jun NH2-terminal kin

75 Hypoxia (1% O2) stimulated strongly the stress-activated protein kinases, c-Jun NH2-terminal kin

76 xtracellular signal-regulated kinase 1/2 and stress activated protein kinase/c-jun N-terminal kinase,

77 he extracellular signal-regulated kinase and stress-activated protein kinase/c-Jun amino-terminal kin

78 r transcription factors NF-kappa B and AP-1, stress-activated protein kinase/c-Jun amino-terminal kin

79 tracellular signal-regulated kinase 1/2, and stress-activated protein kinase/c-Jun N-terminal kinase

80 ich specifically associated with decrease in stress-activated protein kinase/c-Jun N-terminal kinase

81 llular signal-regulated kinase 1/2, p38, and stress-activated protein kinase/c-Jun N-terminal kinase

82 maging agents and regulates induction of the stress-activated protein kinase/c-Jun N-terminal kinase

83 otein kinase (MAPK), but not p44/42 (ERK) or stress-activated protein kinase/c-Jun N-terminal kinase

84 Phosphorylated stress-activated protein kinase/c-Jun N-terminal kinase

85 ase-1 (MEKK-1), an upstream activator of the stress-activated protein kinase/c-Jun N-terminal kinase

86 induced apoptosis was evaluated by measuring stress-activated protein kinase/c-jun N-terminal kinase

87 thout effect on the related kinase SAPK/JNK (stress-activated protein kinase/c-Jun N-terminal protein

88 The stress-activated protein kinase/c-Jun N-terminal protein

89 pable of activating c-jun expression include stress-activated protein kinase/c-Jun N-terminal protein

90 inhibition of PI3 kinase with wortmannin or stress-activated protein kinase/c-Jun NH(2)-terminal kin

91 However, TNF activation of p38 or stress-activated protein kinase/c-Jun NH(2)-terminal kin

92 nhibitors wortmannin and LY294002 stimulated stress-activated protein kinase/c-Jun NH(2)-terminal kin

93 and RANKL markedly potentiate NF-kappaB and stress-activated protein kinase/c-Jun NH(2)-terminal kin

94 and dominant negative mutants involving both stress-activated protein kinase/c-Jun NH(2)-terminal kin

95 ar regulated kinase 1 and 2 (ERK1/ERK2), and stress-activated protein kinase/c-Jun NH(2)-terminal kin

96 signal-regulated kinases 1/2, p38 MAPK, and stress-activated protein kinase/c-Jun NH(2)-terminal kin

97 gen-activated protein kinase, phosphorylated stress-activated protein kinase/c-Jun NH(2)-terminal kin

98 urative PDT increased phosphorylated p38 and stress-activated protein kinase/c-Jun NH(2)-terminal kin

99 llular signal-regulated kinase, p38 MAPK, or stress-activated protein kinase/c-Jun NH2-terminal kinas

100 kinase kinase kinases in pathways leading to stress-activated protein kinase/c-Jun NH2-terminal kinas

101 c stress response involves activation of the stress-activated protein kinase/c-Jun NH2-terminal kinas

102 ellular signal-regulated protein kinase 1/2, stress-activated protein kinase/c-JUN NH2-terminal kinas

103 signal-regulated kinase (ERK), p38 MAPK, and stress-activated protein kinase/c-Jun-N terminal kinase)

104 lar signal-regulated kinase 1 and 2, p38 and stress-activated protein kinase/c-Jun-NH(2)-kinase were

105 ited the phosphorylation of Akt, Erk1/2, and stress-activated protein kinase/c-jun-NH(2)-kinase, with

106 EK) > SAPK/c-Jun NH(2)-terminal kinase (JNK) stress-activated protein kinase cascade and coordinately

107 ling via the JNK (c-Jun NH2-terminal kinase)/stress-activated protein kinase cascade to stimulate or

108 iated factor 2 (TRAF2) and activation of the stress-activated protein kinase cascade.

109 and of the JNK/SAPK (c-Jun N-terminal kinase/stress-activated protein kinase) cascade through inhibit

110 tor protein connection to IkappaB kinase and stress-activated protein kinases (CIKS)/Act1.

111 onse to HO-induced oxidative stress in a p38 stress-activated protein kinase-dependent manner.

112 previously reported substrate for DSP18, the stress-activated protein kinase, does not localize to mi

113 optosis, including Ceramide and c-Jun Kinase/Stress-activated Protein Kinase'; Dr Barbara Hoffman (Fe

114 NO activated the extracellular-regulated and stress-activated protein kinases ERK, c-Jun NH(2)-termin

115 and Raf mutants but not by dominant-negative stress-activated protein kinase/ERK kinase and mitogen-a

116 ity and demonstrated an inhibitory effect on stress-activated protein kinase/ERK kinase-1 (SEK1); oth

117 nd -p90(RSK) and activation of p34(cdc2) and stress-activated protein kinase/ERK kinase/c-Jun N-termi

118 detected in the normal embryonic lens, this stress-activated protein kinase exhibited a robust activ

119 Both stress-activated protein kinase/extracellular signal-reg

120 tant of JNK kinase 2 and RNA interference of stress-activated protein kinase/extracellular signal-reg

121 Concomitantly, Akt1 interacts with stress-activated protein kinase/extracellular signal-reg

122 vity; enhanced phosphorylation of SEK1/MKK4 (stress-activated protein kinase/extracellular signal-rel

123 d protein kinase, NH(2)-terminal Jun kinases/stress-activated protein kinases, hexosamines, and other

124 We conclude that inhibitors of stress-activated protein kinases improve the outcome of

125 .4 is the closest homolog of tobacco Osmotic Stress-Activated Protein Kinase in Arabidopsis (Arabidop

126 ation of hepatic c-Jun NH(2)-terminal kinase/stress-activated protein kinase in Cd39/Entpd1 mice afte

127 regulating the c-Jun N-terminal kinase (JNK)/stress-activated protein kinase in cultured neurons.

128 ltistep phosphorelay system to the Spc1/Sty1 stress-activated protein kinase in the fission yeast Sch

129 n of c-Jun N-terminal protein kinases (JNKs) stress-activated protein kinases in mast cells.

130 The function of PKN, a stress-activated protein kinase, in the heart is poorly

131 Activation of stress-activated protein kinases, including the p38 and

132 increased the protein but not mRNA level of stress-activated-protein kinase interacting protein 1 (S

133 p38 is an important stress activated protein kinase involved in gene regulat

134 The c-Jun N-terminal kinase (JNK)/stress activated protein kinase is preferentially activa

135 The stress-activated protein kinase JNK plays an important r

136 Activation of stress-activated protein kinases JNK (and, to a lesser e

137 C3 toxin stimulated the stress-activated protein kinases JNK and p38 and potenti

138 slation and the coordinate activation of the stress-activated protein kinases JNK and p38 MAPK.

139 Inhibition of c-Jun N-terminal kinase/stress-activated protein kinase (JNK) activity with the

140 in activation of the c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) and ERK1/2 MA

141 lated kinase Erk1/2, c-Jun-N-terminal kinase/stress-activated protein kinase (JNK/SAPK) and p38 MAPK,

142 ctivation of the c-Jun NH(2)-terminal kinase/stress-activated protein kinase (JNK/SAPK) and p38(MAPK)

143 monstrated to induce c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) cascade, whil

144 has been reported to activate the JUN kinase/stress-activated protein kinase (JNK/SAPK) pathway in mo

145 The c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) pathway is ac

146 duction of the Jun N-terminal protein kinase/stress-activated protein kinase (JNK/SAPK) pathway was s

147 APK) and c-Jun NH(2)-terminal protein kinase-stress-activated protein kinase (JNK/SAPK) pathways upon

148 l also activates the c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) signal transd

149 ors, and activates the Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) signaling pat

150 chicine activate the c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) signaling pat

151 hrough activation of c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK), a signaling

152 nduced activation of c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK), and TRAF2 ca

153 s p38 MAP kinase and c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK).

154 ich binds to the c-Jun-NH(2)-terminal kinase/stress-activated protein kinases (JNK/SAPK).

155 genes c-jun and c-fos and activation of the stress-activated protein kinases, JNK/SAPK and p38, in t

156 Chemical inhibition of stress-activated protein kinase/JNK showed that this MAP

157 in the pathway leading to c-Jun activation [stress-activated protein kinase/Jun N-terminal kinase (J

158 ated the phosphorylation of P38, P44/42, and stress-activated protein kinase/Jun N-terminal kinase (S

159 Analysis of the stress-activated protein kinase/Jun N-terminal kinase (S

160 Stress-activated protein kinase kinase (SEK1), an upstre

161 n was blocked by transfections with either a stress-activated protein kinase kinase dominant-negative

162 JNK/stress-activated protein kinase kinase inhibition abroga

163 A for the dominant-negative mutant JNK-KR or stress-activated protein kinase kinase-1 Lys-->Arg mutan

164 (UV-B) triggers the activation of a group of stress-activated protein kinases known as c-Jun NH(2)-te

165 SK1 from 14-3-3zeta, and ASK1 then activates stress-activated protein kinases, leading to cell death.

166 o explants blocked activation of the p38 and stress-activated protein kinase MAPK and nuclear factor-

167 Hypoxia-reoxygenation activated the p38 and stress-activated protein kinase mitogen-activated protei

168 f c-Jun amino-terminal kinase (also known as stress-activated protein kinase), mitogen-activated prot

169 However, p38-stress-activated protein kinase-MK2 activation did not a

170 d human TTP function in both resting and p38-stress-activated protein kinase-MK2-activated cells.

171 netic approaches, we identified mitogen- and stress-activated protein kinases (MSKs) as primary media

172 s studies have established that mitogen- and stress-activated protein kinases (MSKs) regulate IL-10 p

173 tivation of c-Jun N-terminal kinase (JNK), a stress-activated protein kinase, on stress stimulation.

174 RGS7 by TNF-alpha requires activation of the stress-activated protein kinase p38 and the presence of

175 ygen species and mediates stimulation of the stress-activated protein kinase p38 MAPK downstream of t

176 The stress-activated protein kinase p38 plays a central role

177 activity by genotoxic stress is mediated by stress-activated protein kinase p38.

178 h increasing p62-dependent activation of the stress-activated protein kinase p38.

179 MKP5 dephosphorylates/inactivates the stress-activated protein kinase p38.

180 Here we report that stress-activated protein kinases p38 and JNK trans-activ

181 The inhibitor of the 38-kDa stress-activated protein kinase (p38(mapk)), SB 203580,

182 ulated kinase (ERK), c-Jun N-terminal kinase/stress-activated protein kinase, p38, and Big MAP kinase

183 nner, but was sensitive to inhibition of the stress-activated protein kinase, p38.

184 sponse to insult occurs by signaling via the stress-activated protein kinases, p38, and c-Jun NH(2)-t

185 mitogen-activated protein kinase cascade and stress-activated protein kinase/p38, respectively, but i

186 The compound BIRB796 inhibits the stress-activated protein kinases p38alpha and p38beta an

187 e of immature MoDCs to LPS activates the p38 stress-activated protein kinase (p38SAPK), extracellular

188 2mapk/erk2, 2) p46 c-Jun NH2-terminal kinase/stress-activated protein kinase (p46 JNK/SAPK), and 3) p

189 ear Pmk1 by MAPK phosphatases induced by the stress activated protein kinase pathway is important for

190 lammation that include the activation of the stress-activated protein kinase pathway that, together w

191 by modulation of the c-Jun N-terminal kinase/stress-activated protein kinase pathway.

192 eceptors control the c-Jun N-terminal kinase/stress-activated protein kinase pathway.

193 ctivate the c-Jun N-terminal kinase (JNK, or stress-activated protein kinase) pathway.

194 GTPases, phosphatidylinositol 3-kinase, and stress-activated protein kinase pathways are not involve

195 tracellular K(+) and subsequent induction of stress-activated protein kinase pathways.

196 has been implicated in the activation of the stress-activated protein kinase pathways.

197 eIF2alpha) by the endoplasmic reticulum (ER) stress-activated protein kinase PERK modulates protein s

198 ibitors tested, only the inhibitor of JNK, a stress-activated protein kinase, potently blocked E. col

199 s 2/3, p38 mitogen-activated protein kinase, stress-activated protein kinase, protein kinase B, extra

200 d resulted in strong activation of eIF2alpha stress-activated protein kinase R-like endoplasmic retic

201 o study the mechanisms by which mitogen- and stress-activated protein kinases regulate cell cycle re-

202 The Hog1 stress-activated protein kinase regulates both stress re

203 ith Shiga toxin, synergistically upregulates stress-activated protein kinases, resulting in superindu

204 ukemia (CML), activates Ras and triggers the stress-activated protein kinase (SAPK or Jun NH2-termina

205 kinase kinases and an authentic substrate of stress-activated protein kinase (SAPK) 2a/p38.

206 hort-lived phosphorylation and activation of stress-activated protein kinase (SAPK) and cellular diff

207 Interestingly, DMS treatment induced the p38 stress-activated protein kinase (SAPK) and expression of

208 The stress-activated protein kinase (SAPK) and mitogen-activ

209 n kinases, c-Jun amino-terminal kinase (JNK)/stress-activated protein kinase (SAPK) and p38 mitogen-a

210 after treatment, and significantly activated stress-activated protein kinase (SAPK) and p38 mitogen-a

211 is accompanied by the activation of both the stress-activated protein kinase (SAPK) and p38 mitogen-a

212 al activators, c-Jun N-terminal kinase (JNK)/stress-activated protein kinase (SAPK) and p38 mitogen-a

213 ate DNA damage responses mediated by the p38/stress-activated protein kinase (SAPK) axis of signaling

214 We characterized participation of the stress-activated protein kinase (SAPK) cascade in the le

215 ytes by activating an upstream kinase in the stress-activated protein kinase (SAPK) cascade.

216 We tested if mitogen- and stress-activated protein kinase (SAPK) cascades are invo

217 Lyn is required in part for induction of the stress-activated protein kinase (SAPK) in the response t

218 se C (PKC) betaII-mediated activation of the stress-activated protein kinase (SAPK) pathway.

219 Stress-activated protein kinase (SAPK) pathways are evol

220 rs, the sphingomyelin and c-Jun Kinase (JNK)/Stress-activated Protein Kinase (SAPK) pathways have bee

221 minal kinase and p38 pathways, also known as stress-activated protein kinase (SAPK) pathways, are sig

222 Therefore, not surprisingly stress-activated protein kinase (SAPK) pathways, pathway

223 In contrast, c-jun-NH2-terminal kinase (JNK)/stress-activated protein kinase (SAPK) phosphorylation w

224 In eukaryotes, activation of JNK/p38 stress-activated protein kinase (SAPK) signaling pathway

225 restingly, SB203580, an inhibitor of the p38 stress-activated protein kinase (SAPK), and overexpressi

226 environmental stress stimuli is also called stress-activated protein kinase (SAPK), which has crucia

227 external hyperosmolarity activates the Hog1 stress-activated protein kinase (SAPK), which is a key p

228 alone had no effect on expression of phospho-stress-activated protein kinase (SAPK), wild-type p53, o

229 ignal-regulated kinase (ERK) kinase (MEKK) > stress-activated protein kinase (SAPK)-ERK kinase (SEK)

230 well as c-Jun amino-terminal kinase 1 (JNK1)/stress-activated protein kinase (SAPK).

231 of the c-Abl protein tyrosine kinase and the stress-activated protein kinase (SAPK).

232 or DLK activity and subsequent activation of stress-activated protein kinase (SAPK).

233 yeloma (MM) is associated with activation of stress-activated protein kinase (SAPK).

234 y a mechanism dependent on activation of the stress-activated protein kinase (SAPK).

235 tatin 1 was associated with the induction of stress-activated protein kinase (SAPK).

236 knockdowns reduce TNF-mediated activation of stress-activated protein kinase (SAPK).

237 h RhoA-mediated actin remodeling and the p38 stress-activated protein kinase (SAPK).

238 sponse kinase (ERK), p-38 kinase (p-38), and stress-activated protein kinase (SAPK).

239 increased baseline levels of phosphorylated stress-activated protein kinase (SAPK)/c-jun NH(2)-termi

240 Here we describe the rapid activation of the stress-activated protein kinase (SAPK)/JNK pathway in BA

241 Chemotaxis toward ETs did not involve p38 or stress-activated protein kinase (SAPK)/Jun N-terminal ki

242 8 mitogen-activated protein (MAP) kinase and stress-activated protein kinase (SAPK)/Jun N-terminal ki

243 ceptor 2 (TNFR2), and the signaling proteins stress-activated protein kinase (SAPK)/Jun NH(2)-termina

244 )1/2, p38, and c-Jun N-terminal kinase (JNK)/stress-activated protein kinase (SAPK)] as well as serin

245 rotein kinase domain and that stimulates the stress-activated protein kinase (SAPK, also referred to

246 The stress-activated protein kinase (SAPK, alternatively JNK

247 Activation of the stress-activated protein kinase (SAPK/JNK) by genotoxic

248 lta is required in part for induction of the stress-activated protein kinase (SAPK/JNK) in cells trea

249 C-terminus, has a specific activation of the stress-activated protein kinase (SAPK/JNK) pathway.

250 estly increased expression/activation of the stress-activated protein kinase (SAPK/JNK); moreover, SA

251 th the nuclear factor kappaB (NF-kappaB) and stress-activated protein kinase (SAPK; also known as c-J

252 In response to extracellular stimuli, stress-activated protein kinases (SAPK) modulate gene ex

253 es the N-terminal phosphorylation of ATF2 by stress-activated protein kinases (SAPK).

254 have identified a new Dictyostelium kinase (stress-activated protein kinase [SAPK]alpha), which is r

255 includes c-jun NH2-terminal kinase (JNK) or stress-activated protein kinase (SAPK1) and extracellula

256 ase cascades leading to enhanced activity of stress activated protein kinases (SAPKs), including JNK

257 TNF-induced activation of stress activated protein kinases (SAPKs, Jun NH2-termina

258 ases (GCKs) and the subsequent activation of stress-activated protein kinases (SAPKs and c-Jun NH(2)-

259 Stress-activated protein kinases (SAPKs) are known to re

260 Stress-activated protein kinases (SAPKs) are stimulated

261 Regulation of gene expression by stress-activated protein kinases (SAPKs) is essential fo

262 The objective was to determine whether stress-activated protein kinases (SAPKs) mediated the tr

263 SR is known to involve the activation of the stress-activated protein kinases (SAPKs) p38 and JNK.

264 implex virus type 1 (HSV-1) can activate the stress-activated protein kinases (SAPKs) p38 and JNK.

265 Importantly, the stress-activated protein kinases (SAPKs) were not signif

266 t of the immediate upstream activator of the stress-activated protein kinases (SAPKs), abrogates the

267 c-Jun N-terminal kinases (JNKs), also called stress-activated protein kinases (SAPKs), belong to the

268 inase (JNK) activity, which involves JNKs or stress-activated protein kinases (SAPKs), is dependent o

269 In eukaryotic species from yeast to human, stress-activated protein kinases (SAPKs), members of a M

270 Stress-activated protein kinases (SAPKs), members of a m

271 fluorescence, resulting in the activation of stress-activated protein kinases (SAPKs), p38 MAPK, and

272 The relationship between PKR and the stress-activated protein kinases (SAPKs), such as p38 mi

273 nt on stress-induced nuclear accumulation of stress-activated protein kinases (SAPKs).

274 ed in 293 cells, MKK7 specifically activated stress-activated protein kinases (SAPKs)/c-Jun N-termina

275 Induction of gene 33 requires the stress-activated protein kinases (SAPKs)/c-Jun NH(2)-ter

276 The stress-activated protein kinases (SAPKs, also called c-J

277 whereas that of AP-1/ATF can be mediated by stress-activated protein kinases (SAPKs; also named Jun

278 in kinase (MAPK) ERK-2, but did activate the stress-activated protein kinases (SAPKs; c-jun NH2-termi

279 r levels of hydroperoxide, and activates the stress-activated protein kinase (SEK) pathway.

280 tein kinase kinase 4 (MKK4), a member of the stress-activated protein kinase signaling cascade, has b

281 est that c-Jun and c-Jun NH2-terminal kinase/stress-activated protein kinase signaling may be involve

282 ively regulating the c-Jun N-terminal kinase/stress-activated protein kinase signaling pathway during

283 The effects of oxalate on mitogen- and stress-activated protein kinase signaling pathways were

284 t by the NF-kappaB and Jun N-terminal kinase/stress-activated protein kinase signaling pathways, two

285 attern of activation and deactivation of the stress-activated protein kinase signalling molecules c-J

286 Here we show that the fission yeast stress-activated protein kinase Sty1, a homolog of mamma

287 quired both Cdr1 autophosphorylation and the stress-activated protein kinase Sty1.

288 Other stress-activated protein kinases, such as ERK and p38, p

289 Jun N-terminal kinase (JNK) is a stress-activated protein kinase that can be induced by i

290 t PIKK family member, hSMG-1, as a genotoxic stress-activated protein kinase that displays some funct

291 AMP-activated protein kinase (AMPK) is a stress-activated protein kinase that is regulated by hyp

292 JNK is a stress-activated protein kinase that modulates pathways

293 Indeed, if JNK is considered as a stress-activated protein kinase, there appear to be mult

294 racellular-signal regulated kinases-1/-2 and stress-activated protein kinases, these enzymes did not

295 8 mitogen-activated protein (MAP) kinase nor stress-activated protein kinase was activated by [Ca(2+)

296 Activation of p38 stress-activated protein kinase was required for LPS- or

297 Phosphorylation of the stress-activated protein kinases was increased in both i

298 ctivity of the proapoptotic signal mediator, stress-activated protein kinase, was increased severalfo

299 nal kinase (JNK1) is a member of a family of stress-activated protein kinases which are activated by

300 DEP chemicals also induced the activation of stress-activated protein kinases, which play a role in c