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1 ceptive afferents to prevent recovery of the stretch reflex.
2 -reflex, the electrical analog of the spinal stretch reflex.
3 a strong peripheral amplifier of the spinal stretch reflex.
4 jor contribution to the phase advance of the stretch reflex.
5 SPs in motoneurons, and the disappearance of stretch reflexes.
6 e input favours relatively tonic and diffuse stretch reflexes.
7 limb position sensation (proprioception) and stretch reflexes.
8 to influence the gain of artificially evoked stretch reflexes.
9 e 'natural' inputs involved in transcortical stretch reflexes.
10 contribute to maintain or even increase the stretch reflex after nerve crush and by difference to ne
13 gests that twitches trigger the monosynaptic stretch reflex and, by doing so, contribute to its activ
14 This pattern cannot be produced by muscle stretch reflexes and can only arise from the anticipator
16 are likely to convey long-latency jaw-muscle stretch reflexes and may contribute to stiffness regulat
17 nerve transection, Ia afferent synapses and stretch reflexes are permanently lost, even after regene
18 he first demonstration that the long-latency stretch reflex can be modified by repeated, precisely ti
20 otoneurons (MNs), the central portion of the stretch reflex circuit, are highly specific, but the mec
26 ese findings are difficult to reconcile with stretch reflex control of the pendulum and are of partic
27 -reflex, the electrical analog of the spinal stretch reflex, creates a memory trace that includes cha
28 LUT1 synapses are not re-established and the stretch reflex does not recover; however, electrically e
29 velocity dependent, increase in muscle tonic stretch reflexes, due to the amplified reactivity of mot
31 s an electrical analogue of the monosynaptic stretch reflex, elicited by bypassing the muscle spindle
33 that reinnervated muscles failed to generate stretch reflexes, extending observations of areflexia to
40 s on locomotion, the phase-dependency of the stretch reflexes implies a dynamically fluctuating role
41 eparate studies, we examined recovery of the stretch reflex in decerebrate cats, and found that it re
42 trate a previously uncharacterized nonneural stretch reflex in gastric muscles and provide physiologi
49 l disparity in recovery between strEPSPs and stretch reflex led us to conclude that factors in additi
52 evels (1 microM) decreased the monosynaptic "stretch" reflex (MSR) amplitude in WT animals and increa
53 bthreshold corticospinal conditioning of the stretch reflex of biceps and quadriceps was abnormal in
55 loops appear to add flexibility to the human stretch reflex, once considered to be immutable, allowin
58 tap to Tri evoked its own homonymous phasic stretch reflex, providing neurophysiological evidence fo
59 udy was designed to estimate the fraction of stretch reflex recovery attributable to functional recov
60 anding, and they produced no evidence of any stretch reflex response in soleus, or gastrocnemius.
61 o how the nervous system centrally modulates stretch reflex responses.A common measure of H-reflex ga
62 -and-hold perturbations used to elicit tonic stretch reflexes revealed significantly prolonged EMG re
64 tryptamine) from enterochromaffin cells, and stretch reflexes that determine the site of origin and p
67 ted that rapid motor responses (i.e., muscle-stretch reflexes) to mechanical perturbations can be mod
68 stationarity the amplitude of biceps phasic stretch reflex varied <10% in the first six repeats of t
69 both methods the magnitude of biceps phasic stretch reflex varied linearly with tap force over the r
70 troke the threshold of the homonymous phasic stretch reflex was low, but it had a normal onset latenc
71 elements in the sensorimotor circuit of the stretch reflex were examined in both the PNS and CNS.
74 ent low thresholds for the homonymous phasic stretch reflex, which had abnormally short onset latenci
75 as calf muscles shortened in contrast to the stretch reflex whose amplitude decreases as muscle short
76 exes, such as the vestibulo-ocular reflex or stretch reflex, whose gains adapt in response to novel a
77 Paradoxical movements cannot be generated by stretch reflexes with constant intrafusal drive but migh
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