ライフサイエンスコーパス: stricture

1 ic emptying, pancreatic fistula, and biliary strictures).

2 NA was positively correlated with intestinal stricture.

3 nce of postoperative cholangitis and biliary stricture.

4 vere mucosal disease activity and intestinal stricture.

5 age, and five patients (6.0%) had esophageal stricture.

6 ceptance for the treatment of benign biliary strictures.

7 ocation and relatively short length of these strictures.

8 d 19 (3.3%) bile leaks and 27 (4.6%) biliary strictures.

9 agnostic algorithm for indeterminate biliary strictures.

10 d in the evaluation of indeterminate biliary strictures.

11 t is best for primary and recurrent urethral strictures.

12 invasive options to manage men with urethral strictures.

13 ve to bowel wall fibrosis as occurs in Crohn strictures.

14 ses in the evaluation and therapy of biliary strictures.

15 aintenance of wide urethral calibres without strictures.

16 detected esophageal narrowing and localized strictures.

17 dence on the management of anterior urethral strictures.

18 my), and 22% were reoperations for recurrent strictures.

19 risk score, and lower occurrence of dominant strictures.

20 evaluation of indeterminate pancreatobiliary strictures.

21 phosphorylation were increased 205%-292% in strictures.

22 There have been no reoperations for biliary strictures.

23 diagnosis of malignancy in pancreatobiliary strictures.

24 erformed during 112 treatments of 84 biliary strictures.

25 her risk for salvage therapy and anastomotic strictures.

26 C with intrapancreatic vs proximal bile duct strictures.

27 performed in 85 patients with benign biliary strictures.

28 steroid withdrawal, especially with proximal strictures.

29 atients, nine had ureterovesical anastomotic strictures.

30 ifestations of fibrosis and gross esophageal strictures.

31 trated wide, patent vaginal calibers without strictures.

32 ancreatography for the evaluation of biliary strictures.

33 to help identify malignant pancreatobiliary strictures.

34 on, early allograft dysfunction, and biliary strictures.

35 brotic strictures from mixed or inflammatory strictures.

36 eta1, collagen, and CTGF production in ileal strictures.

37 hagia/odynophagia/chest pain (9% versus 2%), strictures (0% versus 0%), and mortality (7% versus 7%)

38 Overall, of the eight strictures 10 mm or shorter, there was success rate in s

39 langitis:11%, 12%, and 10%; P = 0.96, NS; re-stricture:18%,5%, and 29%; P = 0.01; nonsurgical 14%, 5%

40 complication indication (15.6% fistula, 7.4% stricture, 3.0% bleeding, 5.8% other).

41 oesophageal reflux disease (26%), esophageal stricture (39%), or both (15%) does not account for all

42 ons (4.3% vs 5.6%; P = .03), and anastomotic stricture (5.8% vs 14.0%; P < .001).

43 Of patients with stricture, 5 of 9 in the jejunal cohort required percuta

44 malities on endoscopy, including anastomotic strictures (53%), marginal ulcers (16%), functional obst

45 for recurrent cholangitis [50% and 27%], re-stricturing (75% and 61%), redo reconstructions (31% and

46 For penile strictures, a ventral onlay procedure using skin can be

47 ng cases (OR 0.53), and certain indications (strictures, active pancreatitis).

48 aged 51+/-11 years) with anastomotic biliary stricture after LDLT.

49 liary ducts for the treatment of anastomosis stricture after LDLT.

50 eased occurrence of late biliary anastomotic stricture after liver transplantation.

51 milar to other long tubularised tissues, can stricture after reconstruction.

52 FBD for anastomotic strictures after esophageal atresia repair is feasible a

53 afe for the treatment of anastomotic biliary strictures after LDLT.

54 llenging use in the treatment of anastomosis strictures after live donor liver transplantation (LDLT)

55 as no difference in the incidence of biliary strictures after resection for benign (n = 10, 2.6%) or

56 sed on the current approaches to anastomotic stricture and RUF following radical prostatectomy.

57 al of 115 patients (17.2%) developed BCs (83 strictures and 44 leaks).

58 Among patients with benign biliary strictures and a bile duct diameter 6 mm or more in whom

59 8 consecutive patients with pancreatobiliary strictures and analyzed by RC, DIA, and FISH as per stan

60 l series as treatment for posterior urethral strictures and bladder neck contractures resulting from

61 therapy by ERCP for the treatment of biliary strictures and chronic pain in chronic pancreatitis will

62 modality used in the evaluation of urethral strictures and fistulas in case of 'watering can penis'.

63 ammatory phase, whereas features like ductal strictures and focal masslike swelling are predictive of

64 y tissue remodeling that leads to esophageal strictures and food impactions.

65 n muscle cells of B2 phenotype patients from strictures and normal intestine in the same patient and

66 Many patients with IAC present with biliary strictures and obstructive jaundice, making cholangiocar

67 Proximally, ureteral strictures and symptomatic retrocaval ureters have been

68 ged: high STAT3(S727) and low STAT3(Y705) in strictures and the opposite in unaffected intestine.

69 o either resolution or development of ductal strictures and/or focal masslike swelling.

70 time of referral, most had a combination of stricturing and fistulizing disease.

71 Stricturing and penetrating complications account for su

72 ive inflammation from fibrosis, characterize stricturing and penetrating complications, and diagnose

73 f stent with ureteral complications (leak or stricture) and urinary tract infections (UTI).

74 tions (leak, cholangitis, bile gastritis, or stricture), and the secondary end points were overall co

75 diographic evidence of postoperative biliary stricture, and all patients underwent successful endosco

76 ality, whereas anastomotic leak, anastomotic stricture, and recurrent laryngeal nerve palsy rate was

77 and miscellaneous surgical complications and strictures, and similar postoperative use of additional

78 formation on previous operations, predefined strictures, and/or penetrating lesions of maximal severi

79 Strictures are more frequently associated with an inadeq

80 IC among centers, the importance of biliary strictures as a risk factor for graft failure, and does

81 or dilatation in the management of urethral strictures as first-line therapy in selected patients.

82 C should be suspected in unexplained biliary strictures associated with increased serum IgG4 and unex

83 Stricturing associated with endoscopic submucosal resect

84 cant restenosis after the first PBBD between strictures at anastomotic and nonanastomotic sites (P =

85 significant restenosis after PBBD of biliary strictures at anastomotic and nonanastomotic sites.

86 iagnostic delay was the only risk factor for strictures at the time of EoE diagnosis (odds ratio = 1.

87 isk factor for the development of esophageal strictures, Barrett esophagus, and esophageal adenocarci

88 Benign biliary strictures (BBS) respond to placement of multiple large-

89 ificantly more prevalent in CD patients with stricturing behaviour (B2) and perianal disease (7/11, p

90 younger age, with ileocolonic location, and stricturing behaviour with perianal disease.

91 structurally coordinates a three-dimensional stricture between intragenic elements of CFTR bound by s

92 thrombosis, primary nonfunction, and biliary stricture between the two groups.

93 duct obstruction, Bismuth- Corlette type IV stricture, biliary obstruction caused by gallbladder can

94 cations from chronic complications (fistula, stricture, bleeding) or the number of previously treated

95 Outcomes, including stricture, bleeding, hospitalization, and complete eradi

96 ilation was technically successful in all 52 strictures, but stenosis recurred in 10 patients and was

97 Smooth muscle cell hyperplasia in stricturing CD is regulated by increased endogenous IGF-

98 lls were isolated from muscularis propria of stricturing CD or normal margins.

99 nds in regulating muscle cell hyperplasia in stricturing CD.

100 On first diagnosis, most patients had a stricturing CD.

101 of the biliary tree with low rates of leak, stricture, cholangitis, and bile gastritis.

102 R] 0.30, 95% CI 0.10-0.89; p=0.0296) but not stricturing complication (1.13, 0.51-2.51; 0.76) than we

103 Ruminococcus was implicated in stricturing complications and Veillonella in penetrating

104 h Crohn's disease experienced penetrating or stricturing complications within 90 days after diagnosis

105 ient demographics, presence of urinary leak, stricture, compression, or vesicoureteral reflux, and ho

106 come was development of intrahepatic biliary strictures consistent with IC.

107 The prevalence of esophageal strictures correlates with the duration of untreated dis

108 and muscle cell hyperplasia are features of stricturing Crohn's disease (CD); however, the role of I

109 PBBD of benign strictures demonstrates long-term effectiveness.

110 Symptomatic strictures developed in 9% of patients and were treated

111 ression of stricture formation; we evaluated stricture development over time in the absence of treatm

112 ith kappa values of 0.76, 0.85, and 0.98 for strictures, dilatation, and choledocholithiasis, respect

113 e respective sensitivity and specificity for strictures, dilatation, and intraductal filling defects

114 endent readers evaluated the MRCP images for strictures, dilatation, and intraductal filling defects.

115 Ureteral stricture disease commonly affects the cancer patients.

116 tion urethroplasty for the treatment of male stricture disease is often accompanied by subsequent tis

117 Urethral stricture disease is poorly understood in prostate cance

118 xist for the management of anterior urethral stricture disease.

119 teropelvic junction obstruction and ureteric stricture disease.

120 LY75 loci were associated with a complicated stricturing disease course (Pcombined = 2.01 x 10(-8)),

121 stricturing nonpenetrating disease, 4.6% had stricturing disease, and 14.0% had penetrating disease.

122 ith corrosive-induced upper gastrointestinal strictures, either ES or GOO alone and simultaneous occu

123 Esophageal stricture (ES) and gastric outlet obstruction (GOO) can

124 stic stents or a single cSEMS, stratified by stricture etiology and with endoscopic reassessment for

125 MR and PET information performed better for stricture evaluation than either modality alone.

126 e review the cause and incidence of urethral strictures (excluding bladder neck contracture) in this

127 of disease-related complications, including strictures, fistulae, and abscesses.

128 al response was seen in patients with ductal stricture formation (two of 13 patients) and in those in

129 The pathogenesis of intrahepatic biliary stricture formation in patients with primary sclerosing

130 Stricture formation in the distal ureter is a common con

131 Intestinal remodeling and stricture formation is a complication of inflammatory bo

132 disease (CD); however, the role of IGF-I in stricture formation is unknown.

133 Long term sequelae included esophageal stricture formation requiring dilatation, persistent eso

134 ial complications of bleeding, protein loss, stricture formation, and perforation.

135 he prostate also carry high risk of urethral stricture formation, particularly in the salvage setting

136 treatment and investigated risk factors for stricture formation.

137 Adverse events include migration and new stricture formation.

138 CD14 is crucial during biliary infection and stricture formation.

139 by subsequent tissue fibrosis and secondary stricture formation.

140 ablative therapies are at risk for urethral stricture formation.

141 Little is known about progression of stricture formation; we evaluated stricture development

142 Stricture free rates from urethra dilatation and DVIU va

143 Indeterminate biliary strictures frequently present as a diagnostic conundrum

144 formance for differentiation of inflammatory strictures from fibrotic strictures was assessed.

145 e for the differentiation of purely fibrotic strictures from mixed or inflammatory strictures.

146 gastrectomy include postoperative leaks and strictures, gastric dilation, and gastroesophageal reflu

147 e any learning curve with regard to risks of stricture, gastrointestinal bleeding, perforation, or ho

148 mmon: 1 (2%) patient developed an esophageal stricture (grade 2) and 1 (2%) grade 4 esophagitis.

149 Two (3%) patients developed a bronchial stricture (grade 2), and 1 (2%) a grade 4 bronchial fist

150 ne neoplasms with associated pancreatic duct stricture had prominent stromal fibrosis.

151 hybridization in brushing samples of biliary strictures had a sensitivity of 50%, 57%, and 86%, speci

152 d classification of pCLE findings of biliary strictures has been proposed.

153 Palliation of malignant strictures has improved with advanced endoscopic techniq

154 Biliary strictures have a negative effect on HCV fibrosis severi

155 as associated with increased rate of biliary strictures (hazard ratio [HR], 1.67; 95% confidence inte

156 HR 3.711, P=0.008), Bismuth-Corlette type IV stricture (HR 2.082, P=0.008), obstruction due to gallbl

157 Biliary strictures (HR = 2.25, P = 0.0006), creatinine at LT (HR

158 for doubling of AST, P = 0.005) and biliary strictures (HR = 2.68, P = 0.0001) were associated with

159 than 400 mL/min was associated with biliary strictures (HR, 1.53; 95% CI, 1.04-2.24; P = 0.0297) on

160 imited (diseased terminal ileum <40 cm), non-stricturing, ileocaecal Crohn's disease in whom conventi

161 the Netherlands and the UK, adults with non-stricturing, ileocaecal Crohn's disease, in whom convent

162 ding ureteral leak in 10 (2.1%) and ureteral stricture in 8 (1.6%).

163 All cases with biliary stricture in DCD group finally led to graft loss, and al

164 The development of a biliary stricture in patients who have undergone PD for malignan

165 ith corrosive-induced upper gastrointestinal strictures in a tertiary hospital were recruited into th

166 el of evidence upon which to base therapy of strictures in children is low and consists mostly of cas

167 to present the current literature regarding strictures in children to provide an evidence-based reco

168 addition, management of biliary anastomotic strictures in liver transplant patients, role of cholang

169 he intraoperative HA flow to prevent biliary strictures in such patients.

170 inal inflammation, and long gross intestinal strictures in Tl1a transgenic compared to wild-type litt

171 The management of ureteral strictures in transplanted kidney is challenging.

172 re associated with increased rate of biliary strictures in younger donors (<50 years old), and in pat

173 n was associated with higher rate of biliary strictures in younger donors with duct-to-duct reconstru

174 and this gene signature was associated with stricturing in the risk model (HR 1.70, 95% CI 1.12-2.57

175 s) were determined for the rate of bile duct strictures, incomplete ablation, and tumor recurrence.

176 Similarly, the prevalence of esophageal strictures increased with duration of diagnostic delay,

177 dditional complications often ensue, such as strictures, infection, hair growth, graft shrinkage, div

178 for acute urinary retention, and another had stricture interventions requiring hospital admission.

179 on and normalized expression of TGF-beta1 in strictured intestinal muscle.

180 e STAT3(S727E) phenocopied muscle cells from strictured intestine.

181 on the presence of any remaining fistulas or strictures involving the urethra.

182 rrent obstruction due to tissue in-growth or stricture is similar between urethral stent placement an

183 Ureteric stricture is the most common urological complication fol

184 Development of strictures is a major concern for patients with eosinoph

185 Endoscopic dilation of strictures is possible.

186 sorders, such as biliary atresia or ischemic strictures, is restricted by the lack of biliary tissue

187 first-line treatment for most benign biliary strictures; it is possible that fully covered, self-expa

188 In muscle cells from strictures, Ki67 immunoreactivity and [(3)H]thymidine in

189 pecial reference to late biliary anastomotic strictures (LBAS).

190 Multivariate analysis showed that stricture length was significantly associated with highe

191 ssment of structural bowel damage, including stricturing lesions, penetrating lesions, and surgical r

192 athy (IC), a disease of diffuse intrahepatic stricturing limits broader DCDD use.

193 tion and DVIU remain widely used in urethral stricture management but high-level comparative evidence

194 gastric bypass include anastomotic leaks and strictures, marginal ulcers, jejunal ischemia, small bow

195 esophageal ulcer in the presence of proximal stricture may be indicative of underlying lichenoid esop

196 e postcystectomy patient, obstruction due to stricture must be differentiated from dysfunctional void

197 1) and a benign chronic inflammatory biliary stricture (n = 1), respectively.

198 ncluded benign stricture (n = 18), malignant stricture (n = 12), choledochal cyst (n = 5), choledocho

199 Indications included benign stricture (n = 18), malignant stricture (n = 12), choled

200 h high-grade dysplasia (n = 6), recalcitrant stricture (n = 8), gastrointestinal stromal tumor (n = 3

201 ess (n = 21), rectovaginal fistula (n = 19), strictures (n = 10), prior Hartmann procedures (n = 13),

202 atients with treatment-naive, benign biliary strictures (N = 112) due to orthotopic liver transplant

203 Biliary anastomotic strictures occurred in 1 DCD patient and 3 DBD patients.

204 The most common adverse event was stricture, occurring in 8 patients receiving ablation (1

205 Stricture occurs in 0-8% and is amenable to endoscopic d

206 ent 2-stage surgery had a lower risk of anal stricture (odds ratio = 8.21; P = .01) and no difference

207 0.76 (95% CI, 0.10-5.43) per million MCs for stricture of male genital organs to 703.23 (95% CI, 659.

208 The incidence and course of stricture of the hepaticojejunostomy have not been docum

209 hould be a first line treatment for ureteral strictures of length 10 mm or shorter in kidney transpla

210 aneous approaches to palliation of malignant strictures of the bile duct.

211 c cholestatic liver disease characterized by strictures of the biliary tree complicated by cirrhosis

212 c cholestatic liver disease characterized by strictures of the biliary tree complicated by cirrhosis

213 we aimed to assess its long-term success for strictures of transplant kidney ureters.

214 nt, 162 (21.8%) patients developed a biliary stricture, of which 88 (11.8%) exhibited intrahepatic st

215 For strictures, one CSEMS is inserted without need for dilat

216 outcome measures were the effect of time of stricture onset on graft survival, complications, and ri

217 avored with pretest probabilities of biliary stricture or malignancy 0%-73% for patients aged 20-44 y

218 s, and 4 (3%) had superior mesenteric artery stricture or spasm.

219 on dilatation for ureterovesical anastomotic strictures or ureteropelvic junction obstruction.

220 5%-CI 0.203-0.835, P = 0.014) and esophageal stricture (OR 2.666, 95%-CI 1.259-5.645, P = 0.01).

221 95%-CI 0.4668-2.835, P = 0.761), esophageal stricture (OR 2.832, 95%-CI 1.508-5.321, P = 0.001), pea

222 gh-volume MIRP experienced fewer anastomotic strictures (OR, 0.93; 95% CI, 0.87 to 0.99) and less sal

223 95% CI, -3.45 to -2.53) but more anastomotic strictures (OR, 1.40; 95% CI, 1.04 to 1.87) and higher r

224 presence of complications (abscess, fistula, stricture, or perforation), and severity of the inflamma

225 of white exudates, moderate or severe rings, strictures, or combination of furrows and edema.

226 ureteropelvic junction obstruction, ureteral strictures, or ureteral polyps.

227 ceptor phosphorylation was increased 320% in strictured over normal muscle, and basal Erk1/2, p70S6 k

228 reased 1.8- to 3.4-fold in muscle cells from strictures over normal margins.

229 IAC patients (P = 0.009), while an isolated stricture (P = 0.038), a biliary mass (P = 0.006), and n

230 nificantly higher incidence of postprocedure strictures (P = 0.006).

231 iated with development of dominant bile duct strictures (P = 0.02).

232 varying etiologies (including benign biliary stricture, papillary stenosis, choledocholithiasis, extr

233 reased GM-CSF Ab levels were associated with stricturing/penetrating behavior (odds ratio, 2.2; P=.01

234 negative predictive values of GM-CSF Ab for stricturing/penetrating behavior were comparable with th

235 Pharyngo-oesophageal stricture (PES) is a serious complication that occurs in

236 by whitish exudates, furrows, and edema), a stricturing phenotype (characterized by rings and stenos

237 In PSC patients, the presence of a dominant stricture plus FISH polysomy has a specificity of 88% fo

238 PDT appears to have a higher stricture rate and to be more expensive than RFA.

239 The stricture rate appears higher, and rates of complete rev

240 Stricture rates differ for various modalities of radiati

241 Refractory benign esophageal strictures (RBESs) have been treated with the temporary

242 countries, 187 patients with benign biliary strictures received FCSEMS.

243 ties are often at increased risk of urethral stricture recurrence brought upon in-part by delayed vas

244 erquartile range, 12.9-24.3 mo), the rate of stricture recurrence was 14.8% (95% CI, 8.2%-20.9%).

245 s limited to those patients with obstructing stricture-related disease, and even liver transplantatio

246 ial infarction (n = 1), and duodenoileostomy stricture requiring endoscopic dilation (n = 1).

247 stomotic stenting >3 months, and 3 developed strictures requiring percutaneous dilation.

248 stent placement, three underwent esophageal stricture resection, and four underwent esophageal recon

249 Steroid-sensitive biliary strictures resembling primary sclerosing cholangitis but

250 Primary outcome was stricture resolution after no more than 12 months of end

251 Long-term stricture resolution is achieved in up to 92%.

252 Primary stricture resolution rate was achieved in 17 (89.5%) of

253 atients were followed up for 12 months after stricture resolution to assess for recurrence.

254 success of FCSEMS after extended indwell and stricture resolution were achieved for approximately 75%

255 Stricture resolution without restenting upon FCSEMS remo

256 plastic stents after 12 months in achieving stricture resolution.

257 indwell and the frequency and durability of stricture resolution.

258 effect was primarily related to reduction in stricture risk (IRR, 0.23; P<0.05; 95% CI, 0.05-0.99).

259 f untreated disease is the best predictor of stricture risk.

260 Stricture severity was graded based on the degree of dif

261 lications (ie, erosive esophagitis or peptic stricture) should take a PPI for short-term healing, mai

262 LBAS was defined as stricture that occurred 30 days or more after LT.

263 dilemma because they harbor chronic biliary strictures that are difficult to distinguish from CCA.

264 pithelium, resulting in multifocal bile duct strictures that can affect the entire biliary tree.

265 ed in 19 patients with Crohn disease who had strictures that underwent surgical resection with pathol

266 e endoscopic management of malignant biliary strictures, the broadening therapeutic indications inclu

267 angiocarcinoma, choledochal cysts, or benign strictures; the procedures included 59 duodenal reconstr

268 thrombosis, small bowel obstruction, urinary stricture, urine leak, hernia formation, and delayed gra

269 inst the formation of nonanastomotic biliary strictures versus CC/CT patients (12.6%; P = 0.01).

270 ncidence of leak was 9.6% and of anastomotic stricture was 26%.

271 red in 4 of 119 subjects (3.4%); the rate of stricture was 7.6%.

272 Biliary anastomotic stricture was confirmed by endoscopic retrograde cholang

273 PBBD of the stricture was performed with a noncompliant balloon (8-1

274 Anastomotic stricture was the most common biliary complication (DCD

275 cidental removal, urine leakage, or urethral stricture was then pooled using random-effects models.

276 ion of inflammatory strictures from fibrotic strictures was assessed.

277 The incidence of anastomotic strictures was higher in patients with no T-tube.

278 esence of proximal extrahepatic/intrahepatic strictures was predictive of relapse.

279 Additionally, longer strictures were associated with a higher risk of recurre

280 At presentation, biliary strictures were confined to the intrapancreatic bile duc

281 Just a decade ago, intestinal strictures were considered to be an inevitable consequen

282 All strictures were dilated under direct visualization by us

283 holangiopancreatography for pancreatobiliary strictures were examined by all 3 (cytology, DIA, and FI

284 Fewer anastomotic strictures were found in the T-tube group (n = 2, 2.1%)

285 Strictures were stratified as proximal (n = 33) or dista

286 eloped in 6.5% of subjects treated with RFA; strictures were the most common complication.

287 elated complications (leaks, cholangitis, or strictures) were fewer in the duodenal than the jejunal

288 system in a patient with transplant ureteric stricture when antegrade stent placement or surgical rec

289 ne APC treatment developed a late esophageal stricture, which required endoscopic dilation, and 2 pat

290 n's disease (CD) patients develop intestinal strictures, which are difficult to prevent and treat.

291 ications were also noted, including urethral strictures, which occurred in 3.4% (CI, 1.0% to 7.0%) of

292 History of esophageal stricture who have recurrent symptoms of dysphagia.

293 In those patients with bulbar strictures who fail or are not suitable for these proced

294 Both strictures with failure were longer than 10 mm.

295 s in only one (14 months follow-up) and both strictures with failure were shorter than 10 mm.

296 ation (FISH) can be used to evaluate biliary strictures with greater accuracy than conventional cytol

297 Distinguishing benign from malignant biliary strictures with routine biliary cytology in this populat

298 stasis, but many develop progressive biliary strictures with time, leading to recurrent cholangitis,

299 y has durable outcomes for radiation-induced strictures, with a preference for excision and primary a

300 bacteremia and an 18% incidence of bile duct stricture within 6 months.