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1 ng the prognostic effect of age at onset and stridor.
2 ness, with no associated pain, dysphagia, or stridor.
3 , and 32 subjects experienced postextubation stridor.
4 erval, 36.8-99) in predicting postextubation stridor.
5  the airleak test may predict postextubation stridor.
6 asis, 44% vs. 73% (p < .001); postextubation stridor, 22% vs. 53% (p < .001); and withdrawal syndrome
7 ion patients experienced more postextubation stridor (7% vs 4%; P = .03) and fewer stage 2 or worse i
8 lt-onset asthmatic patients who lack a clear stridor and show prolonged coughs and chest discomfort c
9 ed subglottic cyst presented with persistent stridor and voice hoarsening in a baby diagnosed with Wi
10 showed features of obstructive sleep apnoea, stridor, and abnormal sleep architecture (undifferentiat
11  but are deemed high risk for postextubation stridor, and be administered systemic steroids for at le
12 aria), the respiratory tract (cough, wheeze, stridor, and dyspnea), and/or the gastrointestinal tract
13 , airleak values, presence of postextubation stridor, and extubation failure secondary to upper airwa
14 d events, mortality, antibiotic utilization, stridor, and reintubations to better understand the net
15 scharge (aOR, 4.57; 95% CI, 1.30-16.10), and stridor (aOR, 2.63; 95% CI, 1.17-5.90).
16 Dependent variables included the presence of stridor, Croup Score, and pulsus paradoxus at 10 mins, 6
17 dexamethasone group had a lower frequency of stridor, Croup Score, and pulsus paradoxus measurement a
18 in 77 patients (14.4%), and postextubational stridor developed in 28 patients (6.4%) (P = 0.20 and 0.
19 rade 4 anaphylactic reaction and one grade 3 stridor) during the first treatment cycle.
20 ulmonary function in patients with asthma or stridor following antireflux pharmacotherapy or surgery.
21 her the airleak test predicts postextubation stridor in children and if age affects its sensitivity a
22 s a screening test to predict postextubation stridor in young children (<7 yrs old), whereas in older
23 ion coefficient = 0.080), and postextubation stridor (intraclass correlation coefficient = 0.078).
24 y (one in 400 procedures was associated with stridor, laryngospasm, wheezing or apnea).
25  This may be due to subjective assessment of stridor or inability to differentiate supraglottic from
26 no significant differences between groups in stridor or reintubations.
27 if postextubation problems occurred, such as stridor requiring treatment or reintubation.
28 e esophagus and are causing symptoms (cough, stridor, respiratory distress, drooling or pain) are man
29  disturbances, including insomnia, laryngeal stridor, sleep breath disturbance, and sleep-related inv
30 and HPIV-2 (24.2%) were more associated with stridor than HPIV-3 (6.6%) and HPIV-4 (0%) (P < .01).
31 ic recipients developed a persistent audible stridor that did not occur in the syngeneic experimental
32 nting with an encephalomyopathy, inspiratory stridor, ventilator failure, progressive hypotonia, and
33  yrs of age, the incidence of postextubation stridor was greater in patients with an airleak at >20 m
34  yrs of age, the incidence of postextubation stridor was similar in patients with or without an airle

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