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1 ing properties of a native chemokine ligand (stromal cell-derived factor 1alpha).
2 hat they do not contribute to the binding of stromal cell-derived factor 1alpha.
3 r 1, vascular endothelial growth factor, and stromal cell-derived factor-1alpha.
4 ough both monocyte chemotactic protein-1 and stromal cell-derived factor-1alpha.
5 ng and biological responses to the chemokine stromal cell-derived factor-1alpha.
6 macrophage inflammatory protein-1alpha, and stromal cell-derived factor-1alpha.
7 tes to MIP-1alpha, but not their response to stromal cell-derived factor-1alpha.
8 ded EPCs underwent ex vivo modification with stromal cell-derived factor-1alpha (100 ng/mL) to potent
9 by a monoclonal antibody against CD4 and by stromal cell-derived factor 1alpha, a natural ligand of
10 irus type 1 (HIV-1) and of the CXC chemokine stromal cell-derived factor 1alpha against T-cell-tropic
11 stration of endothelial progenitor cells and stromal cell-derived factor-1alpha analog) could perform
12 rts have identified a role for the chemokine stromal cell-derived factor 1alpha and its receptor CXC
13 Human colon epithelial cells stimulated with stromal cell-derived factor 1alpha and macrophage inflam
14 and CSCs further enhanced the production of stromal cell-derived factor-1alpha and vascular endothel
15 s involved in the signaling cascade of IL-2, stromal cell-derived factor 1alpha, and type I interfero
16 L5551 inhibited CXCR4 binding to its ligand, stromal cell-derived factor-1alpha, and reduced hypoxia-
17 roteinase (MMP) -2, soluble VEGF receptor 1, stromal cell-derived factor-1alpha, and soluble Tek/Tie2
20 d growth factor, epidermal growth factor, or stromal cell-derived factor-1alpha, but not basic fibrob
22 and signaling studies with the CXCR4 agonist stromal cell-derived factor-1alpha (chemokine (CXC motif
23 e inflammatory protein-1beta (MIP-1beta) and stromal cell-derived factor-1alpha, chemokine ligands fo
24 f trans-endothelial migration in response to stromal cell-derived factor-1alpha (CXC chemokine ligand
26 okine receptor CXCR4 and its cognate ligand, stromal cell-derived factor-1alpha (CXCL12), regulate ly
29 is induced by constitutively active Rac1 and stromal cell-derived factor 1alpha-induced ERM dephospho
32 CXCR4, a receptor for the chemokine CXCL12 (stromal-cell derived factor-1alpha), is a G-protein-coup
33 ived factor-1alpha, and reduced hypoxia- and stromal cell-derived factor-1alpha-mediated migration do
34 neurotoxic natural ligands of CXCR4, such as stromal cell-derived factor-1alpha or viral macrophage i
35 (SMM)-chemokine analogs derived from natural stromal cell-derived factor-1alpha or viral macrophage i
36 of vascular endothelial growth factor D and stromal cell-derived factor 1alpha (P = .037 and .025, r
40 tor for the normal physiological function of stromal cell-derived factor 1alpha (SDF-1alpha) and the
41 gration toward formyl-Met-Leu-Phe (fMLP) and stromal cell-derived factor 1alpha (SDF-1alpha) as well
42 s the ability to cleave the chemokine CXCL12/stromal cell-derived factor 1alpha (SDF-1alpha) at its p
43 droxypropyl-beta-cyclodextrin (BCD) inhibits stromal cell-derived factor 1alpha (SDF-1alpha) binding
45 inhibitors for monocyte migration induced by stromal cell-derived factor 1alpha (SDF-1alpha) or fMLP.
47 g to Jurkat cells was blocked by addition of stromal cell-derived factor 1alpha (SDF-1alpha), as was
48 e inflammatory protein 1beta (MIP-1beta) and stromal cell-derived factor 1alpha (SDF-1alpha), chemoki
49 otent chemotactic responses to the chemokine stromal cell-derived factor 1alpha (SDF-1alpha), indicat
53 gulating secretion of the angiogenic factors stromal cell-derived factor 1alpha (SDF-1alpha)/CXCL12,
54 lly, exogenous administration of recombinant stromal cell-derived factor-1alpha (SDF) enhances neovas
55 is process, particularly when activated with stromal cell-derived factor-1alpha (SDF), known to be th
59 ignificantly reduce the extent of subsequent stromal cell-derived factor-1alpha (SDF-1alpha [CXCL12])
61 ction by examining chemotaxis in response to stromal cell-derived factor-1alpha (SDF-1alpha) as well
63 through the trophic effects of the chemokine stromal cell-derived factor-1alpha (SDF-1alpha) binding
64 rred after a necessary switch in bone marrow stromal cell-derived factor-1alpha (SDF-1alpha) expressi
66 ression of HIF-1alpha, CXCR4, and its ligand stromal cell-derived factor-1alpha (SDF-1alpha) in heman
70 To investigate the effects of the chemokine stromal cell-derived factor-1alpha (SDF-1alpha) on VLA-4
73 in diabetes is due to decreased wound level stromal cell-derived factor-1alpha (SDF-1alpha), a chemo
74 of the cells in response to the CXCR4 ligand stromal cell-derived factor-1alpha (SDF-1alpha), accompa
75 and plasma levels of its endogenous ligand, stromal cell-derived factor-1alpha (SDF-1alpha), correla
76 otein-coupled receptor for the ligand CXCL12/stromal cell-derived factor-1alpha (SDF-1alpha), plays a
77 mulation of cells with the ligand for CXCR4, stromal cell-derived factor-1alpha (SDF-1alpha), resulte
81 ophage inflammatory protein II (vMIP-II) and stromal cell-derived factor-1alpha (SDF-1alpha), were sy
82 we show that the secreted signaling protein stromal cell-derived factor-1alpha (SDF-1alpha), which a
83 appeared additive and distinct from that of stromal cell-derived factor-1alpha (SDF-1alpha), which i
84 s secrete several chemo-cytokines, including stromal cell-derived factor-1alpha (SDF-1alpha), which t
86 lls by expression of SPA1 or Rap1GAP blocked stromal cell-derived factor-1alpha (SDF-1alpha)-stimulat
91 the ligand for the CXCR4 chemokine receptor stromal cell-derived factor-1alpha (SDF-1alpha/CXCL12),
94 y, the migration of MKs toward a gradient of stromal cell-derived factor 1alpha (SDF1alpha) and the f
96 eptor CXCR4 expressed by the CTL and CXCL12 (stromal cell-derived factor 1alpha) secreted by tumor ce
99 rleukin-10, Tumor Necrosis Factor alpha, and stromal cell-derived factor-1alpha were reduced in the i
100 the cognate CXC chemokine receptor 4 ligand stromal cell-derived factor-1alpha were unchanged in bot
101 at, in contrast to the natural CXCR4 ligand, stromal cell-derived factor-1alpha, which promotes the e
102 er isoform, CXCR-4A or CXCR-4B, responded to stromal cell-derived factor-1alpha with a rise in intrac
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