ライフサイエンスコーパス: stromelysin 3

コーパス検索結果 (１語後でソート)

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1 erial specimens (n = 5) contain little or no stromelysin-3.

2 ed in atheroma, induced de novo synthesis of stromelysin-3.

3 andidate AP-1 target genes, collagenase-1 or stromelysin-3.

4 ar enzyme activation mechanism observed with stromelysin-3.

5 Osteoblasts express stromelysin-3, a matrix metalloproteinase associated wit

6 However, the induction of stromelysin-3 after prolonged treatment with FGF-2 resul

7 atalytic domains are most closely related to stromelysin-3 and contain the consensus HEXXH zinc-bindi

8 mRNAs for several other MMPs (stromelysin-1, stromelysin-3 and gelatinase A) and MMP inhibitors (TIMP

9 Differences in the expression patterns of stromelysin-3 and the delayed response proteinases in th

10 In normal mice, the MMPs MT1-MMP, stromelysin 3, and gelatinase B were expressed at low le

11 Expression of stromelysin-1, stromelysin-3, and two different membrane type-MMPs was

12 ne MC3T3 was used to study the regulation of stromelysin-3 by FGF-2.

13 This study demonstrates dual regulation of stromelysin-3 by FGF-2: acute destabilization of stromel

14 Many family members, including stromelysin-3, gelatinase A, MT-MMP, interstitial collag

15 ts reveal a stromal gene expression pattern (stromelysin-3, IGF-II and TIMP-1) identical to that obse

16 gulation may be important in the function of stromelysin-3 in bone and in remodeling processes, such

17 sion of the unusual matrix metalloproteinase stromelysin-3 in human atherosclerotic lesions and impli

18 uman atherosclerotic plaques (n = 7) express stromelysin-3 in situ, whereas fatty streaks (n = 5) and

19 tumor necrosis factor alpha did not augment stromelysin-3 in vascular wall cells.

20 Stromelysin-3 is an unusual matrix metalloproteinase, be

21 Stromelysin-3 is up-regulated by TH with early kinetics,

22 E4 substrates, such as the metalloproteinase stromelysin 3, is required for tumor cell invasion.

23 Furin processes stromelysin-3, membrane type 1 matrix metalloproteinase

24 In addition, stromelysin-3 mRNA and protein colocalized with CD40L an

25 Stromelysin-3 mRNA and protein colocalized with endothel

26 Acutely, FGF-2 decreased stromelysin-3 mRNA levels, whereas prolonged treatment c

27 ed that acute treatment with FGF-2 decreased stromelysin-3 mRNA stability but did not alter gene tran

28 melysin-3 by FGF-2: acute destabilization of stromelysin-3 mRNA, followed by induction of gene transc

29 s prolonged treatment caused an induction of stromelysin-3 mRNA.

30 The MMP-11 proteinase, also known as stromelysin-3, probably plays an important role in human

31 n of the matrix metalloproteinase (MMP) gene stromelysin-3 ( ST3 ) has been shown to be tightly assoc

32 ro array analysis found that TH up regulates stromelysin 3 (ST3, matrix metalloproteinase 11) in the

33 The matrix metalloproteinase (MMP) stromelysin-3 (ST3) (MMP11) was first isolated as a brea

34 involvement of the matrix metalloproteinase stromelysin-3 (ST3) in tissue remodeling and pathogenesi

35 The MMP stromelysin-3 (ST3) is induced by T3, and its expression

36 We demonstrate here that three MMPs-stromelysin-3 (ST3), collagenases-3 (Col3), and collagen

37 Stromelysin-3 (STR-3) is a recently characterized matrix

38 Stromelysin-3 then cleaves and activates Notch.

39 stromelysin-3, TIMP-2 and TIMP-3 mRNA while stromelysin-3, TIMP-2 and gelatinase A were seen in the

40 The skeletal muscle of the tongue expressed stromelysin-3, TIMP-2 and TIMP-3 mRNA while stromelysin-

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