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1 h 5 to 10 mm in 4 reference patients without structural disease.
2 is of ischemic heart disease had surgery for structural disease.
3  validate this strategy in eyes with macular structural disease.
4 ncement scar, indicating a relationship with structural disease.
5   We extended its application to hearts with structural disease and examined its ability to detect an
6 was no association between progressive RV/LV structural disease and newly developed ECG TFC.
7    Alarm symptoms suggest the possibility of structural disease, but do not necessarily negate a diag
8 icular arrhythmia; proband status; extent of structural disease; cardiac syncope; male sex; the prese
9 cular measure that may aid the prediction of structural disease evolution and represents a potential
10                                              Structural disease found infrequently in patients with I
11 ordance between symptoms and the severity of structural disease has not been explained.
12  that electrical abnormalities precede overt structural disease in arrhythmogenic right ventricular c
13                     To identify subthreshold structural disease in normal-appearing lung regions in s
14                             The incidence of structural disease in patients with concerning symptoms
15 reatening arrhythmias even in the absence of structural disease.It is believed that mutations in desm
16 h CF, lung clearance index is insensitive to structural disease (kappa = -0.03 [95% confidence interv
17 e evaluated the efficacy of these agents for structural disease modification.
18 rdial leads, V1-V3 (unrelated to ischemia or structural disease), normal QT intervals, apparent right
19                                      Rate of structural disease progression, symmetry between eyes, a
20  in the disease course to arrest and prevent structural disease progression.
21 stry were retrospectively compared to assess structural disease progression.

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