ライフサイエンスコーパス: strut

1 (energy transfer between adjacent porphyrin struts).

2 ell-arranged one-dimensional units (nanotube struts).

3 rosclerosis, uncovered strut, and malapposed strut.

4 trut and extends drug delivery between stent struts.

5 phenyl)benzene and trans-1,2-dipyridylethene struts.

6 ST was the ratio of uncovered to total stent struts.

7 tely healed neointimal layer overlying stent struts.

8 n had a clot extending over the aortic valve struts.

9 gold coated of the same thickness as control struts.

10 ion, serving as motors, brakes, springs, and struts.

11 act areas and contact stresses between stent struts.

12 on the relative positioning of drug-eluting struts.

13 Malapposed struts (1.2% versus 0.3%; P=0.02) and malapposition leng

14 4.5%), neoatherosclerosis (27.6%), uncovered struts (12.1%), and stent underexpansion (6.9%).

15 Thick-strutted (162 mum) stents were 1.5-fold more thrombogeni

16 was neoatherosclerosis (31.3%) and uncovered struts (20.2%).

17 he maximal length of malapposed or uncovered struts (3.40 mm; 95% confidence interval, 2.55-4.25; ver

18 e most common dominant finding was uncovered struts (33.3%) and severe restenosis (19.1%); and for ve

19 At 5 years, uncovered struts (4.1% versus 1.0%; P<0.01), length of uncoverage

20 at maximum interstrut angle, and fewer stent struts (4.9+/-1.0 versus 6.0+/-0.5; P<0.01) even when no

21 ation of a symmetrical urea tetracarboxylate strut, 4,4'-bipyridine, and Zn(NO(3))(2).6H(2)O under so

22 significantly lower proportion of uncovered struts; 4.3% [interquartile range, 1.2-9.8] versus 9.0%

23 .02 to 0.09 mm), the prevalence of uncovered struts (49%; IQR, 16% to 96%), fibrin deposition (63+/-2

24 e tomography end point (percentage uncovered struts 5.64+/-9.65% in BMS+DEB versus 4.93+/-9.29% in DE

25 OF that are composed of two Zn(II) porphyrin struts [5,15-dipyridyl-10,20-bis(pentafluorophenyl)porph

26 ndomly assigned (2:1) to either an ultrathin strut (60 mum) bioresorbable polymer sirolimus-eluting s

27 e most common dominant finding was uncovered struts (61.7%) and underexpansion (25.5%); for late ST,

28 nding adjudicated for acute ST was uncovered struts (66.7% of cases); for subacute ST, the most commo

29 demonstrated a higher percentage of embedded struts (71.0% [47.6, 89.1] compared with BVS 40.3% [20.5

30 The TAXUS Element is a novel thin-strut (81 microm), platinum chromium alloy PES designed

31 e thrombogenic than otherwise identical thin-strutted (81 mum) devices in ex vivo flow loops (P<0.001

32 m [IQR, 0.07 to 0.21 mm], P=0.008; uncovered struts: 9% [IQR, 0% to 39%], P=0.01; fibrin: 36+/-27%, P

33 The number and distribution of stent struts affect the amount of neointima after SES implanta

34 minimal in stents with 180/100 and 70/70 mum struts after embedding.

35 ng"--incomplete apposition of SB or MV stent struts against the MV wall proximal to the carina--was s

36 At all time points, the struts along the patent portions of the aneurysm necks h

37 and H2N-Cys-His-Asp-CONHL (where L = organic struts) amino acid sequences by covalently attaching the

38 A strut analysis of trabeculae was also performed and the

39 Strut analysis revealed significant reductions in trabec

40 ing individuals with sickle cell anemia than strut analysis.

41 e drug pellet no longer being affixed to the strut and categorized as spontaneous or surgically relat

42 e load of drug immediately around each stent strut and extends drug delivery between stent struts.

43 Protruding strut and strut malapposed with moderate detachment (ISA

44 viscous solutions of metal node and organic strut and subsequent evaporation of a plasticizer-modula

45 ion-based correlations between the converter/strut and the nucleotide-binding pocket, revealing a sur

46 gative density-based correlation between the strut and the nucleotide-binding pocket, which is consis

47 n intricate network of highly interconnected struts and channels that not only ensure extraordinary s

48 Composed of discontinuous struts and continuous cables, such systems are only stru

49 Similarly, uncovered struts and fibrin deposition was significantly greater a

50 her than concentrating drug around the stent struts and for its ability to match coating erosion with

51 tal-organic framework (MOF) with porphyrinic struts and Hf6 nodes is reported.

52 ge with extensive segments of double-layered struts and inappropriately apposed struts at the bifurca

53 th larger amount of uncovered and malapposed struts and similar rate of neoatherosclerosis as compare

54 Malapposed struts and stent underexpansion were more frequently dem

55 The percentages of malapposed struts and struts being both uncovered and malapposed at

56 antation results in less inflammation around struts and thinner neointima at 28 days in this pig mode

57 ime intervals from index stenting: uncovered struts and underexpansion in acute/subacute ST and neoat

58 vered cells were found attached to the stent struts and were also distributed within the adjacent den

59 dings, such as neoatherosclerosis, uncovered strut, and malapposed strut.

60 vidence of blood flow (speckling) behind the strut, and where the immediate postimplantation IVUS rev

61 Neointimal area, uncovered struts, and fibrin deposition were significantly higher

62 IH at maximum interstrut angle, fewer stent struts, and larger maximum interstrut angle.

63 malapposition, neoatherosclerosis, uncovered struts, and stent underexpansion without differences bet

64 MRI provided anatomic confirmation of stent strut apposition and functional corroboration of aneurys

65 We used long organic struts (approximately 2 nanometers) incorporating 34- an

66 Tensegrity structures with detached struts are naturally suitable for deployable application

67 mplete circumferential encroachment of stent struts around the catheter, suggesting multidirectional

68 can dislocate spontaneously from the suture strut as a late event that typically occurs after 3 year

69 Neointimal thickness was greater for struts associated with medial damage than struts in cont

70 pontaneously dislocated away from the suture strut at a mean postimplantation time of 77.4 months (ra

71 solution; (4) percentage of malapposed stent struts at 6 months; (5) 6-month restenosis; and (6) 6-mo

72 th BMS was associated with greater uncovered struts at flow divider sites, which is likely due to flo

73 e-layered struts and inappropriately apposed struts at the bifurcation level in 3 of 5 cases.

74 ciple that these stents are superior to thin-strut bare-metal stents for preventing repeat revascular

75 The percentages of malapposed struts and struts being both uncovered and malapposed at follow-up

76 vered within, adjacent, or without the stent struts being present in the RA.

77 largely comparable at all time points, with struts being sequestered within the neointima.

78 pocket, the relay helix, the SH1 helix, the strut between the upper 50 kDa and the lower 50 kDa subd

79 interlocked molecule (MIM) as the pillaring strut between two periodic Zn-carboxylate layers.

80 , thick-strut fully bioabsorbable EES, thick-strut biodegradable polymer metallic biolimus-eluting st

81 enicity and re-endothelialization among thin-strut biodegradable polymer metallic everolimus eluting

82 specified subgroup analysis of the Ultrathin Strut Biodegradable Polymer Sirolimus-Eluting Stent Vers

83 Ultrathin strut biodegradable polymer sirolimus-eluting stents (BP

84 size can be expanded with the long molecular struts biphenyl, tetrahydropyrene, pyrene, and terphenyl

85 red with DP-DES and more effective than thin-strut BMS, but without evidence for better safety nor lo

86 -(4-carboxyphenyl)porphyrin]Co(III) (CoTCPP) struts bound by linear trinuclear Co(II)-carboxylate clu

87 beneath regions of arterial contact with the strut but surprisingly also beneath standing drug pools

88 diac cycles were measured to describe the 3D strut, cell, section, and stent configuration.

89 Damage is therefore likely to accumulate in strut centers making cancellous bone more tolerant of st

90 The strut chordae were encircled with exteriorized wire snar

91 utions of the anterior leaflet second-order "strut" chordae are unknown.

92 adable polymer drug-eluting stents: the thin-strut cobalt-chromium sirolimus-eluting Orsiro stent and

93 Regarding this, the thin-strut CoCr-EES has consistently been associated with the

94 Simulations that coupled strut configurations with flow dynamics correlated with

95 ough both PES and SES showed nearly complete strut coverage after 12 months for on-label use, the maj

96 In this randomized trial, strut coverage and neointimal proliferation of a therapy

97 Strut coverage at 3 months of single implanted stents (n

98 is optical frequency domain imaging-assessed strut coverage at 3 months.

99 myocardial infarction would provide improved strut coverage at 6 months in comparison with angiograph

100 biolimus-eluting stent implantation improves strut coverage at 6-month follow-up in comparison with a

101 achment <100 mum at baseline showed complete strut coverage at follow-up, whereas segments with a max

102 -wall ISA distance on the risk of incomplete strut coverage at follow-up.

103 Flow disturbances and risk of delayed strut coverage both increase with ISA detachment distanc

104 Incomplete strut coverage has been documented an important histopat

105 Good stent strut coverage of >94% was found in both therapy groups.

106 Strut coverage of combined single and overlapped stents

107 ISCOVERY 1TO3 study (Evaluation With OFDI of Strut Coverage of Terumo New Drug Eluting Stent With Bio

108 In vivo impact on strut coverage was assessed retrospectively using optica

109 Nearly complete strut coverage was observed in this complex population v

110 CoCr-EES demonstrated greater strut coverage with less inflammation, less fibrin depos

111 ants in which fibrin deposition, endothelial strut coverage, inflammatory response, and mechanism(s)

112 luting technology allows complete very early strut coverage.

113 as healed or healing, characterized by stent struts covered by a thin neointima, overlying endothelia

114 at had developed in the place of side-branch struts, creating a neo-carina.

115 updates in the hardware itself, focusing on strut design.

116 ristic sample sizes (film thickness, wire or strut diameter, ribbon width, particle diameter, etc), a

117 ains with dimensions similar to the fiber or strut diameters and in thin plates where grain diameters

118 bable scaffolds (BRS) is highly dependent on strut dimensions and polymer features.

119 However, strut dimensions and positioning relative to the vessel

120 Struts discernible on optical coherence tomography at 6

121 cases, malapposition by OCT in 5 of 9 cases, strut discontinuity in 2 of 9 cases, and underexpansion

122 also beneath standing drug pools created by strut disruption of flow.

123 To assess stent strut distribution, the maximum interstrut angle was mea

124 assessed noninferiority of a novel ultrathin strut drug-eluting stent releasing sirolimus from a biod

125 rae (window-like openings) separated by bony struts (e.g., lizards, tuatara, dinosaurs and crocodiles

126 designed metallic stent contains honeycombed strut elements with inlaid stacked layers of drug and po

127 ated inflammatory cells compared with 44% of struts embedded in a lipid core and 36% of struts in con

128 form a complete neointimal layer over stent struts) extends the window during which stents are prone

129 l-type nodes and 1,3,5-benzenetricarboxylate struts, features accessible Cu(II) sites to which solven

130 Meta-analyses have shown that the thin-strut, fluoropolymer-coated cobalt-chromium everolimus-e

131 r sternum, and internal support with a steel strut for 6 months.

132 provide trophic support for neurons, act as struts for migrating neurons and growing axons, form bou

133 tached to the capsid inner surface as hinged struts, forming a mobile array, an arrangement with impl

134 Stent fracture was graded as I (single-strut fracture), II (> or =2 struts), III (> or =2 strut

135 By December 2003, outlet strut fractures (OSFs), often with fatal outcomes, had b

136 ld distortion at the bifurcation with single strut fractures in 4 of 5 and double fractures in 1 of 5

137 in 3 of 5 T-and protrusion procedures single strut fractures were noted.

138 On microcomputed tomography, no strut fractures were present after modified-T, whereas i

139 ion, which is separation of at least 1 stent strut from the arterial wall intima that does not overla

140 e 27 procedures, dissociation of the implant strut from the drug-containing cup occurred in 11 eyes (

141 allic everolimus eluting stents (EES), thick-strut fully bioabsorbable EES, thick-strut biodegradable

142 tery contact stress and area depend on stent-strut geometry, balloon compliance, and inflation pressu

143 lialization and neointimal coverage on stent struts has been put forward as the main underlying mecha

144 rials comprising inorganic nodes and organic struts, have potential application in many areas due to

145 a more favorable biomechanical behavior and strut healing profile compared with BVS in normal porcin

146 Scaffold strut healing was evaluated in vivo using weekly optical

147 nd surfaces, as demonstrated with thin stent struts, help reduce the potential for thrombosis despite

148 ingly different between platforms; localized strut hypersensitivity was exclusive to SES, whereas mal

149 ther had the coating applied to the standard strut, ie, gold coated thicker than controls, or had the

150 trols, or had the coating applied to thinned struts, ie, gold coated of the same thickness as control

151 ed as I (single-strut fracture), II (> or =2 struts), III (> or =2 struts with deformation), IV (with

152 ign of organic building blocks, which act as strut-impervious scaffolds, can be exploited to generate

153 s been developed and the arene employed as a strut in the synthesis of P5A-MOF-1, which has been demo

154 unded by thrombus in 7.1%, 9.0%, and 8.9% of struts in cases 1, 2, and 4, respectively.

155 f struts embedded in a lipid core and 36% of struts in contact with damaged media (P<0.001).

156 n stents implanted for </=3 days, only 3% of struts in contact with fibrous plaque had >20 associated

157 or struts associated with medial damage than struts in contact with plaque (P<0.0001) or intact media

158 cute ST and neoatherosclerosis and uncovered struts in late/very late ST.

159 ic order and site isolation of the catalytic struts in MOFs facilitate the studies of their activitie

160 ug-induced fibrin deposition surrounding DES struts in porcine coronary arteries.

161 Scaffold discontinuity with struts in the lumen center was the cause of malappositio

162 by covalently attaching them to the organic struts in the MOFs, without losing porosity or crystalli

163 6 months, the percentage of malapposed stent struts in the MTA arm was higher than in the RT arm (2.7

164 Adjacent and overlapping stent struts increased computed arterial drug deposition by fa

165 e in risk for LST as the number of uncovered struts increased.

166 Others, with thicker, high-atomic-number struts, induced cold spots in the dose distribution adja

167 al thickening independently of the extent of strut-induced injury.

168 ced glycation end products compared with the strut interior.

169 he 24C6 macrocyclic ring of the pillared MIM strut is now free enough to undergo full rotation.

170 is not repeated in a given octahedron, each strut is uniquely addressable by the appropriate sequenc

171 Because the base-pair sequence of individual struts is not repeated in a given octahedron, each strut

172 Strut junction interactions affect local directional per

173 Reaction of a ditopic urea "strut" (L1) with cis-(tmen)Pd(NO3)2 yielded a [3+3] self

174 e bifurcation coverage and in minimal double-strut layers at the neocarina.

175 Overlapping stent struts lead to localized peaks of drug concentration tha

176 (4%), and tissue protrusion within the stent struts leading to lumen compromise lumen (4%).

177 heart valve, in which one of the two outlet strut legs separates from the flange before the other, p

178 cal coherence tomography, which also enables strut-level assessment due to its higher axial resolutio

179 Strut-level neointimal thickness was 0.19+/-0.09 mm and

180 A-binding domains located at each end of its strut-like structure.

181 , and neutrophils were associated with stent struts </=11 days after deployment.

182 Protruding strut and strut malapposed with moderate detachment (ISA detachmen

183 The most frequent findings were strut malapposition (34.5%), neoatherosclerosis (27.6%),

184 Extensive strut malapposition was the presumed cause for ScT in 1

185 ere related to (1) stent underexpansion, (2) strut malapposition, (3) edge dissection(s), and (4) res

186 The MGuard is a novel thin-strut metal stent with a polyethylene terephthalate micr

187 biphasic relationship between cell speed and strut modulus and also indicated that mechanical factors

188 s been hypothesized to serve as a "molecular strut", most likely playing a role in ribosome assembly

189 in-cap NA), II (thick-cap NA), and III (peri-strut NA).

190 is identified the number of visualized stent struts normalized for the number of stent cells and maxi

191 The process of crack propagation within the struts of a foam is not well understood and is complicat

192 via the midline central stem and the lateral struts of the vault cartilages.

193 nds to reduce ISA, with the malapposed stent struts often integrated completely into the vessel wall,

194 that higher inflation pressures, wider stent-strut openings, and more compliant balloon materials cau

195 t the DNA strands fold successfully, with 12 struts or edges joined at six four-way junctions to form

196 Formation of a novel bone strut, or a bone bridge connecting the Op and BR togethe

197 ith the central helix serving as a molecular strut, or perhaps a spring, linking the two widely space

198 fectly aligned fibers giving rise to fibrous strut orientation, variable inter-strut pore size and co

199 nts were percent of uncovered and malapposed struts over time.

200 architectures composed of closed-cell porous struts patterned in the form of hexagonal and triangular

201 Stent strut penetration into a lipid core was associated with

202 incomplete apposition; 4) restenosis; and 5) strut penetration into a necrotic core.

203 iate analysis revealed that maximal depth of strut penetration, % strut with medial tear, and % strut

204 Stents designed with 12 struts per cross section had 50% to 60% less mural throm

205 timal area than identical stents with only 8 struts per cross section.

206 mm; P=0.02), and ratio of uncovered to total struts per cross-section >/=30% (35.5% versus 9.7%; P=0.

207 e to compare the ratio of uncovered to total struts per cross-section >/=30% and other optical cohere

208 ile range] of uncovered and malapposed stent struts per lesion was 0 [0 to 0.35], 2.84 [0 to 6.63], a

209 ent with a ratio of uncovered to total stent struts per section >30% is 9.0 (95% CI, 3.5 to 22).

210 ion versus ratio of uncovered to total stent struts per section demonstrated a marked increase in ris

211 on was the ratio of uncovered to total stent struts per section.

212 ppearance of tip embedding, degree of filter strut perforation, and distance of filter tip from the n

213 Inhomogeneous strut placement influenced hydrophilic drugs more negati

214 to fibrous strut orientation, variable inter-strut pore size and controlled film width (via layering)

215 o drug delivery depends on clot geometry and strut position in clot relative to the vessel wall.

216 01, its pseudorotaxanes, and their molecular strut precursors.

217 s are a blueprint for applying the 'node and strut' principles of reticular synthesis to molecular cr

218 my is improved by muscles that act as active struts rather than working machines.

219 Medial injury and lipid core penetration by struts result in increased inflammation.

220 nd 2012 were reviewed for drug pellet-suture strut separation, observed before surgery in clinic or o

221 that depends on the stent design, with each strut serving as a vertex.

222 de a framework (MOF-177) devoid of polyether struts showed negligible uptake of PQT2+, indicating the

223 eneration everolimus-eluting DP-DES, or thin-strut silicon-carbide-coated BMS in 8 European centers.

224 The thin-strut sirolimus-eluting Orsiro stent was noninferior to

225 Mean neointimal thickness at stent strut sites was reduced 49% (P<0.0003) and 36% (P<0.007)

226 mately 40 square meters per gram and pore or strut sizes of approximately 20 nanometers.

227 Each connector has up to four struts, so that a single connector can link up to four M

228 phorylcholine polymer on a cobalt alloy thin-strut stent has shown promising experimental and early c

229 , the crush technique with the use of a thin-strut stent may result in improved immediate hemodynamic

230 Moreover, both studies used thick-strut stents known to have high restenosis rates as cont

231 Struts still recognizable on optical coherence tomograph

232 mum and >300 mum had 6.1% and 15.7% of their struts still uncovered at follow-up, respectively (P<0.0

233 osomes, suggesting that microtubules act as "struts" stretching the spindle matrix.

234 ribution of junction points between scaffold struts strongly modulates motility.

235 The thin-strut structure of the stent platform, the thromboresist

236 dition, drug eluted from the abluminal stent strut surface accounted for only 11% of total deposition

237 ug load or arterial wall contact with coated strut surfaces.

238 rties associated with increased ductility of strut surfaces.

239 ne more tolerant of stress concentrations at strut surfaces.

240 OCT showed malapposed scaffold struts surrounded by thrombus in 7.1%, 9.0%, and 8.9% of

241 induced more uncovered and malapposed stent struts than BMS, but less than after DES.

242 nsive regulatory segments form a coiled-coil strut that blocks peptide and ATP binding to the otherwi

243 ance to blood flow as compared with floating strut that has more significant ISA distance.

244 proposed to act mechanically as compressive struts that resist both actomyosin contractile forces an

245 panding endovascular stent was designed with strut thickness of 70 mum x 70 mum width.

246 Despite a similar scaffold strut thickness, the Magmaris sirolimus-eluting bioabsor

247 rformed to study the efficacy of stents with struts (thickness/width) 70/70, 180/100 and 300/150 mum

248 cobalt porphyrin catalysts linked by organic struts through imine bonds, to prepare a catalytic mater

249 separated, but not dislocated away, from the strut; time to exchange of dislocated or dissociated pel

250 ts suggest that Cypher functions as a linker-strut to maintain cytoskeletal structure during contract

251 Upon reduction of the metalloporphyrin struts to (Co(I)TCPP)CoPIZA, the CoPIZA thin film demons

252 cified noninferiority margin of 5% uncovered struts versus DES (difference between treatment means, 0

253 on in different cases of ISA with increasing strut-wall detachment distance (ranging from 100 to 500

254 age revealed an important impact of baseline strut-wall ISA distance on the risk of incomplete strut

255 support strut was used for 66 patients; the strut was placed anterior to the sternum in 9 patients u

256 The strut was routinely removed within 6 months.

257 A substernal support strut was used for 66 patients; the strut was placed ant

258 que and/or thrombus protrusion through stent struts was initially present in 70.4% of PES and 64.8% o

259 ogenicity of polymer-coated stents with thin struts was lowest in all configurations and remained ins

260 uction in the inflammatory infiltrate around struts was observed in untouched, compared with handled,

261 complete endothelialization over the device struts was present.

262 The percentage of uncovered struts was similar between SES and PES including stents

263 groups, whereas the percentage of uncovered struts was strikingly lower in CoCr-EES (median=2.6%) ve

264 The percentage of acutely malapposed struts was substantially lower in the OCT-guided group (

265 y distal to individual isolated drug-eluting struts was twice as great as in the proximal area and fo

266 ed as ISA volume or maximum ISA distance per strut) was an independent predictor of ISA persistence a

267 erations and location of drug elution on the strut were far more important in determining arterial wa

268 In lesions B, uncovered struts were 2.91+/-5.47% at 6-months.

269 Malapposed struts were 3.55+/-5.16% at post-procedure, 1.51+/-3.52%

270 In lesions A, uncovered struts were 3.77+/-4.94% and 3.02+/-4.35% at 3 versus 9

271 Malapposed struts were 4.94+/-6.70% post-procedure and 1.01+/-3.11%

272 nts presenting very late ST, uncovered stent struts were a common dominant finding in drug-eluting st

273 al hyperplasia and inflammation around stent struts were also assessed in the pig in-stent restenosis

274 actor centered on the stent, and the visible struts were counted and normalized for the number of ste

275 n patients with ST, uncovered and malapposed struts were frequently observed with the incidence of bo

276 channel density (P<0.0001) were greater when struts were in contact with a ruptured arterial media co

277 In all patients, BVS struts were integrated in the vessel and were not discer

278 Uncovered and malapposed struts were more frequent in thrombosed compared with no

279 At 5 years, struts were no longer discernable by OCT and IVUS.

280 Uncovered and malapposed struts were observed in 70.5% (43/61) and 62.3% (38/61)

281 At 21 days, uncovered struts were still present in the BVS group (3.8% [2.1, 1

282 nations, only small remnants of the original struts were visible, well embedded into the intima.

283 med" myosin heads may function as "transient struts" when attached to the thin filaments.

284 x (perimysial coils, pericellular weaves and struts), which were often arranged in disorganized patte

285 on and scattering of beta-particles by stent struts will cause significant perturbations in the unifo

286 be coupled to prepare the requisite organic strut with four metal-binding sites in the form of four

287 d that maximal depth of strut penetration, % strut with medial tear, and % struts with incomplete app

288 d by the lack of contact of at least 1 stent strut with the vessel wall in a segment not overlying a

289 fracture), II (> or =2 struts), III (> or =2 struts with deformation), IV (with transection without g

290 penetration, % strut with medial tear, and % struts with incomplete apposition were the primary indic

291 Uncovered scaffold struts with superimposed thrombus were the predominant f

292 in particular, at the contacts of the stent struts with the artery.

293 ssociations of both uncovered and malapposed struts with thrombus were consistent among early- and ne

294 f patients in the irradiated group had stent struts with undetectable neointimal versus only 27% in t

295 s over a net distance of up to ~45 porphyrin struts within its lifetime in DA-MOF (but only ~3 in F-M

296 where the protein functions as a "molecular strut" within the ribosome.

297 Pellets also may be dissociated from the strut without dislocation, when separation occurs at the

298 n Fe-decorated Hf6 node and the Fe-porphyrin strut work in concert.

299 Directly testing the effects of varying strut Young's modulus on cell motility showed a biphasic

300 ic framework with Lewis acidic (porphyrin)Zn struts, ZnPO-MOF, can be made in high yields.