ライフサイエンスコーパス: stunt

1 g through a mediator rather than directly on stunting).

2 nitive scores than did children who remained stunted.

3 le cases where sinus venosus angiogenesis is stunted.

4 an children, aged 12-59 mo, of whom 62% were stunted.

5 e strongly and significantly associated with stunting.

6 ng neonatal and under-5 mortality, and child stunting.

7 ed that low-grade inflammation may influence stunting.

8 s observed in boys might have been caused by stunting.

9 CI, 1.22-2.55) were the major predictors of stunting.

10 a noninvasive biomarker of future childhood stunting.

11 ) compared with 5.48 (4.62, 6.50) for severe stunting.

12 0) for underweight and 57.1% (51.9-62.4) for stunting.

13 nowledge of any associated risks for EED and stunting.

14 eonatal mortality, and prevalence of under-5 stunting.

15 rth-weight (LBW) infants are at high risk of stunting.

16 on between any of the markers and subsequent stunting.

17 ut children of older mothers had less 2-year stunting (0.64 [0.54-0.77]) and failure to complete seco

18 ratio (OR) of 0.993 (95% CI 0.989-0.995) for stunting, 0.986 (0.982-0.990) for underweight, and 0.984

19 t of covariates were 0.996 (0.993-1.000) for stunting, 0.989 (0.985-0.992) for underweight, and 0.983

20 wealth quintile were 0.997 (0.990-1.004) for stunting, 0.999 (0.991-1.008) for underweight, and 0.991

21 )], preterm birth (1.26 [1.03-1.53]), 2-year stunting (1.46 [1.25-1.70]), and failure to complete sec

22 o high prevalences of anemia (8.1-27.5%) and stunting (13.2%).

23 LNS-PLs reduced the risk of newborn stunting (18.7% compared with 22.6%; RR: 0.83; 95% CI: 0

24 3-0.32; 2415 children) and less likely to be stunted (35% vs 41%, PR 0.86, 95% CI 0.74-1.0) than chil

25 Sample sizes were 462,854 for stunting, 485,152 for underweight, and 459,538 for wasti

26 cines (17%), reductions in wasting (11%) and stunting (9%), facility birth care (7%), and prevention

27 was associated with 3% higher odds of being stunted (95% CI of ORs: 1.00, 1.06; P = 0.04), 5% higher

28 9 wk of age were 1.8 times more likely to be stunted (95% CI: 1.1, 2.8; P = 0.03) irrespective of eth

29 However, a key limitation which has stunted a full development of high-performance nanophoto

30 ian children aged 12-59 mo, of whom 62% were stunted.A low height-for-age z score (HAZ) was associate

31 whereas there was no effect modification by stunting across length quartiles (P = 0.32).

32 Stunting affects 1 in 5 young children.

33 Stunting affects approximately 25% of children <5 y of a

34 Stunting affects one-third of children under 5 y old in

35 Choline status has been associated with stunting among young children.

36 amples consisted of 390,328 children for the stunting analysis, 397,080 children for the underweight

37 chool-aged children, 0.1% were classified as stunted and obese and 1.4% were both anemic and overweig

38 the SlSHN3-OE leaf tissue resulted in shiny, stunted and permeable leaves.

39 is We report that Tctp deficiency results in stunted and splayed retinotectal projections that fail t

40 4 (95% CI: 2.6, 4.3) among children who were stunted and underweight but not wasted; 4.7 (95% CI: 3.1

41 eceiving microbiota from healthy or severely stunted and underweight infants; age- and growth-discrim

42 cceptably high (30.0%, 95% CI 27.0-33.0, for stunting and 22.1%, 19.4 to 24.8, for underweight).

43 0.4 million in these regions) are at risk of stunting and adult-onset metabolic conditions.

44 Child stunting and anemia are intractable public health proble

45 In Mexico, stunting and anemia have declined but are still high in

46 hygiene and improved infant feeding on child stunting and anemia in Zimbabwe.

47 t the impact of SHINE interventions on child stunting and anemia will be modified by these maternal c

48 on (EED) is a major underlying cause of both stunting and anemia, that chronic inflammation is the ce

49 er, sanitation, and hygiene interventions on stunting and anemia.

50 We assessed the prevalence of childhood stunting and both child and adult female overweight and

51 where there was also a greater prevalence of stunting and DBM at the individual level, both in women

52 ice, we excluded children jointly exposed to stunting and extreme poverty from children living in ext

53 Hookworms infect over 400 million people, stunting and impoverishing them.

54 shows a cross-sectional association between stunting and low serum omega-3 and omega-6 long-chain PU

55 children <2 y showed an association between stunting and obesity (OR: 2.0; 95% CI: 1.2, 3.6).

56 vel, double burden was defined as concurrent stunting and overweight and obesity in children aged 5-1

57 he individual level, prevalences were 1% for stunting and overweight or obesity and 2.9% for anemia a

58 ated risk for compromised development due to stunting and poverty are likely to forgo about a quarter

59 New estimates, based on proxy measures of stunting and poverty, indicate that 250 million children

60 We estimated the prevalence of stunting and the number of stunting cases that were attr

61 den of malnutrition and continue to focus on stunting and to a lesser extent on overweight, without a

62 indicated that the region had high rates of stunting and underweight (40% and 31% of children aged <

63 MI >/=25) and year of survey on the risks of stunting and underweight in children.

64 The prevalence of stunting and underweight remained unacceptably high (30.

65 to longer-run child-growth outcomes such as stunting and underweight.

66 All degrees of underweight, stunting and wasting were associated with significantly

67 rates of obesity with very low frequency of stunting and, although more information is needed, the d

68 As a result, Mars's growth is stunted, and nothing large in the asteroid belt can accu

69 ant gut microbiota, infant feeding, EED, and stunting, and discuss the potential causal pathways rela

70 uring infancy, ascertain its contribution to stunting, and investigate the impact of the randomized i

71 of long-term neurodevelopmental impairment, stunting, and non-communicable conditions.

72 to increase survival and reduce disability, stunting, and non-communicable diseases.

73 ith persistent diarrhea, cognitive deficits, stunting, and nutrient deficiencies have demonstrated co

74 e that mycotoxin exposure is associated with stunting, and propose that the causal pathway may be thr

75 ry outcome), height for age, weight for age, stunting, and underweight.

76 hose who were wasted and underweight but not stunted; and 12.3 (95% CI: 7.7, 19.6) in those who were

77 iations between mycotoxin exposure and child stunting; and to investigate EED as a potential pathway

78 objective was to describe the coexistence of stunting, anemia, and overweight and obesity at the nati

79 evalence and distribution of undernutrition (stunting, anemia, and zinc deficiency), overweight, and

80 ividual levels, the coexistence of OW/OB and stunting, anemia, or zinc deficiency was found in 2.8%,

81 that RAM1 regulates expression of EXO70I and Stunted Arbuscule, two genes whose loss of function impa

82 t lines show compromised AM colonization and stunted arbuscules.

83 The causes of stunting are complex but likely include prenatal effects

84 onmental enteric dysfunction (EED) and child stunting are lacking.

85 the HS) were significantly less likely to be stunted at 12 mo than were controls (OR: 0.35; 95% CI: 0

86 First, 36% of children stunted at age 8 y managed to catch up with their peers

87 12% of children were stunted at birth with 57% stunted by 2 years.

88 for-age z score of -1.3 +/- 1.2 and 22% were stunted at birth.

89 Supplementation reduced the prevalence of stunting at 1 (RR: 0.95; 95% CI: 0.92, 0.98) and 3 (RR:

90 second half of infancy or the likelihood of stunting at 12 mo postpartum.

91 e proportion of children with underweight or stunting at 2 years of age halved during four decades of

92 Stunting at 6 months was associated with a higher risk o

93 Early life Giardia was a risk factor for stunting at age 2 but not poor weight gain.

94 Prevalence of stunting at birth [length for age z score (LAZ): <-2] wa

95 seasonality, and temperature, and childhood stunting at the household level (12 of 15 studies, 80%).

96 Integrating EED, chemical exposure, and stunting at various ages during childhood will enhance o

97 Maternal undernutrition and newborn stunting [birth length-for-age z score (LAZ) <-2] are co

98 under greenhouse conditions, they revealed a stunted bushy appearance that could be rescued by gibber

99 omes than did children who were persistently stunted but were not generally different from children w

100 ransition, as evidenced by very low rates of stunting, but the country shows a high prevalence of obe

101 % of children were stunted at birth with 57% stunted by 2 years.

102 translation of nucleic acids drugs has been stunted by limited delivery options.

103 sk of neonatal deaths and, for survivors, of stunting by 2 years of age.

104 and about a fifth of the existing burden of stunting can be averted using these approaches, if acces

105 We estimated the number of stunting cases among children aged 24-35 mo (i.e., at th

106 the prevalence of stunting and the number of stunting cases that were attributable to each risk facto

107 manent neuromotor and cognitive deficits and stunts cerebellar growth.

108 thers with excess body weight coexist with a stunted child <5 y old.

109 roximately 5% of households had at least one stunted child aged <5 y and an overweight mother compare

110 ed the prevalence of DBM at the household (a stunted child and an overweight mother) and individual (

111 An obese mother with a stunted child are present in 6.3% of households; 1.9% of

112 d are present in 6.3% of households; 1.9% of stunted children are obese at age 6 y and 3.1% are obese

113 Around 30% of the world's stunted children live in India.

114 ntake explains low circulating choline among stunted children living in low-income countries and whet

115 In 2008, the prevalence of stunted children was 28.8 percentage points higher and o

116 5, 698.2) pg/mL urine, and the prevalence of stunted children was 44%, 55%, and 56%, respectively.

117 , and riboflavin (P = .01).The proportion of stunted children was significantly higher among children

118 1R) that locks Arl6 in the GDP form leads to stunted cilia and inhibition of PC1 on primary cilia.

119 stunting, the total number of children with stunting continues to rise in sub-Saharan Africa.

120 The prevalence of stunting decreased from 60% in 2000 to 40% in 2014.

121 n 1998 and 2008, the prevalence of childhood stunting decreased in both indigenous and nonindigenous

122 mmation during the second postnatal week are stunted dendrites of the cerebellum's principal neurons,

123 (+) neurons over time-but with significantly stunted dendrites-suggesting an uncoupling of neuron sur

124 ckdown of FOXP1 in cultured cortical neurons stunts dendritic outgrowth and this phenotype cannot be

125 to explore relations between linear growth (stunting), diarrhea, and age at the first introduction o

126 eased proliferation and branching, producing stunted ducts filled with luminal cells showing altered

127 Mutant pups were severely stunted during the suckling period, but many recovered a

128 ture, they show enhanced leaf senescence and stunted ear development.

129 ence), little to no difference in proportion stunted (eight fewer per 1000 children, 95% CrI -48 to 3

130 infections have been associated with growth stunting, even in the absence of diarrhea.

131 a; (ii) atrophic cerebellar hemispheres with stunted folia, profound granule cell depletion, Bergmann

132 , we used the effect size of risk factors on stunting from meta-analyses of epidemiological studies a

133 s had the second largest estimated impact on stunting globally and in the South Asia, sub-Saharan Afr

134 icy platform, integrating child survival and stunting goals within macro-level policies and programme

135 utritional supplementation for children with stunted growth (CBGM+NS), and control.

136 ersity reduce maternal investment leading to stunted growth (developmental constraints).

137 yr), spruce/fir forest (33.8 mug/m2-yr), and stunted growth alpine/fir forest (44.0 mug/m2-yr).

138 oss of editing at the five sites resulted in stunted growth and accumulation of apparent photodamage.

139 peroxisomal CATALASE2 (cat2-2) that display stunted growth and cell death lesions under ambient air,

140 d bioenergy crops, but frequently results in stunted growth and developmental abnormalities, the mech

141 alnutrition and recurrent diarrhoea leads to stunted growth and longer-term effects on cognitive deve

142 t during the 1st week of life they exhibited stunted growth and suffered desquamation, with most dyin

143 larger positive effects among children with stunted growth at baseline, with estimated interaction e

144 found that ZxAKT1-silenced plants exhibited stunted growth compared to wild-type Z. xanthoxylum.

145 Typical double mutants showed stunted growth of aerial and root tissue, formation of m

146 he repression of lignin biosynthesis and the stunted growth of the phenylpropanoid pathway mutant red

147 cases, the whole-plant stomatal conductance, stunted growth phenotype, and leaf ABA level were restor

148 s ccc knockout mutant abolished the mutant's stunted growth phenotypes and reduced shoot Cl(-) and Na

149 e glycosylation, are likely the cause of the stunted growth seen both in our model and in PMM2-CDG pa

150 The stunted growth was associated with lower PSII efficiency

151 report that mouse embryos lacking OLA1 have stunted growth, delayed development leading to immature

152 ) and MED5b (also known as RFR1) rescues the stunted growth, lignin deficiency and widespread changes

153 (MPI) that impair glycosylation and lead to stunted growth, liver dysfunction, coagulopathy, hypogly

154 The Hv-CBF2A overexpressing lines exhibited stunted growth, poor yield, and greater freezing toleran

155 4 mo of age, when nearly 50% of children had stunted growth.

156 is along with pale green leaves and severely stunted growth.

157 had modest positive effects on children with stunted growth.

158 renatally, while survivors had significantly stunted growth.

159 Children with and without stunting had median (25th, 75th percentile) serum cholin

160 lative contribution of these risk factors to stunting has not been examined across countries.

161 A large number of risk factors for stunting have been identified in epidemiological studies

162 ide, continued high rates of early childhood stunting have put the global applicability of internatio

163 In children who were stunted (HAZ < -2) at baseline, cotrimoxazole use was no

164 ldren in the sample were classified as being stunted (HAZ <-2).

165 t-for-age outcomes were also seen for severe stunting (HAZ < -3) and mean HAZ.

166 In 2011 the prevalence of stunting (height-for-age < -2 z scores) was 1.9% for chi

167 elevated AGP was positively associated with stunting (height-for-age z score <-2) in 7 of 10 surveys

168 challenged recipients initially retained the stunted IFN-gamma production found prior to transfer, an

169 in ameliorating long-term sequelae, notably stunting, immune dysfunction, and neurocognitive deficit

170 logical effects ranging from anaemia, growth stunting, impaired cognition, and decreased physical fit

171 ptors and B cell receptor (BCR) signaling is stunted in atypical MBCs resulting in impaired B cell re

172 lls throughout the hippocampus were markedly stunted in both homozygous and heterozygous Gdf5 null mu

173 hippocampal excitatory neurons were markedly stunted in Cd40(-/-) mice, whereas those of striatal inh

174 ngs suggest that although salt perception is stunted in samples containing a random coil, ionic, muco

175 The prevalence of stunting in children aged <2 y is 10.9%.

176 ousehold level, the prevalence of concurrent stunting in children aged <5 y and overweight and obesit

177 estimated and defined as the coexistence of stunting in children aged <5 y and overweight or obesity

178 Growth stunting in children under 2 years of age in low-income

179 We used country-level prevalence of stunting in children younger than 5 years based on the 2

180 anemia, malnutrition, growth and development stunting in children, and severe morbidity and mortality

181 chronic disease can cause malnourishment and stunting in children.

182 sanitation are the leading risk factors for stunting in developing countries.

183 d vitamin-enhanced MNP significantly reduced stunting in FT-LBW infants in this high-risk setting.

184 ctions and improve nutrient intake to reduce stunting in FT-LBW infants.

185 uction of aguitas and the high prevalence of stunting in Guatemala, longitudinal studies are urgently

186 Stunting in linear growth occurs mainly during the first

187 ability, is a major contributor to childhood stunting in low-income countries.

188 pite its high morbidity and association with stunting in the developing world, current therapies for

189 per arm circumference, and the prevalence of stunting in the LNS group was 8.9%, compared with 13.7%

190 Stunting in this community is high, whereas acute undern

191 t on linear growth in newborns suggests that stunting in utero is unlikely to be reduced by supplemen

192 ous studies from Vellore slums have reported stunting in well over a third of children, comparable to

193 es, betaine and TMAO, with linear growth and stunting in young children.

194 childhood linear growth faltering (known as stunting) in low-income and middle-income countries rema

195 ge on childhood undernutrition (particularly stunting) in subsistence farmers in low- and middle-inco

196 ximity to these positively influenced higher stunting incidence.

197 oled models, child age, iron deficiency, and stunting independently predicted anemia and severe anemi

198 icantly less abundant in those with severely stunted infants.

199 eason, admission criteria, or baseline FFMI, stunting, inflammation, or breastfeeding (p > 0.05).

200 Stunting is common in young children in developing count

201 supplements (CFSs) can enhance growth where stunting is common, but substitution for the usual diet

202 Stunting is its presumed major consequence.

203 The pathogenesis of stunting is poorly understood.The purpose of this study

204 issue layer encapsulating the myocardium and stunting its growth, and which is typically associated w

205 that gave psychosocial stimulation to growth-stunted Jamaican toddlers.

206 Mutant septal tips were stunted, lacked elastin-positive tips, exhibited reduced

207 , respectively, and 12% of the children were stunted (LAZ <-2).

208 nutrition (kwashiorkor), and 1221 (69%) were stunted (length-for-age Z score <-2).

209 and 12.1% (P = 0.856), respectively; newborn stunting (length-for-age z score < -2) was 19.2%, 14.0%,

210 tlements and infrastructures explained lower stunting levels.

211 alyzed the data for the outcome variables of stunting (low height-for-age), underweight (low weight-f

212 d from the fecal microbiota of a 6-month-old stunted Malawian infant and fed recipient animals a prot

213 Stunting malnutrition (suboptimal linear growth) also ha

214 atively hypoplastic uteri, abnormal ovaries, stunted mammary gland ductal development, and abnormal p

215 and Neto2(-/-) neurons from adult mice have stunted neurite outgrowth.

216 Despite this, stunting of children, a measure of human malnutrition, w

217 HA's interaction with M1 protein, while the stunting of fusion by deacylated HA acting in isolation

218 or metabolism disturbances (leptin, afamin), stunting of growth (growth hormone binding protein), and

219 in CTGF secretion that non-cell autonomously stunts oligodendrocyte development and decreases the tot

220 ld Bank to estimate children who were either stunted or lived in extreme poverty for 141 low-income a

221 defined as the coexistence of overweight and stunting or anemia in the same person or household.

222 en aged younger than 5 years were exposed to stunting or extreme poverty in 2004.

223 n reducing the number of children exposed to stunting or poverty between 2004 and 2010, but this is s

224 id not reduce the incidences of underweight, stunting, or wasting in Tanzanian infants.

225 n cases (95% CI 9.1 million-12.6 million) of stunting (out of 44.1 million) were attributable to it,

226 ions of the 2 conditions, the coexistence of stunting, overweight or obesity, and anemia at the natio

227 n utilization and assimilation resulted from stunted plant growth but not the complete inhibition of

228 des, Purkinje cells of the mutants displayed stunted, poorly branched dendritic arbors.

229 Animals lacking muscle ERK1/2 displayed stunted postnatal growth, muscle weakness, and a shorter

230 Mice lacking Klhl31 exhibited stunted postnatal skeletal muscle growth, centronuclear

231 ere found between low development scores and stunting, poverty, male sex, rural residence, and lack o

232 Stunting prevalence (LAZ <-2) was lower in the LNS-LNS g

233 Stunting prevalence in Pakistan's districts ranged betwe

234 Stunting prevalence remained stable at around 30% until

235 on actual supplement provided at enrollment, stunting prevalences were 8.9% compared with 15.1% and 1

236 6% (95% CI 35.4-35.9) of young children were stunted (ranging from 8.7% [7.6-9.7] in Jordan to 51.1%

237 Similar means, SDs, and stunting rates were found when less restrictive definiti

238 However, length stunting remains common.

239 digenous communities where the prevalence of stunting remains high.

240 Reducing the burden of stunting requires a paradigm shift from interventions fo

241 Zinc deficiency is closely associated with stunting, respiratory infections, diarrhea, and dermatit

242 eight mothers had a 21% higher risk of being stunted (RR: 1.21; 95% CI: 1.13, 1.30).

243 VP1 knockdown (pCoYMV::RNAiAVP1) resulted in stunted seedlings in sucrose-deprived medium.

244 al ablation of SoxC genes in vivo results in stunted sensory organs of the inner ear and loss of hair

245 y develop small nodule primordia and display stunted shoot growth, and show that the aberrant nodulat

246 ld and an overweight mother) and individual (stunting/short stature and overweight or anemia and over

247 Mutations of the molecular motor myosin 15 stunt stereocilia growth and cause deafness.

248 production-related genes were upregulated in stunted subcultures compared with the Fgmcm1 mutant, whi

249 suppressor mutations occurred frequently in stunted subcultures to recover growth rate.

250 as Fgmcm1 mutants were unstable and produced stunted subcultures, Fgmcm1 mat1-1-1 but not Fgmcm1 fst1

251 ducts, but Fsp-Cre;SmoM2 outgrowths were not stunted, suggesting that the histology might be mediated

252 In teleosts, and some others, a stunted tail is eclipsed by the upward-expanding caudal

253 weight mothers had a 5% higher risk of being stunted than those born to healthy-BMI mothers (RR: 1.05

254 rimary data to investigate the proportion of stunting that can be attributed to climate/weather varia

255 es completely inhibited tubule formation and stunted the growth of acini.

256 to a very slow decline in the prevalence of stunting, the total number of children with stunting con

257 The prevalence of stunting, thinness, and wasting was 49.2%, 27.8%, and 59

258 lted in improvements in LAZ and reduction in stunting through 3 mo of age, but not thereafter and had

259 or estimating the impact of risk factors on stunting through a mediator rather than directly on stun

260 pathway linking mycotoxin exposure to child stunting, to inform potential areas for intervention.

261 Previous work with Tomato bushy stunt tombusvirus (TBSV) in model host yeast has reveale

262 ses, have been found to inhibit Tomato bushy stunt tombusvirus (TBSV) replication in a Saccharomyces

263 y 50-70% in alpha(M)(-/-) mice, resulting in stunted tumor growth as compared with WT mice.

264 ne the associations between maternal BMI and stunting, underweight, and wasting in U5s over time.

265 0-35 months, and the outcome variables were stunting, underweight, and wasting.

266 oup.Iron deficiency, malaria, breastfeeding, stunting, underweight, inflammation, low socioeconomic s

267 dth of sequelae: hematuria, anemia, dysuria, stunting, uremia, bladder cancer, urosepsis, and human i

268 Childhood stunting usually begins in utero and continues after bir

269 differentiated poorly and exhibited severely stunted villi.

270 The VRCs built by Tomato bushy stunt virus (TBSV) are enriched with phosphatidylethanol

271 Tomato bushy stunt virus (TBSV) co-opts cellular ESCRT (endosomal sor

272 Overall, the works on Tomato bushy stunt virus (TBSV) have revealed intriguing and complex

273 med complete RNA replication of Tomato bushy stunt virus (TBSV) in yeast cell-free extracts and in pl

274 ilarly to other (+)RNA viruses, tomato bushy stunt virus (TBSV) induces major changes in infected cel

275 on, we show that replication of Tomato bushy stunt virus (TBSV) leads to the formation of double-stra

276 lication of the closely related Tomato bushy stunt virus (TBSV) or Cucumber necrosis virus (CNV) in a

277 ssay that the activation of the Tomato bushy stunt virus (TBSV) RdRp requires a soluble host factor(s

278 ethanolamine (PE) vesicle-based Tomato bushy stunt virus (TBSV) replication assay.

279 as brome mosaic virus (BMV) and tomato bushy stunt virus (TBSV).

280 did not influence the spread of Tomato bushy stunt virus and Potato virus X.

281 hanical stability of individual tomato bushy stunt virus nanoparticles (TBSV-NPs).

282 -infecting hypovirus, or p19 of tomato bushy stunt virus.

283 ants with mutated protein kinase OST1 showed stunted VPD-induced responses.

284 d analysis of surveys that measured malaria, stunting was associated with elevated AGP but not CRP in

285 Moreover, stunting was higher away from protected areas.

286 d 12.3 (95% CI: 7.7, 19.6) in those who were stunted, wasted, and underweight.

287 gregate--including fetal growth restriction, stunting, wasting, and deficiencies of vitamin A and zin

288 We estimated the prevalences of stunting, wasting, and underweight among children and of

289 tify the association between combinations of stunting, wasting, and underweight and mortality among c

290 At 19 mo of age, prevalences of stunting, wasting, and underweight were 19.8%, 6.0%, and

291 ds models were used to compare incidences of stunting, wasting, and underweight.

292 in the height-for-age z score or on rates of stunting, wasting, or underweight.

293 red metabolic pathways associated with child stunting.We measured 677 serum metabolites using liquid

294 The effects of LNS-PL on newborn stunting were greatest in infants born before a 10-wk in

295 ma infections, cellular iron deficiency, and stunting were significantly negatively associated with h

296 nal inequalities in coverage, mortality, and stunting were substantially reduced.

297 Weight loss and stunting were the main reported symptoms at diagnosis (4

298 ack of PSII SCs, mutant plants were severely stunted when cultivated with light of variable intensity

299 emperature showed weak radial swelling, were stunted with reduced root elongation, and contained redu

300 <5 y old was mildly, moderately, or severely stunted worldwide in 2010.