ライフサイエンスコーパス: subacute

1 models of Parkinsonism produced by acute or subacute 1-methy-4-phenyl-1,2,3,6-tetrahydropyridine (MP

2 eir occurrence as acute (</=24 h after PCI), subacute (1 day to 30 days), and late (30 days to 1 year

3 port on the use of acute (1 hour, n = 7) and subacute (1 week, n = 6) resting first-pass perfusion an

4 The rates of subacute (1-30 days) ST were similar with bivalirudin an

5 CVEs were classified as acute (</=24 hours), subacute (1-30 days), or late (>30 days).

6 tion and cognitive function at various acute/subacute (1-7 day post-injury) and chronic (14-60 days p

7 he early (5 weeks) increase in edema and the subacute (10 weeks) increase in fibrosis (r=0.90; P<0.00

8 lesions were detected on pmMRI (chronic: 25; subacute: 16; acute: 30; and peracute: 53).

9 ionts, we investigated acute (7 to 10 days), subacute (2 to 3 weeks), and chronic (4 to 6 weeks) phas

10 Limited data for treatment of subacute (2 to 6 weeks after onset) type B aortic dissec

11 antation intervals: acute (</=30 days; n=7), subacute (31 to 90 days; n=23), chronic (91 to 300 days;

12 (>30 days; HR, 0.48; 95% CI, 0.24-0.96) and subacute (4 hours-30 days; HR, 0.60; 95% CI, 0.39-0.93)

13 Subacute (48 hours) exposure to bifenthrin commencing 2

14 radiography of radioligands was performed at subacute (5-6 d) and chronic (40-42 d) time points after

15 or cells and two treatment schedules, either subacute (7 days) or early chronic (21 days) neural stem

16 .2%-77.5%]) even when symptoms were acute or subacute (89.0% [95% CI, 81.0%-93.9%]).

17 itis) or by prominent barrier abnormalities (subacute allergic contact dermatitis, atopic dermatitis)

18 ration in the lung parenchyma in response to subacute alveolar hypoxia.

19 Patients presenting with subacute amnesia are frequently seen in acute neurologic

20 the stent implantation was 8.9+/-8.5 days in subacute and 152.7+/-100.4 days in late thrombosis cases

21 e predictors and patient stratifiers in both subacute and chronic clinical trials.

22 riable clinical manifestations indicative of subacute and chronic disease.

23 C) disaster has been associated with several subacute and chronic health effects, but whether excess

24 Importantly, subacute and chronic HP may mimic several interstitial l

25 i-implantitis was in most cases a mixture of subacute and chronic inflammation dominated by plasma ce

26 ention had a sustained effect on troublesome subacute and chronic low-back pain at a low cost to the

27 d cine MR images allows for the diagnosis of subacute and chronic MI with high accuracy.

28 re analysis (TA) allows for the diagnosis of subacute and chronic myocardial infarction (MI) on nonco

29 Subacute and chronic myocardial infarction could be dete

30 Effect sizes for subacute and chronic neurobehavioral endpoints in other

31 al changes (P-Tau and GFAP induction) in the subacute and chronic phase as well as development of chr

32 on of leukocytes and macrophages, but in the subacute and chronic phases of injury the glial scar is

33 yperacute setting, and superior to CT in the subacute and chronic settings.

34 revealed increased anxiety-like behavior at subacute and chronic time-points, respectively.

35 Patients with subacute and Discoid Lupus Erythematous had elevated Int

36 history of infantile liver involvement, and subacute and remitting course simulating multiple sclero

37 the hippocampus is rapid and severe, but is subacute and significantly slower in the ipsilateral sep

38 Chronic, subacute, and acute infarction cases correlated excellen

39 3-T pmMRI visualizes chronic, subacute, and acute myocardial infarction in situ.

40 Medical treatment of uncomplicated acute, subacute, and chronic type B aortic dissection is manage

41 oth imaging modalities at each stage (acute, subacute, and chronic) and it is important that physicia

42 ecular pathways that contribute to early and subacute axonal degeneration after stroke.

43 In a longitudinal brain imaging study, subacute back pain (SBP) patients were followed over the

44 n activity for back pain in the early, acute/subacute back pain group is limited to regions involved

45 hed in time, whereas in the persistent acute/subacute back pain group, activity diminished in acute p

46 In the recovered acute/subacute back pain group, brain activity diminished in t

47 istory of back pain for 1 year (early, acute/subacute back pain group, n = 94), to subjects who have

48 We tracked brain properties in subacute back pain patients longitudinally for 3 years a

49 In a subset of subacute back pain patients, we followed brain activity

50 or children with attention deficit disorder, subacute bacterial endocarditis prophylaxis and follow-u

51 teins as those recognized by sera from acute/subacute, blood culture-positive brucellosis patients bu

52 The results suggest that in a subacute canine infarct model, the LBs are a source of a

53 itted infection etiologies in this community subacute care setting.

54 transfer of undocumented patients in need of subacute care to their country of origin.

55 chy areas of air trapping, are seen in acute/subacute cases, whereas reticular opacities, volume loss

56 the differential diagnosis of patients with subacute cerebellar ataxia of unknown cause.

57 and traction bronchiectasis superimposed on subacute changes are observed in chronic cases.

58 ly and invasively, lesions developing in the subacute-chronic phase can be managed with medical treat

59 ute self-limited necrotizing arteritis (NA), subacute/chronic (SA/C) vasculitis, and luminal myofibro

60 Our results reveal that subacute/chronic axon loss induced by vincristine occurs

61 e form and by Th1 and likely Th17 T cells in subacute/chronic cases.

62 to the injured spinal cord parenchyma at the subacute/chronic phase.

63 uced peripheral neuropathy, is the result of subacute/chronic processes that may be regulated differe

64 abnormalities are observed in the acute and subacute/chronic stages.

65 erosis (MS) in 85% of young adults is with a subacute clinically isolated syndrome (CIS) of the optic

66 lts with multiple sclerosis (MS), onset is a subacute clinically isolated syndrome (CIS) of the optic

67 r antagonists have therapeutic potential for subacute CNS axonal injuries such as spinal cord trauma.

68 e industrial exposure have been implemented, subacute cobalt-related cardiomyopathy had become rare.

69 clinical symptoms in each of the seven were subacute cognitive decline or seizure rather than hemorr

70 tandard deviations were generally <10% at CW subacute concentrations levels.

71 logy can detect a wide range of compounds at subacute concentrations with no sample preparation and a

72 vated intracranial pressure in children with subacute conditions, such as craniosynostosis or tumor,

73 t ST-segment shifts associated with acute or subacute coronary occlusion in a porcine model.

74 emperature >38 degrees C [>100.4 degrees F], subacute course, large effusion or tamponade, and failur

75 ents with lupus erythematosus (LE) including subacute cutaneous lupus erythematosus (SCLE) and discoi

76 Cutaneous neonatal lupus resembles subacute cutaneous lupus erythematosus (SCLE), and photo

77 rapeutic agents have been reported to induce subacute cutaneous lupus erythematosus (SCLE).

78 Evaluation of discoid and subacute cutaneous lupus erythematosus lesions showed si

79 c valve implantation, the rates of acute and subacute CVEs were 2.7% and 2.4%, respectively.

80 2.76; 95% CI, 1.11-6.83) was a predictor of subacute CVEs.

81 largely was attributable to a lower risk of subacute definite stent thrombosis: 0.1% versus 0.6% (ra

82 An acute or subacute demyelinating polyneuropathy should be consider

83 All eight had an acute or subacute deterioration following a long asymptomatic or

84 ed blood-brain barrier (BBB) was involved in subacute diaschisis.

85 Classical AAG is a subacute disorder associated with antibodies against gan

86 Relative to TAA without dissection, acute or subacute dissection (OR=2.9; 95% CI, 1.6-5.3), but not c

87 hronic dissection and 109 (10%) had acute or subacute dissection.

88 ngin (MFG) for the treatment of experimental subacute disseminated candidiasis.

89 The clinical presentation was subacute during a period of weeks in 13 patients (38%).

90 g; CCC of 0.995 and 95% CI: 0.993, 0.997 for subacute DW imaging).

91 n children with brain tumors, or a transient subacute effect characterized by an intralesional or per

92 , and most patients had repeated episodes of subacute encephalopathy with motor regression, irritabil

93 minent clinical manifestations are headache, subacute encephalopathy, optic papillitis, inflammatory

94 rder with childhood onset that presents as a subacute encephalopathy, with confusion, dysarthria, and

95 METHODS AND We used subacute endotoxemia in the rodent macrophage-to-feces R

96 (4.4%), including 28 acute events (0.9%), 49 subacute events (1.6%), 32 late events (1.0%), and 33 ve

97 al fibrillation determined a higher risk for subacute events.

98 Subacute exposure (19 d after initiating ascent to Evere

99 elper T-cell-type inflammatory response, and subacute fibrosis were recognized.

100 Acute/subacute flumazenil positron emission tomography studies

101 with the ail mutant resulted in an atypical, subacute form of bubonic plague associated with extensiv

102 are all moderately effective for chronic or subacute (>4 weeks' duration) low back pain.

103 Subacute, hematogenous, and extrapulmonary disease tends

104 inhibition (P = 0.021, n = 5/group) without subacute hematologic toxicity (n = 3/group).

105 Differences in the subacute hemodynamic effects of amlodipine and lisinopri

106 hat lipid accumulation in the liver leads to subacute hepatic 'inflammation' through NF-kappaB activa

107 HIT antibodies indicates that patients with subacute HIT undergoing repeated TPE before heparin reex

108 ts who have serologically-confirmed acute or subacute HIT; for this situation, a negative platelet ac

109 rial serum samples in a patient with recent (subacute) HIT who underwent serial TPE precardiac surger

110 (nGD), a mouse model (4L;C*), an analogue to subacute human nGD, was investigated for global profiles

111 rgical lung biopsy that were consistent with subacute hypersensitivity pneumonitis (and IgG positive)

112 We suggest that during subacute hypoxia, mitochondria might be protected from o

113 ed into 3 phases: the acute viral phase, the subacute immune phase, and the chronic cardiac remodelin

114 The early increase in edema and subacute increase in fibrosis are strongly linked and ar

115 phalopathy, metachromatic leukodystrophy and subacute infarct.

116 (VSMC) were injected into the border zone of subacute infarcted syngeneic Fischer rat hearts and comp

117 ster urethane urea (PEUU) cardiac patch onto subacute infarcts and to examine the resulting cardiac v

118 Subacute infection was more frequent (median, 4 weeks [r

119 ligaments, and is also implicated in causing subacute infective endocarditis in humans.

120 Streptococcus oralis is a leading cause of subacute infective endocarditis.

121 We have matched this state of chronic, subacute 'inflammation' by low-level activation of NF-ka

122 expansion in the liver and can precipitate a subacute inflammatory hepatitis with stellate cell activ

123 etwork over-stimulation that may explain the subacute inflammatory presentation.

124 ntribute to endogenous protection during the subacute injury phase.

125 ificant inflammation, indicative of acute or subacute injury.

126 ular, patients with BVS had a higher risk of subacute, late, and very late ST, whereas the risk of ac

127 Of note, BVS had a higher risk of subacute, late, and very late ST, whereas the risk of TL

128 r parietal cortex in patients suffering from subacute left spatial neglect, in a randomized, double-b

129 d false negative rates for concentrations at subacute levels.

130 six from a single institution, who developed subacute limbic encephalitis initially considered of unc

131 ting neutrophils in 25 patients with ALF and subacute liver failure (SALF).

132 utants show a marked susceptibility to acute/subacute liver injury.

133 evidence of moderate efficacy for chronic or subacute low back pain are cognitive-behavioral therapy,

134 on 1: Given that most patients with acute or subacute low back pain improve over time regardless of t

135 line approach for management of non-specific subacute low back pain in primary care.

136 ck pain, spinal manipulation; for chronic or subacute low back pain, intensive interdisciplinary reha

137 ths in patients consulting primary care with subacute low back pain.

138 ate lumbar imaging in patients with acute or subacute low-back pain and without features suggesting a

139 Results are most applicable to acute or subacute low-back pain assessed in primary-care settings

140 e the current state of managing chronic (and subacute) low back pain as reflected in recently publish

141 role for SP-D in response to noninfectious, subacute lung injury via modulation of oxidative-nitrati

142 has the potential to help reliably quantify subacute microinfarction.

143 is an important factor in several acute and subacute models of vascular injury.

144 partial sparing of striatal DA levels in the subacute MPTP model but not in the acute MPTP model.

145 because of concerns of its association with subacute myelo-optic neuropathy in Japan; therefore, any

146 market when it was linked to an epidemic of subacute myelo-optico-neuropathy (SMON).

147 Whereas increased tissue heterogeneity after subacute myocardial infarction creates a highly arrhythm

148 n of a novel biodegradable PEUU patch onto a subacute myocardial infarction promoted contractile phen

149 findings show acute myocardial ischaemia and subacute myocardial microinfarction after intracoronary

150 Necrotizing Myopathy is a poorly studied subacute myopathy triggered by toxic, viral, or autoimmu

151 For participants with acute and subacute neck pain, SMT was more effective than medicati

152 to guide the choice of therapy for acute and subacute neck pain.

153 Leigh syndrome (LS) is a subacute necrotizing encephalomyelopathy with gliosis in

154 ng normal liver function and Leigh syndrome (subacute necrotizing encephalomyelopathy) seen in associ

155 nts without neglect, the group including all subacute neglect patients had decreased fractional aniso

156 rial disease is Leigh syndrome, an episodic, subacute neurodegeneration that can lead to death within

157 receptor was examined in vivo in a model of subacute neuroinflammation induced by administration of

158 an with erosive rheumatoid arthritis in whom subacute neurologic and psychiatric symptoms developed a

159 All patients but one presented with subacute neurological deterioration in infancy or childh

160 nts with this disorder present with acute or subacute neurological deterioration, with MRI showing on

161 sera from approximately 75000 patients with subacute neurological disorders that were suspected to b

162 All patients had manifested acute or subacute neurological symptoms; the brain MRIs indicatin

163 Between November, 2006, and May, 2008, a subacute neurological syndrome affected workers from two

164 ed as a possible cause of diffuse unilateral subacute neuroretinitis in humans.

165 al signs and diagnosis of diffuse unilateral subacute neuroretinitis in its early stage, followed by

166 with early- or late-stage diffuse unilateral subacute neuroretinitis were included.

167 es the strongest correlation with a score of subacute on modified Rankin scale at discharge.

168 e teenage years and is often associated with subacute onset and marked muscle inflammation.

169 The patient developed subacute onset of bilateral blindness following his 44th

170 Predictors of clinical response included subacute onset of CA (odds ratio [OR], 0.50; 95% CI, 0.2

171 a-myalgia syndrome (EMS) is characterized by subacute onset of myalgias and peripheral eosinophilia,

172 rolizumab or nivolumab), 10 (2.9%) developed subacute onset of neurological complications.

173 ere, we describe six HMSN VI families with a subacute onset of optic atrophy and subsequent slow reco

174 p of disorders characterised by the acute-to-subacute onset of painful sensory and motor deficits tha

175 Patients present with the subacute onset of severe heart failure, which is accompa

176 In patients with CA and GAD65-Abs, subacute onset of symptoms and prompt immunotherapy are

177 gth dependence, motor predominance, acute or subacute onset, and prominent autonomic involvement shou

178 Atypical neuropathy features, such as acute/subacute onset, asymmetry, and/or motor predominant sign

179 or an asymmetric polyneuropathy of acute or subacute onset.

180 ted at age 7 years with cerebellar ataxia of subacute onset.

181 Four patients had a subacute-onset and sensory ataxia.

182 bulatory visits were primarily for acute- or subacute-onset diplopia.

183 Symptomatic disease is subacute or chronic and diverse in presentation and outc

184 2) or small (enhanced volume </=20%, n = 48) subacute or chronic ischemic scars were included.

185 aken in England, 701 adults with troublesome subacute or chronic low-back pain were recruited from 56

186 ng repair and restoration of function in the subacute or chronic phase after stroke.

187 elpful in evaluating ongoing symptoms in the subacute or chronic setting.

188 ng G-CSF in patients with hyperacute, acute, subacute or chronic stroke, and asked Investigators to s

189 emisphere, or bilateral; (2) recovery stage: subacute or chronic stroke; (3) stimulation timing: tDCS

190 traneurologic morbidity, particularly in the subacute or chronic variants.

191 canalization used in the treatment of acute, subacute or even chronic occlusions or stenosis of perip

192 No myocardial infarction, subacute or late thrombosis, or death occurred.

193 HP may present as acute, subacute, or chronic clinical forms but with frequent ov

194 tient population includes adults with acute, subacute, or chronic low back pain.

195 mplantation regardless of the timing (acute, subacute, or late).

196 eart structures may be associated with worse subacute outcomes but typically normalize within 1 year.

197 separate groups of dogs with pacing-induced subacute overt HF complicated by renal dysfunction.

198 In contrast, subacute ozone exposure did not enhance inflammation in

199 superior longitudinal fasciculus II/III for subacute patients and in its caudal portion for chronic

200 81.5% (22 of 27 patients) in the context of subacute pelvic pain for readers 1, 2, and 3, respective

201 All patients with acute or subacute pelvic pain who were undergoing MR examination

202 ients who have an adnexal mass with acute or subacute pelvic pain.

203 Reperfusion was assessed using subacute perfusion MRI coregistered to baseline imaging.

204 ive phase of axonal injury occurs during the subacute period and damages axons that survive the initi

205 r how PMO, a phenomenon limited to the acute/subacute period of MI, drives adverse remodeling in chro

206 tive testing for neglect twice, first at the subacute phase (<3 months after onset) and then at the c

207 levels while GGA1 levels are restored in the subacute phase (7 d) after injury.

208 ), the ADBR was greater than the ADIR in the subacute phase (LSMD: -0.39%; 95% CI: -0.58% to -0.20%;

209 y occluded infarct-related artery during the subacute phase after myocardial infarction compared with

210 and without severe inducible ischemia in the subacute phase after myocardial infarction.

211 ous-scaly as the condition progresses to the subacute phase and papular-hyperkeratotic in the chronic

212 Our results showed that the subacute phase of cerebral infarction in patients was ch

213 , the effect of IRL-1620 is not known in the subacute phase of cerebral ischemia, where development o

214 damage and depletion in the liver during the subacute phase of CLP sepsis.

215 d BACE1 solely depends on GGA3 levels in the subacute phase of injury.

216 We conclude that in the subacute phase of myocardial infarction, the peri-infarc

217 and in vivo regulation of miRNAs during the subacute phase of stroke has not yet been proposed.

218 nying motor abnormalities at least up to the subacute phase, advances this cortical hemorrhage model

219 terial soft tissue injury is greatest in the subacute phase, and least in the chronic phase, suggesti

220 or evaluating infarct growth and size in the subacute phase.

221 We sequenced tissue samples from acute and subacute phases (2 days and 7 days after injury) and sys

222 raction between MA and mTBI during acute and subacute phases after injury.

223 the bifurcation point at both the acute and subacute phases.

224 n presented to the emergency department with subacute poorly localized abdominal pain that was increa

225 This negative regulation was mediated by subacute postseizure increases in mammalian target of ra

226 Subacute potentially catastrophic illnesses in three div

227 Patients in ICUs are susceptible to subacute potentially catastrophic illnesses such as resp

228 Acute/subacute presentations accounted for the majority of pri

229 The natural history of LHON is not a subacute process, as previously believed, but progresses

230 , newer understandings suggest that acute or subacute processes are involved, and with proper diagnos

231 The subacute response exhibited granulation tissue with earl

232 ntrast to its remarkable effect on acute and subacute SCI.

233 Subacute sclerosing panencephalitis (SSPE) is a fatal co

234 Subacute sclerosing panencephalitis (SSPE) is a progress

235 severe sequela of measles virus infection is subacute sclerosing panencephalitis (SSPE), a fatal dise

236 measles virus infection of the brain causes subacute sclerosing panencephalitis (SSPE), a progressiv

237 essive fatal neurodegenerative complication, subacute sclerosing panencephalitis (SSPE), occurs durin

238 lasma cells from the brain of a patient with subacute sclerosing panencephalitis (SSPE).

239 lasma cells from the brain of a patient with subacute sclerosing panencephalitis, and single-cell RT-

240 assay in 7 of 14 cases with MS and 1 case of subacute sclerosing panencephalitis, but not in IgG from

241 IgGs recognized measles virus, the cause of subacute sclerosing panencephalitis.

242 s the in vivo response to acute (sepsis) and subacute (skin) inflammatory challenge.

243 In patients with subacute spinal cord injury (SCI), the motor system unde

244 uncovered struts and underexpansion in acute/subacute ST and neoatherosclerosis and uncovered struts

245 and out-of-hospital ST groups indicated that subacute ST had the highest mortality.

246 T was uncovered struts (66.7% of cases); for subacute ST, the most common dominant finding was uncove

247 ussive blast injury (n = 50) in the acute to subacute stage and combat-deployed control individuals (

248 ncy and CR2-fH treatment is sustained in the subacute stage of infarct development, adding to the cli

249 al hemorrhage (ICH) at hyperacute, acute and subacute stages by comparing with susceptibility weighte

250 singly viewed as a new therapeutic target in subacute stages of brain infarction.

251 RI could detect ICH at hyperacute, acute and subacute stages.

252 intensities of ICH at hyperacute, acute and subacute stages.

253 r for detecting ICH at hyperacute, acute and subacute stages.

254 A chronic, subacute state of inflammation often accompanies the acc

255 flammation can be better differentiated in a subacute state.

256 death (26 [3.5%] vs 18 [1.7%]; P = .01), and subacute stent thrombosis (8 [1.1%] vs 3 [0.3%]; P = .03

257 e investigated whether patients who suffered subacute stent thrombosis (SAT) have higher post-treatme

258 alences of successful reperfusion, acute and subacute stent thrombosis, 30-day death or combined endp

259 independent predictors of reduced acute and subacute stent thrombosis, respectively.

260 1), with nonsignificantly different rates of subacute stent thrombosis.

261 s following other disasters, suggesting that subacute stress may have served to promote this arrhythm

262 Forty patients with subacute stroke (3-14 days after neurological symptom on

263 Here, for the first time, a large sample of subacute stroke patients (N = 667) was assessed without

264 nt (BOLD) data in detecting hypoperfusion of subacute stroke patients through comparison with dynamic

265 ve to DSC-PWI for detecting hypoperfusion in subacute stroke patients who had obvious MRA lesions wit

266 Twelve subacute stroke patients with a subcortical ischemic les

267 Twenty-eight hospitalized subacute stroke patients with severe motor deficits were

268 ficantly better motor functional outcomes in subacute stroke patients with severe motor impairments.

269 ed in a randomized controlled pilot study in subacute stroke patients.

270 pharmacological blockade of the NgR promotes subacute stroke recovery by facilitating axonal plastici

271 tidepressants may provide a novel target for subacute stroke therapy.

272 cy medical services; acute stroke treatment; subacute stroke treatment and secondary prevention; reha

273 cular ischemic events (acute stroke, n = 20; subacute stroke, n = 2; chronic stroke, n = 3; transient

274 ubsequent MTX without recurrence of acute or subacute symptoms.

275 es (n = 16), acute ataxia (n = 4), and other subacute syndromes (transverse myelitis [n = 1], opsoclo

276 traumatic cervical spinal cord lesions in 24 subacute tetraplegic patients.

277 yocardial infarction (3.5% versus 4.7%), and subacute thrombosis (0.6% versus 0.8%) were similar betw

278 , myocardial infarction (3.2% vs. 6.4%), and subacute thrombosis (0.6% vs. 1.2%) were comparable betw

279 Administration recently issued a warning of subacute thrombosis and hypersensitivity reactions to si

280 olecular" MRI for the detection of acute and subacute thrombosis using a novel fibrin-binding MRI con

281 e feasibility of direct imaging of acute and subacute thrombosis using MRI together with a novel fibr

282 an animal model of atherosclerosis and acute/subacute thrombosis.

283 of the work presented here was to develop a subacute to chronic caprine model of melioidosis and to

284 Most of these diseases have a subacute to chronic course and they can become recalcitr

285 op systemic acute infections that can become subacute to chronic infections.

286 d be restricted to patients with progressive subacute to chronic neurological symptoms due to congest

287 ecovery by promoting brain plasticity in the subacute to chronic phase after ischaemic stroke.

288 spontaneous partial recovery of functions in subacute to chronic phases.

289 e to the diffusion tensor imaging changes in subacute to chronic traumatic brain injury.

290 echniques, particularly in the assessment of subacute to early chronic stroke evolution.

291 ) and 85.1% (23 of 27 patients) in acute and subacute torsion, respectively.

292 n hypothesized that cytoplasmic dopamine has subacute toxic effects in Parkinson Disease (PD) leading

293 quires further careful assessment, acute and subacute toxicities are generally acceptable.

294 se anticonvulsant studies, being nontoxic in subacute toxicity studies.

295 Only the subacute transplant of neural stem/precursor cells enhan

296 e imaging (MRI) protocol to 14 patients with subacute traumatic SCI at baseline, 2 months, 6 months,

297 Fifteen had subacute vision loss, swollen optic discs, and field def

298 We report the occurrence of subacute WMLs detected by magnetic resonance imaging (MR

299 Subacute zinc deficiency significantly increases systemi

300 We hypothesized that subacute zinc deficiency would amplify immune responses