ライフサイエンスコーパス: subarachnoid

1 brain and spinal cord, as well as aneurysm, subarachnoid and cerebral haemorrhage, carotid dissectio

2 of the model showed severe brain injury with subarachnoid and hemorrhage together with glial cell act

3 as associated with poor clinical grade, more subarachnoid and intraventricular blood seen on admissio

4 The importance of convective flow in the subarachnoid and paravascular spaces has long been recog

5 venteen neonates with ICHs (16 subdural, two subarachnoid, and six parenchymal hemorrhages) were iden

6 Previous analyses of the International Subarachnoid Aneurysm Trial (ISAT) cohort have reported

7 eurysm in the follow-up of the International Subarachnoid Aneurysm Trial (ISAT).

8 (2) The International Subarachnoid Aneurysm Trial compared endovascular and su

9 ng of the applicability of the International Subarachnoid Aneurysm Trial data and the roles of surgic

10 Intracranial Aneurysms and the International Subarachnoid Aneurysm Trial have provided data on the na

11 uptured Aneurysm Trial and the International Subarachnoid Aneurysm Trial II, are aimed at improving u

12 hemorrhage developed from the International Subarachnoid Aneurysm Trial in a retrospective unselecte

13 an observed probabilities, the International Subarachnoid Aneurysm Trial prediction model needs to be

14 n continues to emerge from the International Subarachnoid Aneurysm Trial, with a neuropsychological s

15 Our results demonstrate that subarachnoid blood causes a delayed increase in the ampl

16 tin, could inhibit the vasospasm provoked by subarachnoid blood in a rat subarachnoid hemorrhage mode

17 Subarachnoid hemorrhage, defined as (1) subarachnoid blood on computed tomography scan; (2) xant

18 We report that subarachnoid blood profoundly impacts communication with

19 ampled CSF for 6 hours through an indwelling subarachnoid catheter in PTSD patients before, during, a

20 These results suggest that subarachnoid clots in sulci/fissures are sufficient to i

21 innervation and function that correlate with subarachnoid CN3 hypoplasia, occasional abducens nerve h

22 nd the associated inflammatory milieu in the subarachnoid compartment plays a role in the pathogenesi

23 d exits through paravenous spaces again into subarachnoid compartments.

24 ne in the efficiency of exchange between the subarachnoid CSF and the brain parenchyma.

25 king transforming growth factor-beta-induced subarachnoid fibrosis and protected against hydrocephali

26 nal fluid flow and drainage is obstructed by subarachnoid fibrosis in which the potent fibrogenic cyt

27 minal aortic aneurysm (0.46 [0.35-0.59]) and subarachnoid haemorrhage (0.48 [0.26-0.89]), and not ass

28 ominal aortic aneurysm (0.46, 0.35-0.59) and subarachnoid haemorrhage (0.48, 0.26-0.89).

29 hage (hazard ratio 1.44 [95% CI 1.32-1.58]), subarachnoid haemorrhage (1.43 [1.25-1.63]), and stable

30 egree relative affected (FDRA) by aneurysmal subarachnoid haemorrhage (aSAH) are at a higher lifetime

31 erebral vasospasm (cVSP) in human aneurysmal subarachnoid haemorrhage (aSAH).

32 INTRODUCTION: Acute non-traumatic convexity subarachnoid haemorrhage (cSAH) is increasingly recognis

33 rial hypertension (HTN) is a risk factor for subarachnoid haemorrhage (SAH).

34 ogical deterioration occurs frequently after subarachnoid haemorrhage (SAH).

35 aemic), transient ischaemic attack (TIA) and subarachnoid haemorrhage (SAH).

36 t plays important roles in migraine, stroke, subarachnoid haemorrhage and brain injury.

37 hort have reported on the risks of recurrent subarachnoid haemorrhage and death or dependency for a m

38 with confirmatory evidence of an aneurysmal subarachnoid haemorrhage and presenting less than 96 h f

39 of global as opposed to focal deficits after subarachnoid haemorrhage and traumatic brain injury in h

40 pants in six prospective cohort studies with subarachnoid haemorrhage as outcome.

41 Patients with subarachnoid haemorrhage benefit from multidisciplinary

42 e complex treatment strategies applied after subarachnoid haemorrhage call for interdisciplinary coll

43 The management of patients with aneurysmal subarachnoid haemorrhage demands expertise to anticipate

44 The prognosis of subarachnoid haemorrhage depends on the severity of the

45 how that axonal injury also occurs following subarachnoid haemorrhage in an animal model.

46 Here we studied acute sequelae of subarachnoid haemorrhage in the gyrencephalic brain of p

47 Aneurysmal subarachnoid haemorrhage is a potentially devastating di

48 of statins in patients with acute aneurysmal subarachnoid haemorrhage is unclear.

49 33 patients had a recurrent subarachnoid haemorrhage more than 1 year after their in

50 ety concerns, we conclude that patients with subarachnoid haemorrhage should not be treated routinely

51 Prognosis seems worse in patients with subarachnoid haemorrhage than in those without.

52 Most patients with subarachnoid haemorrhage undergo surgical or endovascula

53 ated ipsilateral basal ganglia bleeding with subarachnoid haemorrhage with no aetiology is uncommon.

54 tudy (cardiac arrest, pneumonia, sepsis, and subarachnoid haemorrhage), none were deemed treatment re

55 Subarachnoid haemorrhage, a particularly deadly form of

56 ars or older with hypertension, a history of subarachnoid haemorrhage, and a giant-sized (>20 mm) pos

57 schaemia (DCI) which occurs after aneurysmal subarachnoid haemorrhage, and often leads to cerebral in

58 redictors were age, hypertension, history of subarachnoid haemorrhage, aneurysm size, aneurysm locati

59 cal clipping or endovascular coiling after a subarachnoid haemorrhage, assuming treatment equipoise,

60 nts with ischaemic and haemorrhagic strokes, subarachnoid haemorrhage, cerebrovascular malformations,

61 e intensive care management of patients with subarachnoid haemorrhage, emphasizing the detection and

62 atment (one retroperitoneal haemorrhage, one subarachnoid haemorrhage, one respiratory distress, and

63 can present with headache, ischaemic stroke, subarachnoid haemorrhage, or symptoms associated with ma

64 s also a functionally significant feature of subarachnoid haemorrhage, raising the prospect of common

65 With the exception of subarachnoid haemorrhage, there is little evidence of se

66 TCH might be the first sign of subarachnoid haemorrhage, unruptured intracranial aneury

67 human brain injured by trauma or aneurysmal subarachnoid haemorrhage, we used DC electrode recording

68 is also a prominent feature of experimental subarachnoid haemorrhage.

69 mmon in poor-grade patients after aneurysmal subarachnoid haemorrhage.

70 ort-term outcome in patients with aneurysmal subarachnoid haemorrhage.

71 antiplatelet therapy or in the setting of a subarachnoid haemorrhage.

72 neurological injury from trauma, stroke and subarachnoid haemorrhage.

73 rebral vasospasm in patients with aneurysmal subarachnoid haemorrhage; (4) the use in the biomechanic

74 n-Hispanic black patients aged 45 to 54 with subarachnoid hemorrhage (13.2/10000 to 10.3/10000 hospit

75 c stroke (19%), 936 ventilated patients with subarachnoid hemorrhage (32%), and 1,404 ventilated pati

76 Of 383 patients enrolled, there were 128 subarachnoid hemorrhage (33.4%), 134 subdural hematoma (

77 The most common diagnoses were subarachnoid hemorrhage (38%), intracerebral hemorrhage

78 Patients with coma attributable to subarachnoid hemorrhage (4/80; 5%) or global hypoxic-isc

79 and 131 met CT criteria for PM nonaneurysmal subarachnoid hemorrhage (53 women; mean age, 53 years [r

80 and unstable angina, and inverse (0.69) for subarachnoid hemorrhage (all P<0.001).

81 ribute to poor outcomes following aneurysmal subarachnoid hemorrhage (aSAH).

82 al hemorrhage (HR, 1.9; 95% CI, 1.5-2.4) and subarachnoid hemorrhage (HR, 2.4; 95% CI, 1.7-3.5) than

83 (odds ratio [OR], 2.494), the indication of subarachnoid hemorrhage (OR, 2.523), and the comorbidity

84 hemorrhage (RR: 0.96; 95% CI: 0.84, 1.10) or subarachnoid hemorrhage (RR: 1.01; 95% CI: 0.90, 1.14).

85 Subarachnoid hemorrhage (SAH) carries a 50% mortality ra

86 Patients with subarachnoid hemorrhage (SAH) frequently develop delayed

87 Cerebral vasospasm following aneurysmal subarachnoid hemorrhage (SAH) has devastating consequenc

88 in acute brain injury but an involvement in subarachnoid hemorrhage (SAH) has not been investigated.

89 mic neurological damage following aneurysmal subarachnoid hemorrhage (SAH) have remained elusive.

90 -OPN) could prevent cerebral vasospasm after subarachnoid hemorrhage (SAH) in rats.

91 Acute aneurysmal subarachnoid hemorrhage (SAH) is a complex multifaceted

92 Aneurysmal subarachnoid hemorrhage (SAH) is a potentially devastati

93 gy of early ischemic injury after aneurysmal subarachnoid hemorrhage (SAH) is not understood.

94 soconstriction in brain slices obtained from subarachnoid hemorrhage (SAH) model rats.

95 rospective observational study of aneurysmal subarachnoid hemorrhage (SAH) patients, we explored the

96 eficit (DIND) contributes to poor outcome in subarachnoid hemorrhage (SAH) patients.

97 Aneurysmal subarachnoid hemorrhage (SAH) remains a devastating cond

98 Subarachnoid hemorrhage (SAH) results in significant ner

99 ness (LOC) is a common presenting symptom of subarachnoid hemorrhage (SAH) that is presumed to result

100 Subarachnoid hemorrhage (SAH) usually results from ruptu

101 stoperative ICU management of patients after subarachnoid hemorrhage (SAH), especially with regards t

102 m is usually associated with the presence of subarachnoid hemorrhage (SAH), SAH is not required for v

103 Significance statement: Aneurysmal subarachnoid hemorrhage (SAH)--strokes involving cerebra

104 re accounts for the majority of nontraumatic subarachnoid hemorrhage (SAH).

105 us, especially in the early period following subarachnoid hemorrhage (SAH).

106 merous neuropathological processes including subarachnoid hemorrhage (SAH).

107 More than 30% of patients with RCVS develop subarachnoid hemorrhage (SAH).

108 ere and pervasive consequences of aneurysmal subarachnoid hemorrhage (SAH).

109 data regarding anemia and transfusion after subarachnoid hemorrhage (SAH).

110 Medical complications occur frequently after subarachnoid hemorrhage (SAH).

111 The aggregate traumatic subarachnoid hemorrhage (tSAH) component of the Stockhol

112 patients: adjusted odds ratios (95% CI) for subarachnoid hemorrhage 0.17 (0.06-0.45) and intracerebr

113 % vs. 32%), and more intracranial pathology (subarachnoid hemorrhage 62% vs. 44%; intraparenchymal le

114 Retrospective analysis of all subarachnoid hemorrhage admissions.

115 ology of types of stroke, such as aneurysmal subarachnoid hemorrhage and cerebral vein thrombosis, th

116 gh intracranial pressure peaked 3 days after subarachnoid hemorrhage and declined after day 7.

117 protective against early brain injury after subarachnoid hemorrhage and determined whether this effe

118 seizure like activity found to have diffuse subarachnoid hemorrhage and extensive dural venous sinus

119 rebral vasospasm in patients with aneurysmal subarachnoid hemorrhage and for guiding transfusion ther

120 Conclusion In patients with PM nonaneurysmal subarachnoid hemorrhage and initial DSA negative for ane

121 Hemorrhagic stroke (HS), ie, subarachnoid hemorrhage and intracerebral hemorrhage, is

122 O administered for 8h improved recovery from subarachnoid hemorrhage and reduced the inflammatory res

123 ocity were improved between acute aneurysmal subarachnoid hemorrhage and stable state (p </= .005); c

124 Subarachnoid hemorrhage and surgical obliteration of rup

125 Recent findings in both subarachnoid hemorrhage and traumatic brain injured pati

126 ever is sustained for longer durations after subarachnoid hemorrhage and traumatic brain injury.

127 lactate and glucose levels after aneurysmal subarachnoid hemorrhage are associated with delayed cere

128 chemic stroke, intracerebral hemorrhage, and subarachnoid hemorrhage as defined by validated diagnosi

129 2000 to 2013, 252 consecutive patients with subarachnoid hemorrhage at computed tomography (CT) and

130 All patients with a diagnosis of subarachnoid hemorrhage between 2009 and 2014.

131 redict 60-day case fatality after aneurysmal subarachnoid hemorrhage developed from the International

132 rted on a case of a 16-year-old patient with subarachnoid hemorrhage diagnosed due to a ruptured cere

133 dministration of sodium nitrite after severe subarachnoid hemorrhage differentially influences quanti

134 However, patients with subarachnoid hemorrhage due to rupture of a dissecting a

135 nterventions for intracerebral hemorrhage or subarachnoid hemorrhage generally hinge on whether they

136 nt ameliorating early brain injury following subarachnoid hemorrhage has been nonexistent.

137 The care of patients with aneurysmal subarachnoid hemorrhage has evolved significantly with t

138 The indication for DSA was subarachnoid hemorrhage in 71 patients (87.6%), stroke o

139 ix glycoprotein, on early brain injury after subarachnoid hemorrhage in rats.

140 common complication in the first week after subarachnoid hemorrhage in severe cases admitted to ICU.

141 e young age and high prevalence of traumatic subarachnoid hemorrhage in this cohort may limit its gen

142 One hour after subarachnoid hemorrhage induction via endovascular perfo

143 eated with an equal volume (1 microL) of pre-subarachnoid hemorrhage intracerebroventricular administ

144 l role in the development of vasospasm after subarachnoid hemorrhage is accumulating.

145 s primary cerebral vasculitis and aneurysmal subarachnoid hemorrhage is common because of overlapping

146 imal timing of tracheostomy in patients with subarachnoid hemorrhage is controversially debated.

147 that the primary cause of poor outcome after subarachnoid hemorrhage is not only cerebral arterial na

148 Thus, subarachnoid hemorrhage may constitute an important exce

149 Argon application after experimental subarachnoid hemorrhage met the primary endpoint of redu

150 pasm provoked by subarachnoid blood in a rat subarachnoid hemorrhage model.

151 ssociated with inferior outcomes, to compare subarachnoid hemorrhage mortality with other neurologica

152 tion with isoflurane in patients with severe subarachnoid hemorrhage not having intracranial hyperten

153 Aneurysmal subarachnoid hemorrhage often leads to death and poor cl

154 defined as the presence of intracerebral or subarachnoid hemorrhage on computed tomography or magnet

155 als were euthanized 6, 24, or 72 hours after subarachnoid hemorrhage or sham surgery.

156 Subarachnoid hemorrhage or sham-operated rats were treat

157 Subarachnoid hemorrhage patients admitted to ICU in Aust

158 Pregnant subarachnoid hemorrhage patients also had a higher likel

159 ly and particularly to vulnerable regions in subarachnoid hemorrhage patients at risk for delayed cer

160 omparison with other neurological diagnoses, subarachnoid hemorrhage patients had significantly great

161 udy was to describe in-hospital mortality in subarachnoid hemorrhage patients requiring ICU admission

162 lity physiological recordings in 48 comatose subarachnoid hemorrhage patients to better characterize

163 A total of 11,327 subarachnoid hemorrhage patients were identified in the

164 Pregnant versus nonpregnant subarachnoid hemorrhage patients were less impaired at a

165 Nevertheless, subarachnoid hemorrhage patients who are at risk for vas

166 neurological outcome in good-grade surgical subarachnoid hemorrhage patients, as assessed by the Gla

167 ence of acute kidney injury in patients with subarachnoid hemorrhage patients.

168 rial in a retrospective unselected cohort of subarachnoid hemorrhage patients.

169 time points: on admission (acute aneurysmal subarachnoid hemorrhage phase) and at least 21 days late

170 determinant of outcome with intracranial or subarachnoid hemorrhage predicting a extremely high mort

171 ation rates for intracerebral hemorrhage and subarachnoid hemorrhage remained stable, with the except

172 e polycystic kidney disease, presenting with subarachnoid hemorrhage secondary to a ruptured intracra

173 ment behavioral assessments of patients with subarachnoid hemorrhage shortly after the injury.

174 Critically ill patients with subarachnoid hemorrhage show a strong association betwee

175 ncreased due to sympathetic activation after subarachnoid hemorrhage similar to critically ill patien

176 diagnoses, and to explore the variability in subarachnoid hemorrhage standardized mortality ratios.

177 nd at least 21 days later (stable aneurysmal subarachnoid hemorrhage state).

178 luation of neuropsychological function after subarachnoid hemorrhage to date.

179 Consecutive patients with aneurysmal subarachnoid hemorrhage treated with clipping or coiling

180 apeutic potential for the management of post-subarachnoid hemorrhage vasospasm.

181 At baseline, the severity of aneurysmal subarachnoid hemorrhage was assessed clinically (Hunt an

182 CT evidence of subarachnoid hemorrhage was associated with a multivaria

183 ng manual PVI (1%), an SCL with asymptomatic subarachnoid hemorrhage was detected; the bleeding compl

184 Subarachnoid hemorrhage was induced in mice by endovascu

185 In 18 patients (30%), subarachnoid hemorrhage was present.

186 The endovascular perforation model of subarachnoid hemorrhage was produced.

187 spital between 2006 and 2011 with poor-grade subarachnoid hemorrhage were prospectively entered into

188 omatose patients with high-grade spontaneous subarachnoid hemorrhage who underwent continuous surface

189 r delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage with hemoglobin 7-13 g/dL.

190 al aneurysm (CA) rupture is a major cause of subarachnoid hemorrhage with high morbidity and mortalit

191 ctive properties of argon after experimental subarachnoid hemorrhage with mortality as the primary en

192 tients (25 traumatic brain injured and seven subarachnoid hemorrhage) were included.

193 SH score (Functional Recovery Expected after Subarachnoid Hemorrhage).

194 erated, subarachnoid hemorrhage-vehicle, and subarachnoid hemorrhage+2% isoflurane.

195 with traumatic brain injury, 10% to 14% with subarachnoid hemorrhage, 1% to 21% with intracerebral he

196 ing and 2) an open-field test 24 hours after subarachnoid hemorrhage, 3) protein analysis of hippocam

197 ange of disorders including ischemic stroke, subarachnoid hemorrhage, and brain trauma, and suggest a

198 ies in patients with traumatic brain injury, subarachnoid hemorrhage, and intracranial hemorrhage hav

199 f cerebral ischemia, traumatic brain injury, subarachnoid hemorrhage, and spinal cord injury.

200 idase outside the subventricular zone (SVZ), subarachnoid hemorrhage, and ventriculomegaly.

201 For patients with subarachnoid hemorrhage, autoregulation reactivity index

202 For patients with subarachnoid hemorrhage, autoregulation reactivity index

203 feasible in patients with intracerebral and subarachnoid hemorrhage, but has yet to be tested in a p

204 (brain infarction, intracerebral hemorrhage, subarachnoid hemorrhage, coronary heart disease and deat

205 Subarachnoid hemorrhage, defined as (1) subarachnoid blo

206 In stroke, traumatic brain injury or subarachnoid hemorrhage, endothelin-1 (ET-1) is induced

207 luding migraine, ischemic stroke, aneurysmal subarachnoid hemorrhage, intracerebral hematoma, and tra

208 Subarachnoid hemorrhage, intracerebral hemorrhage, and i

209 Patients were included when subarachnoid hemorrhage, intracranial hemorrhage, ischem

210 hereas gabapentin/pregabalin were favored in subarachnoid hemorrhage, intracranial hemorrhage, spine,

211 ular outcomes, including ischemic stroke and subarachnoid hemorrhage, leading to long-term physical a

212 tal cardiac arrest, 86 were included (mainly subarachnoid hemorrhage, n = 73).

213 ated with poor outcome, but after aneurysmal subarachnoid hemorrhage, this has not been investigated.

214 brain, including traumatic brain injury and subarachnoid hemorrhage, thus improvement in outcome may

215 Urgent surgery patients and patients with a subarachnoid hemorrhage, trauma, acute renal failure, or

216 se in the prevalence of hospitalizations for subarachnoid hemorrhage, whereas females aged 5 to 14 ye

217 ointensive care," "neurological," "stroke," "subarachnoid hemorrhage," "intracerebral hemorrhage," or

218 als were randomly assigned to sham-operated, subarachnoid hemorrhage-vehicle, and subarachnoid hemorr

219 Federation of Neurosurgical Societies, 3-5) subarachnoid hemorrhage.

220 an, and ischemic lesion within 72 hours from subarachnoid hemorrhage.

221 safe procedure for patients with poor-grade subarachnoid hemorrhage.

222 eadache requiring investigations to rule out subarachnoid hemorrhage.

223 27.5% (95% CI, 25.6%-29.5%) specificity for subarachnoid hemorrhage.

224 d with LRDA were intracranial hemorrhage and subarachnoid hemorrhage.

225 f the 2131 enrolled patients, 132 (6.2%) had subarachnoid hemorrhage.

226 AH Rule was highly sensitive for identifying subarachnoid hemorrhage.

227 n the CA group, 1 patient died at 1 month of subarachnoid hemorrhage.

228 s occur during the early phase of aneurysmal subarachnoid hemorrhage.

229 efficacy of RIPC in protecting brain against subarachnoid hemorrhage.

230 d brain edema at 24 hrs but not 72 hrs after subarachnoid hemorrhage.

231 prevent cerebral vasospasm after aneurysmal subarachnoid hemorrhage.

232 ales aged 5 to 14 years showed increases for subarachnoid hemorrhage.

233 ents had intracerebral hemorrhage, and 4 had subarachnoid hemorrhage.

234 entified as an important cause of stroke and subarachnoid hemorrhage.

235 nt is effective for early brain injury after subarachnoid hemorrhage.

236 scular contraction that follows experimental subarachnoid hemorrhage.

237 s 22 yrs, and a majority (63%) had traumatic subarachnoid hemorrhage.

238 from patients with traumatic brain injury or subarachnoid hemorrhage.

239 the patient died after an acute large-volume subarachnoid hemorrhage.

240 rkedly suppressed basilar artery spasm after subarachnoid hemorrhage.

241 ts with perimesencephalic (PM) nonaneurysmal subarachnoid hemorrhage.

242 he argon group was discovered 24 hours after subarachnoid hemorrhage.

243 lmark of delayed cerebral ischemia following subarachnoid hemorrhage.

244 assessment of consciousness in patients with subarachnoid hemorrhage.

245 ncluding stroke, traumatic brain injury, and subarachnoid hemorrhage.

246 beneficial effect of argon application after subarachnoid hemorrhage.

247 The primary objective was mortality after subarachnoid hemorrhage.

248 rmed of 83 consecutively treated adults with subarachnoid hemorrhage.

249 ool for risk stratification after aneurysmal subarachnoid hemorrhage.

250 ot in control patients with intracerebral or subarachnoid hemorrhage.

251 % vs 28.2%, p<0.001), with larger amounts of subarachnoid (Hijdra Sum Score 17 vs 14, p<0.001) and in

252 Subarachnoid infusion of 1-2 ml of fresh blood at 200 mi

253 The subarachnoid portions of the CN3s were imaged with a 1.5

254 g (PSCO)-and the anterior-most aspect of the subarachnoid space (ASAS), within digital three-dimensio

255 d increased cerebrospinal fluid (CSF) in the subarachnoid space (i.e., extra-axial CSF) from 6 to 24

256 llowing spinal delivery into the lumbosacral subarachnoid space (intrathecal; i.t.).

257 ted CD4(+) T cells gain direct access to the subarachnoid space and become reactivated on encounter w

258 mphatic vessels absorb CSF from the adjacent subarachnoid space and brain interstitial fluid (ISF) vi

259 e of the central nervous system involves the subarachnoid space and indicate that the leptomeninges p

260 The subarachnoid space around the optic nerve was identified

261 (CRV), peripapillary choroid and sclera, and subarachnoid space around the optic nerve, were investig

262 omeningeal coverings of blood vessels in the subarachnoid space as potential access points allowing s

263 The mean (SD) size of the subarachnoid space before and 1 hour after IV fluid bolu

264 aphy evaluation to determine the size of the subarachnoid space before and after IV fluids.

265 e small, dense fat droplets scattered in the subarachnoid space corresponding to a dermoid cyst ruptu

266 oduce Th1/Th17 cytokines, accumulated in the subarachnoid space early during the course of experiment

267 l processes that affect ventricular outflow, subarachnoid space function, or cerebral venous complian

268 Free hemoglobin (Hb) within the subarachnoid space has been implicated in the pathogenes

269 therapeutic transgene, IL-10, to the spinal subarachnoid space have yielded promising results in ani

270 (90-100 microg/kg) was administered into the subarachnoid space in anesthetized cats 28-30 h before t

271 s included the difference in the size of the subarachnoid space in millimeters squared before and 1 h

272 Instead, viral spread into the subarachnoid space in rCDV(SH)-infected animals was trig

273 brain was attenuated and transport from the subarachnoid space into dcLNs was abrogated.

274 l molecules can diffuse through the subdural/subarachnoid space into the underlying neocortex and spr

275 ngeal cells ensheathing blood vessels in the subarachnoid space may provide unique entry sites into t

276 Interobserver consistency for the subarachnoid space measurement between attending radiolo

277 ned-rank test was used to examine changes in subarachnoid space measurements (millimeters squared).

278 ne (90-100 mug/kg) was administered into the subarachnoid space of cats 30 h prior to EA or sham-oper

279 ecrosis factor and interferon gamma into the subarachnoid space of female Dark Agouti rats pre-immuni

280 hromaffin cells transplanted into the spinal subarachnoid space significantly reduced pain-related be

281 e hypothesized that IV fluids would increase subarachnoid space size.

282 pressure induces forces in the retrolaminar subarachnoid space that can deform ONH structures, parti

283 inal fluid (CSF) barriers to spread into the subarachnoid space to induce dramatic viral meningoencep

284 skull base, with direct communication of the subarachnoid space to the extracranial space, usually a

285 jor histocompatibility class II+ APCs in the subarachnoid space were investigated using flow cytometr

286 CSF column extending extracranially from the subarachnoid space with or without brain/ meningeal hern

287 n lead to locally increased pressures in the subarachnoid space within the orbit, which impinges on t

288 med to be caused by elevated pressure in the subarachnoid space, does not correlate with the amount o

289 racerebral tissue (scalp, skull, dura mater, subarachnoid space, etc.) and the bottom layer (layer 2)

290 ized by excessive cerebrospinal fluid in the subarachnoid space, particularly over the frontal lobes.

291 fluid connecting lateral ventricle with the subarachnoid space.

292 ial bones and dura, and those located in the subarachnoid space.

293 ix molecules, laminin and fibronectin in the subarachnoid space.

294 periaqueductal gray (PAG) or into the spinal subarachnoid space.

295 ocytes from meningeal blood vessels into the subarachnoid space.

296 pial arteries and cerebrospinal fluid in the subarachnoid space.

297 local proliferation of CD4(+) T cells in the subarachnoid space.

298 nographic measure of the neonatal and infant subarachnoid space.

299 reach the outer surface of the brain in the subarachnoid spaces from where it drains into venous blo

300 onset is restricted to IL-1R1(+) subpial and subarachnoid vessels.

301 Ophthalmoplegia occurred only when the subarachnoid width of CN3 was <1.9 mm.