ライフサイエンスコーパス: subarachnoid haemorrhage

1 mmon in poor-grade patients after aneurysmal subarachnoid haemorrhage.

2 antiplatelet therapy or in the setting of a subarachnoid haemorrhage.

3 is also a prominent feature of experimental subarachnoid haemorrhage.

4 ort-term outcome in patients with aneurysmal subarachnoid haemorrhage.

5 neurological injury from trauma, stroke and subarachnoid haemorrhage.

6 ible exception of magnesium for treatment of subarachnoid haemorrhage.

7 physiology of migraine, cluster headache and subarachnoid haemorrhage.

8 tion, primary intracerebral haemorrhage, and subarachnoid haemorrhage.

9 minal aortic aneurysm (0.46 [0.35-0.59]) and subarachnoid haemorrhage (0.48 [0.26-0.89]), and not ass

10 ominal aortic aneurysm (0.46, 0.35-0.59) and subarachnoid haemorrhage (0.48, 0.26-0.89).

11 47, 0.27-0.83, p=0.01) but was unchanged for subarachnoid haemorrhage (0.83, 0.44-1.57, p=0.57).

12 hage (hazard ratio 1.44 [95% CI 1.32-1.58]), subarachnoid haemorrhage (1.43 [1.25-1.63]), and stable

13 rebral vasospasm in patients with aneurysmal subarachnoid haemorrhage; (4) the use in the biomechanic

14 Subarachnoid haemorrhage, a particularly deadly form of

15 t plays important roles in migraine, stroke, subarachnoid haemorrhage and brain injury.

16 cation for many patients who have aneurysmal subarachnoid haemorrhage and can lead to delayed ischaem

17 hort have reported on the risks of recurrent subarachnoid haemorrhage and death or dependency for a m

18 with confirmatory evidence of an aneurysmal subarachnoid haemorrhage and presenting less than 96 h f

19 between the incidence of ischaemic stroke or subarachnoid haemorrhage and temperature.

20 of global as opposed to focal deficits after subarachnoid haemorrhage and traumatic brain injury in h

21 ntracranial pathologies (such as meningitis, subarachnoid haemorrhage and tumour) have been considere

22 trokes, 3% of strokes in young adults, 9% of subarachnoid haemorrhages and, of all primary intracereb

23 ars or older with hypertension, a history of subarachnoid haemorrhage, and a giant-sized (>20 mm) pos

24 schaemia (DCI) which occurs after aneurysmal subarachnoid haemorrhage, and often leads to cerebral in

25 redictors were age, hypertension, history of subarachnoid haemorrhage, aneurysm size, aneurysm locati

26 pants in six prospective cohort studies with subarachnoid haemorrhage as outcome.

27 egree relative affected (FDRA) by aneurysmal subarachnoid haemorrhage (aSAH) are at a higher lifetime

28 erebral vasospasm (cVSP) in human aneurysmal subarachnoid haemorrhage (aSAH).

29 cal clipping or endovascular coiling after a subarachnoid haemorrhage, assuming treatment equipoise,

30 Patients with subarachnoid haemorrhage benefit from multidisciplinary

31 e complex treatment strategies applied after subarachnoid haemorrhage call for interdisciplinary coll

32 nts with ischaemic and haemorrhagic strokes, subarachnoid haemorrhage, cerebrovascular malformations,

33 INTRODUCTION: Acute non-traumatic convexity subarachnoid haemorrhage (cSAH) is increasingly recognis

34 The management of patients with aneurysmal subarachnoid haemorrhage demands expertise to anticipate

35 The prognosis of subarachnoid haemorrhage depends on the severity of the

36 ulopathy, central hypoventilation, recurrent subarachnoid haemorrhage, depression, seizures and perio

37 e intensive care management of patients with subarachnoid haemorrhage, emphasizing the detection and

38 how that axonal injury also occurs following subarachnoid haemorrhage in an animal model.

39 Here we studied acute sequelae of subarachnoid haemorrhage in the gyrencephalic brain of p

40 sk of developing intracranial aneurysms, and subarachnoid haemorrhage is a major cause of death and d

41 Aneurysmal subarachnoid haemorrhage is a potentially devastating di

42 Subarachnoid haemorrhage is often followed by haemolysis

43 of statins in patients with acute aneurysmal subarachnoid haemorrhage is unclear.

44 33 patients had a recurrent subarachnoid haemorrhage more than 1 year after their in

45 tudy (cardiac arrest, pneumonia, sepsis, and subarachnoid haemorrhage), none were deemed treatment re

46 atment (one retroperitoneal haemorrhage, one subarachnoid haemorrhage, one respiratory distress, and

47 can present with headache, ischaemic stroke, subarachnoid haemorrhage, or symptoms associated with ma

48 ved with vasoactive cerebrospinal fluid from subarachnoid haemorrhage patients.

49 s also a functionally significant feature of subarachnoid haemorrhage, raising the prospect of common

50 The incidence of subarachnoid haemorrhage (SAH) is 6-8 per 100 000 person

51 Relatives of people with aneurysmal subarachnoid haemorrhage (SAH) may be at increased risk

52 Patients with aneurysmal subarachnoid haemorrhage (SAH) secondary to ruptured ant

53 rial hypertension (HTN) is a risk factor for subarachnoid haemorrhage (SAH).

54 ogical deterioration occurs frequently after subarachnoid haemorrhage (SAH).

55 aemic), transient ischaemic attack (TIA) and subarachnoid haemorrhage (SAH).

56 people with no personal or family history of subarachnoid haemorrhage should be left untreated.

57 ety concerns, we conclude that patients with subarachnoid haemorrhage should not be treated routinely

58 Prognosis seems worse in patients with subarachnoid haemorrhage than in those without.

59 With the exception of subarachnoid haemorrhage, there is little evidence of se

60 Most patients with subarachnoid haemorrhage undergo surgical or endovascula

61 TCH might be the first sign of subarachnoid haemorrhage, unruptured intracranial aneury

62 human brain injured by trauma or aneurysmal subarachnoid haemorrhage, we used DC electrode recording

63 s for patients who did not have a history of subarachnoid haemorrhage with aneurysms located in inter

64 ated ipsilateral basal ganglia bleeding with subarachnoid haemorrhage with no aetiology is uncommon.