ライフサイエンスコーパス: subarachnoid hemorrhage

1 SH score (Functional Recovery Expected after Subarachnoid Hemorrhage).

2 27.5% (95% CI, 25.6%-29.5%) specificity for subarachnoid hemorrhage.

3 d with LRDA were intracranial hemorrhage and subarachnoid hemorrhage.

4 f the 2131 enrolled patients, 132 (6.2%) had subarachnoid hemorrhage.

5 AH Rule was highly sensitive for identifying subarachnoid hemorrhage.

6 n the CA group, 1 patient died at 1 month of subarachnoid hemorrhage.

7 s occur during the early phase of aneurysmal subarachnoid hemorrhage.

8 efficacy of RIPC in protecting brain against subarachnoid hemorrhage.

9 d brain edema at 24 hrs but not 72 hrs after subarachnoid hemorrhage.

10 prevent cerebral vasospasm after aneurysmal subarachnoid hemorrhage.

11 ales aged 5 to 14 years showed increases for subarachnoid hemorrhage.

12 ents had intracerebral hemorrhage, and 4 had subarachnoid hemorrhage.

13 entified as an important cause of stroke and subarachnoid hemorrhage.

14 nt is effective for early brain injury after subarachnoid hemorrhage.

15 scular contraction that follows experimental subarachnoid hemorrhage.

16 s 22 yrs, and a majority (63%) had traumatic subarachnoid hemorrhage.

17 rkedly suppressed basilar artery spasm after subarachnoid hemorrhage.

18 outcome among patients with acute aneurysmal subarachnoid hemorrhage.

19 -fold in patients after TBI and nontraumatic subarachnoid hemorrhage.

20 my among good-grade patients with aneurysmal subarachnoid hemorrhage.

21 tion]) were routinely referred for DSA after subarachnoid hemorrhage.

22 rction and for evaluation of vasospasm after subarachnoid hemorrhage.

23 eurysms, in patients with nontraumatic acute subarachnoid hemorrhage.

24 from patients with traumatic brain injury or subarachnoid hemorrhage.

25 the patient died after an acute large-volume subarachnoid hemorrhage.

26 an adverse effect on cerebral ischemia after subarachnoid hemorrhage.

27 cephalus, one hemorrhagic contusion, and one subarachnoid hemorrhage.

28 ts with perimesencephalic (PM) nonaneurysmal subarachnoid hemorrhage.

29 he argon group was discovered 24 hours after subarachnoid hemorrhage.

30 assessment of consciousness in patients with subarachnoid hemorrhage.

31 ncluding stroke, traumatic brain injury, and subarachnoid hemorrhage.

32 beneficial effect of argon application after subarachnoid hemorrhage.

33 The primary objective was mortality after subarachnoid hemorrhage.

34 rmed of 83 consecutively treated adults with subarachnoid hemorrhage.

35 ool for risk stratification after aneurysmal subarachnoid hemorrhage.

36 lmark of delayed cerebral ischemia following subarachnoid hemorrhage.

37 ot in control patients with intracerebral or subarachnoid hemorrhage.

38 Federation of Neurosurgical Societies, 3-5) subarachnoid hemorrhage.

39 an, and ischemic lesion within 72 hours from subarachnoid hemorrhage.

40 safe procedure for patients with poor-grade subarachnoid hemorrhage.

41 eadache requiring investigations to rule out subarachnoid hemorrhage.

42 patients: adjusted odds ratios (95% CI) for subarachnoid hemorrhage 0.17 (0.06-0.45) and intracerebr

43 with traumatic brain injury, 10% to 14% with subarachnoid hemorrhage, 1% to 21% with intracerebral he

44 n-Hispanic black patients aged 45 to 54 with subarachnoid hemorrhage (13.2/10000 to 10.3/10000 hospit

45 erated, subarachnoid hemorrhage-vehicle, and subarachnoid hemorrhage+2% isoflurane.

46 Forty-one percent had subarachnoid hemorrhage, 24% had traumatic brain injury,

47 ing and 2) an open-field test 24 hours after subarachnoid hemorrhage, 3) protein analysis of hippocam

48 c stroke (19%), 936 ventilated patients with subarachnoid hemorrhage (32%), and 1,404 ventilated pati

49 Of 383 patients enrolled, there were 128 subarachnoid hemorrhage (33.4%), 134 subdural hematoma (

50 The most common diagnoses were subarachnoid hemorrhage (38%), intracerebral hemorrhage

51 Patients with coma attributable to subarachnoid hemorrhage (4/80; 5%) or global hypoxic-isc

52 and 131 met CT criteria for PM nonaneurysmal subarachnoid hemorrhage (53 women; mean age, 53 years [r

53 Diagnoses included subarachnoid hemorrhage (60%), cerebral infarction (23%)

54 % vs. 32%), and more intracranial pathology (subarachnoid hemorrhage 62% vs. 44%; intraparenchymal le

55 ident strokes were documented, including 119 subarachnoid hemorrhages, 62 intraparenchymal hemorrhage

56 Retrospective analysis of all subarachnoid hemorrhage admissions.

57 The subarachnoid hemorrhage age-adjusted risk ratio was 1.57

58 and unstable angina, and inverse (0.69) for subarachnoid hemorrhage (all P<0.001).

59 Fourteen patients were hospitalized after subarachnoid hemorrhage and 2 patients were hospitalized

60 to determine the proportion of patients with subarachnoid hemorrhage and acute lung injury who a rece

61 ology of types of stroke, such as aneurysmal subarachnoid hemorrhage and cerebral vein thrombosis, th

62 l vasospasm is a frequent complication after subarachnoid hemorrhage and contributes to overall morbi

63 gh intracranial pressure peaked 3 days after subarachnoid hemorrhage and declined after day 7.

64 protective against early brain injury after subarachnoid hemorrhage and determined whether this effe

65 seizure like activity found to have diffuse subarachnoid hemorrhage and extensive dural venous sinus

66 rebral vasospasm in patients with aneurysmal subarachnoid hemorrhage and for guiding transfusion ther

67 Conclusion In patients with PM nonaneurysmal subarachnoid hemorrhage and initial DSA negative for ane

68 Hemorrhagic stroke (HS), ie, subarachnoid hemorrhage and intracerebral hemorrhage, is

69 Acute lung injury is common in patients with subarachnoid hemorrhage and is independently associated

70 O administered for 8h improved recovery from subarachnoid hemorrhage and reduced the inflammatory res

71 ocity were improved between acute aneurysmal subarachnoid hemorrhage and stable state (p </= .005); c

72 Subarachnoid hemorrhage and surgical obliteration of rup

73 lar hemoglobin decreases hypoperfusion after subarachnoid hemorrhage and that sustained hemodilution

74 Recent findings in both subarachnoid hemorrhage and traumatic brain injured pati

75 ever is sustained for longer durations after subarachnoid hemorrhage and traumatic brain injury.

76 ange of disorders including ischemic stroke, subarachnoid hemorrhage, and brain trauma, and suggest a

77 ies in patients with traumatic brain injury, subarachnoid hemorrhage, and intracranial hemorrhage hav

78 f cerebral ischemia, traumatic brain injury, subarachnoid hemorrhage, and spinal cord injury.

79 stroke, intracerebral hemorrhage, aneurysmal subarachnoid hemorrhage, and traumatic brain injury have

80 idase outside the subventricular zone (SVZ), subarachnoid hemorrhage, and ventriculomegaly.

81 ved a single treatment of hemodilution after subarachnoid hemorrhage; and, for eight animals, treatme

82 lactate and glucose levels after aneurysmal subarachnoid hemorrhage are associated with delayed cere

83 chemic stroke, intracerebral hemorrhage, and subarachnoid hemorrhage as defined by validated diagnosi

84 ng injury in a large cohort of patients with subarachnoid hemorrhage as well as determine the risk fa

85 ribute to poor outcomes following aneurysmal subarachnoid hemorrhage (aSAH).

86 2000 to 2013, 252 consecutive patients with subarachnoid hemorrhage at computed tomography (CT) and

87 For patients with subarachnoid hemorrhage, autoregulation reactivity index

88 For patients with subarachnoid hemorrhage, autoregulation reactivity index

89 All patients with a diagnosis of subarachnoid hemorrhage between 2009 and 2014.

90 to prevent early rebleeding after aneurysmal subarachnoid hemorrhage, but anticoagulation and induced

91 feasible in patients with intracerebral and subarachnoid hemorrhage, but has yet to be tested in a p

92 (brain infarction, intracerebral hemorrhage, subarachnoid hemorrhage, coronary heart disease and deat

93 ed protein levels in humans after aneurysmal subarachnoid hemorrhage correlate with the degree of cer

94 Subarachnoid hemorrhage, defined as (1) subarachnoid blo

95 redict 60-day case fatality after aneurysmal subarachnoid hemorrhage developed from the International

96 rted on a case of a 16-year-old patient with subarachnoid hemorrhage diagnosed due to a ruptured cere

97 dministration of sodium nitrite after severe subarachnoid hemorrhage differentially influences quanti

98 However, patients with subarachnoid hemorrhage due to rupture of a dissecting a

99 In stroke, traumatic brain injury or subarachnoid hemorrhage, endothelin-1 (ET-1) is induced

100 intracerebral hemorrhage for 17 percent, and subarachnoid hemorrhage for 6 percent.

101 nterventions for intracerebral hemorrhage or subarachnoid hemorrhage generally hinge on whether they

102 nt ameliorating early brain injury following subarachnoid hemorrhage has been nonexistent.

103 The care of patients with aneurysmal subarachnoid hemorrhage has evolved significantly with t

104 morbidity and mortality following aneurysmal subarachnoid hemorrhage; however, the effect of acute lu

105 al hemorrhage (HR, 1.9; 95% CI, 1.5-2.4) and subarachnoid hemorrhage (HR, 2.4; 95% CI, 1.7-3.5) than

106 The indication for DSA was subarachnoid hemorrhage in 71 patients (87.6%), stroke o

107 ix glycoprotein, on early brain injury after subarachnoid hemorrhage in rats.

108 f nonaneurysmal perimesencephalic pattern of subarachnoid hemorrhage in ruptured vertebrobasilar aneu

109 common complication in the first week after subarachnoid hemorrhage in severe cases admitted to ICU.

110 e young age and high prevalence of traumatic subarachnoid hemorrhage in this cohort may limit its gen

111 One hour after subarachnoid hemorrhage induction via endovascular perfo

112 luding migraine, ischemic stroke, aneurysmal subarachnoid hemorrhage, intracerebral hematoma, and tra

113 Subarachnoid hemorrhage, intracerebral hemorrhage, and i

114 al centers were eligible if they a) suffered subarachnoid hemorrhage, intracerebral hemorrhage, ische

115 ointensive care," "neurological," "stroke," "subarachnoid hemorrhage," "intracerebral hemorrhage," or

116 eated with an equal volume (1 microL) of pre-subarachnoid hemorrhage intracerebroventricular administ

117 Patients were included when subarachnoid hemorrhage, intracranial hemorrhage, ischem

118 hereas gabapentin/pregabalin were favored in subarachnoid hemorrhage, intracranial hemorrhage, spine,

119 l role in the development of vasospasm after subarachnoid hemorrhage is accumulating.

120 s primary cerebral vasculitis and aneurysmal subarachnoid hemorrhage is common because of overlapping

121 imal timing of tracheostomy in patients with subarachnoid hemorrhage is controversially debated.

122 The heme released following subarachnoid hemorrhage is metabolized by heme-oxygenase

123 that the primary cause of poor outcome after subarachnoid hemorrhage is not only cerebral arterial na

124 ular outcomes, including ischemic stroke and subarachnoid hemorrhage, leading to long-term physical a

125 Thus, subarachnoid hemorrhage may constitute an important exce

126 Argon application after experimental subarachnoid hemorrhage met the primary endpoint of redu

127 pasm provoked by subarachnoid blood in a rat subarachnoid hemorrhage model.

128 ssociated with inferior outcomes, to compare subarachnoid hemorrhage mortality with other neurologica

129 , intracerebral hemorrhage (n = 97,709), and subarachnoid hemorrhage (n = 27,334) among Hispanics, Bl

130 tal cardiac arrest, 86 were included (mainly subarachnoid hemorrhage, n = 73).

131 r III ("good-grade patients"), who had had a subarachnoid hemorrhage no more than 14 days before plan

132 tion with isoflurane in patients with severe subarachnoid hemorrhage not having intracranial hyperten

133 confidence interval 0.82-0.90), diagnosis of subarachnoid hemorrhage (odds ratio 2.44, confidence int

134 Aneurysmal subarachnoid hemorrhage often leads to death and poor cl

135 defined as the presence of intracerebral or subarachnoid hemorrhage on computed tomography or magnet

136 Five patients had subarachnoid hemorrhage, one patient had traumatic brain

137 als were euthanized 6, 24, or 72 hours after subarachnoid hemorrhage or sham surgery.

138 Subarachnoid hemorrhage or sham-operated rats were treat

139 onsecutive volume-resuscitated patients with subarachnoid hemorrhage or traumatic brain injury, witho

140 h an increase in Pbto2 in most patients with subarachnoid hemorrhage or traumatic brain injury.

141 ost patients with hydrocephalus secondary to subarachnoid hemorrhage or traumatic brain injury.

142 (odds ratio [OR], 2.494), the indication of subarachnoid hemorrhage (OR, 2.523), and the comorbidity

143 Subarachnoid hemorrhage patients admitted to ICU in Aust

144 Pregnant subarachnoid hemorrhage patients also had a higher likel

145 ly and particularly to vulnerable regions in subarachnoid hemorrhage patients at risk for delayed cer

146 omparison with other neurological diagnoses, subarachnoid hemorrhage patients had significantly great

147 udy was to describe in-hospital mortality in subarachnoid hemorrhage patients requiring ICU admission

148 lity physiological recordings in 48 comatose subarachnoid hemorrhage patients to better characterize

149 A total of 11,327 subarachnoid hemorrhage patients were identified in the

150 Pregnant versus nonpregnant subarachnoid hemorrhage patients were less impaired at a

151 Nevertheless, subarachnoid hemorrhage patients who are at risk for vas

152 neurological outcome in good-grade surgical subarachnoid hemorrhage patients, as assessed by the Gla

153 ence of acute kidney injury in patients with subarachnoid hemorrhage patients.

154 rial in a retrospective unselected cohort of subarachnoid hemorrhage patients.

155 time points: on admission (acute aneurysmal subarachnoid hemorrhage phase) and at least 21 days late

156 determinant of outcome with intracranial or subarachnoid hemorrhage predicting a extremely high mort

157 ation rates for intracerebral hemorrhage and subarachnoid hemorrhage remained stable, with the except

158 hemorrhage (RR: 0.96; 95% CI: 0.84, 1.10) or subarachnoid hemorrhage (RR: 1.01; 95% CI: 0.90, 1.14).

159 onin I (cTI) release occurs frequently after subarachnoid hemorrhage (SAH) and has been associated wi

160 netically altered mice include occurrence of subarachnoid hemorrhage (SAH) and variability of infarct

161 Subarachnoid hemorrhage (SAH) carries a 50% mortality ra

162 Patients with subarachnoid hemorrhage (SAH) frequently develop delayed

163 Cerebral vasospasm following aneurysmal subarachnoid hemorrhage (SAH) has devastating consequenc

164 in acute brain injury but an involvement in subarachnoid hemorrhage (SAH) has not been investigated.

165 mic neurological damage following aneurysmal subarachnoid hemorrhage (SAH) have remained elusive.

166 -OPN) could prevent cerebral vasospasm after subarachnoid hemorrhage (SAH) in rats.

167 Cerebral aneurysm rupture and subarachnoid hemorrhage (SAH) inflict disability and dea

168 Acute aneurysmal subarachnoid hemorrhage (SAH) is a complex multifaceted

169 Aneurysmal subarachnoid hemorrhage (SAH) is a potentially devastati

170 Symptomatic vasospasm after subarachnoid hemorrhage (SAH) is associated with a high

171 gy of early ischemic injury after aneurysmal subarachnoid hemorrhage (SAH) is not understood.

172 FFA) are elevated in the first minutes after subarachnoid hemorrhage (SAH) is tested.

173 y and morbidity can be reduced if aneurysmal subarachnoid hemorrhage (SAH) is treated urgently.

174 soconstriction in brain slices obtained from subarachnoid hemorrhage (SAH) model rats.

175 rospective observational study of aneurysmal subarachnoid hemorrhage (SAH) patients, we explored the

176 eficit (DIND) contributes to poor outcome in subarachnoid hemorrhage (SAH) patients.

177 Aneurysmal subarachnoid hemorrhage (SAH) remains a devastating cond

178 Subarachnoid hemorrhage (SAH) results in significant ner

179 ness (LOC) is a common presenting symptom of subarachnoid hemorrhage (SAH) that is presumed to result

180 Subarachnoid hemorrhage (SAH) usually results from ruptu

181 Subarachnoid hemorrhage (SAH) was induced in CD-1 mice (

182 dysfunction has been reported in humans with subarachnoid hemorrhage (SAH), and its underlying pathop

183 stoperative ICU management of patients after subarachnoid hemorrhage (SAH), especially with regards t

184 studies have shown that for the treatment of subarachnoid hemorrhage (SAH), outcomes are improved but

185 m is usually associated with the presence of subarachnoid hemorrhage (SAH), SAH is not required for v

186 To begin to examine HO activity following subarachnoid hemorrhage (SAH), the expression of HO-1 an

187 Significance statement: Aneurysmal subarachnoid hemorrhage (SAH)--strokes involving cerebra

188 More than 30% of patients with RCVS develop subarachnoid hemorrhage (SAH).

189 merous neuropathological processes including subarachnoid hemorrhage (SAH).

190 data regarding anemia and transfusion after subarachnoid hemorrhage (SAH).

191 Medical complications occur frequently after subarachnoid hemorrhage (SAH).

192 ere and pervasive consequences of aneurysmal subarachnoid hemorrhage (SAH).

193 cerebrospinal fluid (CSF) of patients after subarachnoid hemorrhage (SAH).

194 l changes in the cerebral arteries following subarachnoid hemorrhage (SAH).

195 re accounts for the majority of nontraumatic subarachnoid hemorrhage (SAH).

196 us, especially in the early period following subarachnoid hemorrhage (SAH).

197 tic meningitis only (six patients with acute subarachnoid hemorrhage [SAH] excluded), the sensitivity

198 e polycystic kidney disease, presenting with subarachnoid hemorrhage secondary to a ruptured intracra

199 ment behavioral assessments of patients with subarachnoid hemorrhage shortly after the injury.

200 Critically ill patients with subarachnoid hemorrhage show a strong association betwee

201 ncreased due to sympathetic activation after subarachnoid hemorrhage similar to critically ill patien

202 diagnoses, and to explore the variability in subarachnoid hemorrhage standardized mortality ratios.

203 nd at least 21 days later (stable aneurysmal subarachnoid hemorrhage state).

204 should be maintained even if the pattern of subarachnoid hemorrhage suggests a nonaneurysmal origin

205 w-onset stroke, intracerebral hemorrhage, or subarachnoid hemorrhage support these observations.

206 ts with TBI, five patients with nontraumatic subarachnoid hemorrhage, ten nonneurologic controls, and

207 rity populations had higher death rates from subarachnoid hemorrhage than did Whites.

208 organ donor had been healthy before having a subarachnoid hemorrhage that led to his death.

209 d of operation in patients with nontraumatic subarachnoid hemorrhage they were 104 +/- 68 ng/mL (p =

210 ated with poor outcome, but after aneurysmal subarachnoid hemorrhage, this has not been investigated.

211 brain, including traumatic brain injury and subarachnoid hemorrhage, thus improvement in outcome may

212 luation of neuropsychological function after subarachnoid hemorrhage to date.

213 Urgent surgery patients and patients with a subarachnoid hemorrhage, trauma, acute renal failure, or

214 atients with acute brain injury secondary to subarachnoid hemorrhage, traumatic brain injury, primary

215 Consecutive patients with aneurysmal subarachnoid hemorrhage treated with clipping or coiling

216 The aggregate traumatic subarachnoid hemorrhage (tSAH) component of the Stockhol

217 apeutic potential for the management of post-subarachnoid hemorrhage vasospasm.

218 als were randomly assigned to sham-operated, subarachnoid hemorrhage-vehicle, and subarachnoid hemorr

219 At baseline, the severity of aneurysmal subarachnoid hemorrhage was assessed clinically (Hunt an

220 CT evidence of subarachnoid hemorrhage was associated with a multivaria

221 ng manual PVI (1%), an SCL with asymptomatic subarachnoid hemorrhage was detected; the bleeding compl

222 In 1 patient, subarachnoid hemorrhage was diagnosed on CT but not on M

223 Subarachnoid hemorrhage was induced by injecting 0.3 mL

224 Subarachnoid hemorrhage was induced in mice by endovascu

225 In 18 patients (30%), subarachnoid hemorrhage was present.

226 The endovascular perforation model of subarachnoid hemorrhage was produced.

227 spital between 2006 and 2011 with poor-grade subarachnoid hemorrhage were prospectively entered into

228 y-five consecutive adult patients with acute subarachnoid hemorrhage were recruited into the institut

229 tients (25 traumatic brain injured and seven subarachnoid hemorrhage) were included.

230 se in the prevalence of hospitalizations for subarachnoid hemorrhage, whereas females aged 5 to 14 ye

231 y identification of patients with aneurysmal subarachnoid hemorrhage who are at high risk for symptom

232 omatose patients with high-grade spontaneous subarachnoid hemorrhage who underwent continuous surface

233 e consecutive patients with acute (<14 days) subarachnoid hemorrhage who underwent GDC embolization w

234 r delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage with hemoglobin 7-13 g/dL.

235 al aneurysm (CA) rupture is a major cause of subarachnoid hemorrhage with high morbidity and mortalit

236 ctive properties of argon after experimental subarachnoid hemorrhage with mortality as the primary en