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1 lassified as nuclear, cortical, or posterior subcapsular.
2 eneously, 109 had a smooth surface, 101 were subcapsular, 89 had ill-defined margins, and 62 had a ce
3             As opposed to the clear DNA-free subcapsular and cortical areas of young adult mouse lens
4                           In aged mice, most subcapsular and cortical cataracts colocalize with accum
5 nset of heart failure and the development of subcapsular and cortical cataracts is observed in Mbnl3(
6 vital dyes, to determine whether age-related subcapsular and cortical cataracts were linked to the fa
7 oped that initially appeared to be posterior subcapsular and gradually matured to involve the entire
8 lipopolysaccharide, were very visible in the subcapsular and medullary sinuses but were largely exclu
9 a recta in the renal medulla, the lymph node subcapsular and medullary sinuses, and some capillaries
10 ly associated with the severity of posterior subcapsular and nuclear cataracts, which was the only fe
11 immature nuclear sclerotic, mature posterior subcapsular, and mature nuclear.
12 s; nanoparticles were found in the cortical, subcapsular, and medullary sinuses.
13                      NK cells located in the subcapsular area exhibited reduced motility and were fou
14 sults in the accumulation of NK cells in the subcapsular area of the draining lymph node and their ac
15 gions in the medullary, interfollicular, and subcapsular areas where viral infection was initially co
16 itating reticulum cells of T zones, cells in subcapsular areas, and cells of the reticular network we
17 rotic diseases in the lens, such as anterior subcapsular cataract (ASC) formation.
18 ciated with a higher prevalence of posterior subcapsular cataract (PSC) (OR, 1.29; 95% CI, 1.07-1.55)
19 te a possible relationship between posterior subcapsular cataract (PSC) formation and expression of t
20 cities formed during recovery from posterior subcapsular cataract (PSC) in Royal College of Surgeons
21  presence of cortical, nuclear, or posterior subcapsular cataract (PSC) opacification in at least one
22 were correlated to the severity of posterior subcapsular cataract (r = 0.4, P = .0006).
23 ty, except for variable mild local posterior subcapsular cataract and local retinal toxicity with hig
24 lation was evident between risk of posterior subcapsular cataract and size at birth.
25 95% CI, 1.27-2.87; P = 0.002); and posterior subcapsular cataract increase of 5% or more versus less
26 aract, and any role in cortical or posterior subcapsular cataract is scarcely measurable.
27 ers (OR, 1.28; 95% CI, 0.79-2.08); posterior subcapsular cataract occurred in 3.0% of statin users an
28 I 0.96-1.63) cataracts, but not to posterior subcapsular cataract or cataract surgery.
29 act, 1.95 (95% CI: 0.48, 7.95) for posterior subcapsular cataract, 1.82 (95% CI: 0.91, 3.66) for soft
30 95% CI, 0.78-1.65; P = 0.519); and posterior subcapsular cataract, 3.05 (95% CI, 1.79-5.19; P < 0.001
31 The associations of lens features (posterior subcapsular cataract, nuclear color, nuclear white scatt
32 .82, 95% CI: 0.68, 0.97; primarily posterior subcapsular cataract, RR = 0.90, 95% CI: 0.71, 1.13).
33 ear white scatter, cortical spokes, anterior subcapsular cataract, vacuoles, waterclefts, coronary fl
34 iations were found between PXS and posterior subcapsular cataract.
35 n 50% of NF2 patients also develop posterior subcapsular cataracts (PSCs).
36 es regarding the pathophysiology of anterior subcapsular cataracts secondary to posterior chamber pIO
37                                    Posterior subcapsular cataracts were present in 97.5% of patients.
38 ar incident nuclear, cortical, and posterior subcapsular cataracts, but was related to incident catar
39 .23-2.27), but not to cortical and posterior subcapsular cataracts.
40 t laxity, skin hyperelasticity and bilateral subcapsular cataracts.
41 ce from the four families exhibited anterior subcapsular cataracts.
42 raphs were graded for cortical and posterior subcapsular cataracts.
43 resembled the plaques seen in human anterior subcapsular cataracts.
44 al dominant "progressive childhood posterior subcapsular" cataracts segregating in a white family to
45           We observed that mice with adrenal subcapsular cell hyperplasia (SCH), a common histologica
46 is triggered by the interaction of bacterial subcapsular components and bone marrow-derived DC (likel
47 fr2(UB-/-) kidneys have abnormally thickened subcapsular cortical stromal mesenchyme.
48 th hydrocortisone induced both medullary and subcapsular cortical TE cells to express CK6, a differen
49 gitidis and that in each case, the bacterial subcapsular domain markedly influences the Ig response t
50 osition and/or architecture of the bacterial subcapsular domain.
51 mains unresolved whether different bacterial subcapsular domains can exert differential effects on PS
52 sues, high endothelial venules and basal and subcapsular epithelia are CD164 class II-positive, while
53 le negative thymoctyes and laminin 5 made by subcapsular epithelial cells is required for the surviva
54  to the cell surface of thymic medullary and subcapsular epithelium.
55 differentiating fibers of the bow region and subcapsular fibers of the central zone, whereas the lens
56 oneal implants, intratumoral hemorrhage, and subcapsular fluid, showed a significant association with
57                              When grown as a subcapsular graft, the Gli2(-/-) UGS exhibited prostatic
58 icities of these two important streptococcal subcapsular group polysaccharides to fully understand th
59                       CT showed a voluminous subcapsular hematoma compressing the hepatic parenchyma,
60 s, ureteropelvic junction obstruction, renal subcapsular hematoma, cholelithiasis, medullary calcinos
61  acute liver failure occurs in patients with subcapsular hematoma.
62 including abruptio placentae, renal failure, subcapsular hematomas, and hepatic rupture.
63        Gross liver lesions, characterized by subcapsular hemorrhages or enlargement of the right inte
64 l vascular abnormalities, including aberrant subcapsular hepatic veins, enlarged glomeruli, intestina
65 murine model of chronic kidney disease, with subcapsular hydrogel injections acting as a delivery dep
66 aft model of kidney cancer, characterized by subcapsular implantation of Caki-1 clear cell human kidn
67                   Moreover, renal restricted subcapsular infusion of Snx5-specific siRNA (vs. mock si
68 ix pancreatic cancer cell lines and a spleen subcapsular inoculation nude mouse model were also used.
69 ion although they survived longer than renal subcapsular islet allografts.
70                                    The renal subcapsular islet graft was easily detectable on T2*-wei
71 ll to glucose challenge, comparable to renal subcapsular islet grafts, despite a marginal islet dose,
72 4 to prevent recurrence of diabetes in renal subcapsular islet isografts in DR-BB (RT1uu) rats with e
73 awbacks of the conventional method of kidney subcapsular islet transplantation.
74 normoglycemia faster than animals with renal subcapsular islet transplants.
75  tolerance of intratesticular, but not renal subcapsular, islet allografts.
76 onsive (>120 days) Lewis recipients of renal subcapsular islets underwent nephrectomy of the islet be
77 ed reading center for cortical and posterior subcapsular lens opacities and for AMD severity.
78 age-related nuclear, cortical, and posterior subcapsular lens opacities.
79 ted with an increased risk of mild posterior subcapsular lens opacity development.
80  receptor CCR7 for their transition from the subcapsular lymph node sinus into the parenchyma, a migr
81                                 Depletion of subcapsular macrophages (SCMsmall ef, Cyrillic) or abrog
82 affected by the loss of F4/80(+) or CD169(+) subcapsular macrophages.
83 d.HGF-transduced NHP islets in vivo, a renal subcapsular marginal mass islet transplant model was dev
84                           CD169(+) SIGNR1(+) subcapsular medullary macrophages are the primary cells
85                                          (2) Subcapsular necrotic areas in the liver suggestive of pr
86 ocal inflammation, and large grossly visible subcapsular necrotic foci.
87  anticipated from earlier studies, posterior subcapsular, nuclear, and cortical cataracts were associ
88 d to UVB exhibited accelerated anterior lens subcapsular opacification, which was more pronounced in
89 ngle-strand breaks, as well as lens anterior subcapsular opacification.
90 aract (RR = 1.39), and preexisting posterior subcapsular opacities (RR = 6.67).
91 d cortical opacities as grade > or = 0.5 and subcapsular opacities as grade > or =0.3 of the Lens Opa
92                       Cortical and posterior subcapsular opacities increased with age, but scores for
93 tations, 2 eyes had progression of posterior subcapsular opacities, although neither required surgery
94  graded for nuclear, cortical, and posterior subcapsular opacities, and the amount of liquid vitreous
95 iring nearwork, nuclear opacities, posterior subcapsular opacities, glaucoma, and ocular hypertension
96 tical opacity as grade >/=1.0, and posterior subcapsular opacity as grade >/=0.5 according to the Len
97 OL showed either stage 5 (complete posterior subcapsular opacity) or stage 6 (mature) cataracts, wher
98 ryl CoA reductase inhibitor, developed frank subcapsular opacity.
99  (OR, 1.57; 95% CI, 1.13-2.20) and posterior subcapsular (OR, 1.73; 95% CI, 1.10-2.72) cataract, but
100 stant cell renewal, involving recruitment of subcapsular progenitors to ZG fate and subsequent lineag
101  graded for nuclear, cortical, and posterior subcapsular (PSC) cataract, following the Wisconsin Cata
102  presence of nuclear, cortical, or posterior subcapsular (PSC) cataract, from standardized grading of
103 , including nuclear, cortical, and posterior subcapsular (PSC) cataract.
104 cipants had nuclear, cortical, and posterior subcapsular (PSC) cataracts, respectively.
105 the odds of developing cortical or posterior subcapsular (PSC) cataracts.
106 entage involvement of cortical and posterior subcapsular (PSC) lens opacities within the central 5 mm
107 viduals with nuclear, cortical, or posterior subcapsular (PSC) opacities and individuals with no cata
108 ens for presence of nuclear (NSC), posterior subcapsular (PSC), and cortical cataract (CC), using the
109 tes assemble in clusters in the cords of the subcapsular red pulp and are distinct from macrophages a
110 lomeruli identified located in the immediate subcapsular region (P<0.001).
111 nsplanted, but most biopsies sample only the subcapsular region and may not accurately represent the
112  revealed laminin 5 expression mostly in the subcapsular region of the adult thymus.
113                            Gp96 accesses the subcapsular region of the draining lymph node, and it is
114 ghout the cortex rather than confined to the subcapsular region of the thymus.
115 butes to pre-T cell development, as does the subcapsular region of the thymus.
116 onized by newly produced antibodies from the subcapsular region to the germinal center, and affinity
117         Naive T lymphocyte locomotion in the subcapsular region was 38% slower and had higher turning
118  a predominance of sclerosis in the kidney's subcapsular region, the area predominantly sampled by th
119 ry junction in lymphoid follicles and in the subcapsular region.
120     MBs were larger and more numerous in the subcapsular region.
121 /delta TCR expression especially high in the subcapsular region.
122 elop cataracts in the anterior and posterior subcapsular regions as well as punctate opacities in the
123 n DCs from the early stage in the lymph node subcapsular regions, and COX-2 inhibition markedly suppr
124 lant survival and blood glucose control in a subcapsular renal graft model in immuno-incompetent diab
125        The tissue recombinants were grown as subcapsular renal grafts and treated from the time of gr
126 tain innate-like lymphocytes that survey the subcapsular sinus (SCS) and associated macrophages for p
127 CD169+MHCII+ macrophages on the floor of the subcapsular sinus (SCS) and in the medulla of lymph node
128              The layer of macrophages at the subcapsular sinus (SCS) captures pathogens entering the
129 inate via lymphatics and preferentially bind subcapsular sinus (SCS) CD169(+) macrophages in tumor-dr
130 ) alpha1beta2, which maintained a protective subcapsular sinus (SCS) macrophage phenotype within viru
131 were found associated with CD169(+)-positive subcapsular sinus (SCS) macrophages and collagen fibers.
132                                              Subcapsular sinus (SCS) macrophages capture antigens fro
133 y than naive T cells, relocalized toward the subcapsular sinus (SCS) near invaded macrophages, and en
134 b(+)CD169(+) macrophages, which populate the subcapsular sinus (SCS) of LNs, are critical for the cle
135 in-water emulsion adjuvant MF59 localizes in subcapsular sinus and medullary macrophage compartments
136 Our findings identify macrophages lining the subcapsular sinus as an important site of B cell encount
137 , small numbers of beads were present in the subcapsular sinus as early as 6 h after inhalation.
138  CD1d-dependent manner in close proximity to subcapsular sinus CD169(+) macrophages.
139                                 Depletion of subcapsular sinus CD169-positive macrophages by clodrona
140 ng by lymphatic endothelial cells lining the subcapsular sinus ceiling stabilizes interfollicular CCL
141 borne antigens and chemoattractants from the subcapsular sinus directly to the B cell follicles.
142 ure that macrophages of the mouse lymph node subcapsular sinus facilitate B cell activation in vivo b
143 broblastic reticular cells that connects the subcapsular sinus floor and the HEVs by intertwining wit
144                                 In contrast, subcapsular sinus macrophages (SSMs) exposed to lymph-bo
145 t contain the unprocessed Ag are captured by subcapsular sinus macrophages and are transferred onto f
146 resulting from S. flexneri interactions with subcapsular sinus macrophages and dendritic cells, and r
147    By contrast, large antigens were bound by subcapsular sinus macrophages and subsequently transferr
148 culate antigens relies on antigen capture by subcapsular sinus macrophages of the lymph node.
149 cate that NK-cell recruitment is mediated by subcapsular sinus macrophages, IFN-gamma, and CXCR3 duri
150 acquisition did not require dendritic cells, subcapsular sinus macrophages, or B cell movement to the
151 nate-loaded liposomes, indicating a role for subcapsular sinus macrophages.
152 cles must be translocated into follicles via subcapsular sinus macrophages.
153 rticulate antigens and large IC are bound by subcapsular sinus macrophages.
154 s (Mo) and B cell areas in the spleen and to subcapsular sinus Mo in lymph nodes of naive mice (CR-Fc
155 ymph node, exosomes were not retained in the subcapsular sinus of CD169(-/-) mice but penetrated deep
156  cells were identified in the paracortex and subcapsular sinus of the draining internal iliac lymph n
157 n the marginal zone of the spleen and in the subcapsular sinus of the lymph node.
158 ith the removal of macrophages that line the subcapsular sinus of the lymph node.
159 in the DLN, most of the LPS was found in the subcapsular sinus or medulla, near or within lymphatic e
160  antigens diffuse directly from lymph in the subcapsular sinus to be acquired by antigen-specific B c
161 rt of small antigens and chemokines from the subcapsular sinus to follicular B cells.
162 ymph-borne soluble molecules travel from the subcapsular sinus to the HEVs is unclear.
163 luding chemokines, traveled rapidly from the subcapsular sinus to the HEVs using the reticular networ
164 njection, first in the region closest to the subcapsular sinus where lymph enters the lymph node.
165  endothelial cells lining the ceiling of the subcapsular sinus, but not those lining the floor, expre
166 luorophore-labeled molecules highlighted the subcapsular sinus, the reticular fibers, and the ablumin
167 h the lymph to macrophages in the lymph node subcapsular sinus.
168 tion of lymphoid stromal cells lining the LN subcapsular sinus.
169  vessels at the junction with the lymph node subcapsular sinus.
170 ion into the LN parenchyma from lymph in the subcapsular sinus.
171 sinus macrophages, or B cell movement to the subcapsular sinus.
172 spite the microspheres being confined to the subcapsular sinus.
173 and infected cells residing just beneath the subcapsular sinus.
174 tor 1 (S1P1)-expressing CD68+ macrophages in subcapsular sinuses of FTY-P-treated MLNs.
175 cessed islets were transplanted at the renal subcapsular site in rats.
176     In addition, the spatially limited renal subcapsular site restricts the mass of islet tissue that
177 r allografts were transplanted to the kidney subcapsular site.
178 st, porcine islets transplanted to the liver subcapsular space do not survive, although autologous is
179  2500, were transplanted into the left renal subcapsular space of a syngeneic adult mouse made diabet
180 -2d) islets were transplanted into the renal subcapsular space of diabetic c-Rel-/- C57BL/6 (H-2b) mi
181 ncreatic islets of Langerhans into the renal subcapsular space of immunodeficient BALB/c.rag2(-/-).cg
182  regeneration of prostatic structures in the subcapsular space of the kidney was observed within 4-8
183 ically, its expression was restricted to the subcapsular space of the LN during early inflammation, w
184 mokidney by thymic autografting to the renal subcapsular space results in normal thymic growth and fu
185                  Control dogs received liver subcapsular space transplants of porcine islets and auto
186 n as few as 10 islets implanted in the renal subcapsular space, intrahepatic, intraabdominal, and sub
187 re recovered and transplanted into the renal subcapsular space.
188 cells flowed from medullary sinuses into the subcapsular space.
189 sorganized TEBs characterized by exaggerated subcapsular spaces, breaks in basal lamina, dissociated
190 These results suggest the presence of common subcapsular surface antigens, such as outer membrane pro
191 m-immunosuppressed TMX recipients with renal subcapsular syngeneic thymic grafts.
192 cified schedule starting 2 days before renal subcapsular transplantation of an islet isograft.
193          Twelve (75%) of 16 hemangiomas were subcapsular, two (12%) of 16 demonstrated exophytic grow
194         The nuclear, cortical, and posterior subcapsular types of cataracts did not show different re
195  fat planes obscured, retroperitoneal fluid (subcapsular vs extracapsular), ascites beyond the cul-de
196 tes downregulate CCR9 and migrate toward the subcapsular zone where they recombine their TCR beta-cha
197 across the cortex before accumulation in the subcapsular zone.

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