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1 iations were found between PXS and posterior subcapsular cataract.
2 .23-2.27), but not to cortical and posterior subcapsular cataracts.
3 t laxity, skin hyperelasticity and bilateral subcapsular cataracts.
4 ce from the four families exhibited anterior subcapsular cataracts.
5 raphs were graded for cortical and posterior subcapsular cataracts.
6 resembled the plaques seen in human anterior subcapsular cataracts.
7 act, 1.95 (95% CI: 0.48, 7.95) for posterior subcapsular cataract, 1.82 (95% CI: 0.91, 3.66) for soft
8 95% CI, 0.78-1.65; P = 0.519); and posterior subcapsular cataract, 3.05 (95% CI, 1.79-5.19; P < 0.001
9 ty, except for variable mild local posterior subcapsular cataract and local retinal toxicity with hig
12 ar incident nuclear, cortical, and posterior subcapsular cataracts, but was related to incident catar
13 95% CI, 1.27-2.87; P = 0.002); and posterior subcapsular cataract increase of 5% or more versus less
15 The associations of lens features (posterior subcapsular cataract, nuclear color, nuclear white scatt
16 ers (OR, 1.28; 95% CI, 0.79-2.08); posterior subcapsular cataract occurred in 3.0% of statin users an
18 ciated with a higher prevalence of posterior subcapsular cataract (PSC) (OR, 1.29; 95% CI, 1.07-1.55)
19 te a possible relationship between posterior subcapsular cataract (PSC) formation and expression of t
20 cities formed during recovery from posterior subcapsular cataract (PSC) in Royal College of Surgeons
21 presence of cortical, nuclear, or posterior subcapsular cataract (PSC) opacification in at least one
24 .82, 95% CI: 0.68, 0.97; primarily posterior subcapsular cataract, RR = 0.90, 95% CI: 0.71, 1.13).
25 es regarding the pathophysiology of anterior subcapsular cataracts secondary to posterior chamber pIO
26 al dominant "progressive childhood posterior subcapsular" cataracts segregating in a white family to
27 ear white scatter, cortical spokes, anterior subcapsular cataract, vacuoles, waterclefts, coronary fl
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