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1 hylogenomic approach, we have identified and subclassified a new family of carotenoid-binding protein
2 development and internal validation sets and subclassified according to height-adjusted TKV (HtTKV) r
3             Cases without SMN1 mutations are subclassified according to phenotype.
4 sected PDAC patients with follow-up data was subclassified according to predominant growth pattern, a
5 , the action tremors of the upper limbs were subclassified according to the predominant site and stat
6                                Patients were subclassified according to the presence of donor coronar
7 s with acute pancreatitis, of which 107 were subclassified according to the revised Atlanta criteria
8                                Patients were subclassified according to the Verhaak and Phillips clas
9                        Tonic S neurones were subclassified, according to their post-stimulus response
10 ory data were collected on patients who were subclassified and serially revalidated based on publishe
11                                       AR was subclassified as antibody (Ab)-treated AR or other manag
12                              Lobar CMBs were subclassified as cortical or subcortical.
13                G:C-A:T transitions were also subclassified as CpG or non-CpG.
14  CP to a more advanced phase, which has been subclassified as either accelerated phase or blastic pha
15                          These reactions are subclassified as either type I or type II depending on h
16 tologically aggressive component was further subclassified as frank LCL or as L&H-cell-rich, but not
17                 The follicular component was subclassified as grade 3a (FL3a) or grade 3b (FL3b) acco
18                           PAH is now further subclassified as idiopathic PAH, familial PAH, and assoc
19 receptors (EP(1)-EP(4)), that can be further subclassified as low-affinity (EP(1) and EP(2)) or high-
20 tive emphysema), panlobular, and paraseptal (subclassified as mild or substantial).
21    Lobulation, atrophy, and hypertrophy were subclassified as mild-moderate or severe.
22 linical disease severity (Atlanta criteria); subclassified as multiorgan dysfunction (MOF), pancreati
23 ic macular edema [DME]) or "any DR" (further subclassified as NPDR or PDR, without or with DME).
24 which was not significant when patients were subclassified as producers and non-producers of cytochro
25 nodule) and were otherwise classified as NS (subclassified as pure or heterogeneous).
26                             The 263 MIs were subclassified as spontaneous MI (n=78; 29.7%), secondary
27    Emphysema is classified as centrilobular (subclassified as trace, mild, moderate, confluent, and a
28                    All cases were clinically subclassified at presentation as amnestic AD dementia ve
29                 Poised enhancers can also be subclassified based on presence or absence of H3K27me3 a
30 ous group of neoplasms and are traditionally subclassified based on type and degree of differentiatio
31 cally, we found that active enhancers can be subclassified based on varying levels of H3K4me1, H3K27a
32 re stained with subtype-specific markers and subclassified by a pathologist.
33 tudy confirms that most so-called MFH can be subclassified by defined criteria; it provides evidence
34            Incidence of colorectal carcinoma subclassified by F nucleatum status in tumor tissue, det
35 t-naive patients with achalasia, defined and subclassified by high-resolution esophageal pressure top
36 ssified as Hispanic or non-Hispanic and then subclassified by race forming six race-specific subgroup
37               Full-thickness macular hole is subclassified by size of the hole as determined by OCT a
38                Vitreomacular traction can be subclassified by the diameter of vitreous attachment to
39                     This set of peptides was subclassified by their capacity to sensitize targets whe
40     Cell-line-defined super-enhancers can be subclassified by their somatic alteration status into so
41 ally unclassifiable by the LPD analyses were subclassified by this signature.
42 ians who were blinded to the study treatment subclassified causes of death as cardiovascular (cardiac
43 amples by CD40 activation status and further subclassified CD40-activated CLL cells from healthy B ce
44                         Forty-nine HCAs were subclassified into 14 inflammatory, 20 hepatocyte nuclea
45 xylase (GAD)-positive interneurons that were subclassified into at least four groups based on nAChR s
46  mouse lungs are not homogeneous, but can be subclassified into capsaicin-sensitive and capsaicin-ins
47                            The patients were subclassified into categories of body mass index (BMI) d
48        Primary chronic daily headache can be subclassified into disorders of short duration (<4 h/att
49 a single disease entity, but one that can be subclassified into distinct clinical prognostic stages,
50 ents with esophageal hypersensitivity may be subclassified into distinct phenotypic subclasses based
51 s of stay were compared with body mass index subclassified into five groups: underweight, normal weig
52 tive and EpCAM-negative HCC could be further subclassified into four groups with prognostic implicati
53 ffuse large B-cell lymphomas (DLBCLs) can be subclassified into germinal center B-cell (GCB)-like and
54 a serovars, and some of these can be further subclassified into groups of strains that differ profoun
55 a that develops in adults, is pathologically subclassified into monophasic spindle synovial sarcoma a
56                             Each subject was subclassified into one of five groups: obstructive physi
57                                Mortality was subclassified into overall mortality (during 10 years of
58 a suggest that most patients with MFH can be subclassified into specific STS types, but the clinical
59                        Assemblage A has been subclassified into subtypes A-I to A-IV: A-I has been re
60                        Group I patients were subclassified into those undergoing MVR with chordal pre
61             Our data indicate that BC can be subclassified into three subtypes, on the basis of their
62                             Regarding MM, we subclassified ISS stages into clusters based on shared f
63            Recent collaborative efforts have subclassified malignant glioblastomas into 4 clinical re
64 es mostly confirmed the cytogenetic data and subclassified patients according to 14q32 translocations
65                                      We have subclassified the molecular substrates by system, focusi
66 trahepatic from extrahepatic tumors and have subclassified this latter group into proximal, middle, a
67                                   Cases were subclassified using CD10, bcl-6, and MUM1 expression, an
68                                Patients were subclassified (using the Kiel classification) as having

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