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1 hile, Brain computed tomography demonstrated subdural abscess in right parietal area.
2                                        Using subdural and depth recordings from epileptic patients, w
3 ence of diffuse cerebral oedema, presence of subdural and extradural hematoma; however in isolation t
4                          A sensor was tested subdural and in vitro, simulating a supine infant with a
5  Although complications such as intracystic, subdural, and extradural hematomas are well known after
6 n death was induced by sudden inflation of a subdural balloon catheter with continuous monitoring of
7    Brain death was induced by inflation of a subdural balloon in ten mongrel dogs weighing 23 to 30 k
8 s were skull fractures (36% of cases), acute subdural bleeding (72%) and retinal haemorrhages (71%);
9 correlated with the increasing volume of the subdural blood clot (sham: 9+/-3 mm3; 200 microl: 81+/-1
10 ter and again at 2 h after completion of the subdural blood infusion.
11 e (ICP) was monitored in all patients with a subdural catheter (Camino Systems, San Diego, CA) for up
12 g studies revealed a large mixed-attenuation subdural collection in the right frontal region with pro
13 aminations showed no white matter changes or subdural collections.
14 sies, 12 intracranial cyst evaluations, four subdural drainages, and five transsphenoidal pituitary r
15 e and interictal spike frequency measures on subdural ECoG recording may both be useful in predicting
16                   High density scalp EEG and subdural ECoG recordings provide an opportunity to map t
17                                              Subdural ECoG signals were recorded while each patient v
18 ured local cortical activity using arrays of subdural electrocorticographic (ECoG) electrodes in huma
19                                              Subdural electrocorticographic (ECoG) recordings in pati
20  human functional brain mapping, we recorded subdural electrocorticographic (ECoG) signals in five cl
21 wed by 2-stage epilepsy surgery with chronic subdural electrocorticographic monitoring, and were seiz
22 quency measures obtained from extraoperative subdural electrocorticography (ECoG) recording could pre
23 sequently recorded cortical physiology using subdural electrocorticography during a spatial-attention
24 tentials from hand sensorimotor cortex using subdural electrocorticography during a visually cued, in
25 ation surgery, we use the novel technique of subdural electrocorticography in combination with subtha
26                 To address this, we utilized subdural electrocorticography to study cortical oscillat
27                      We do so using invasive subdural electrode arrays from a population of 16 patien
28 imeslicing), in a subject in whom indwelling subdural electrode arrays had been placed for clinical p
29  with motor movement across 22 subjects with subdural electrode arrays placed for identification of s
30 graphy (ECoG) signals measured directly from subdural electrode arrays that were implanted in patient
31                            It can help guide subdural electrode implantation and narrow the search fo
32              Thirteen patients had undergone subdural electrode implantation for the surgical managem
33                                        After subdural electrode implantation, we used DCS to localize
34  propofol-anaesthetized juvenile swine using subdural electrode strips (electrocorticography) and int
35 preading depolarizations were monitored with subdural electrode strips and regional cerebral blood fl
36 corticography [duration: 54 h (34, 66)] from subdural electrode strips was analysed over Days 0-3 aft
37 electrocorticographic recordings obtained by subdural electrode-strip monitoring during intensive car
38                                 ECoG from 63 subdural electrodes (500 Hz/channel) chronically implant
39 cy oscillations were seen in recordings from subdural electrodes adjacent to the microelectrode array
40 s by stimulating a part of the brain through subdural electrodes and recording the cortical evoked po
41   These patients had chronic implantation of subdural electrodes covering part of the lateral and med
42 42 years) underwent invasive monitoring with subdural electrodes for epilepsy surgery.
43                             EEG studies from subdural electrodes have demonstrated a face-specific ev
44 ecorded high gamma (62-100 Hz) activity from subdural electrodes implanted for seizure monitoring.
45 ctrical stimulation of chronically implanted subdural electrodes in 34 patients (mean age, 12.2 years
46  cortex (M1), using electocorticography from subdural electrodes in four patients while they performe
47 re recorded from human temporal cortex using subdural electrodes in order to investigate in greater a
48  face perception in a patient implanted with subdural electrodes in the right inferior temporal lobe.
49  using electrocorticographic recordings from subdural electrodes over frontal and temporal cortices.
50 ed brain-computer interface that consists of subdural electrodes placed over the motor cortex and a t
51 ctrodes, and the precise localization of the subdural electrodes was defined by MRI co-registration.
52                    The exact location of the subdural electrodes was determined on high-resolution th
53 nd verb generation in 11 humans in whom 1210 subdural electrodes were implanted.
54                                              Subdural electrodes were placed over the prefrontal cort
55 lated visual cortex in humans implanted with subdural electrodes while recording from other brain sit
56 ored by invasive electrocorticography (ECoG; subdural electrodes) and noninvasive scalp EEG during in
57 ified with intracranial recordings (depth or subdural electrodes).
58 s performed by electrical stimulation of the subdural electrodes, and the precise localization of the
59 rect cortical stimulation (DCS) of implanted subdural electrodes.
60 on that a greater proportion of patients had subdural empyema and hemiparesis in 2011-2013.
61 %), two intraventricular masses (0.05%), two subdural fluid collections (0.05%), and two other tumors
62  magnetic resonance imaging findings include subdural fluid collections, enhancement of the pachymeni
63 ing of the brain, pituitary enlargement, and subdural fluid collections.
64                                      Chronic subdural haematoma causes serious morbidity and mortalit
65 ients aged 18 years and older with a chronic subdural haematoma for burr-hole drainage were assessed
66  a drain after burr-hole drainage of chronic subdural haematoma is safe and associated with reduced r
67    The results indicate that following acute subdural haematoma, a rapid cellular redistribution of a
68 ors, if any, are associated with presence of subdural haematoma.
69  2 h or 4 h following production of an acute subdural haematoma.
70                                              Subdural haematomas are thought to be uncommon in babies
71                                              Subdural haematomas occurred in two patients.
72 onates; to study the natural history of such subdural haematomas; and to ascertain which obstetric fa
73         Presence of unilateral and bilateral subdural haemorrhage is not necessarily indicative of ex
74                              All babies with subdural haemorrhage were assessed clinically but no int
75 lso to have a skull fracture, a thin film of subdural haemorrhage, but lack extracranial injury.
76 s in symptomatic neonates and babies in whom subdural haemorrhages are detected fortuitously.
77       We aimed to establish the frequency of subdural haemorrhages in asymptomatic term neonates; to
78                     They tend to have larger subdural haemorrhages, and where traumatic axonal injury
79                              Nine babies had subdural haemorrhages: three were normal vaginal deliver
80 in >/=2% of patients were hematuria (2%) and subdural hematoma (2%).
81 ere 128 subarachnoid hemorrhage (33.4%), 134 subdural hematoma (35.0%), and 121 intraparenchymal hemo
82  agonist, BAY X3702, in a rat model of acute subdural hematoma (ASDH).
83                    Among 10010 patients with subdural hematoma (mean age, 69.2 years; 3462 women [34.
84                                      The rat subdural hematoma (SDH) model produces a zone of ischemi
85          In that series, one patient died of subdural hematoma 380 days after implant.
86 ceiving apixaban who developed a spontaneous subdural hematoma and declining mental status that impro
87                        There were 6 cases of subdural hematoma and intracranial injury reported in fo
88 ticularly among those >80 yrs of age (36% of subdural hematoma cohort), in lower income patients, in
89               Neurosurgical intervention for subdural hematoma decreased from 41% in 1998 to 31% in 2
90                                              Subdural hematoma evacuation was associated with decreas
91                   The increased incidence of subdural hematoma from 2000 to 2015 appears to be associ
92                                 Incidence of subdural hematoma has been reported to be increasing.
93            The prevalence and total cost for subdural hematoma has increased significantly in the las
94                                              Subdural hematoma incidence and antithrombotic drug use
95 bdural hematoma risk and determine trends in subdural hematoma incidence and antithrombotic drug use
96                                  The overall subdural hematoma incidence rate increased from 10.9 per
97 dural hematoma with antithrombotic drug use, subdural hematoma incidence rate, and annual prevalence
98                         Hospitalizations for subdural hematoma increased from 59,373 (30 per 100,000
99                            The prevalence of subdural hematoma increased with age (p < .001), particu
100                                              Subdural hematoma is a common type of intracranial hemor
101              Health resource consumption for subdural hematoma is increasing without clear evidence t
102                                              Subdural hematoma occurred in 18% of HI (5% of TP), with
103                                              Subdural hematoma occurred in 8 patients (2 in the core
104 of various ages, particularly rib fractures, subdural hematoma of the brain, and retinal hemorrhages.
105 rge disposition, length of stay, and cost of subdural hematoma over time.
106 tients aged 20 to 89 years with a first-ever subdural hematoma principal discharge diagnosis from 200
107 tion between use of antithrombotic drugs and subdural hematoma risk and determine trends in subdural
108 ritical care unit with an acute nontraumatic subdural hematoma that required emergent surgical evacua
109 f subdural hematoma; and the highest odds of subdural hematoma was associated with combined use of a
110                                  The risk of subdural hematoma was highest when a VKA was used concur
111                               Association of subdural hematoma with antithrombotic drug use, subdural
112 rmatory cranial CT scan revealed a worsening subdural hematoma with midline shift, a single dose of f
113 echanical fall with head trauma resulting in subdural hematoma with no associated neurological defici
114  (43%, 26/60); central pontine myelinolysis, subdural hematoma, acute infarcts, and Aspergillus brain
115 in injury, primary intracerebral hemorrhage, subdural hematoma, brain tumor, central nervous system i
116 ciousness or amnesia for more than 24 hours, subdural hematoma, or brain contusion).
117 for later seizures were brain contusion with subdural hematoma, skull fracture, loss of consciousness
118 8-4.03]) were associated with higher risk of subdural hematoma.
119             A CT scan of his head revealed a subdural hematoma.
120 s old, 66% were male patients, and 62.6% had subdural hematoma; admission Glasgow Coma Scale score wa
121  drug use was associated with higher risk of subdural hematoma; and the highest odds of subdural hema
122 ognostic factors following surgery for acute subdural hematomas (ASDHs) in England and Wales over a 2
123                                              Subdural hematomas (SDH) can induce ischemia and neurona
124 morrhagic contusions or underlying evacuated subdural hematomas was studied.
125 djacent to cerebral contusions or underlying subdural hematomas, even brief periods of hyperventilati
126  may shorten detection time for epidural and subdural hematomas, increase sensitivity (especially for
127                Major discrepancies were four subdural hematomas, one pneumocephalus, one hemorrhagic
128 ury, only one (1%) of 70 children had spinal subdural hemorrhage at presentation; this patient had di
129 icance of the proportion of the spinal canal subdural hemorrhage in abusive head trauma versus that i
130       The comparison of incidences of spinal subdural hemorrhage in abusive head trauma versus those
131  21 years were tabulated for histopathology: subdural hemorrhage in the optic nerve sheath, intrascle
132                                 Spinal canal subdural hemorrhage was present in more than 60% of chil
133 maging, and 24 (63%) of 38 had thoracolumbar subdural hemorrhage.
134 t are characteristic of abusive head trauma--subdural hemorrhages, optic nerve sheath hemorrhages, an
135                 As a conclusion, spontaneous subdural hygroma is a rare complication of the arachnoid
136                                              Subdural infusion of CA-I in rats induced cerebral vascu
137 tempted to mimic the actions of glutamate by subdural infusion of the selective glutamate receptor ag
138                         Initiation of EAE or subdural injection of IL-1beta induces a similar cytokin
139 Our findings provide proof-of-principle that subdural intraspinal pressure at the injury site can be
140                                Perivascular, subdural meningeal and choroid plexus macrophages are no
141 xtraction had a significantly higher rate of subdural or cerebral hemorrhage (odds ratio, 2.7; 95 per
142 dence interval, 1.8 to 3.4), but the rate of subdural or cerebral hemorrhage associated with vacuum e
143 heart failure, chest pain, other angina, and subdural or extradural haemorrhage.
144 ve patients with refractory epilepsy in whom subdural or intracerebral electrodes were implanted for
145 ither spontaneous intracerebral, spontaneous subdural, or postoperative.
146 obes were inserted to simultaneously monitor subdural pressure below the injury and extradural pressu
147  pressure at the injury site was higher than subdural pressure below the injury or extradural pressur
148 k of high frequency oscillations in adjacent subdural recording sites, despite the presence of a stro
149                  When applied to the broader subdural recording, this measure consistently predicted
150                             Here we describe subdural recordings from epileptic patients learning to
151 ing depolarizations were first identified in subdural recordings, and EEG was then examined visually
152  recordings in conjunction with intracranial subdural recordings, we asked whether fine duration disc
153 ction of 100 or 200 microl of blood into the subdural space (SDH) or into the caudate nucleus (ICH) o
154 isation to receive a drain inserted into the subdural space and 107 to no drain after evacuation.
155              The injection of blood into the subdural space or into the brain parenchyma induced bloo
156 n of 400 microl of autologous blood into the subdural space.
157                                 Insertion of subdural spinal pressure probe.
158 ble, small molecules can diffuse through the subdural/subarachnoid space into the underlying neocorte
159 tal high frequency activity at the cortical (subdural) surface.
160             Seventeen neonates with ICHs (16 subdural, two subarachnoid, and six parenchymal hemorrha

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