ライフサイエンスコーパス: subendothelial matrix

1 its adhesion to endothelial cells and to the subendothelial matrix.

2 were present as a maturation stimulus in the subendothelial matrix.

3 heparan sulfate, laminin, or collagen in the subendothelial matrix.

4 the retention of plasma lipoproteins in the subendothelial matrix.

5 nt of highly sulfated heparin-like HS in the subendothelial matrix.

6 ding of LDL to artery derived decorin and to subendothelial matrix.

7 ce of TN on the surface of HBMECs and in the subendothelial matrix.

8 Although the biology of platelet adhesion on subendothelial matrix after vascular injury is well char

9 This enhances deposition of Fg in subendothelial matrix and interstitium making the immobi

10 Binding of lipoprotein (a) [Lp(a)] to both subendothelial matrix and Matrigel(R) increased 2-10-fol

11 oB100- containing lipoproteins with heparin, subendothelial matrix, and artery wall purified proteogl

12 ha2(VIII) collagen, a major component of the subendothelial matrix, and examined the ability of and m

13 ver, RBC interactions with components of the subendothelial matrix are not well-characterized.

14 Rather than penetrate deeply into the subendothelial matrix, as is seen with untreated control

15 Monocytes that remained in the subendothelial matrix became macrophages.

16 Most migrated cells remained in the subendothelial matrix, but ~10% underwent spontaneous ba

17 a containing apoB17 decreased LDL binding to subendothelial matrix by 42%.

18 itive erythrocytes adhere to endothelium and subendothelial matrix components.

19 On subendothelial matrix, endogenous vWF and adsorbed plasm

20 Subendothelial matrix, fibronectin, or collagen I stimul

21 cal step in the adhesion of platelets to the subendothelial matrix following endothelial cell damage,

22 young thrombocytes, they adhere first to the subendothelial matrix, get activated rapidly, release ag

23 tribute to erythrocyte interactions with the subendothelial matrix, hereby participating in the patho

24 However, in subendothelial matrix in blood vessels and in the baseme

25 the ability to sustain protrusions into the subendothelial matrix in contrast with control cells.

26 telets rapidly adhere to the site of exposed subendothelial matrix in the vessel wall, become activat

27 Platelet microparticles bind to subendothelial matrix in vitro and in vivo and can act a

28 cells (RBCs) to the vascular endothelium and subendothelial matrix likely plays a significant role in

29 vascular HS(act) predominantly occurs in the subendothelial matrix, mice were subjected to a carotid

30 lipoprotein B lipoproteins with the specific subendothelial matrix molecules that mediate retention a

31 at mediates the adhesion of platelets to the subendothelial matrix of injured blood vessels.

32 turns to baseline; the basement membrane and subendothelial matrix of the inner wall appear to remain

33 urned to baseline; the basement membrane and subendothelial matrix of the inner wall remained intact.

34 e incorporated dying autologous cells in the subendothelial matrix of the model.

35 Herein, we show that MPO concentrates in the subendothelial matrix of vascular tissues by a transcyto

36 ligand, von Willebrand factor (vWF), in the subendothelial matrix or plasma.

37 On interaction with the subendothelial matrix, platelets are transformed into ba

38 Cs) have enhanced adhesion to the plasma and subendothelial matrix protein thrombospondin-1 (TSP) und

39 epositing them to collagen and other exposed subendothelial matrix proteins.

40 ere to HUVECs, whereas only spores adhere to subendothelial matrix proteins.

41 Inhibition of LOX-dependent subendothelial matrix stiffening alone suppressed HG-ind

42 s of tunable stiffness, we demonstrated that subendothelial matrix stiffening is necessary and suffic

43 that HG significantly enhances LOX-dependent subendothelial matrix stiffness in vitro, which correlat

44 lymorphonuclear leukocytes [PMNs]) encounter subendothelial matrix substrates that may require additi

45 IIb/IIIa) to interact with components of the subendothelial matrix, such as fibronectin (Fn), exposed

46 Platelets interact with collagen in the subendothelial matrix that is exposed by vascular damage

47 elets initially adhere on vWF affixed to the subendothelial matrix through the glycoprotein (GP) Ib-I

48 W) VWF is targeted basolaterally, toward the subendothelial matrix, using the adaptor protein complex

49 To mimic the subendothelial matrix, vWF was microarrayed over sonicat