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1  for surgical resection or other therapy for subependymal giant-cell astrocytoma.
2 arised tumours including angiomyolipomas and subependymal giant cell astrocytomas.
3 me of 50% or greater relative to baseline in subependymal giant cell astrocytomas.
4 ally and significantly reduced the volume of subependymal giant cell astrocytomas.
5 s (3q and 9p), one astrocytoma (6q), and two subependymal giant cell astrocytomas (16p and 21q).
6 cm or greater, and either serial growth of a subependymal giant cell astrocytoma, a new lesion of 1 c
7 disease was more severe than TSC1, with more subependymal giant cell astrocytomas and angiomyolipomas
8 iated with marked reduction in the volume of subependymal giant-cell astrocytomas and seizure frequen
9 ent study, 25 TSC-related cortical tubers or subependymal giant cell astrocytomas, as well as tissue
10 eaningful reduction in volume of the primary subependymal giant-cell astrocytoma, as assessed on inde
11 se results support the use of everolimus for subependymal giant cell astrocytomas associated with tub
12 cy and safety of everolimus in patients with subependymal giant cell astrocytomas associated with tub
13 se results support the use of everolimus for subependymal giant cell astrocytomas associated with tub
14 ficacy end point was the change in volume of subependymal giant-cell astrocytomas between baseline an
15                      In the brain, growth of subependymal giant cell astrocytomas can cause life-thre
16 the molecular and developmental basis of the subependymal giant cell astrocytoma in humans.
17 erruption of therapy resulted in regrowth of subependymal giant cell astrocytomas in one patient.
18 ical resection is the standard treatment for subependymal giant-cell astrocytomas in patients with th
19 ects with clinically definite TSC and either subependymal giant cell astrocytomas (n = 4) or a pilocy
20 erved, which may be the murine equivalent of subependymal giant cell astrocytomas or tubers commonly
21 findings are more likely to have concomitant subependymal giant cell astrocytomas, renal angiomyolipo
22  and CNS lesions include cortical tubers and subependymal giant cell astrocytomas, resulting in menta
23 Aug 10, 2009, more than 35% of patients with subependymal giant cell astrocytoma (SEGA) associated wi
24 ed expression of these proteins in tuber and subependymal giant cell astrocytoma (SEGA) specimens in
25 xpression was assessed in tuber (n = 16) and subependymal giant cell astrocytoma (SEGA; n = 6) specim
26              Subependymal nodules (SENs) and subependymal giant cell astrocytomas (SEGAs) are common
27 rtical dysplasias, subependymal nodules, and subependymal giant cell astrocytomas (SEGAs).
28 s in TSC patients, renal angiomyolipomas and subependymal giant cell astrocytomas (SEGAs).
29  had at least 50% reduction in the volume of subependymal giant cell astrocytomas versus none in the
30  years of age or older with serial growth of subependymal giant-cell astrocytomas were eligible for t
31  Five to 15% of affected individuals display subependymal giant cell astrocytomas, which can lead to

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