ライフサイエンスコーパス: subepidermal

1 Subepidermal autoimmune blistering dermatoses (AIBD) are

2 logic hallmark of bullous pemphigoid (BP), a subepidermal autoimmune blistering disease characterized

3 Anti-p200 pemphigoid is a rare subepidermal autoimmune blistering disease characterized

4 s, lateral line-dependent alterations of the subepidermal basement membrane, and a general elaboratio

5 idth, delayed palatal shelf development, and subepidermal blebbing.

6 The phenotypes of these embryos, including subepidermal blebs flanking the neural tube at E8 and bl

7 bullous disease of the skin characterized by subepidermal blister formation due to tissue-bound and c

8 -dependent pathway play an essential role in subepidermal blister formation in experimental BP, and s

9 sical and alternative pathways is crucial in subepidermal blister formation in experimental BP.

10 ential role in neutrophil recruitment during subepidermal blister formation in experimental BP.

11 e, resulting in an inflammatory response and subepidermal blister formation.

12 racterized by intraepidermal (pemphigus) and subepidermal blistering (pemphigoid diseases and dermati

13 ed hallmark RDEB disease features, including subepidermal blistering and anchoring fibril defects.

14 Bullous pemphigoid (BP) is an inflammatory subepidermal blistering disease associated with an IgG a

15 Bullous pemphigoid (BP) is an inflammatory subepidermal blistering disease associated with an IgG a

16 Bullous pemphigoid (BP) is an autoimmune subepidermal blistering disease associated with autoanti

17 Bullous pemphigoid (BP) is a subepidermal blistering disease associated with autoanti

18 Epidermolysis bullosa acquisita is a subepidermal blistering disease associated with tissue-b

19 Bullous pemphigoid (BP) is a subepidermal blistering disease characterized by autoant

20 Bullous pemphigoid (BP) is an autoimmune subepidermal blistering disease characterized by deposit

21 Bullous pemphigoid (BP) is a subepidermal blistering disease characterized by IgE and

22 gainst type VII collagen into mice induces a subepidermal blistering disease dependent upon activatio

23 Bullous pemphigoid (BP) is an autoimmune subepidermal blistering disease seen primarily in elderl

24 BP180 were shown to trigger an inflammatory subepidermal blistering disease that closely mimics bull

25 dermolysis bullosa is an autosomal recessive subepidermal blistering disease typified by null mutatio

26 nesis of autoantibody-induced, cell-mediated subepidermal blistering diseases and identified new ther

27 ry forms of EB among the "cell-poor" list of subepidermal blistering disorders.

28 high levels of p38 MAPK phosphorylation and subepidermal blistering in MC-deficient mice.

29 e pathogenic anti-BP180 IgG failed to induce subepidermal blistering in NE-null (NE(-/-)) mutant mice

30 st cells, and neutrophils play a key role in subepidermal blistering in this animal model.

31 In an experimental BP model in mice, the subepidermal blistering is mediated by antibodies direct

32 the IgG passive transfer mouse model of BP, subepidermal blistering is triggered by anti-BP180 antib

33 In experimental BP, subepidermal blistering is triggered by rabbit anti-muri

34 In patients with subepidermal blistering skin disease but no oral or ocul

35 A biopsy specimen showed subepidermal blistering with prominent inflammatory cell

36 P) is an autoimmune disease characterized by subepidermal blistering.

37 ceded neutrophil infiltration and subsequent subepidermal blistering.

38 an autoimmune skin disease characterized by subepidermal blisters and autoantibodies against 2 hemid

39 pathogenic anti-BP180 IgG failed to develop subepidermal blisters and exhibited a drastic reduction

40 dermal fragility as well as noninflammatory, subepidermal blisters in grafted human (but not adjacent

41 antibodies against type VII collagen create subepidermal blisters in mice that mimic those seen in p

42 Anti-laminin 5 IgG also induced subepidermal blisters in: adult complement-, mast cell-,

43 rine C3 in epidermal basement membranes, and subepidermal blisters of skin and mucous membranes.

44 gG to neonatal mice has been shown to induce subepidermal blisters that mimic those in patients, it h

45 or mast cell-deficient (n = 3) mice produced subepidermal blisters with the same clinical, histologic

46 d neutrophil-rich infiltrates, dermal edema, subepidermal blisters, and deposits of immunoreactants i

47 tes, histologic evidence of noninflammatory, subepidermal blisters, and deposits of rabbit IgG and mu

48 BP180 IgG developed delayed and less intense subepidermal blisters.

49 ic anti-murine BP180 IgG exhibited extensive subepidermal blisters.

50 OL7 and the human COL7 transgene and induced subepidermal blisters.

51 l autoimmune disease characterized by severe subepidermal bullous eruptions and renal insufficiency w

52 s, ovules early in development, and a set of subepidermal cells near the abaxial surface of the anthe

53 EX FORM1 show similar expression patterns in subepidermal cells of pistils just before abortion.

54 wering plants requires the commitment of the subepidermal cells of the ovules and anthers to enter th

55 dent pathway that regulates proliferation of subepidermal cells.

56 nsible for triggering periclinal division of subepidermal cells.

57 gnal, ENOD40 expression was seen in dividing subepidermal cortical cells.

58 Male-sterile mutants with subepidermal defects--mac1 (excess meiocytes), ms23 (def

59 gher in the tail than in the body within the subepidermal fibroblast layer, further indicating that t

60 ysis implicates the single layer of invasive subepidermal fibroblasts as crucial in tail resorption.

61 ndary of delayed response class genes in the subepidermal fibroblasts in the body correlates with epi

62 chord and lie beneath the epidermal lamella (subepidermal fibroblasts).

63 protein 1 (GRIP1), leads to the formation of subepidermal hemorrhagic blisters, renal agenesis, synda

64 In scz mutants, the subepidermal layer (ground tissue) develops root hairs.

65 ain a subclass of trichomes derived from the subepidermal layer.

66 istinct cell types around the vasculature in subepidermal layers of both root and shoots, with cell t

67 imeras with wild-type fim expression only in subepidermal layers.

68 mmary bud, but is found generally within the subepidermal mesenchyme.

69 Subepidermal microinjection of function-blocking antibod

70 There was also severe nerve loss from the subepidermal neural plexus (SNP) and deep dermis.

71 generation of epidermal nerve fibers and the subepidermal neural plexus in capsaicin-treated skin, as

72 ugh another mechanism, perhaps by increasing subepidermal osmolarity.

73 cetic acid was associated with a decrease in subepidermal pH to a level that has been shown to activa

74 In separate experiments, subepidermal pH was measured in vitro while these same s

75 id evokes this wiping response by decreasing subepidermal pH.

76 voked the wiping response without decreasing subepidermal pH.

77 ould evoke the wiping response by decreasing subepidermal pH.

78 f the Manduca sexta larva is supplied with a subepidermal plexus of approximately 350 multidendritic

79 uses a circular incision that transects the subepidermal plexus, resulting in Wallerian degeneration

80 formation of subcorneal, intraepidermal, and subepidermal pustules.

81 een days 21 and 24 and was restricted to the subepidermal regions at the junction of the opposing eye

82 the short form was present along the entire subepidermal regions both during fetal and neonatal deve

83 med at characterization of the epidermal and subepidermal responses to environmental changes.

84 Mice injected with immune IgG developed subepidermal skin blisters and erosions, IgG deposits at

85 oss the epidermal basement membrane into the subepidermal space.

86 sulfide isomerase was found primarily in the subepidermal tissues.

87 as only nine were found predominantly in the subepidermal tissues.

88 g at age 2 months and then develop erosions, subepidermal vesicles, eosinophil-rich skin infiltrates,