ライフサイエンスコーパス: subepithelial

1 tions of patients with CRSwNP in parallel to subepithelial accumulation of IgA.

2 eveloped marked epithelial hyperplasia and a subepithelial acellular zone.

3 osition of Charcot-Leyden-like crystals, and subepithelial airway fibrosis were also prominently note

4 Asthma can progress to subepithelial airway fibrosis, mediated in large part by

5 ay epithelium can express factors that drive subepithelial airway remodeling.

6 modality is capable of in vivo diagnosis of subepithelial airway wall pathology as signs of intubati

7 Despite subepithelial alpha-actin positive myofibroblast prolife

8 Subepithelial and intraepithelial lymphocytes of human a

9 ute rejection and lymphocytic bronchitis, to subepithelial and intraluminal fibrotic lesions of bronc

10 tase and dipeptidyl peptidase 4) and visible subepithelial and smooth muscle layers when compared wit

11 cluded fusion of podocyte foot processes and subepithelial and subendothelial deposition.

12 h asthma, a disease characterized in part by subepithelial angiogenesis.

13 CRSwNP, and with increased IgA deposition in subepithelial areas.

14 the GBM that became most prominent along the subepithelial aspect at maturity; labeling was greatly r

15 rity; labeling was greatly reduced along the subepithelial aspect in agrin-deficient and conditional

16 ripheral cornea in a single patient showed a subepithelial, avascular pannus.

17 This increased subepithelial B7-2 expression may contribute to the sust

18 % [25th-75th IQR], 3.5% to 10.1%, P < .001), subepithelial basement membrane thickening (4.4 mum [25t

19 ssessed for airway smooth muscle (ASM) area, subepithelial basement membrane thickness, nerve fibers,

20 remodeling (measured as the thickness of the subepithelial basement membrane) (r=0.51, P=0.003).

21 correlated with severity, thickening of the subepithelial basement membrane, and pulmonary function.

22 RECENT FINDINGS: MMP is an uncommon, subepithelial blistering conjunctivitis that is commonly

23 OCP is a subepithelial, blistering, autoimmune disease that mainl

24 noreactivities (IRs) in supporting cells and subepithelial Bowman's gland acinar cells, two OE non-ne

25 cally positioned in proximity to a number of subepithelial cell types, including endothelia.

26 al cell density and the presence of abnormal subepithelial cells (presumed fibroblasts).

27 ative central corneal thicknesses when these subepithelial cells were and were not visible were 652 (

28 sity and number and the presence of abnormal subepithelial cells were determined from confocal images

29 Abnormal subepithelial cells were visible in 9%, 19%, and 30% of

30 Abnormal subepithelial cells, presumably fibroblasts, are present

31 gnant colonic tissue is probably confined to subepithelial cells, such as fibroblasts.

32 Abnormal subepithelial cells, which formed reticular networks dee

33 n of mechanical state between epithelial and subepithelial cells.

34 hereas IL-33 was expressed in epithelial and subepithelial cells.

35 including intraluminal mucus production and subepithelial collagen deposition, but did not alter eos

36 ed increase in airway smooth muscle mass and subepithelial collagen deposition.

37 avage fluid, mucin production, ASM mass, and subepithelial collagen deposition.

38 re respectively characterized by a thickened subepithelial collagen plate and increased intraepitheli

39 erial products that have penetrated into the subepithelial compartment.

40 as compared in three distinct epithelial and subepithelial compartments isolated from biopsies of nor

41 s in the portal tracts and in epithelial and subepithelial compartments of extrahepatic bile ducts, w

42 n and bacterial invasion of perivascular and subepithelial compartments.

43 Subepithelial connective tissue (CT)-based procedures an

44 epithelialized free soft tissue autografts, subepithelial connective tissue autografts, coronally po

45 eive either a coronally advanced flap with a subepithelial connective tissue graft (control) or a cor

46 ing and a coronally advanced flap (CAF) to a subepithelial connective tissue graft (CTG) in combinati

47 The subepithelial connective tissue graft (CTG) is a popular

48 The most common treatment is the subepithelial connective tissue graft (CTG), but good ou

49 e similarities between collagen membrane and subepithelial connective tissue graft (SCTG) have made c

50 The subepithelial connective tissue graft (SCTG) is a predic

51 The subepithelial connective tissue graft (SCTG) is one of t

52 Most clinicians adopt two versions of the subepithelial connective tissue graft (SCTG) procedure,

53 Subepithelial connective tissue graft (SCTG) procedures

54 perative morbidity, and esthetic outcomes of subepithelial connective tissue graft (SCTG) technique w

55 The first paper in this series evaluated the subepithelial connective tissue graft and the coronally

56 lap resulted in root coverage similar to the subepithelial connective tissue graft but without the mo

57 ng excision of the lesion, by placement of a subepithelial connective tissue graft concurrently with

58 and patient-reported discomfort, whereas the subepithelial connective tissue graft demonstrated great

59 The subepithelial connective tissue graft has markedly broad

60 Most of the literature suggests that the subepithelial connective tissue graft has the highest pe

61 The treatment consisted of a subepithelial connective tissue graft in conjunction wit

62 ate the healing and revascularization of the subepithelial connective tissue graft in dogs.

63 The subepithelial connective tissue graft in this study was

64 eth that were randomized to receive either a subepithelial connective tissue graft or a coronally adv

65 It is concluded that: 1) the subepithelial connective tissue graft procedure provides

66 Histological evaluation of the subepithelial connective tissue graft revealed a connect

67 ed to eliminate the lesion, beginning with a subepithelial connective tissue graft to increase tissue

68 and submitted for histologic examination; a subepithelial connective tissue graft was harvested and

69 y advanced flap with EMD was superior to the subepithelial connective tissue graft with regard to ear

70 coverage of 73% (collagen membrane) and 84% (subepithelial connective tissue graft) was achieved.

71 re, including a laterally positioned flap, a subepithelial connective tissue graft, and a coronally p

72 d flap with EMD and 79% of the time with the subepithelial connective tissue graft.

73 ce after free soft tissue grafting (FSTG) or subepithelial connective tissue grafting (SCTG) procedur

74 Subepithelial connective tissue grafts (CTGs) and free g

75 pare their soft tissue healing with those of subepithelial connective tissue grafts (SCTG).

76 Subepithelial connective tissue grafts (SCTGs), matrix g

77 All were treated surgically with subepithelial connective tissue grafts and followed for

78 acid root demineralization in the outcome of subepithelial connective tissue grafts performed to cove

79 oethylene (ePTFE) membranes and conventional subepithelial connective tissue grafts, respectively.

80 r cigarette smoking affects wound healing of subepithelial connective tissue grafts.

81 ve placed under a coronally advanced flap to subepithelial connective tissue placed under a coronally

82 The collagen bundles reached into the subepithelial connective tissue where elongated rete-peg

83 Oral subepithelial connective tissues were harvested aseptica

84 Peripheral hypertrophic subepithelial corneal degeneration is an uncommon, usual

85 0.05% cyclosporine A (CsA) in patients with subepithelial corneal infiltrates (SEI).

86 Existing evidence suggests that early subepithelial corneal infiltrates are composed of polymo

87 NPD1 increased subepithelial corneal nerve area three times compared wi

88 [91%]), elevated circumferential peripheral subepithelial corneal opacities and adjacent abnormal li

89 Subepithelial CT graft-based procedures provided the bes

90 ying the expansion of the mucosal epithelial/subepithelial DC network in allergic inflammation.

91 hans' cells did not express DC-SIGN, whereas subepithelial DCs in the lamina propria expressed modera

92 rix metalloproteinase-dependent migration of subepithelial DCs into the FAE, but not into villus epit

93 rix metalloproteinase-dependent migration of subepithelial dendritic cells (DCs) into the FAE, better

94 us, MV infection of alveolar macrophages and subepithelial dendritic cells in the airways precedes in

95 erial within nonciliated cells and increased subepithelial deposition of collagen.

96 Increased subepithelial deposition of extracellular matrix protein

97 ptors did not ameliorate injury, implicating subepithelial deposition of immune complexes and consequ

98 4/19 showed absence of deposits or rare tiny subepithelial deposits by electron microscopy (EM).

99 omplex deposition, and absence of glomerular subepithelial deposits compared with MRL/lpr mice of any

100 buminuria and nephrotic syndrome, because of subepithelial deposits of mouse IgG and C3 with correspo

101 Rare subepithelial deposits were found in the glomeruli.

102 sease involving the eye and characterized by subepithelial detachment resulting from an immunologic r

103 sigma1, but not sigmaNS, was detected in the subepithelial dome (SED) in association with CD11c(+)/CD

104 se the chemokine receptor CCR6 to access the subepithelial dome (SED) of PPs.

105 molecules, particles, and pathogens into the subepithelial dome (SED) region of Peyer's patch mucosa,

106 oid DCs were determined to be present in the subepithelial dome (SED) region, whereas CD8alpha(+) lym

107 orted from the mucosal surface of PPs to the subepithelial dome (SED), through the specialized epithe

108 pressing CD11c and CD11b are absent from the subepithelial dome of Peyer's patches.

109 al bacteria targeting dendritic cells in the subepithelial dome region of PPs represents a mechanism

110 )CD8(-) dendritic cell subset located in the subepithelial dome region of PPs, confirming that the ho

111 t reduction of the CD11b(+) DC number in the subepithelial dome regions of Peyer's patches of both wi

112 (+) dendritic cells (DCs) distributed in the subepithelial dome regions of the Peyer's patches, and m

113 ular regions and in clusters of cells in the subepithelial dome regions.

114 8alpha(-) (myeloid) DCs are localized in the subepithelial dome, CD11b(-)/CD8alpha(+) (lymphoid) DCs

115 ollicle-associated epithelium and DCs of the subepithelial dome.

116 ry to biliary ductal epithelium and 99.3% to subepithelial ductal glands.

117 Subepithelial EC were smaller, more irregular in shape,

118 on molecules on EC nearest the airway lumen (subepithelial EC) were different from those on the oppos

119 rane, especially in regions corresponding to subepithelial electron dense deposits.

120 ts without any sputum eosinophils had normal subepithelial eosinophil numbers (< 1.2%; NPV, 89%).

121 PPV of any sputum eosinophils for increased subepithelial eosinophilia was only 36.4%.

122 his condition from other conditions in which subepithelial, eosinophilic, amorphous materials are dep

123 H & E-stained sections showed subepithelial, eosinophilic, amorphous, acellular deposi

124 antigen-specific CD8+ T cells were intra- or subepithelial, expressed alphaE-integrin CD103, produced

125 agen VIII is localized in subendothelial and subepithelial extracellular matrices.

126 We established a panel of 33 primary subepithelial fibroblast strains from human colonoscopic

127 get gene Gli1 is preferentially expressed in subepithelial fibroblast-like cells, one of four prostat

128 gically, interstitial monocytes/macrophages, subepithelial fibroblast-like interstitial cells, and ad

129 in the mesenchyme caused progressive loss of subepithelial fibroblasts and abbreviated gut length, re

130 induction of cyclooxygenase 2 expression in subepithelial fibroblasts and in villous, but not crypt,

131 m with functional cilia, mucus secretion and subepithelial fibroblasts within type I collagen.

132 ecovery, induced IL-1 receptor expression in subepithelial fibroblasts, and activated de novo inflamm

133 ibrotic lungs, induces production of TN-C by subepithelial fibroblasts.

134 metaplasia (r = 0.36, P = 0.03) and corneal subepithelial fibrosis (r = 0.5, P = 0.0006).

135 aplasia, B and T cell-rich inflammation, and subepithelial fibrosis and augmented the levels of mRNA

136 Findings were correlated with subepithelial fibrosis and clinical CT score.

137 ucus hypersecretion, goblet cell metaplasia, subepithelial fibrosis and enhanced airway hyperreactivi

138 In addition to these features, subepithelial fibrosis and goblet cell hyperplasia chara

139 tear EGF may promote development of corneal subepithelial fibrosis and lid margin changes.

140 n, dye staining, and the presence of corneal subepithelial fibrosis and meibomian gland (MG) orifice

141 ployed in cases wherein visually significant subepithelial fibrosis and scarring become evident after

142 l remodeling of the airways characterized by subepithelial fibrosis and smooth muscle hyperplasia.

143 ammation and significantly reduced levels of subepithelial fibrosis as assessed by either trichrome s

144 Early intervention prior to subepithelial fibrosis can lead to good visual outcomes

145 x protein, has been localized to deposits of subepithelial fibrosis in asthmatic patients, and perios

146 EMT likely contributes to subepithelial fibrosis in subjects with EoE and resolves

147 anisms underlying esophageal remodeling with subepithelial fibrosis in subjects with eosinophilic eso

148 cus plugging, smooth muscle hyperplasia, and subepithelial fibrosis in the OVA-sensitized/challenged

149 whether the secretin/SR axis plays a role in subepithelial fibrosis observed during cholestasis.

150 corneas were vascularized or had pronounced subepithelial fibrosis on results of slitlamp examinatio

151 -mesenchymal signaling, leading to increased subepithelial fibrosis or hyperplasia of smooth muscle.

152 ia, smooth muscle cell layer thickening, and subepithelial fibrosis present on Day 73 persisted at Da

153 ith subsequent epithelial transformation and subepithelial fibrosis that could not be reversed with i

154 yperreactivity, goblet cell hyperplasia, and subepithelial fibrosis that is initiated by the intratra

155 e, significantly fewer goblet cells and less subepithelial fibrosis were observed around large airway

156 ion, hyper-responsiveness to spasmogens, and subepithelial fibrosis were significantly enhanced in CC

157 cytokine that induces tissue remodeling with subepithelial fibrosis when expressed in the airway.

158 e of chronic asthma that often culminates in subepithelial fibrosis with variable airway obstruction.

159 l hyperplasia, basement membrane thickening, subepithelial fibrosis, airway smooth muscle hyperplasia

160 ypersecretion leading to airway obstruction, subepithelial fibrosis, airway smooth muscle hyperplasia

161 ed pulmonary inflammation, mucus metaplasia, subepithelial fibrosis, and airway remodeling are signif

162 ncrease in peribronchial smooth muscle mass, subepithelial fibrosis, and angiogenesis.

163 osinophilic inflammation, mucin composition, subepithelial fibrosis, and corticosteroid responsivenes

164 plasia, smooth muscle cell layer thickening, subepithelial fibrosis, and levels of T helper type 2 ce

165 tion was increased in eyes with MGD, corneal subepithelial fibrosis, and MG orifice metaplasia.

166 g, including increased airway smooth muscle, subepithelial fibrosis, and mucus.

167 athophysiologic aspects of human asthma (ie, subepithelial fibrosis, angiogenesis, neural biology, an

168 features of airway remodeling, in particular subepithelial fibrosis, by reducing the production of eo

169 tion did not affect epithelial thickening or subepithelial fibrosis, despite significantly inhibiting

170 diagnosis, eosinophil counts, and indices of subepithelial fibrosis, eosinophil peroxidase, and TGF-b

171 eactivity, and remodeling of the airway (eg, subepithelial fibrosis, goblet cell metaplasia, and smoo

172 elial mosaic, cystic epithelial changes, and subepithelial fibrosis, in the eyes affected by partial

173 rway smooth muscle cell layer thickening and subepithelial fibrosis, key allergen-induced airway stru

174 tion, eosinophil infiltration of the airway, subepithelial fibrosis, mucus metaplasia, and airway-hyp

175 with significantly reduced mucus secretion, subepithelial fibrosis, smooth muscle thickness, and per

176 ia, smooth muscle cell layer thickening, and subepithelial fibrosis.

177 n of leukocytes, goblet cell hyperplasia and subepithelial fibrosis.

178 ng of respiratory epithelium and significant subepithelial fibrosis.

179 lasia of goblet cells and smooth muscle, and subepithelial fibrosis.

180 ced tissue hyaluronic acid accumulation, and subepithelial fibrosis.

181 n of inflammatory cells, and by promotion of subepithelial fibrosis.

182 ling including smooth muscle hyperplasia and subepithelial fibrosis.

183 e absence of clinically obvious preoperative subepithelial fibrosis.

184 tural analysis revealed occasional knob-like subepithelial GBM thickening but intact podocyte foot pr

185 pithelium in the chilled saline group and to subepithelial glands in both the room-temperature and ch

186 e degree of injury to biliary epithelium and subepithelial glands was assessed on a scale of 0%-100%.

187 of 52.9% ductal epithelial injury and 12.1% subepithelial glandular injury.

188 characterized by (1) the formation of large, subepithelial glomerular immune deposits, which stain fo

189 d, as well as the problems of epithelial and subepithelial graft rejection.

190 mm on 2 separate teeth were treated with the subepithelial graft technique.

191 atal connective tissue (CT) were compared as subepithelial grafts for the treatment of gingival reces

192 uble immunofluorescence staining showed that subepithelial granular deposits contained rhASB colocali

193 with BCVA (r = 0.59; P<0.001; n = 27), with subepithelial haze (r = 0.41; P = 0.01; n = 25), and wit

194 .001 and r = 0.46, P = 0.003), postoperative subepithelial haze (r = 0.43, P = 0.004 and r = 0.39, P

195 e correlations between BCVA and preoperative subepithelial haze (r = 0.61, P < 0.001 and r = 0.46, P

196 The study confirmed corneal subepithelial haze and interface haze as important facto

197 The initiation and spread of subepithelial haze begins shortly after reepithelializat

198 thelial, stromal, and corneal thickness, and subepithelial haze following photorefractive keratectomy

199 of Diabetic Retinopathy Study protocol, and subepithelial haze was measured from the brightness of c

200 Relationships between HOAs, BCVA, subepithelial haze, and recipient age were determined.

201 g best corrected visual acuity (BCVA), IVCM (subepithelial haze, interface haze, graft thickness) and

202 d virions did not infect intraepithelial and subepithelial HIV-susceptible cells.

203 lts show that FcRn promotes the formation of subepithelial immune complexes and subsequent glomerular

204 astructurally, there were subendothelial and subepithelial immune deposits and extensive podocyte foo

205 nvestigators induced formation of glomerular subepithelial immune deposits and tubular lesions in pig

206 pathy (MN) have shown that IgG antibodies in subepithelial immune deposits initiate complement activa

207 icroscopy were present in subendothelial and subepithelial immune deposits, whereas WT kidneys in WT

208 visual acuity (BCVA), evaluation of corneal subepithelial infiltrate scores (CSIS), intraocular pres

209 ical analyses of the oral lesions revealed a subepithelial infiltrate that was primarily composed of

210 ukin-8 may play a role in the development of subepithelial infiltrates in adenovirus keratitis.

211 adenovirus replication and the formation of subepithelial infiltrates in the Ad5/New Zealand White r

212 ication, nor did it prevent the formation of subepithelial infiltrates in the rabbit model.

213 and intrastromally to study the formation of subepithelial infiltrates.

214 among all groups (I-IV) in the formation of subepithelial infiltrates.

215 No goblet cells or subepithelial inflammation were present.

216 Vaginal subepithelial inoculation of estrogen-treated animals re

217 the gut and contribute substantially to the subepithelial intestinal myofibroblast population in the

218 We recently demonstrated that normal human subepithelial intestinal myofibroblasts (IMFs) express M

219 on in the intestine mediated specifically by subepithelial intestinal myofibroblasts (IMFs).

220 This study reviews current concepts in laser subepithelial keratectomy (LASEK), variations in LASEK t

221 man cornea results in a chronic, multifocal, subepithelial keratitis.

222 Laser subepithelial keratomileusis (LASEK) is a relatively new

223 photorefractive keratectomy (PRK) and laser subepithelial keratomileusis (LASEK).

224 hotorefractive keratectomy or laser-assisted subepithelial keratomileusis.

225 dividual cells or small cell clusters in the subepithelial lamina propria of monkeys infected with ei

226 ) DCs proliferate in both the epithelial and subepithelial layers of the airway mucosa as well as in

227 from patients with SSc and were localized to subepithelial layers of the distal airways.

228 ut defense mechanisms against C. albicans in subepithelial layers such as the dermis.

229 s of remodeling that included epithelial and subepithelial layers, as well as mucus production, were

230 line the recommended course of action when a subepithelial lesion is encountered during upper endosco

231 a severe inflammatory response hallmarked by subepithelial leukocyte infiltration.

232 of the esophageal epithelium with a striking subepithelial lichenoid lymphocytic infiltrate extending

233 led granular antigen-antibody complexes in a subepithelial location along the glomerular filtration b

234 has been shown to be useful in evaluation of subepithelial masses of the colon and rectum and evaluat

235 gen-sampling mechanism capable of activating subepithelial mast cells.

236 nd traffics across the cell to exit into the subepithelial matrix.

237 rum, and correlated with asthma severity and subepithelial membrane thickness.

238 it, and anti-C1q IgG4 was mainly detected in subepithelial membranous deposits.

239 Hedgehog (Hh) signals promote aggregation of subepithelial mesenchymal clusters that drive villus eme

240 pithelium, with underlying thickening of the subepithelial mucosal layers.

241 helped to determine the HSV-1 distribution: subepithelial myeloid cells provided a route of spread f

242 show that PGE(2) activated human intestinal subepithelial myofibroblasts (18Co) through Gs protein-c

243 racrine, from epithelium to Ptch1-expressing subepithelial myofibroblasts (ISEMFs) and smooth muscle

244 ate that alpha-smooth muscle actin(+), CD90+ subepithelial myofibroblasts (stromal cells) constitutiv

245 1RL1 associated with two stromal cell types, subepithelial myofibroblasts and mast cells, in Apc(Min/

246 Whereas GREM1 is expressed in intestinal subepithelial myofibroblasts in controls, GREM1 is predo

247 In vitro, IL-33 stimulation of human subepithelial myofibroblasts induced the expression of e

248 The association between COX-2 expression and subepithelial myofibroblasts was also noted in tumors de

249 Smooth muscle cells, subepithelial myofibroblasts, and fibroblasts have tradi

250 ignal leads to mislocalization of intestinal subepithelial myofibroblasts, loss of smooth muscle in v

251 ical functions previously ascribed solely to subepithelial myofibroblasts.

252 uscle actin, which is a molecular marker for subepithelial myofibroblasts.

253 irway smooth muscle, platelet activation and subepithelial myofibroblasts.

254 mal tumors (GIST) are stromal or mesenchymal subepithelial neoplasms affecting the gastrointestinal t

255 and IgG3kappa; electron microscopy revealed subepithelial nonorganized deposits.

256 x adult patients developed bilateral diffuse subepithelial opacifications in the central and paracent

257 A distinctive feature is the appearance of subepithelial opacities in adult life, accompanied by a

258 bullosa hemorrhagica (ABH) describes benign subepithelial oral blood blisters not attributable to a

259 tea mediterranea generates porphyrins in its subepithelial pigment cells under physiological conditio

260 propria thickness (defined as the extent of subepithelial portion of the biopsy containing </=25% or

261 vity in overlying epithelium and with marked subepithelial proteolytic activity.

262 scripts with the cells expressing KGF in the subepithelial, rather than the deeper, connective tissue

263 Ragweed pollens were also found in the subepithelial region of the small intestine 24h after po

264 gastritis, COX-2 expression localizes to the subepithelial region, with variable levels in the epithe

265 proliferation in distinct submesothelial and subepithelial regions.

266 periostin are hypothesized to be involved in subepithelial remodeling and are overexpressed in adult

267 ad adequate lamina propria for evaluation of subepithelial remodeling.

268 epithelial and smooth muscle thickening, and subepithelial reticular fiber deposition in the distal a

269 osteroids and was positively correlated with subepithelial reticular membrane thickening.

270 type of afferent, the crypt afferent, forms subepithelial rings of varicose processes encircling the

271 Two sensory-cell types, subepithelial sensory cells (SSCs) and intraepithelial s

272 immune blistering disorders characterized by subepithelial separation and the deposition of immunoglo

273 nty patients with histologic confirmation of subepithelial separation with or without direct immunofl

274 numbers and thickness of the epithelium and subepithelial smooth muscle layer, which was accompanied

275 Most TG nerve endings are subepithelial, so this colonization implies subepithelia

276 A third type of sensory cells, with subepithelial somata and tufts of stiff-cilia (TSCs, pre

277 n, indicating that cell types inhabiting the subepithelial space can provide such an activity to the

278 and intestinal epithelial cells occur in the subepithelial space of the gastrointestinal tract.

279 peribronchial smooth muscle, epithelium, and subepithelial space were measured quantitatively.

280 important to process immune complexes in the subepithelial space, where it also limits complement act

281 ent, such as that of the inflamed intestinal subepithelial space.

282 interactions occur in the epithelium and the subepithelial space.

283 onjunctival and oral/pharyngeal lesions with subepithelial splitting were found in 80% and 100% of mi

284 roblasts were present at high density in the subepithelial stroma of rabbit eyes that had -9.0D PRK,

285 nce of abnormally large proteoglycans in the subepithelial stroma.

286 B ligand (RANKL) is selectively expressed by subepithelial stromal cells in PP domes.

287 in epithelial cells as well as apoptosis in subepithelial stromal cells.

288 ype XVIII collagen immunolocalization to the subepithelial stromal wound region peaked at 1 week afte

289 Subepithelial T-cell and macrophage infiltration on post

290 the timing of innervation of epithelial and subepithelial targets.

291 nd blood eosinophils, higher serum IgE, more subepithelial thickening, and higher expression of Th2 s

292 s in rat trachea, whereas basal cells in the subepithelial tissue displayed heavy, non-polarized stai

293 Infiltration of B lymphocytes into the subepithelial tissue of the lungs has been demonstrated

294 Gastric subepithelial tumors are usually asymptomatic and observ

295 therapies for achalasia and gastrointestinal subepithelial tumors originating from the muscularis pro

296 SD with enucleation for treatment of gastric subepithelial tumors originating from the muscularis pro

297 ique with enucleation for removal of gastric subepithelial tumors originating from the muscularis pro

298 ed a modified ESD with enucleation for their subepithelial tumors.

299 subepithelial, so this colonization implies subepithelial viral spread, where myeloid cells provide

300 ls were present in the epithelium and in the subepithelial zones of both tonsils and adenoids.