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1  lower testis weight, lower sperm count, and subfertility.
2 le mice heterozygous for Gpx4_U46S presented subfertility.
3 nic anomalies, diminished sperm motility and subfertility.
4  hypoplasia, eyelid abnormalities and female subfertility.
5 efficiency in mice, resulting in severe male subfertility.
6 number of cycles before reaching a metric of subfertility.
7 ly depletion of ovarian follicles and female subfertility.
8  of RAMP2 (but not RAMP3) display remarkable subfertility.
9 disease, appendectomy, and interventions for subfertility.
10           Finally, associations between male subfertility and a spectrum of health-threatening condit
11 ee analysis to estimate threshold values for subfertility and fertility with respect to the sperm con
12 ated loss of the ovarian reserve, leading to subfertility and infertility.
13 in the range of reproductive failure linking subfertility and late pregnancy complications and has al
14  has important implications for the roles of subfertility and manipulation by nest mates in the evolu
15                                              Subfertility and severe pelvic pains are symptoms associ
16 yndrome, ophthalmoplegia, parkinsonism, male subfertility and, in a transgenic mouse model, premature
17 ion in women that results in pelvic pain and subfertility, and has been associated with decreased bod
18                              Infertility and subfertility are important and pervasive reproductive pr
19  ofWnt5a(but notWnt11) results in the female subfertility associated with increased follicular atresi
20 gral to treating prostate and breast cancer, subfertility, blood pressure, and other diseases.
21 s impairs spermatogenesis, resulting in male subfertility, but causes no meiotic arrest.
22 nsgenic animals demonstrate reduced fitness, subfertility, defective meiotic pairing, and other germ-
23                   These results suggest that subfertility exhibited by female mice following Chst10 l
24 rt that HELQ helicase-deficient mice exhibit subfertility, germ cell attrition, ICL sensitivity and t
25 er associated with menstrual irregularities, subfertility, hirsutism, acne, and a range of endocrine
26  increased risks of spontaneous abortion and subfertility (i.e., taking more than 1 year of unprotect
27 gest FOXA3 as a potential candidate gene for subfertility in man.
28 hic sperm motility defects resulting in male subfertility in the human population.
29  for some of the unexplained infertility (or subfertility) in the general population.
30 ivation of the Alox15 gene might rescue male subfertility induced by heterozygous expression of catal
31  diagnostic evaluation and treatment of male subfertility is beneficial for most physicians.
32                                         Male subfertility is often correctable, may be genetically tr
33 in, it is currently unclear whether diabetic subfertility is the result of deficiency of pancreatic i
34 from which a couple is drawn and the precise subfertility metric which is most relevant, for example
35 productive technologies for the treatment of subfertility, more men are fathering children at advance
36 there was a nonsignificant increased risk of subfertility (odds ratio in the high exposure group = 1.
37 8; 95% confidence interval (CI) 1.4-5.6) and subfertility (odds ratio in the high exposure group = 4.
38 ially how selection would favor sterility or subfertility of most individuals within a highly social
39 eifers, classified as having high fertility, subfertility or infertility, were selected for further s
40 osis are chronic intolerable pelvic pain and subfertility or infertility, which profoundly affect the
41 emature depletion of the PF reserve leads to subfertility or infertility.
42 n age at first birth, increased treatment of subfertility, or changes in oral contraceptive use.
43 ious retinal pathology but leads to a severe subfertility phenotype in agreement with minor endogenou
44 d Ca(2+) responses, reduced AE, and a strong subfertility phenotype.
45             Our results demonstrate that the subfertility seen in male PLAG1-deficient mice is, at le
46 n-54 can induce egg maturation in women with subfertility undergoing in vitro fertilization therapy.
47  cycles of non-conception as an indicator of subfertility was found to be reasonably robust, though a
48                                              Subfertility was manifested in a reduced number of litte

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