ライフサイエンスコーパス: subglottic

1 ost-extubation UAO and 49 (12%) of whom were subglottic.

2 mastoid cell fluid (25%), sinus fluid (83%), subglottic airway fluid (92%), and pulmonary ground-glas

3 s (50%) had high-attenuation sediment in the subglottic airways.

4 G) activity was also recorded, together with subglottic and esophageal pressures.

5 inflammatory infiltrates were present in the subglottic and proximal tracheal regions, whereas respir

6 We describe a case of an acquired subglottic cyst presented with persistent stridor and vo

7 nts with the syndrome and the formation of a subglottic cyst.

8 inability to differentiate supraglottic from subglottic disease.

9 Subglottic edema and acquired subglottic stenosis are po

10 Subglottic edema is the most common cause of pediatric e

11 he incidences of failed extubation caused by subglottic edema or acquired subglottic stenosis in neon

12 that the use of prophylactic probiotics and subglottic endotracheal tubes are cost-effective for pre

13 ion, penetration, extralaryngeal spread, and subglottic extension were correlated with pathologic fin

14 r airway obstruction (UAO) and differentiate subglottic from supraglottic UAO.

15 osal necrosis, submucosal edema, swelling of subglottic glands, and submucosal infiltration of inflam

16 Studies of subglottic hemangioma have compared the outcomes of trea

17 allergic bronchopulmonary aspergillosis, and subglottic hemangioma.

18 mations (port wine stains) and cutaneous and subglottic hemangiomas is reviewed within the context of

19 aid in the early diagnosis of postintubation subglottic injury and help reduce the incidences of fail

20 incomplete and is more commonly seen at the subglottic level, resulting in a spectrum of characteris

21 e dynamic MRI, for supraglottic, glottic and subglottic location, was 100%, 80%, and 92%; 100%, 85%,

22 and esophageal manometry) and classified as subglottic or supraglottic based on airway maneuver resp

23 yngeal (P < 0.001; multiple r(2) = 0.44) and subglottic (P < 0.001; multiple r(2) = 0.55) airway wall

24 Peak subglottic pressure occurred during glottic narrowing an

25 rmine the intraabdominal, intrathoracic, and subglottic pressure, control of which is necessary for g

26 arts of the airway including the larynx, the subglottic region, or the more peripheral aspects of the

27 ative included head-of-bed elevation, use of subglottic secretion drainage endotracheal tubes, oral c

28 Subglottic secretion drainage is associated with fewer v

29 Subglottic secretion drainage is associated with lower v

30 ic review and meta-analysis of the impact of subglottic secretion drainage on duration of mechanical

31 guidelines recommend endotracheal tubes with subglottic secretion drainage to prevent ventilator-asso

32 Subglottic secretion drainage was associated with lower

33 erved significantly less antibiotic use with subglottic secretion drainage whereas a third did not.

34 on, patient position, sinusitis prophylaxis, subglottic secretion drainage, tracheal cuff monitoring)

35 are required to demonstrate the benefits of subglottic secretion drainage.

36 ve of the study was to confirm the effect of subglottic secretion suctioning on ventilator-associated

37 Subglottic secretion suctioning resulted in a significan

38 ents intubated with a tracheal tube allowing subglottic secretion suctioning were randomly assigned t

39 Continuous aspiration of subglottic secretions (CASS) is believed to lower the in

40 intestinal tract bleeding, and aspiration of subglottic secretions and oscillating beds in select pat

41 Aspiration of subglottic secretions and oscillating beds may be useful

42 Microaspiration of subglottic secretions plays a pivotal role in ventilator

43 Leakage of fluid from the subglottic space to the lungs occurs along the longitudi

44 HVLP cuffs for leakage of dye placed in the subglottic space to the tracheobronchial tree in a rigid

45 illustrated by studies on Wegener's-related subglottic stenosis and endobronchial involvement, it ha

46 Subglottic edema and acquired subglottic stenosis are potentially airway-compromising

47 One patient with severe subglottic stenosis developed pneumococcal tracheobronch

48 ation caused by subglottic edema or acquired subglottic stenosis in neonates.

49 Some forms of damage (such as subglottic stenosis or renal insufficiency) occur as the

50 asthma and otolaryngological complications (subglottic stenosis, laryngitis, pharyngitis, or cancer)

51 or outcomes, with less risk of posttreatment subglottic stenosis.

52 recurrent pneumonias, asthma, sinusitus, or subglottic stenosis.

53 ategy from the hospital perspective included subglottic suction endotracheal tubes, probiotics, and t

54 tioning, minimization of gastric distension, subglottic suctioning, avoidance of ventilator circuit c

55 Twenty-six subjects (93%) had fluid in the subglottic trachea and main bronchi.

56 Microspheres were instilled into the subglottic trachea to assess pulmonary aspiration.

57 ings of pan sinus fluid, mastoid cell fluid, subglottic tracheal and bronchial fluid, and ground-glas

58 sophageal manometry can objectively identify subglottic UAO after extubation.

59 leak pressures or cuff leak volumes predict subglottic UAO in children, but only if the ETT is cuffe

60 Risk factors independently associated with subglottic UAO included low cuff leak volume or high pre

61 Objective 2 was to identify risk factors for subglottic UAO, stratified by cuffed versus uncuffed end

62 9, with 14 (41%) of these 34 attributable to subglottic UAO.

63 f preextubation leak was not associated with subglottic UAO.

64 a positive correlation between laryngeal and subglottic wall thickness and duration of intubation, su