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1 s encoding alpha-gal A and transplanted into sublethally and lethally irradiated alpha-gal A-deficien
2 , cotransplantation of donor B6 T-reg's into sublethally conditioned BALB/c recipients supported sign
7 ound in the majority of analyzed organs, and sublethally infected animals developed virus-specific ne
15 -containing peptide adhere to the surface of sublethally injured MPT cells but not to control, dextro
17 tes the repair of physiological processes in sublethally injured renal proximal tubular cells (RPTC).
18 e 1 day after kidney ischemia/reperfusion in sublethally injured tubular cells and that the protein i
22 Tcm, support donor chimerism (> 6 months) in sublethally irradiated (5.5Gy) mice, without GVHD sympto
23 complex (MHC) class-I incompatible marrow in sublethally irradiated (550 cGy) bm1 recipients, whereas
24 TCR-transgenic donors were transplanted into sublethally irradiated (750 cGy) Ld+ or Ld- recipients.
25 or CD28(+/+) T cells were transplanted into sublethally irradiated (750 cGy), MHC class-I incompatib
26 or CD28(+/+) T cells were transplanted into sublethally irradiated (750 cGy), MHC class-II incompati
27 raftment of C57BL/6 x 129/F2(H-2b) marrow in sublethally irradiated (800 cGy) recipients (AKR/J, H-2k
28 Unexpectedly, reconstitution of sensitized, sublethally irradiated +/+ and -/- mice with bone marrow
30 effect of GCV treatment on alloengraftment, sublethally irradiated AKR mice underwent transplantatio
31 onally derived murine neurosphere cells into sublethally irradiated allogeneic hosts leads to a donor
33 ecule-1 (VCAM-1) using null strains and mice sublethally irradiated and bone marrow (BM) reconstitute
34 nvestigated the role of IFN-gamma in GVHD in sublethally irradiated B6D2F1 recipients of B6 allo-HCT.
35 transfer of C57BL/6-primed lymphoid cells to sublethally irradiated BALB/c mice engrafted with C57BL/
36 CD3 and IL-2, then adoptively transferred to sublethally irradiated BALB/c or C57BL/6 recipients bear
40 l-depleted BALB/c BM cells were infused into sublethally irradiated C57BL/6 mice and administered KGF
42 cells from CBA/J host animals with GVHD into sublethally irradiated CBA/J recipients Depletion of Vbe
44 active CD4(+) and CD8(+) TCR Tg T cells into sublethally irradiated hosts, both Tg T cells population
45 When DC-expanded Tregs were introduced into sublethally irradiated hosts, the T cells suppressed gra
48 usion of parental lymph node (LN) cells into sublethally irradiated hybrid F1 recipients created a mu
49 erism, and early chimerism in particular, in sublethally irradiated immunodeficient and lethally irra
51 e transfer of naive C57BL/6 splenocytes into sublethally irradiated line SV11 mice expressing the SV4
53 of highly purified CD4+ or CD8+ T cells into sublethally irradiated MHC class II or I disparate recip
54 not dependent on lethal irradiation, because sublethally irradiated mice all had elevated serum IgM a
55 -host reaction to promote alloengraftment in sublethally irradiated mice and provide a graft-vs-leuke
56 ing gene-1-deficient (RAG-/-) recipients, in sublethally irradiated mice naive CD8 T cells of donor o
58 Reciprocal bone marrow transplantation in sublethally irradiated mice revealed that enhanced susce
59 eneic bone marrow and result in the death of sublethally irradiated mice treated with costimulation b
60 transplants can be successfully performed in sublethally irradiated mice with and without ex vivo cul
61 robust allogeneic hematopoietic chimerism in sublethally irradiated mice without subsequent GVHD by b
62 s potently increased donor BM engraftment in sublethally irradiated mice, an event occurring independ
66 a, from immunized mice efficiently protected sublethally irradiated naive mice against a subsequent t
67 istance to DEX in a xenogeneic GVHD model in sublethally irradiated NOD-scid IL2rgamma(null) mice.
71 old more human myeloid cells are produced in sublethally irradiated NOD/SCID-IL-2Receptor-gammachain-
72 within 3 weeks after being transplanted into sublethally irradiated NOD/scid-IL-2Rgammac-null mice.
73 eparated into CD34(+) cells and infused into sublethally irradiated nonobese diabetic (NOD)severe com
74 sure to vector stocks and then injected into sublethally irradiated nonobese diabetic-severe combined
75 ytic as well as myeloid/lymphoid lineages in sublethally irradiated nonobese diabetic/severe combined
77 (KO) donor cells to reconstitute DCs/LCs in sublethally irradiated recipients and compared the resul
78 r of splenocytes from KyA-immune donors into sublethally irradiated recipients resulted in a greater
83 umors and bone marrow were transplanted into sublethally irradiated secondary recipients, 10 of these
84 eukemia; (2) GM-AML cells are tumorigenic in sublethally irradiated SJL/J mice but not in Swiss nu/nu
87 gen-primed B and T lymphocytes were given to sublethally irradiated wild-type mice or mice deficient
90 10(6) T-bet(-/-) lymph node (LN) cells into sublethally irradiated, major histocompatibility complex
92 5(+) cells recovered from the bone marrow of sublethally irradiated, transplanted NOD-Scid IL2Rgamma(
93 poietic stem cells (HSCs) were injected into sublethally-irradiated NOD-scid-IL2Rg-/- (NSI) mice.
94 to offset this M1-like polarization process, sublethally ischemic neurons may instead secrete a poten
95 failed to increase GVHD mortality in either sublethally or lethally irradiated animals that received
96 AG1 were compared for efficacy and safety in sublethally preconditioned Rag1(-/-) mice undergoing tra
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