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1 anted, but none developed tumors in the left submandibular gland.
2 ression for all 10 known active genes is the submandibular gland.
3 at is essential for saliva production by the submandibular gland.
4 eptor regulates fluid secretion in the mouse submandibular gland.
5 nd correlated with acute inflammation in the submandibular gland.
6 ndrogens have profound effects on the murine submandibular gland.
7 tubule) cells of the neonatal rat and mouse submandibular gland.
8 zed a stem cell population from adult murine submandibular glands.
9 on of the cannulated main excretory ducts of submandibular glands.
10 crease in NOS and BH4 biosynthetic enzyme in submandibular glands.
11 OC, of acinar and ductal cells in intact rat submandibular glands.
12 diffuse lymphocytic infiltration was seen in submandibular glands, a major target of pSS, by age 6 wk
13 nd the Cl- current were measured in isolated submandibular gland acinar and duct cells to characteriz
14 int of the Ca(2+)-activated Cl(-) current in submandibular gland acinar cells from Best2-deficient mi
16 ected HEK293 cells and from the pancreas and submandibular gland also coimmunoprecipitated EBP50.
17 tive, non-vasomotor sympathetic axons in the submandibular gland and at the base of piloerector hairs
18 1 IFNR (IL14alphaTG.IFNR(-/-)) had the same submandibular gland and lacrimal gland injury as did the
19 lized in the plasma membrane region of human submandibular gland and Madin-Darby canine kidney cells.
20 sly and exogenously expressed TRPC1 in human submandibular gland and Madin-Darby canine kidney cells.
21 glands, specifically in acinar cells of the submandibular gland and palatine minor glands, as well a
22 termined by recording salivary flow from the submandibular gland and temperature changes on the tongu
23 e acinar cells in developing rat parotid and submandibular glands and are also products of the sublin
24 s re-addressed, using isolated, perfused rat submandibular glands and dispersed-cell aggregates from
25 ved in cells isolated from mouse and opossum submandibular glands and rat sublingual and parotid glan
26 ein occurred in the nasal lamina propria and submandibular glands and the frequencies of CD11c+CD8+ d
28 ively influences gene expression in the male submandibular gland, and that many of the sex difference
29 f androgen control of gene expression in the submandibular gland, and to explore the degree to which
30 ences in ABPs secreted by mouse lacrimal and submandibular glands, and in ABPs secreted by male and f
32 cover that decreasing MT-MMP activity during submandibular gland branching morphogenesis decreases pr
40 tro stimulation with a muscarinic agonist of submandibular gland cells isolated from mice treated wit
46 s observed in cultured cells in vitro and in submandibular gland, cortex, and caudate nucleus for as
47 , we report that ex vivo branching of intact submandibular glands decreases when either FGFR2 express
49 renergic receptors) in pancreatic acinar and submandibular gland duct cells, respectively, evoke a Ca
50 and membrane current were measured in human submandibular gland ductal (HSG) cells to determine the
51 PC3 was detected in the apical region of rat submandibular gland ducts, whereas TRPC6 was present in
56 cells as well as lymphocytic infiltrates in submandibular glands from patients with pSS demonstrated
57 show that calcineurin is required for normal submandibular gland function and secretion of digestive
58 the question of a role for 5-HT in mammalian submandibular gland function was re-addressed, using iso
64 ion of adenylyl cyclase and, at least in rat submandibular gland, involved in modifying the volume an
65 t adenoviral-mediated gene transfer to mouse submandibular glands is possible by intraductal cannulat
67 respectively), AdhAQP1 administration to rat submandibular glands led to a two- to threefold increase
69 15 and 5-lipoxygenase) is expressed in mouse submandibular glands (mSMG), using qPCR and Western blot
71 ations of this stem cell population into the submandibular gland of irradiated mice successfully rest
74 an hamsters were transplanted into the right submandibular glands of 50 female hamsters that were or
75 of immunoglobulin A (IgA) was studied in the submandibular glands of anaesthetized rats by stimulatin
77 y of a replication-deficient adenovirus-5 in submandibular glands of C57BL/6 mice through retrograde
78 Temporal expression of IL-17 and IL-23 in submandibular glands of C57BL/6.NOD-Aec1Aec2 mice correl
79 d enriched the number of functional acini in submandibular glands of irradiated animals and enhanced
82 measurements from time-lapse images of mouse submandibular gland organ explants to construct a tempor
83 chain reaction studies in cells of mammalian submandibular gland origin using consensus sequence prim
85 93), spinal cord (PRP, 64%; mean SUV, 2.12), submandibular glands (PRP, 53%; mean SUV, 2.11), parotid
86 caspase-3, lack of leukocyte infiltration of submandibular glands, reduced synthesis of disease-assoc
87 ped large ductal-type adenocarcinomas in the submandibular gland region, where islets were transplant
88 ncreased by LPS-induced periodontitis in the submandibular gland, returned to control values after HU
89 H+ exchanger (NHE) isoforms expressed in the submandibular gland (SMG) acinar and duct cells and thei
90 l-]i and the Cl- current in the rat salivary submandibular gland (SMG) acinar and duct cells was used
91 ned miRNAs expressed in the mouse developing submandibular gland (SMG) and found that miR-200c accumu
92 y studies have been done to understand mouse submandibular gland (SMG) branching morphogenesis, littl
94 Parasympathetic innervation is critical for submandibular gland (SMG) development and regeneration.
95 -inducible Cre recombinase Ela-CreERT in the submandibular gland (SMG) ductal cells, was established
96 Here, our results show that in primary mouse submandibular gland (SMG) epithelial cells, P2X7R activa
98 f the vector to the parotid gland (PTG), the submandibular gland (SMG) or to the liver via the tail v
99 y the latter two develop inflammation in the submandibular gland (SMG), a critical target of Sjogren'
100 ery (BA), middle cerebral artery (MCA)], the submandibular gland (SMG), and pineal gland was quantifi
101 infiltration and caspase-3 activation in the submandibular gland (SMG), production of antinuclear and
103 ctive of this study was to determine whether submandibular glands (SMG) from ALX/FPR2(-/-) mice displ
104 sequently, this DC subset became resident in submandibular glands (SMGs) and nasal passages (NPs) in
106 issues such as the nasal passages (NPs), the submandibular glands (SMGs), and nasopharyngeal-associat
110 ce lacked comparable defects in the lung and submandibular gland, suggesting that MT1-MMP acts via me
111 rt a RCC case with metastasis to parotid and submandibular glands that has the same sonographic and s
112 DNA levels were lower in the spleen than in submandibular glands, the number of individual viral gen
113 Of the different cells isolated from the submandibular gland, this specific population, Lin-CD24+
114 ike the induced pancreatic tumors, all three submandibular gland tumors that were examined had the mu
115 pine-stimulated in vivo fluid secretion from submandibular glands was essentially normal in double-nu
116 ean dose, 0.26 Gy/GBq, was seen in the right submandibular gland, whereas the lowest mean dose, 0.029
117 bulated solid mass was detected in the right submandibular gland with similar sonographic findings.
118 al saliva obtained from both the parotid and submandibular glands, with highest levels of activity pr
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