ライフサイエンスコーパス: submucosa

1 ) layer and dissipated in deeper layers (ie, submucosa).

2 monolayer by an intracellular pathway to the submucosa.

3 of serovar Typhi invasion of the intestinal submucosa.

4 ocation of this microorganism to the gastric submucosa.

5 as injected in a tangential fashion into the submucosa.

6 onella enterica serovar Typhi to the gastric submucosa.

7 ll types (nerves, inflammatory cells) in the submucosa.

8 tubuloacinar glands, and occasionally in the submucosa.

9 rated a few eosinophils within the bronchial submucosa.

10 t affecting the number of eosinophils in the submucosa.

11 from the tips of the villus back toward the submucosa.

12 these cells and subsequent invasion into the submucosa.

13 ntly in the epithelial cells but also in the submucosa.

14 lergic inflammation of the airway mucosa and submucosa.

15 lity to recruit mast cells in the intestinal submucosa.

16 ajority of cells expressing RELM-beta in the submucosa.

17 is distributed primarily within the vaginal submucosa.

18 l tumor cells were seen in the mucosa and/or submucosa.

19 of pericardium, dermis, and small intestine submucosa.

20 of the submucosa, or the final third of the submucosa.

21 ficantly more likely to be invasive into the submucosa.

22 lamina propria, whereas Mf4 was enriched in submucosa.

23 here is no "safe" level of invasion into the submucosa.

24 epth of tumor infiltration of the mucosa and submucosa.

25 in 4 expression in the intestinal mucosa and submucosa.

26 that extended into the deeper layers of the submucosa.

27 c mucosal resection in gastric cancer to the submucosa.

28 sed in colonic muscularis externa and mucosa/submucosa.

29 of wound repair in a model of the intestinal submucosa.

30 etter represents the normal human mucosa and submucosa.

31 ction with macrophages within the intestinal submucosa.

32 in the intestinal epithelium but not in the submucosa.

33 ere largely absent around gland acini in the submucosa.

34 cells compared with 47% of mast cells in the submucosa.

35 d to smooth muscle and isolated cells in the submucosa.

36 Y1 R, P2Y2 R, P2Y4 R, P2Y6 R, and P2Y12 R in submucosa.

37 into the bowel wall layers, particularly the submucosa (29).

38 infected cells were detected in the cervical submucosa 3-4 days after exposure to a primary HIV isola

39 Collagen content of the gastric submucosa 5 mm below the anastomosis was quantified, and

40 ircumferential doughnut of rectal mucosa and submucosa above the dentate line excised and closed with

41 actic gradient to guide neutrophils from the submucosa across epithelia to the luminal site of an inf

42 r patient's resected tumor invaded only into submucosa and all lymph nodes were negative; in that sen

43 tivity within the gastric mucosa and gastric submucosa and also within the tuberomamillary nucleus of

44 stinal (GI) tissue revealed edema within the submucosa and altered mucous cell morphology.

45 responsiveness by infiltrating the bronchial submucosa and disrupting airway smooth muscle (ASM) cell

46 ric gp41; 2) produced by plasma cells in the submucosa and ectopic tertiary lymphoid follicles in the

47 onuclear cell inflammation of the mucosa and submucosa and epithelial cell proliferation and dediffer

48 ificantly more IL-17RA-positive cells in the submucosa and epithelium in children with STRA compared

49 cells develop after birth in the intestinal submucosa and expand around the crypts during the third

50 nfected mice, most jejunal MCs reside in the submucosa and express mMCP-6 and mMCP-7, but not mMCP-9

51 l intestines were processed as whole mounts (submucosa and mucosa removed) to examine CSMG efferent t

52 a conspicuous eosinophilic infiltrate in the submucosa and muscle layers, but the villi were unaffect

53 mRNA and the influx of neutrophils into the submucosa and muscularis propria.

54 ular and longitudinal muscles and within the submucosa and serosa.

55 9, essentially all of the MCs in the jejunal submucosa and spleen of T. spiralis-infected mice expres

56 b reduced eosinophil counts in airway mucosa/submucosa and sputum and suppressed eosinophil counts in

57 the carcinoid tumors of the gastrointestinal submucosa and the islet cell endocrine tumors of the pan

58 es mean+/-SD, lumen-negative with respect to submucosa) and was significantly more lumen-negative tha

59 around smooth muscle cells of the asthmatic submucosa, and (iii) around reserve cells of the asthmat

60 umors were confined to the mucosa, 16 to the submucosa, and 13 to the muscularis propria, and 55 were

61 large and small airways, airway epithelium, submucosa, and alveolar walls.

62 ies inhibited neutrophil infiltration in the submucosa, and cyclosporine A inhibited the tissue expre

63 gammadelta T cells in the thymus, the colon submucosa, and during early tumorigenesis.

64 and histological alterations in the mucosa, submucosa, and serosa.

65 lic inflammation was enumerated in bronchial submucosa, blood, and sputum and related to their body m

66 Vessels were larger and more numerous in the submucosa but not the mucosa.

67 e flow of nonself antigens in the intestinal submucosa can cause both intestinal and extraintestinal

68 umbers of chymase-positive mast cells in the submucosa compared to patients with nonpersistent obstru

69 nd TGF-beta3 in normal tongue and esophageal submucosa compared with gut mucosal tissues, as well as

70 The intestinal mucosa and submucosa contain three types of lymphoid tissues rangin

71 anscriptase polymerase chain reaction of the submucosa demonstrated mRNA for P2Y1R, P2Y2, P2Y4, P2Y6,

72 Similar results were observed with porcine submucosa derived from urinary bladder.

73 Small intestinal submucosa-derived extracellular matrix (SIS-ECM) has bee

74 of cells within the bronchial epithelium and submucosa expressing mRNA for TSLP, TARC/CCL17, MDC/CCL2

75 s in the mucosa and 100% of the cells in the submucosa expressing TNF-alpha mRNA were identified as m

76 pithelioid features involving the mucosa and submucosa (Fig 1A).

77 Bladder submucosa from cured mice contained residual populations

78 ere was widespread injury to tracheal mucosa/submucosa from the use of CASS.

79 ts without involvement of the mucosa and the submucosa had isolated remnants in the muscle layer (5/1

80 limited to the mucosa in 22 (88%) and to the submucosa in 3 (12%).

81 atients with asthma and are increased in the submucosa in a subgroup of obese patients with asthma (O

82 ion of this organism to the gastrointestinal submucosa in transgenic mice that are heterozygous carri

83 Division of the submucosa into thirds did not show a layer with a signif

84 Macrophage accumulation in the submucosa is also an important feature in the pathogenes

85 Invasion of the submucosa is associated with significant risk of LNM.

86 iral infection and chronic inflammation, the submucosa is susceptible to bacterial invasion by lung m

87 P-1 as the cells progressively appear in the submucosa, lamina propria, and epithelium, respectively.

88 The mucosa showed high signal intensity, the submucosa low signal intensity, and the muscularis propr

89 2 cm in diameter, localized to the mucosa or submucosa, may be safely and effectively removed via min

90 associated gastrinomas, which develop in the submucosa, might arise from enteric glial cells through

91 es and eight with gastric wall infiltration (submucosa, n = 4; muscularis propria, n = 3; serosa, n =

92 studies demonstrate that, in contrast to the submucosa, nerve fibers innervating the epithelium of th

93 The proportion of submucosa occupied by ASM was higher in both asthma grou

94 DO11.10 T cells were found in the submucosa of C. parvum-infected, but not uninfected, BAL

95 aled that more CFTR bound to S. typhi in the submucosa of Cftr wild-type mice than in deltaF508 heter

96 ells were frequently observed in the gastric submucosa of Gal3-deficient mice.In vitro, peritoneal ma

97 ers within the smooth muscle, epithelium and submucosa of healthy subjects (n = 10) and well-characte

98 was significantly increased in the bronchial submucosa of human asthmatics compared with controls, an

99 Examination of the intestinal epithelial submucosa of mice after primary and secondary infections

100 t lead to excessive fibrin deposition in the submucosa of NP, which might contribute to the tissue re

101 te to the excessive fibrin deposition in the submucosa of NP, which might contribute to the tissue re

102 al vascular architecture was observed in the submucosa of the jejunum of human patients, cows, and sh

103 l-associated lymphoid organs embedded in the submucosa of the nasal passage, in the initial priming a

104 on-emitting molecular imaging agent into the submucosa of the porcine urinary bladder.

105 ctrodes in the submucosa of the stomach, the submucosa of the small bowel, and the abdominal wall mus

106 from a collagen biomaterial derived from the submucosa of the small intestine and type I bovine colla

107 resuscitation as pH measured directly in the submucosa of the stomach or small bowel.

108 pH was measured with electrodes in the submucosa of the stomach, the submucosa of the small bow

109 ipt and protein were observed in the jejunal submucosa of Trichinella spiralis-infected BALB/c mice.

110 framework of dual involvement of mucosa and submucosa on the one hand, and of the muscularis propria

111 rd of the submucosa, the middle third of the submucosa, or the final third of the submucosa.

112 Not only infiltration of the submucosa (P=0.002) but also lymphatic vessel invasion (

113 subjects (mean [SEM]: 78 [5] vs 22 [3]/mm(2) submucosa, P < .001).

114 In the vas deferens and the ileal submucosa, P2X1 immunoreactivity (ir) was restricted to

115 In Ussing chambered mucosa-submucosa preparations (including the submucosal plexus)

116 h stroking-evoked Isc response in mucosa and submucosa preparations (M-SMP) of rat colon was reduced

117 infiltration of eosinophils into the airway submucosa; proliferation of goblet cells in the airway e

118 , cytokines are released into the mucosa and submucosa propagating and sustaining the inflammatory re

119 iated with tumors confined to the mucosa and submucosa provides justification for more limited and le

120 ing NP retention of plasma proteins in their submucosa remain unclear.

121 lying NP retention of plasma proteins in the submucosa remain unclear.

122 of the villi, the base of the villi, and the submucosa, respectively.

123 btained by lavage or scraping, and the nasal submucosa, sampled by biopsy, are two distinct compartme

124 de metastases once the tumor infiltrates the submucosa seems to necessitate surgery in these cases.

125 rejection primarily involves vessels of the submucosa, serosa, and mesentery, some mucosal alteratio

126 r esophageal cancer, both the mucosa and the submucosa show frequent residual malignant involvement.

127 Porcine small intestinal submucosa (SIS) has been widely used in tunica albuginea

128 Small intestinal submucosa (SIS) is a cell-free collagen matrix derived f

129 Porcine small intestinal submucosa (SIS) is an acellular, naturally derived extra

130 omaterials, such as porcine small-intestinal submucosa (SIS), have been successfully used clinically

131 e of a biologic prosthesis, small intestinal submucosa (SIS), in LPEHR.

132 irds of pT1a = mucosa (m1, m2, m3) or pT1b = submucosa (sm1, sm2, sm3) and number and infiltration of

133 ddle (m2), lower (m3) third of the mucosa or submucosa (sm1, sm2, sm3).

134 86% fewer S. typhi into the gastrointestinal submucosa than wild-type Cftr mice; no translocation occ

135 C populations were transitional forms in the submucosa that expressed mMCP-2 and mMCP-5 without mMCP-

136 e day 3 with lymphocytic infiltration of the submucosa that progressed to the epithelial surface-thes

137 nfined to the mucosa, the first third of the submucosa, the middle third of the submucosa, or the fin

138 polymorphonuclear leukocytes (PMNs) from the submucosa to the lumen of the intestine.

139 ammation, when the jejunal MCs move from the submucosa to the tips of the villus, the MCs briefly exp

140 ad LNM (4%), and 2 of 19 "final third of the submucosa" tumors had LNM (11%).

141 sion showed that 5 of 22 "first third of the submucosa" tumors had LNM (23%), 1 of 24 "middle third o

142 had LNM (23%), 1 of 24 "middle third of the submucosa" tumors had LNM (4%), and 2 of 19 "final third

143 expression for RANTES in the epithelium and submucosa was 26 +/- 9% and 26 +/- 10% in the asthmatic

144 .03), and the amount of smooth muscle in the submucosa was increased 50-83% (p < 0.005).

145 neutrophilic infiltration of the mucosa and submucosa was observed from day 1 until day 14 and was a

146 f the muscular layer, whereas the mucosa and submucosa were normal.

147 Eosinophils and neutrophils in the submucosa were quantified by means of immunohistochemica

148 The mucosa and submucosa were removed and muscle strips were cut and pi

149 The mucosa and submucosa were removed, and strips of longitudinal muscl

150 are key structural cells that reside in the submucosa where they are in contact with large numbers o

151 sed inflammation and bacterial access to the submucosa, which are fundamental aspects of S. flexneri

152 /54, 0%), 65 patients had tumor invading the submucosa with 8 patients having LNM (8/65, 12%).

153 Histology revealed a widening of submucosa, with an increase in inflammatory cells, diffu