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1 fluorescence endoscopy even when tumors were submucosal.
2 d 5-HT release and short-circuit current via submucosal 5-HT3 and 5-HT4 receptors.
3 =250 mug/l) and were higher in patients with submucosal (abdominal, oropharyngeal-laryngeal) attacks
4 nctions connecting the epithelial cells, and submucosal acid sensors, prostaglandins, cytokines, ente
5 he gold standard treatment for patients with submucosal adenocarcinoma who are fit to undergo the pro
6                                              Submucosal and epithelial IL-17A-positive cells and BAL
7 hythms in the smooth muscle originate at the submucosal and myenteric borders, respectively.
8  situ hybridization revealed that subsets of submucosal and myenteric neurons contained mRNA encoding
9                                   Subsets of submucosal and myenteric neurons were also D1, D2, or D3
10                DOR localizes specifically to submucosal and myenteric neurons, which might account fo
11  was expressed in cholinergic neurons in the submucosal and myenteric plexuses, but not in enterocyte
12                             The formation of submucosal and pancreatic ganglia thus involves the attr
13                                              Submucosal and pancreatic ganglia were absent at E12.5,
14  attraction is required for the formation of submucosal and pancreatic ganglia.
15 s, result, respectively, in the formation of submucosal and pancreatic ganglia.
16 oreactive to TRPV1 were increased in muscle, submucosal, and mucosal layers: in the mucosal layer, th
17 e powder air polishing (GPAP) and a repeated submucosal application of Povidone-iodine.
18  powder air polishing (GPAP), and a repeated submucosal application of povidone-iodine.
19 ween some epithelial cells, with an enlarged submucosal area filled with immune cells and sometimes i
20 ast cells and eosinophils (mostly located in submucosal areas) and, in comparison with subcutaneous t
21                                       Intact submucosal arterioles from control, involved, and uninvo
22 in the circular muscle coat, and surrounding submucosal arterioles.
23 fects on eosinophil counts in airway mucosal/submucosal biopsy specimens, sputum, bone marrow, and pe
24 m 13 to 15 months p.i.; invasion of adjacent submucosal blood vessels by glandular epithelia also was
25 ce of MEN1 gastrinomas, which develop within submucosal Brunner's glands.
26                       Patients found to have submucosal cancer at their first endoscopy examination w
27 0% for 70 mucosal carcinomas and 34% for 101 submucosal carcinomas (P = 0.001).
28 y in m3 carcinoma is questionable and in all submucosal carcinomas and lesions > or =2 cm it is not i
29          The rate of lymphatic metastasis in submucosal carcinomas was 21% for sm1 (n = 29), 16% for
30 gerhans cells in the vaginal epithelium, the submucosal CD11b(+) DCs, and the CD8alpha(+) lymph node
31             We showed that EBV first infects submucosal CD14(+) monocytes, which then migrate into th
32 n, the first cells likely to be infected are submucosal CD4(+) T cells and dendritic cells of the low
33 l epithelial cells causes soft palate cleft, submucosal cleft and failure of the primary palate to fu
34                                              Submucosal collagen content in Sham, Delay, and Immediat
35 clusters of epithelial cells residing in the submucosal compartment of extrahepatic bile ducts (EHBDs
36 sion of gel-forming mucins in epithelial and submucosal compartments in CF were similar to normal.
37 s not contain dyskeratotic changes and has a submucosal component, and thus better represents the nor
38 example of differences between myenteric and submucosal components of the enteric nervous system.
39 onstrate a previously unanticipated role for submucosal DCs in the generation of protective Th1 immun
40  lymph nodes revealed that only the CD11b(+) submucosal DCs, but not Langerhans cell-derived or CD8al
41 ation of HSV-2 led to a rapid recruitment of submucosal dendritic cells (DCs) to the infected epithel
42 estinal wall indicated a high preference for submucosal deposition.
43  a novel artificial simulator for endoscopic submucosal dissection (ESD) as a bridge between instruct
44 resection techniques and devices, endoscopic submucosal dissection (ESD) has been considered as an al
45          In early gastric cancer, endoscopic submucosal dissection (ESD) has been shown to be superio
46                                   Endoscopic submucosal dissection (ESD) is a newer procedure that is
47 ng performing lesion marking (p = 0.026) and submucosal dissection (p = 0.037).
48 felt that the most difficult techniques were submucosal dissection and hemostasis.
49  endoscopic mucosal resection and endoscopic submucosal dissection are being applied for the treatmen
50 into a single endoscopic session, endoscopic submucosal dissection for Barrett's esophagus neoplasia,
51    In properly selected patients, endoscopic submucosal dissection has been found to have 100% 5-year
52 ents with gastric adenocarcinoma, endoscopic submucosal dissection is a viable alternative to gastric
53                                              Submucosal dissection techniques are more difficult, ass
54  years, new endoscopic mucosal resection and submucosal dissection techniques have been developed.
55 C risk might be better suited for endoscopic submucosal dissection than for endoscopic mucosal resect
56 (endoscopic mucosal resection and endoscopic submucosal dissection) and translumenal endoscopic proce
57 cosal pre-cutting, circumferential incision, submucosal dissection, and hemostasis.
58  on which patients should undergo endoscopic submucosal dissection, EMR, or surgery.
59 chniques of endoscopic mucosal resection and submucosal dissection, patients will benefit from these
60                 Moreover, the combination of submucosal dissection, use of a fluorescent marker, and
61 ivided into two forms, mucosal resection and submucosal dissection.
62  endoscopic mucosal resection and endoscopic submucosal dissection.
63 as feasible for simulated lesion marking and submucosal dissection.
64 tially distributed between the myenteric and submucosal divisions of the ENS.
65       We sought to investigate the bronchial submucosal DP2 expression in asthmatic patients and heal
66                                   Numbers of submucosal DP2(+) cells were increased in asthmatic pati
67 ion were fund: bowel wall thickening (n=21), submucosal edema (n=8), segment wall hyperenhancement (n
68 ry responses in the gut, including increased submucosal edema and release of extracellular DNA from h
69 blunting with sloughing of epithelial cells, submucosal edema, infiltration of leukocytes, venous con
70 terial attachment, effacement of microvilli, submucosal edema, mucosal heterophile infiltrates, and S
71  and, in particular, as shown in this study, submucosal edema.
72 ckness, mucus gland area, collagen area, and submucosal effector inflammatory cells.
73 ates the latest developments in the field of submucosal endoscopy, focuses on POEM and sheds light on
74 eceptor CCR3 was expressed at high levels on submucosal endothelial cells in patients and a murine mo
75 a, with either low (0-0.45 mm(-2)) ) or high submucosal eosinophil (23.43-46.28 mm(-2) ) counts and h
76                In the bronchoscopy group the submucosal eosinophil number in the subjects with asthma
77                                  Mucosal and submucosal eosinophilia increased over the time course a
78                                              Submucosal eosinophilia is a marker (and possibly a caus
79         In addition there was an increase in submucosal eosinophils (P = 0.06).
80 d the median (interquartile range) number of submucosal eosinophils was increased in obese (19.4 [11.
81                      Group 1 had the highest submucosal eosinophils, as well as high fractional exhal
82                              Sputum IL-5 and submucosal eosinophils, but not sputum eosinophils, are
83 now being proposed as a viable treatment for submucosal esophageal adenocarcinoma.
84                                   A dominant submucosal fibroid and ischemia greater than or equal to
85                                              Submucosal fibrosis due to excessive accumulation of col
86 ased stromal accumulation of macrophages and submucosal fibrosis due to excessive accumulation of col
87                 Crypt epithelial mucin loss, submucosal fibrosis, and length of time to explant were
88                        All CRF-IR neurons in submucosal ganglia expressed vasoactive intestinal pepti
89 h NADPH-diaphorase showed that myenteric and submucosal ganglia formed interconnecting plexuses, simi
90 s also increased, as were cell bodies of the submucosal ganglia immunoreactive to CGRP (p=0.0009).
91 tified in internodal strands, blood vessels, submucosal ganglia, and longitudinal muscle.
92                       CSE was present in all submucosal ganglion neurons and in almost all myenteric
93 f the gastrin gene and induces generation of submucosal gastrin-expressing cell hyperplasia.
94 ignaling, primary cultures of human tracheal submucosal gland (SMG) cells were used to assess EGFR li
95   In human airways, oxidative stress-induced submucosal gland cell hypertrophy and hyperplasia, histo
96 by luminal hyaluronan (HA), and treatment of submucosal gland cells with X/XO induced HA depolymeriza
97             We found that MUC5B emerged from submucosal gland ducts in the form of strands composed o
98 ared with non-CF, MUC5B more often filled CF submucosal gland ducts.
99 ed at mucosal surfaces, including airway and submucosal gland epithelia.
100 aCC conductance in human salivary and airway submucosal gland epithelial cells, and IL-4 treated bron
101 l glands, and they suggest that while murine submucosal gland fluid secretion in response to choliner
102 ding defective airway chloride transport and submucosal gland fluid secretion; variably penetrant mec
103          It has been proposed that defective submucosal gland function in CF airways is a major deter
104    Our results suggest that defective airway submucosal gland function is an early, primary defect in
105                We tested the hypothesis that submucosal gland function is defective early in CF subje
106 osed NHPs developed robust mucus metaplasia, submucosal gland hypertrophy and hyperplasia, airway inf
107                  Goblet cell hyperplasia and submucosal gland hypertrophy are shared with other hyper
108 irway defenses include reflex stimulation of submucosal gland mucus secretion by sensory neurons that
109 hat PDE3A inhibition augments CFTR-dependent submucosal gland secretion and actin skeleton disruption
110  therapeutic approaches to correct defective submucosal gland secretion in CF.
111 IL-4 treated bronchial cells, and stimulated submucosal gland secretion in human bronchi and smooth m
112 teraction was investigated using pig trachea submucosal gland secretion model.
113 ere also exposed to bethanechol to stimulate submucosal gland secretion, when plastered mucus covered
114                                              Submucosal gland serous acinar cells are primary sites o
115                                        Here, submucosal gland serous acinar cells were isolated from
116  Calu-3 human cell line exhibits features of submucosal gland serous cells and secretes HCO(3)(-).
117                                       Airway submucosal gland serous cells express the cystic fibrosi
118                  The mechanism for increased submucosal gland volume in CF deserves further study.
119 c feature in the CF airway is an increase in submucosal gland volume, but serous cell transdifferenti
120 olin-stimulated mucus secretion from porcine submucosal glands (75 glands, 7 pigs).
121 oride channels causes defective secretion by submucosal glands (SMGs), leading to persistent bacteria
122 ithelial-derived appendages including airway submucosal glands (SMGs).
123 onsequent reduction in mucus production from submucosal glands and bronchodilation have been proposed
124 ciliated respiratory epithelium and cells of submucosal glands and ducts.
125 ssociations in airways that contain abundant submucosal glands and goblet cells are uncertain.
126 bsence of goblet cells in tracheal/laryngeal submucosal glands and in the conducting airway epitheliu
127 ted expression of SPLUNC1 in serous cells of submucosal glands and surface epithelial cells of the up
128 way smooth-muscle tone, mucus secretion from submucosal glands and surface epithelial goblet cells, v
129 T is a primary defect in CF, suggesting that submucosal glands and tethered mucus may be targets for
130                 Whereas both mouse and human submucosal glands and their serous acinar cells express
131                                       Airway submucosal glands are important sites of cystic fibrosis
132                                       Airway submucosal glands are sites of high expression of the cy
133                  Here, we studied individual submucosal glands from 1-day-old piglets in situ in expl
134                                       Airway submucosal glands have been proposed as a primary site f
135 logy revealed similar size and morphology of submucosal glands in CF and non-CF specimens.
136                        The potential role of submucosal glands in cystic fibrosis lung disease is dis
137 o measure SubP-mediated secretion from human submucosal glands in lung transplant tissue.
138  airway epithelium and the epithelium of the submucosal glands in the paranasal sinuses.
139 the alveolar epithelium and the epithelia of submucosal glands in the upper airway and nasopharynx.
140 mmunohistochemical data revealed that within submucosal glands of sinonasal tissues, SPLUNC1 and LPLU
141  in ciliated airway epithelial cells and the submucosal glands of the lung.
142                        We found that porcine submucosal glands produce MUC5B, whereas goblet cells pr
143                         The tracheobronchial submucosal glands secrete liquid that is important for h
144  to periciliary liquid depletion; rather, CF submucosal glands secreted mucus strands that remained t
145                                              Submucosal glands supply most of the mucus in upper airw
146                                The volume of submucosal glands was fourfold higher than normal (p = 0
147                   Secretions from individual submucosal glands were visualized by light/fluorescence
148 his distal gap segment (which has esophageal submucosal glands) is actually the dilated distal esopha
149 chanisms of salt and water secretion by lung submucosal glands, and they suggest that while murine su
150 al membrane of the airway epithelium, airway submucosal glands, and type 1 pneumocytes, where it can
151 m, even without hyperviscous secretions from submucosal glands, produces an intrinsically hyperviscou
152 lizes to the surface epithelium and MUC5B to submucosal glands, the finding that Muc5b is secreted by
153                   By analogy to salivary and submucosal glands, where fluid secretion is aquaporin-5
154 reflect a profound decrease in the number of submucosal glands.
155 ononuclear leukocytes and in serous cells of submucosal glands.
156 triggers CFTR-dependent ASL secretion by the submucosal glands.
157      These cells localize to proximal airway submucosal glands/intercartilagenous rings, neuroepithel
158 ities were coincident in subsets of neurons (submucosal > myenteric) in guinea pig and mouse intestin
159  immunocytochemically in neuronal perikarya (submucosal >> myenteric plexus; small intestine > stomac
160 t a clear watershed between deep mucosal and submucosal infiltration does not exist.
161           Histologic analysis of mucosal and submucosal infiltration in thirds has shown an increasin
162                                              Submucosal infiltration of inflammatory cells preceded a
163 t is preceded by precancerous lesions having submucosal inflammation and hyperplastic crypts.
164 ked staining of arginase I, predominantly in submucosal inflammatory lesions.
165                Three female pigs underwent a submucosal injection of the bladder with fluorescent-tag
166  diclofenac HPbetaCD administered as a local submucosal injection prior to lower third molar surgery.
167 ity of diclofenac HPbetaCD used locally as a submucosal injection prior to third molar surgery.
168       Within 10 min of a gastric and colonic submucosal injection, (99m)Tc-DTPA-mannosyl-dextran demo
169 ultiple gas collections in the subserosal or submucosal intestinal wall of the large or small intesti
170 e mucosa (T1a) in 75 patients (60%), whereas submucosal invasion (T1b) was present in 51 patients (40
171 ion are associated with an increased risk of submucosal invasion and lymph node metastases and should
172                                Patients with submucosal invasion are not suitable for endoscopic trea
173                         Subclassification of submucosal invasion showed that 5 of 22 "first third of
174 t is important to identify those at risk for submucosal invasive cancer (SMIC).
175 verall prevalence of NP-CRNs with in situ or submucosal invasive carcinoma was 0.82% (95% CI, 0.46%-1
176 usted association of NP-CRNs with in situ or submucosal invasive carcinoma was also observed in subpo
177                                              Submucosal invasive foci were observed in 9 of 11 WT-inf
178 cal progression from APC-mutant neoplasia to submucosal invasive tumor.
179  acute C1-INH-HAE attacks, particularly with submucosal involvement.
180 NA-positive cells detected at the epithelial-submucosal junction 6 h after virus exposure.
181 f a saline injection, to separate the mucosa-submucosal layer, followed by a cap-assisted snare resec
182 lls could underlie fibrosis of the mucosa or submucosal layers are presented.
183 tis is generally confined to the mucosal and submucosal layers, although Crohn's colitis may be trans
184 n vivo imaging of lung tumors in the mucosal/submucosal layers, providing real-time fluorescence guid
185 g improvement at 3 months were higher with a submucosal leiomyoma location (P =.04); however, this as
186                                            A submucosal leiomyoma location was associated with a grea
187 ound is a key component of the evaluation of submucosal lesions of the gastrointestinal tract, allowi
188 seful in separating these lesions from other submucosal lesions of the gastrointestinal tract.
189 ment of pancreatic disease and evaluation of submucosal lesions.
190          Smaller baseline leiomyoma size and submucosal location are more likely to result in a posit
191                                 Lacteals and submucosal lymphatic capillaries of the intestine were a
192                                              Submucosal mast cell, neutrophil, and lymphocyte counts
193                                              Submucosal mast cells were not affected by corticosteroi
194 on in asthma; although it has been linked to submucosal matrix deposition, its relationship with othe
195                                 Peri-implant submucosal microbial samples were collected from 85 pati
196 y, EndoMT was observed in rectal mucosal and submucosal microvessels in a preclinical model of radiat
197 espect to virulence factors, suggesting that submucosal monocytes/macrophages are the main source of
198 and trefoil factors, protective functions of submucosal mononuclear cells, junctional proteins affect
199 l colonic epithelium, smooth muscle, and the submucosal myenteric plexus.
200 ahexaenoic acid was increased in mucosal and submucosal nasal-biopsy specimens (P<0.001) and rectal-b
201 letion of CGRP in neuroepithelial bodies and submucosal nerve plexuses without altering the overall d
202 mum [25th-75th IQR, 3.7-4.6 mum], P = 0.02), submucosal nerves (1.0 per thousand [25th-75th IQR, 0.7-
203 yenteric neuron to glia ratio, reduced colon submucosal neuron density, and increased colon myenteric
204       The purinergic excitatory input to the submucosal neurones came from neighbouring neurones in t
205                               Removal of the submucosal neurons abolished the gradient.
206 e calcium imaging, we compared activation of submucosal neurons by the TRPV1 agonist capsaicin in rec
207                              The majority of submucosal neurons contained HO-2 immunoreactivity (HO-2
208                 Moreover, TRPV1 responses of submucosal neurons from healthy volunteers could be pote
209                           TRPV1 responses of submucosal neurons from patients with IBS were potentiat
210 Fos-LI dose-dependently in the myenteric and submucosal neurons of the duodenum, but not jejunum and
211                                  Cholinergic submucosal neurons reacted with antibodies against 5-HT(
212  and 71% in the adult myenteric plexus, when submucosal neurons were also OTR-immunoreactive.
213 GAL-R1 myenteric neurons and 70.7% of GAL-R1 submucosal neurons were substance P immunoreactive.
214 in the soma of the majority of myenteric and submucosal neurons, although faint immunoreactivity was
215 mmunoreactivity was found in 48.3% of GAL-R1 submucosal neurons, but not in GAL-R1 myenteric neurons.
216 f Fos-immunoreactive nuclei of myenteric and submucosal neurons, following CCK injection.
217  due to CO, and that the source of CO is the submucosal neurons.
218  elevated excitability in both myenteric and submucosal neurons.
219                                              Submucosal neutrophils were not increased in children wi
220 scopic imaging technique can reliably assess submucosal or lymphangio-invasion.
221  with low-grade and high-grade dysplasia and submucosal or more advanced cancer were excluded.
222    Segments of canine colon were pinned with submucosal or myenteric surface uppermost or cut in cros
223 s according to the 5-FU dose, and in gastric submucosal orthotopic xenografts of MKN45/5FU cells.
224       The aim of the study is to compare the submucosal peri-implant microflora between FES and PES.
225 ificant difference in the composition of the submucosal peri-implant microflora in healthy and peri-i
226 dies reporting in the same article about the submucosal peri-implant microflora of FES and PES were s
227 evels of dissolved titanium were detected in submucosal plaque around implants with peri-implantitis
228 r fluid (GCF) and a selection of subgingival/submucosal plaque bacteria from clinically healthy or di
229 s to compare levels of dissolved titanium in submucosal plaque collected from healthy implants and im
230                                              Submucosal plaque from 20 implants with peri-implantitis
231 haracterized by neutrophils and infiltrating submucosal plasma cells consisting primarily of T cells.
232 ressed in the neurons of human myenteric and submucosal plexi.
233 CRF-IR cell bodies were more abundant in the submucosal plexus (29.9-38.0%) than in the myenteric ple
234  small intestine and ICC associated with the submucosal plexus (ICC-SMP) in the colon.
235 even per cent of CSE positive neurons in the submucosal plexus and 50% of CSE positive neurons in the
236 gic or sympathetic markers terminated in the submucosal plexus and mucosa of the duodenum.
237               TRPC3-IR was found only in the submucosal plexus and was expressed exclusively by neuro
238  Absence of NF145 from ganglion cells in the submucosal plexus is an example of differences between m
239 urons, while being on axonal compartments of submucosal plexus neurons.
240 n morphologically identified neurones in the submucosal plexus of guinea-pig small intestine.
241 ach and small and large intestine and in the submucosal plexus of the small and large intestine.
242 tions of adult (8-12-week old) myenteric and submucosal plexus stained with NADPH diaphorase (neurons
243 trast, human stomachs have a clearly defined submucosal plexus that contains a variety of transmitter
244 mucosa-submucosa preparations (including the submucosal plexus) of rat proximal colon, carbachol (CCh
245                                       In the submucosal plexus, CRF1 receptor immunoreactivity was fo
246                                       In the submucosal plexus, DOReGFP was detected in neuropeptide
247 oglial structures similar to a myenteric and submucosal plexus, had functional interstitial cells of
248 e also observed in the myoenteric plexus and submucosal plexus, involving enteric neurons with enteri
249 nduced slow EPSP-like response in guinea pig submucosal plexus, suggesting that CaMKII activity is re
250                                       In the submucosal plexus, TRPC4/6-IR was expressed exclusively
251 rgic secretomotor/vasodilator neurons in the submucosal plexus.
252 d noncholinergic secretomotor neurons in the submucosal plexus.
253 neurons, but was abundantly expressed in the submucosal plexus.
254 sensory elements did not lie adjacent to the submucosal plexus.
255  immunoreactivity was rarely observed in the submucosal plexus.
256 l as by TTX, hexamethonium or removal of the submucosal plexus.
257 CRF-IR fibers persisted in the myenteric and submucosal plexuses after 7 days in organotypic culture.
258                            The myenteric and submucosal plexuses and DVC were processed for detection
259  the hindgut in large numbers, myenteric and submucosal plexuses in the hindgut almost entirely compo
260  was expressed in both the myenteric and the submucosal plexuses of all regions of the large and smal
261 anglia, and the ganglia of the myenteric and submucosal plexuses of the duodenum following intraperit
262 ally identified neurons in the myenteric and submucosal plexuses of the guinea pig enteric nervous sy
263 e enteric nervous system (ENS; myenteric and submucosal plexuses) of the gastrointestinal (GI) tract
264 rve fibers were present in the myenteric and submucosal plexuses, in the circular muscle coat, and su
265 calized in nerve fibers of the myenteric and submucosal plexuses, muscularis externa and lamina propr
266 or mRNA, but not CRF2, in both myenteric and submucosal plexuses.
267 ressed in nerve fibers in both myenteric and submucosal plexuses.
268 onoclonal antibody fluorescence intensity in submucosal postcapillary venules with the use of intravi
269 status was significantly associated with the submucosal presence of Porphyromonas gingivalis (Pg), Pr
270  high-speed video to infer information about submucosal processes.
271 acetylcholine, two critical mediators in the submucosal reflex pathway.
272 the outer mesenchyme, but is absent from the submucosal region, supporting the presence of both ENCC-
273                                              Submucosal regions with high virally induced BMP4 expres
274       Stricturing associated with endoscopic submucosal resection and circumferential endoscopic muco
275                                   Endoscopic submucosal resection was developed to increase en-bloc r
276 cal features, such as the esophageal mucosal-submucosal separation, pulmonary alveoli and intestinal
277                                              Submucosal smooth muscle cells express bone morphogeneti
278 ) through the ICC-SM network lying along the submucosal surface of the circular muscle (CM).
279 ound epithelial cell (EpC) mucin release and submucosal swelling in the nasal mucosa of mice that dep
280 4 one log lower than that required to elicit submucosal swelling.
281 f lymph node metastases for intramucosal and submucosal (T1) esophageal adenocarcinoma and to analyze
282 copic therapy (ET) for intramucosal (T1a) or submucosal (T1b) esophageal carcinoma.
283 ic viruses that would then be able to infect submucosal target cells, including T cells and macrophag
284 by marked recruitment of inflammatory cells, submucosal thickening, goblet cell metaplasia, and incre
285 e chronic inflammatory response resulting in submucosal tissue remodeling and scarring.
286  self-limiting edema of the subcutaneous and submucosal tissue, due to a temporary increase in vascul
287 ) eosinophils remained TUNEL negative in the submucosal tissue, throughout the 10-day period after Af
288 ux of allergens, toxins, and microbes to the submucosal tissue.
289  prolonged release of nanoparticles into the submucosal tissue.
290               Immature DCs (iDCs) located in submucosal tissues can capture HIV-1 and migrate to lymp
291  leading to enhanced bacterial ingestion and submucosal translocation.
292 osal tumors but increased significantly with submucosal tumor invasion (22%)[P = 0.0003].
293  surface squamous epithelium disguising as a submucosal tumor.
294 ostic measure of choice in the evaluation of submucosal tumors of the GI tract, and EUS criteria have
295 s these tumors closely mimic the mesenchymal submucosal tumors such as lipoma, leiomyoma, and gastroi
296 plasia, epithelial cell Cox-2 expression and submucosal tumour invasion, as well as increased nuclear
297        Peroral endoscopic myotomy (POEM) and submucosal tunneling and endoscopic resection (STER) hav
298                                    A greater submucosal type I and III collagen expression and tenasc
299                         Increased numbers of submucosal vessels are a consistent pathologic component
300 hils also failed to extravasate from gastric submucosal vessels in a murine model of Helicobacter pyl

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