コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 n the globus pallidus, the thalamus, and the substantia nigra pars compacta.
2 ial hypothalamus, and ventral tegmental area/substantia nigra pars compacta.
3 cular regions, but remarkably, little to the substantia nigra pars compacta.
4 e is the loss of dopaminergic neurons of the substantia nigra pars compacta.
5 s and a loss of dopamine (DA) neurons in the substantia nigra pars compacta.
6 of the midbrain dopamine (DA) neurons in the substantia nigra pars compacta.
7 ing the cerebral cortex, caudate-putamen and substantia nigra pars compacta.
8 degeneration of dopaminergic neurons in the substantia nigra pars compacta.
9 c neurons of ventral tegmental area (VTA) or substantia nigra pars compacta.
10 hat are a consequence of degeneration of the substantia nigra pars compacta.
11 metabolites, and dopaminergic neurons in the substantia nigra pars compacta.
12 death with apoptosis and inflammation in the substantia nigra pars compacta.
13 xtensive loss of dopaminergic neurons in the substantia nigra pars compacta.
14 Very dense expression is noted in the substantia nigra pars compacta.
15 the degeneration of dopaminergic neurons in substantia nigra pars compacta.
16 degeneration of dopaminergic neurons in the substantia nigra pars compacta.
17 sive loss of the dopaminergic neurons in the substantia nigra pars compacta.
18 gmental area and, to a lesser extent, in the substantia nigra pars compacta.
19 ghly enriched in the dopamine neurons of the substantia nigra pars compacta.
20 s of the lateral olfactory tract, and within substantia nigra pars compacta.
21 mygdala, the ventral tegmental area, and the substantia nigra pars compacta.
22 arge, presumably dopaminergic neurons of the substantia nigra pars compacta.
23 ch dissimilar cell fields as hippocampus and substantia nigra pars compacta.
24 ranule cells and dopaminergic neurons in the substantia nigra pars compacta.
25 idal neurons and dopaminergic neurons of the substantia nigra pars compacta.
26 hanism within the ventral tegmental area and substantia nigra pars compacta.
27 preferential loss of dopamine neurons in the substantia nigra pars compacta.
28 striatum and dopamine neuron activity in the substantia nigra pars compacta.
29 action potentials in dopamine neurons of the substantia nigra pars compacta.
30 ially innervated dopaminergic neurons in the substantia nigra pars compacta.
31 tely dissociated dopamine neurons from mouse substantia nigra pars compacta.
32 ne (6-OHDA) in the dorsal GL or in the right substantia nigra pars compacta.
33 as striatal cholinergic interneurons and the substantia nigra pars compacta.
34 show high levels of LRRK2 expression in the substantia nigra pars compacta.
35 ce in tyrosine hydroxylase expression in the substantia nigra pars compacta.
36 nificant loss of dopaminergic neurons in the substantia nigra pars compacta 60 days after infection.
37 the degeneration of dopamine neurons of the substantia nigra pars compacta, a deficit in dopamine ne
38 (DRN), a major source of serotonin, and the substantia nigra pars compacta, a major source of dopami
39 d neurons and in dopaminergic neurons of the substantia nigra pars compacta, a susceptible brain regi
40 irk2-positive cells are most numerous in the substantia nigra, pars compacta, a region badly affected
41 within the ventral tegmental area (VTA) and substantia nigra pars compacta activates inhibitory post
42 ived bilateral injections of 6-OHDA into the substantia nigra pars compacta and 1 week later were tes
43 ressive loss of dopaminergic (DA) neurons in substantia nigra pars compacta and age-dependent L-DOPA-
44 duction of dopamine-producing neurons in the substantia nigra pars compacta and decreased striatal do
45 n is very low in the dopaminergic neurons of substantia nigra pars compacta and in the CA1/2 pyramida
46 ary to a loss of dopaminergic neurons in the substantia nigra pars compacta and intracellular inclusi
47 ted with loss of dopaminergic neurons in the substantia nigra pars compacta and locus ceruleus, witho
48 ctive aggregates in pigmented neurons of the substantia nigra pars compacta and locus coeruleus in al
49 neration of catecholaminergic neurons of the substantia nigra pars compacta and locus coeruleus, amon
50 he activity of dopaminergic neurons from the substantia nigra pars compacta and noradrenergic neurons
51 tes (Lewy-body inclusions) in neurons of the substantia nigra pars compacta and other brain areas.
52 D1-like neurotransmission, particularly the substantia nigra pars compacta and other dopamine-synthe
53 M analogs protected dopamine (DA) neurons in substantia nigra pars compacta and restored DA levels in
54 1 protein is expressed in neurons within the substantia nigra pars compacta and striatum, two regions
55 greater loss of dopaminergic neurons in the Substantia Nigra pars compacta and terminal dopamine fib
56 d by the loss of dopaminergic neurons in the substantia nigra pars compacta and the accumulation of t
57 by the loss of dopaminergic neurons from the substantia nigra pars compacta and the presence, in affe
58 volved in T cell trafficking, in vivo in the substantia nigra pars compacta and the serum of 1-methyl
59 diencephalic dopamine neurons located in the substantia nigra pars compacta and the ventral tegmental
60 ascending dopaminergic projections from the substantia nigra pars compacta and the ventral tegmental
61 dritic release of dopamine by neurons in the substantia nigra pars compacta and ventral tegmental als
63 ne burst and dip responses from cells in the substantia nigra pars compacta and ventral tegmental are
64 dendrites and cell bodies of neurons in the substantia nigra pars compacta and ventral tegmental are
65 dopamine transporter (DAT) mRNA in both the substantia nigra pars compacta and ventral tegmental are
66 Midbrain dopaminergic (DA) neurons in the substantia nigra pars compacta and ventral tegmental are
68 as recognized as homologous to the mammalian substantia nigra pars compacta and was renamed according
69 posterior tuberculum (PT; homologous to the substantia nigra pars compacta and/or ventral tegmental
70 groups of midbrain dopaminergic neurons, A9 (substantia nigra pars compacta) and A10 (ventral tegment
71 structures accumulated within neurons of the substantia nigra pars compacta, and dual labeling and co
72 ypothalamic nucleus, ventral tegmental area, substantia nigra pars compacta, and nucleus of the solit
73 t levels detected in the ventral tier of the substantia nigra pars compacta, and the lowest levels in
74 irst demonstration of Mn accumulation in the substantia nigra pars compacta, and thus, can represent
75 stem, BDNF expression was upregulated in the substantia nigra pars compacta, and trkB was elevated in
76 , nucleus accumbens, subthalamic nucleus and substantia nigra pars compacta, and was present at lower
77 subiculum and the hippocampal CA4 field, the substantia nigra/pars compacta, and the pineal gland, pa
81 xterna (GPe), subthalamic nucleus (STN), and substantia nigra pars compacta, are conserved throughout
82 eurons found in the A9 and A10 region of the substantia nigra pars compacta as well as the technical
83 ic nigrostriatal neurons of the adult rodent substantia nigra pars compacta, as well their developmen
84 P to mice, there was a robust gliosis in the substantia nigra pars compacta associated with significa
86 ne hydroxylase-immunoreactive neurons in the substantia nigra pars compacta (both in total and in sub
87 us degeneration of brain neurons not only in substantia nigra pars compacta but also in other extrast
88 progressive loss of dopamine neurons in the substantia nigra pars compacta, but not in the adjacent
89 28kD is neuroprotective, was observed in the substantia nigra, pars compacta, but not in the ventral
90 ne hydroxylase immunoreactive neurons in the substantia nigra pars compacta by 2-fold (p < 0.05) in a
92 ake stronger projections to the striatum and substantia nigra pars compacta compared with PV-GPe neur
94 of melanized dopaminergic neurons within the substantia nigra pars compacta coupled with depletion of
95 degeneration of dopaminergic neurons in the substantia nigra pars compacta coupled with intracytopla
96 rized by the loss of dopamine neurons in the substantia nigra pars compacta, culminating in severe mo
97 y dopamine-containing neurons of the brain's substantia nigra pars compacta die in Parkinson's diseas
98 n addition, in vivo recordings of identified substantia nigra pars compacta dopamine neurons in R1441
99 an intense expression of torsinA mRNA in the substantia nigra pars compacta dopamine neurons, the loc
100 rial integrity and thereby properly maintain substantia nigra pars compacta dopaminergic neurons and
103 also report that PAR1 is expressed in human substantia nigra pars compacta glia as well as tyrosine
104 ctive TRPC3 channels, forming an ultra-short substantia nigra pars compacta --> SNr dopamine pathway
105 dial geniculate, piriform cortex (layer II), substantia nigra pars compacta, hippocampal CA3 pyramida
107 nd accumulation and neurodegeneration in the substantia nigra pars compacta in aged VMAT2 LO mice.
108 ay also account for the vulnerability of the substantia nigra pars compacta in PD, why the disorder i
109 Degeneration of dopaminergic neurons of the substantia nigra pars compacta is a cardinal feature of
110 reas selective degeneration of DA neurons in substantia nigra pars compacta is a key neuropathologica
111 degeneration of dopaminergic neurons in the substantia nigra pars compacta is the primary cause for
112 gressive loss of dopaminergic neurons of the substantia nigra pars compacta leading to abnormal activ
113 d by the loss of dopaminergic neurons in the substantia nigra pars compacta, leading to nigrostriatal
114 rabrachial pigmented nucleus and dorsomedial substantia nigra pars compacta) mesodiencephalic dopamin
116 ptor kinase type B (trkB) receptor occurs in substantia nigra pars compacta neurons and is required f
118 c currents driving the spontaneous firing of substantia nigra pars compacta neurons, isolated from tr
119 sed within vulnerable dopaminergic (DAergic) substantia nigra pars compacta neurons, only select down
121 ction in TH-positive cell body counts in the substantia nigra pars compacta of 6-OHDA- and free-3NT-t
122 ection to isolate single DA neurons from the substantia nigra pars compacta of controls and subjects
123 hosphorylated p53 in dopamine neurons of the substantia nigra pars compacta of mice following systemi
124 spiking activity of dopamine neurons in the substantia nigra pars compacta of monkeys (Macaca mulatt
125 rapid activation of p21(ras) in vivo in the substantia nigra pars compacta of MPTP-intoxicated mice.
126 nd deleterious effects of glial cells in the substantia nigra pars compacta of Parkinson's disease.
127 bnormal proteins have been identified in the substantia nigra pars compacta of patients with sporadic
128 ound that NF-kappaB was activated within the substantia nigra pars compacta of PD patients and MPTP-i
129 ES and eotaxin were also up-regulated in the substantia nigra pars compacta of post-mortem PD brains
130 sphorylated p53 was also demonstrated in the substantia nigra pars compacta of post-mortem PD brains.
133 /3-ir could be detected in TH-ir soma within substantia nigra pars compacta, or in TH-ir striatal ter
134 etion of Atg7 in the dopamine neurons of the substantia nigra pars compacta, other regions of the mid
135 d), 38% in nucleus A9 (the DA neurons in the substantia nigra pars compacta, pars reticulata, and par
136 degeneration of dopaminergic neurons in the substantia nigra pars compacta portion of the brain.
137 ontal cortex and dopaminergic neurons of the substantia nigra pars compacta preferentially terminate
139 l loss of dopaminergic (DA) neurons in their substantia nigra pars compacta, progressing to bilateral
140 la, basal forebrain, thalamus, hypothalamus, substantia nigra, pars compacta, raphe, and pontine para
141 ng the Purkinje cells of the cerebellum, the substantia nigra pars compacta, red nucleus, dorsal moto
142 a small group of dopaminergic neurons in the substantia nigra pars compacta region of the brain.
143 degeneration of dopaminergic neurons in the substantia nigra pars compacta, reproducing an important
145 P<0.05) and of tyrosine hydroxylase mRNA in substantia nigra pars compacta (SC) (93%; P<0.05) compar
146 icant induction of cell proliferation in the substantia nigra pars compacta (SN(C)) with a time-depen
147 gressive loss of dopaminergic neurons in the substantia nigra pars compacta (SN) leads to debilitatin
148 Neuronal degeneration was observed in the substantia nigra pars compacta (SN), ventral tegmental a
150 The loss of dopaminergic neurons in the substantia nigra pars compacta (SNc) and consequent depl
151 (PD), the ak mouse lacks the majority of the substantia nigra pars compacta (SNc) and experiences str
152 burst firing of dopaminergic neurones in the substantia nigra pars compacta (SNc) and may contribute
155 vulnerability of the dopamine neurons of the substantia nigra pars compacta (SNc) and the importance
156 degeneration of dopaminergic neurons in the substantia nigra pars compacta (SNc) and the presence of
157 Neurons in midbrain dopamine centers, the substantia nigra pars compacta (SNc) and ventral tegment
159 al identification of dopamine neurons in the substantia nigra pars compacta (SNC) and ventral tegment
160 mine whether CeA neurons that project to the substantia nigra pars compacta (SNc) are activated by a
161 rons in the ventral tegmental area (VTA) and substantia nigra pars compacta (SNc) are not significant
162 The rodent ventral tegmental area (VTA) and substantia nigra pars compacta (SNC) contain dopamine ne
163 rons in the ventral tegmental area (VTA) and substantia nigra pars compacta (SNc) convey distinct sig
165 TATEMENT Prior studies have established that substantia nigra pars compacta (SNc) dopamine neurons ar
166 mutant mice displayed increased activity of substantia nigra pars compacta (SNc) dopaminergic neuron
168 on and behavior that depend on the firing of substantia nigra pars compacta (SNc) dopaminergic neuron
169 to be a major factor underlying the loss of substantia nigra pars compacta (SNc) dopaminergic neuron
170 tion of a region homologous to the mammalian substantia nigra pars compacta (SNc) evokes increasing a
172 tanding how dopaminergic (DA) neurons of the substantia nigra pars compacta (SNc) govern movements re
173 se immunoreactive neurons (TH-IR) within the substantia nigra pars compacta (SNc) in both young and a
174 of the ventral tegmental area (VTA) and the substantia nigra pars compacta (SNC) labeled axons and t
175 cuit through which a loss of dopamine in the substantia nigra pars compacta (SNc) leads to increased
177 date the role of this receptor in regulating substantia nigra pars compacta (SNc) neuron physiology i
178 amp microelectrode recordings were made from substantia nigra pars compacta (SNC) neurons in horizont
179 echanism in VTA neurons differs from that in substantia nigra pars compacta (SNc) neurons, where subt
182 ine hydroxylase (TH) positive neurons in the substantia nigra pars compacta (SNc) of C57bl/6J mice fo
183 ts were injected with rAAV2/5 vectors in the substantia nigra pars compacta (SNc) on one side of the
184 ; no such expression was seen in neighboring substantia nigra pars compacta (SNC) or substantia nigra
185 nalling pathways and have been implicated in substantia nigra pars compacta (SNc) pathology in Parkin
186 s unclear to what extent dopamine neurons in substantia nigra pars compacta (SNc) play such roles.
188 ditioned learning, dopaminergic cells in the substantia nigra pars compacta (SNc) respond immediately
189 Somatodendritic dopamine (DA) release in the substantia nigra pars compacta (SNc) shows a limited dep
190 c dopaminergic (mdDA) neurons, including the substantia nigra pars compacta (SNc) subpopulation that
191 ered a type of dopamine neuron in the monkey substantia nigra pars compacta (SNc) that retains past l
192 essed via injection of viral vector into the substantia nigra pars compacta (SNc) to investigate its
193 ease in the ventral tegmental area (VTA) and substantia nigra pars compacta (SNc) to that of axonal d
194 tVTA on the nigrostriatal pathway, from the substantia nigra pars compacta (SNc) to the dorsal stria
196 f either Diamidino Yellow or Fluorogold into substantia nigra pars compacta (SNc) were combined with
197 roxylase (TH)-positive neuron numbers in the substantia nigra pars compacta (SNC) were estimated usin
199 he midbrain superior colliculus (SC), to the substantia nigra pars compacta (SNc) where direct synapt
201 gical criteria were first established in the substantia nigra pars compacta (SNc), but their validity
202 f torsinA in the dopaminergic neurons of the substantia nigra pars compacta (SNc), in addition to man
204 ic inputs and project to dopamine neurons in substantia nigra pars compacta (SNc), whereas matrix neu
217 the tuberculum posterior, a homologue of the substantia nigra pars compacta (SNc)/ventral tegmental a
218 TN DBS) protects dopaminergic neurons of the substantia nigra pars compacta (SNpc) against 6-OHDA and
219 Loss of dopaminergic (DA) neurons in the substantia nigra pars compacta (SNpc) and of noradrenerg
220 effects on firing rates of DA neurons in rat substantia nigra pars compacta (SNPC) and postsynaptic t
221 te (NAA) was significantly diminished in the substantia nigra pars compacta (SNpc) and striatum, regi
222 ic connections onto these neurons in the rat substantia nigra pars compacta (SNpc) and ventral tegmen
223 acterized by loss of dopamine neurons in the substantia nigra pars compacta (SNpc) and widespread agg
224 n travel to higher centers, compromising the substantia nigra pars compacta (SNpc) and, later, the ce
225 therapies to restore dopamine neurons in the substantia nigra pars compacta (SNpc) are greatly needed
226 e (MPTP) damages dopaminergic neurons in the substantia nigra pars compacta (SNpc) as seen in Parkins
227 rotrophic factor for dopamine neurons of the substantia nigra pars compacta (SNpc) by regulating the
228 oxylase-immunoreactive (THir) neurons in the substantia nigra pars compacta (SNpc) compared with sali
229 ions of dopaminergic (DA) neurons within the substantia nigra pars compacta (SNpc) display a differen
230 tain pathogenesis characterized by a loss of substantia nigra pars compacta (SNpc) dopaminergic (DA)
231 rential dysfunction/degeneration of midbrain substantia nigra pars compacta (SNpc) dopaminergic (DA)
232 se signal, while neurons of the ventral tier substantia nigra pars compacta (SNpc) failed to demonstr
233 g is elevated in dopaminergic neurons of the substantia nigra pars compacta (SNpc) of human PD patien
235 n microglia activation has been shown in the substantia nigra pars compacta (SNpc) of PD models when
236 In vivo gene transfer of RGS10 into the substantia nigra pars compacta (SNpc) of rats reduced mi
237 ive dopaminergic (DA) neurons at the ventral substantia nigra pars compacta (SNpc) preferentially deg
239 Dopaminergic neurons containing CR in the substantia nigra pars compacta (SNpc) were relatively sp
241 significant loss of dopaminergic neurons in substantia nigra pars compacta (SNpc), and there was no
242 orous males also had more TH-ir cells in the substantia nigra pars compacta (SNpc), but this differen
243 featuring progressive degeneration of DNs in substantia nigra pars compacta (SNpc), decreased striata
244 otype of dopaminergic neurons located in the substantia nigra pars compacta (SNpc), expression of mon
245 euromelanin signal intensity loss within the substantia nigra pars compacta (SNpc), locus coeruleus,
246 ectiveness of injecting GDF5 into either the substantia nigra pars compacta (SNpc), the lateral ventr
247 ucleolar volume of dopaminergic cells in the substantia nigra pars compacta (SNpc), ventral tegmental
252 elective loss of dopaminergic neurons of the substantia nigra, pars compacta (SNpc) akin to what is o
253 inclusion body formation was detected in the substantia nigra pars compacta, striatum, hippocampus, o
254 or abnormal protein accumulation within the substantia nigra pars compacta, suggesting that nigrostr
255 mice leads to T cell accumulation within the substantia nigra pars compacta, suppression of microglia
256 elective loss of dopaminergic neurons in the substantia nigra pars compacta, the exact mechanism invo
258 eurons of the globus pallidus, thalamus, and substantia nigra pars compacta to various environmental
259 S activity at 2 weeks and a 42% reduction in substantia nigra pars compacta tyrosine hydroxylase-posi
260 nd other Lewy-body-associated disorders, the substantia nigra pars compacta undergoes degeneration, b
261 juvenile dopamine-containing neurons in the substantia nigra pars compacta use pacemaking mechanisms
262 heterogeneous subgroups of neurons, such as substantia nigra pars compacta, ventral tegmental area a
263 CA1 region of hippocampus and dentate gyrus, substantia nigra pars compacta, ventral tegmental area,
264 to PPN cholinergic terminals in the ventral substantia nigra pars compacta (vSNc) or to the ventral
265 ontrast, GluR7 was strongly expressed in the substantia nigra pars compacta, was present at lower lev
266 confirm expression of TRPM2 and TRPM4mRNA in substantia nigra pars compacta.We propose that ICAN is s
268 nt of all dopamine-containing neurons in the substantia nigra pars compacta were located within the c
269 Sections of the post-commissural putamen and substantia nigra pars compacta were processed for tyrosi
271 muli were located more dorsolaterally in the substantia nigra pars compacta, whereas neurons inhibite
272 ssed in the dopaminergic (DA) neurons of the substantia nigra pars compacta, which are preferentially
273 inent expression of the DYT1 gene within the substantia nigra pars compacta, which provides dopaminer
274 and Gli1 lineages specify mDA neurons of the substantia nigra pars compacta while the late Shh and Gl
275 ependent loss of dopaminergic neurons in the substantia nigra pars compacta, with more than 40% of th
276 d A53T mice had reduced neuron counts in the substantia nigra pars compacta, yet striatal medium spin
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。