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1 c reticular nucleus, globus pallidus and the substantia nigra (pars reticulata).
2 t at lower levels in the globus pallidus and substantia nigra pars reticulata.
3 s in PBR expression, as did the striatum and substantia nigra pars reticulata.
4 tly by local application of D-amphetamine in substantia nigra pars reticulata.
5 and ipsilateral ibotenic acid lesions of the substantia nigra pars reticulata.
6 aneously via a second microdialysis probe in substantia nigra pars reticulata.
7 entially innervated GABAergic neurons in the substantia nigra pars reticulata.
8 ns projecting directly to the homolog of the substantia nigra pars reticulata.
9 r via projections to subthalamic nucleus and substantia nigra pars reticulata.
10 ared with inhibition from globus pallidus or substantia nigra pars reticulata.
11 ring substantia nigra pars compacta (SNC) or substantia nigra pars reticulata.
12 rate of neurons in slices from rat or mouse substantia nigra pars reticulata.
13 lize to the GABAergic neurons of the VTA and substantia nigra pars reticulata.
14 cell groups in the brainstem, including the substantia nigra pars reticulata.
15 asolateral amygdala, and caudal parts of the substantia nigra pars reticulata.
16 portions of the interpositus nucleus and the substantia nigra pars reticulata.
17 paramedian pontine reticular formation, and substantia nigra pars reticulata.
18 ons in the neostriatum, globus pallidus, and substantia nigra pars reticulata.
19 activity via basal ganglia output neurons in substantia nigra, pars reticulata.
20 h as the globus and ventral pallidum and the substantia nigra, pars reticulata.
21 o 15-1788 was significantly increased in the substantia nigra pars reticulata (13.5+/-4.1 and 26.3+/-
23 ositive control, 12 GABAergic neurons in the substantia nigra pars reticulata also were recorded and
24 s dopamine D1 receptors (D1+) project to the substantia nigra pars reticulata and are thought to faci
25 eliminate, glutamatergic transmission in the substantia nigra pars reticulata and entopeduncular nucl
26 in globus pallidus, and moderate staining in substantia nigra pars reticulata and entopeduncular nucl
27 f the spike trains from 684 globus pallidus, substantia nigra pars reticulata and subthalamic neurons
28 re taken under urethane anaesthesia from the substantia nigra pars reticulata and subthalamic nucleus
29 as identified as homologous to the mammalian substantia nigra pars reticulata and was also renamed ac
30 e axon collateral, which ramified within the substantia nigra pars reticulata and/or pars compacta.
31 thalamic nucleus and globus pallidus interna/substantia nigra pars reticulata, and infusion of recomb
32 triatal acetylcholine output is initiated in substantia nigra pars reticulata, and this influence con
33 levels in the striatum, globus pallidus and substantia nigra pars reticulata, and was not detectable
34 e tested the hypothesis that D1 receptors in substantia nigra pars reticulata are involved in the exc
35 globus pallidus, entopeduncular nucleus, and substantia nigra pars reticulata, areas of the basal gan
36 rojection neurons in the dorsal striatum and substantia nigra pars reticulata by activating TRPM2 cha
38 lretinin-containing GABAergic neurons of the substantia nigra pars reticulata cannot be differentiate
39 rvated the caudal-dorsal-lateral part of the substantia nigra pars reticulata (cdlSNr), directly or i
40 eld by destruction of a critical zone in the substantia nigra pars reticulata contralateral to a cort
42 the ventral tegmental area, caudate putamen, substantia nigra pars reticulata, entorhinal cortex, cen
43 expression occur in the globus pallidus and substantia nigra pars reticulata following lesions of th
45 ed whole-cell recordings from neurons in the substantia nigra pars reticulata in rat brain slices and
46 ganglia modulates reflex blinking is (1) the substantia nigra pars reticulata inhibits SC neurons, (2
47 opeduncular nucleus, subthalamic nucleus and substantia nigra pars reticulata ipsilateral to the lesi
48 activity measured in the globus pallidus and substantia nigra pars reticulata is caused by abnormal s
49 ifferent response profiles of neurons in the substantia nigra pars reticulata of the basal ganglia an
50 n the mossy fiber region of the hippocampus, substantia nigra pars reticulata, pallidum, and deep cer
51 the output nuclei of the basal ganglia, the substantia nigra pars reticulata, projects via the thala
52 keeping constant the average firing rate of substantia nigra pars reticulata reduces the incidence o
53 d by electrical stimulation was identical in substantia nigra pars reticulata slices prepared from TK
54 s been postulated to inhibit output from the substantia nigra pars reticulata (SNpr) and internal pal
55 n the 25-40 Hz range in LFPs recorded in the substantia nigra pars reticulata (SNpr) and motor cortex
56 7 d after lesion, increases in motor cortex-substantia nigra pars reticulata (SNpr) coherence emerge
57 ction of the superior colliculus (SC) or the substantia nigra pars reticulata (SNpr) contralateral to
58 torticollis) can be evoked by inhibition of substantia nigra pars reticulata (SNpr) in the nonhuman
59 terized the appearance of APP domains in rat substantia nigra pars reticulata (SNpR) neurons targeted
60 in the entopeduncular nucleus (EPN) and the substantia nigra pars reticulata (SNpr) on the intact si
61 the two output nuclei of the basal ganglia, substantia nigra pars reticulata (SNpr), and entopeduncu
62 ons from the pedunculopontine nucleus to the substantia nigra pars reticulata (SNr) act on muscarinic
65 that the zebra finch LSt projects to the GP, substantia nigra pars reticulata (SNr) and pars compacta
66 ntaining neurons expressed GFRalpha-1 in the substantia nigra pars reticulata (SNR) and the ventral t
67 ents, we investigated the involvement of the substantia nigra pars reticulata (SNr) and the ventromed
68 th at synapses with GABAergic neurons in the substantia nigra pars reticulata (SNr) and with distal d
70 haracterize the developmental changes in the substantia nigra pars reticulata (SNr) associated with t
71 taneous firing of GABAergic neurons in mouse substantia nigra pars reticulata (SNr) brain slices.
72 the output of the basal ganglia through the substantia nigra pars reticulata (SNr) controls active a
73 ncrease in excitatory glutamatergic input to substantia nigra pars reticulata (SNr) from the subthala
75 of the primary targets of the striatum, the substantia nigra pars reticulata (SNr) has been hypothes
76 vo recording, we measured BG output from the substantia nigra pars reticulata (SNr) in mice while mon
79 Here we present evidence that the monkey substantia nigra pars reticulata (SNr) in the basal gang
82 injecting muscimol, a GABA agonist, into the substantia nigra pars reticulata (SNr) markedly reduced
83 e-cell patch clamp recordings were made from substantia nigra pars reticulata (SNr) neurones in rat m
84 where they overlapped with non-dopaminergic substantia nigra pars reticulata (SNr) neurons and proxi
85 would be identified altered the activity of substantia nigra pars reticulata (SNr) neurons of the ba
86 to examine impulse activity and responses of substantia nigra pars reticulata (SNr) neurons to GABA,
87 Here we examined the effects of DBS of the substantia nigra pars reticulata (SNr) on amygdala-kindl
89 ABAergic basal ganglia output neurons in the substantia nigra pars reticulata (SNr) receive strong CA
91 umber and denser on dendrites located in the substantia nigra pars reticulata (SNr) than on those loc
92 7 mRNA expression histochemistry, in the rat substantia nigra pars reticulata (SNr) through an acute
93 confirmed a direct GABAergic input from the substantia nigra pars reticulata (SNr) to the deep SC/Me
94 petitive discharge, GABAergic neurons of the substantia nigra pars reticulata (SNr) tonically inhibit
95 m, the adjacent and overlying brain, and the substantia nigra pars reticulata (SNr) were compared wit
96 llmark of the GABA projection neurons of the substantia nigra pars reticulata (SNr), a key basal gang
97 striatal firing pattern is expressed in the substantia nigra pars reticulata (SNr), a main target of
98 pontaneously active GABAergic neurons of the substantia nigra pars reticulata (SNr), a major output o
99 by exerting its excitatory influence on the substantia nigra pars reticulata (SNR), a major output s
100 y the presence of a dual-output nucleus, the substantia nigra pars reticulata (SNr), and the presence
101 RCGU in the entopeduncular nucleus (EP) and substantia nigra pars reticulata (SNr), as does the acut
102 observed to reach their midbrain target, the substantia nigra pars reticulata (SNr), at E14 in the mo
103 y 3-1000 mum ACh in GABAergic neurons of the substantia nigra pars reticulata (SNr), but not in DA ne
104 ithdrawal than background strain mice in the substantia nigra pars reticulata (SNr), subthalamic nucl
106 amic nucleus (STN), globus pallidus (GP) and substantia nigra pars reticulata (SNr), together with th
107 at neurons in gerbil (Meriones unguiculatus) substantia nigra pars reticulata (SNr), whose "neuronal"
112 ized metabolic hyperactivity in both STN and substantia nigra pars reticulata, the main output struct
113 r, the GABAergic output projections from the substantia nigra pars reticulata to the deep layers of t
114 he nucleus accumbens and 5-HT release in the substantia nigra pars reticulata, using a common stimula
115 alamic nucleus and to a lesser extent in the substantia nigra pars reticulata, while no hybridization
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