ライフサイエンスコーパス: subthalamic nucleus

1 tual conditions in the cerebellum, pons, and subthalamic nucleus.

2 emotionally evoked activity to right ventral subthalamic nucleus.

3 overlap the labeled neurons observed in the subthalamic nucleus.

4 ter-related hypotheses about the role of the subthalamic nucleus.

5 acy of bilateral constant-current DBS of the subthalamic nucleus.

6 caudate and left putamen/globus pallidus and subthalamic nucleus.

7 in stimulation of the limbic and associative subthalamic nucleus.

8 in part on neural circuitry inclusive of the subthalamic nucleus.

9 current spread into nonmotor regions of the subthalamic nucleus.

10 tory activation in prefrontal cortex and the subthalamic nucleus.

11 decreased inhibitory GABAergic input to the subthalamic nucleus.

12 he pedunculopontine nucleus, red nucleus and subthalamic nucleus.

13 onnections of the pre-supplementary area and subthalamic nucleus.

14 atory input from the 'limbic' regions of the subthalamic nucleus.

15 ith anterior and posterior subregions of the subthalamic nucleus.

16 des mapped onto a mesial-lateral axis of the subthalamic nucleus.

17 ts localized within the anterior and ventral subthalamic nucleus.

18 atients, after deep brain stimulation of the subthalamic nucleus.

19 in 4 patients with PD undergoing DBS of the subthalamic nucleus.

20 lysis exploring the effect of atrophy in the subthalamic nucleus, a cerebellar input source, confirme

21 average of all the significantly correlated subthalamic nucleus activities accounted for >60% of the

22 The results implicate frequency-specific subthalamic nucleus activities as substantial factors in

23 z) and high-gamma/high-frequency (55-375 Hz) subthalamic nucleus activity and force measures, which e

24 When recording the subthalamic nucleus activity, we found a clear neural re

25 The strength of effective subthalamic nucleus afferents and efferents were reduced

26 rom the substantia nigra pars reticulata and subthalamic nucleus along with cortical electroencephalo

27 We show that VP output pathways to the subthalamic nucleus and also to the ventral tegmental ar

28 chronization and oscillatory activity in the subthalamic nucleus and basal ganglia (BG) output nuclei

29 or performing simultaneous recordings of the subthalamic nucleus and cortex using magnetoencephalogra

30 r treated with deep brain stimulation to the subthalamic nucleus and dopaminergic therapy or managed

31 BAergic transmission within the normal human subthalamic nucleus and evidence of GABA innervation thr

32 served that certain phase alignments between subthalamic nucleus and globus pallidus amplified local

33 ronize a network of model neurons comprising subthalamic nucleus and globus pallidus external and sug

34 ed local field potential recordings from the subthalamic nucleus and globus pallidus of five patients

35 connectivity in the orbitofrontal cortex and subthalamic nucleus and greater local connectivity in th

36 Beta band coupling between the subthalamic nucleus and lateral motor areas was not infl

37 ssed synchronization of activity between the subthalamic nucleus and mesial premotor regions, includi

38 ations, and make stronger projections to the subthalamic nucleus and parafascicular nucleus of the th

39 associated with anterior associative-limbic subthalamic nucleus and right dorsolateral prefrontal fu

40 on in the cardinal nuclei - globus pallidus, subthalamic nucleus and substantia nigra - than in typic

41 and drug-taking behavior via projections to subthalamic nucleus and substantia nigra pars reticulata

42 tudy, 40 received a bilateral implant in the subthalamic nucleus and their data contributed to the pr

43 fied cell classes projected primarily to the subthalamic nucleus and to the striatum, respectively.

44 iven by a neural network, which includes the subthalamic nucleus and ventrolateral thalamus and has b

45 r in all regions except the dentate nucleus, subthalamic nucleus, and corpus callosum of multiple sys

46 ementary motor area, inferior frontal gyrus, subthalamic nucleus, and inferior parietal cortex.

47 subthalamic nucleus, ramping activity of the subthalamic nucleus, and movement-related activity of th

48 Hz, a similar, though weaker, oscillation in subthalamic nucleus, and strong phase coherence between

49 n the external and internal globus pallidus, subthalamic nucleus, and ventral motor thalamic nuclei.

50 eiving more inputs from the globus pallidus, subthalamic nucleus, and zona incerta.

51 The globus pallidus interna and the subthalamic nucleus are accepted targets for this proced

52 The striatum and the subthalamic nucleus are the main entry points for cortic

53 so associated with greater engagement of the subthalamic nucleus area, suggestive of a compensatory m

54 ived bilateral deep brain stimulation of the subthalamic nucleus at the National Institutes of Health

55 a-amplitude are coupled to, and precede, the subthalamic nucleus beta-trough.

56 rdson syndrome had less neuronal loss in the subthalamic nucleus, but more severe neuronal loss in th

57 nd found that therapeutic effects within the subthalamic nucleus can be accounted for by direct selec

58 Accordingly, the subthalamic nucleus can be considered a key component of

59 ect of bilateral delivery of AAV2-GAD in the subthalamic nucleus compared with sham surgery in patien

60 endent upon functioning of the glutamatergic subthalamic nucleus (computing the 'normalization term'

61 sease, and stimulation affecting the ventral subthalamic nucleus (connected to higher order cortical

62 esized that stimulation affecting the dorsal subthalamic nucleus (connected to the motor cortex) woul

63 The identification of a hyperdirect cortico-subthalamic nucleus connection highlighted the important

64 tral striatum to right anterior ventromedial subthalamic nucleus consistent with previous observation

65 internal segment of the globus pallidus and subthalamic nucleus correlated strongly with the number

66 ally and spectrally segregated resting state subthalamic nucleus-cortical networks.

67 oup scanned on and off treatment with either subthalamic nucleus deep brain stimulation (n = 14) or i

68 c efficacy of STN DBS.SIGNIFICANCE STATEMENT Subthalamic nucleus deep brain stimulation (STN DBS) is

69 Subthalamic nucleus deep brain stimulation (STN DBS) pro

70 Bilateral subthalamic nucleus deep brain stimulation (STN-DBS) has

71 Parkinson's Disease patients both on and off subthalamic nucleus deep brain stimulation (STN-DBS), wh

72 onism with severe dyskinesias, and underwent subthalamic nucleus deep brain stimulation 8 years after

73 ly preventable risks for mortality following subthalamic nucleus deep brain stimulation for Parkinson

74 ught to determine the suicide rate following subthalamic nucleus deep brain stimulation for Parkinson

75 Mortality in the first year following subthalamic nucleus deep brain stimulation has been repo

76 Subthalamic nucleus deep brain stimulation improves moto

77 ive-motor declines associated with bilateral subthalamic nucleus deep brain stimulation may be revers

78 Specifically, we show that subthalamic nucleus deep brain stimulation modulates all

79 n advanced Parkinson's disease patients with subthalamic nucleus deep brain stimulation parameters de

80 subthalamic nucleus, respectively, and that subthalamic nucleus deep brain stimulation predominantly

81 results highlight that clinically effective subthalamic nucleus deep brain stimulation suppresses sy

82 rkinson's disease bilaterally implanted with subthalamic nucleus deep brain stimulation systems.

83 ng magnetoencephalography (during concurrent subthalamic nucleus deep brain stimulation).

84 Bilateral subthalamic nucleus deep brain stimulation, however, has

85 y described clinically in some patients with subthalamic nucleus deep brain stimulation.

86 paradigm to test the hypothesis that pairing subthalamic nucleus deep-brain stimulation (STN-DBS) wit

87 beneficial as well as detrimental effects of subthalamic nucleus deep-brain stimulation on impulsive

88 deep brain stimulation to the region of the subthalamic nucleus disrupts decision making when multip

89 oxygen-level-dependent response in the right subthalamic nucleus during an impulsiveness task.

90 eoperatively, (2) the day after insertion of subthalamic nucleus electrodes, (3) three weeks later, p

91 ptimization of deep brain stimulation of the subthalamic nucleus for both motor and cognitive functio

92 The subthalamic nucleus-globus pallidus network is a potenti

93 The subthalamic nucleus has been hypothesized to play a part

94 hat coordinated reset neuromodulation of the subthalamic nucleus has both acute and sustained long-la

95 levations in beta activity (13-35 Hz) in the subthalamic nucleus have been demonstrated to correlate

96 white matter structures in proximity to the subthalamic nucleus have been implicated in the clinical

97 ic) profiles in the dorsolateral part of the subthalamic nucleus (i.e. its sensorimotor territory) wa

98 hods that modulate production of GABA in the subthalamic nucleus improve basal ganglia function in pa

99 ed BCI-controlled adaptive DBS (aDBS) of the subthalamic nucleus in 8 PD patients.

100 In conclusion, our results implicate the subthalamic nucleus in a modulation of outcome value in

101 ecificity of the anterior associative-limbic subthalamic nucleus in decisional impulsivity.

102 Deep brain stimulation of the subthalamic nucleus in individuals with Parkinson's dise

103 ouis Benabid for elucidating the role of the subthalamic nucleus in mediating the motor dysfunction o

104 riatal, and hyperdirect connections with the subthalamic nucleus in modulating waiting and stopping a

105 a better understanding about the role of the subthalamic nucleus in non-motor functions is needed.

106 Recent work has shown that DBS of the subthalamic nucleus in Parkinson's disease greatly reduc

107 Deep brain stimulation (DBS) of the subthalamic nucleus in Parkinson's disease is known to c

108 , and second, the critical importance of the subthalamic nucleus in successful decision making when m

109 etal-brainstem network was coherent with the subthalamic nucleus in the alpha (7-13 Hz) band, whilst

110 ase, suggesting a more pervasive role of the subthalamic nucleus in the control of human decision-mak

111 sibility that cortical connectivity with the subthalamic nucleus in the high and low beta bands may r

112 or regions were predominantly coupled to the subthalamic nucleus in the high beta frequency range, bu

113 tion suppresses synchrony locally within the subthalamic nucleus in the low beta oscillatory range an

114 in the globus pallidus, substantia nigra and subthalamic nucleus, in addition to many axonal spheroid

115 e that the amplitude of beta activity in the subthalamic nucleus increases in proportion to burst dur

116 imulation of estimated predominant non-motor subthalamic nucleus induced hypomanic behaviour.

117 Deep brain stimulation in the subthalamic nucleus is an effective and safe surgical pr

118 Deep brain stimulation of the subthalamic nucleus is an effective treatment for Parkin

119 Deep brain stimulation of the subthalamic nucleus is an established treatment for the

120 We show that stimulation of the subthalamic nucleus is causally implicated in increasing

121 Moreover, the subthalamic nucleus is critical to balance inhibitory an

122 variability, functional organization of the subthalamic nucleus is difficult to investigate in vivo

123 The subthalamic nucleus is implicated in inhibitory function

124 pears at odds with the current view that the subthalamic nucleus is important for adjusting behaviour

125 ese results provide strong evidence that the subthalamic nucleus is involved in response inhibition,

126 These results suggest that the ventral subthalamic nucleus is involved in the balance between a

127 Deep brain stimulation (DBS) of the subthalamic nucleus markedly improves the motor symptoms

128 t spread of current to nonmotor areas of the subthalamic nucleus may be responsible for declines in c

129 that low-frequency neuronal activity in the subthalamic nucleus may encode the information required

130 taucipir uptake in globus pallidus, putamen, subthalamic nucleus, midbrain, and dentate nucleus relat

131 Bilateral deep brain stimulation of the subthalamic nucleus (n = 60) or globus pallidus (n = 61)

132 ms tested, including cortical astrocytes and subthalamic nucleus neurons and in measures of long-term

133 lationship using extracellular recordings of subthalamic nucleus neurons from 19 PD patients undergoi

134 ocal field potentials were recorded from the subthalamic nucleus of 12 patients with advanced Parkins

135 ients received bilateral implantation in the subthalamic nucleus of a constant-current DBS device.

136 ants underwent bilateral implantation in the subthalamic nucleus of a multiple-source, constant-curre

137 ocal field potentials were recorded from the subthalamic nucleus of eight Parkinson's disease patient

138 rded local field potential activity from the subthalamic nucleus of humans undergoing functional neur

139 es by recording the neuronal activity of the subthalamic nucleus of patients with Parkinson's disease

140 We found that the subthalamic nucleus of the basal ganglia has a substanti

141 ests that the modulatory relationship of the subthalamic nucleus on intracerebellar connectivity is l

142 the effects of deep brain stimulation of the subthalamic nucleus on response inhibition tasks.

143 Yet only the change in strength of local subthalamic nucleus oscillations correlates with the deg

144 First that the subthalamic nucleus plays a role in adjusting response t

145 ence of different functional circuits within subthalamic nucleus' portions deemed to be appropriate a

146 -DOPA, and were associated with increases in subthalamic nucleus power over a broad gamma range.

147 , direct, and basal ganglia afferents to the subthalamic nucleus predicted clinical status and therap

148 Motor cortical coupling with subthalamic nucleus predominantly involved driving of th

149 Constant-current DBS of the subthalamic nucleus produced significant improvements in

150 basal ganglia pathways, and the hyperdirect subthalamic nucleus projections.

151 l circuits accompanied by hyperactivation of subthalamic nucleus/putaminal regions.

152 emporally regulated sensory responses of the subthalamic nucleus, ramping activity of the subthalamic

153 patients with deep brain stimulation of the subthalamic nucleus region did not slow down appropriate

154 research, highlights the role of the ventral subthalamic nucleus region in response inhibition and su

155 itive paradigms, but that a wide area of the subthalamic nucleus region is involved in the motor symp

156 tterns in the striatum, globus pallidus, and subthalamic nucleus related to sensory and motor events

157 by the hyperdirect and indirect pathways to subthalamic nucleus, respectively, and that subthalamic

158 ectivity with the ventrolateral thalamus and subthalamic nucleus showed inverse correlation with keta

159 The subthalamic nucleus (STh) is a small subcortical structu

160 iduals with Parkinson's disease treated with subthalamic nucleus stimulation (n = 17) and healthy con

161 These abnormalities improved with subthalamic nucleus stimulation (p < 0.005) but not levo

162 nts with idiopathic Parkinson's disease with subthalamic nucleus stimulation were analysed on externa

163 ssive coupling may be reduced by therapeutic subthalamic nucleus stimulation.

164 analyses revealed two dissociable effects of subthalamic nucleus stimulation.

165 S) of globus pallidus internus (GPi DBS) and subthalamic nucleus (STN DBS) are effective treatment fo

166 Deep brain stimulation of the subthalamic nucleus (STN DBS) has become an accepted tre

167 Deep brain stimulation of the subthalamic nucleus (STN DBS) improves motor symptoms in

168 tential of high-frequency stimulation of the subthalamic nucleus (STN HFS) for heroin addiction.

169 d beta (beta) oscillations (15-30 Hz) in the subthalamic nucleus (STN) accompany movement difficultie

170 such speed-accuracy adjustments by recording subthalamic nucleus (STN) activity and electroencephalog

171 A selective increase in subthalamic nucleus (STN) activity was found when the st

172 Neural synchrony between the subthalamic nucleus (STN) and cortex is critical for pro

173 Reciprocally connected glutamatergic subthalamic nucleus (STN) and GABAergic external globus

174 Recent evidence implicates the subthalamic nucleus (STN) and globus pallidus internus (

175 associated with increasing metabolism in the subthalamic nucleus (STN) and internal globus pallidus (

176 but varies as a function of activity in the subthalamic nucleus (STN) and is further modulated by tr

177 le in the genesis of burst discharges in the subthalamic nucleus (STN) and parkinsonian locomotor sym

178 The GP is reciprocally connected with the subthalamic nucleus (STN) and projects to the SNr and mo

179 We recorded local field potentials in the subthalamic nucleus (STN) and scalp EEG (modified 10/20

180 ease promotes burst firing of neurons in the subthalamic nucleus (STN) and substantia nigra zona reti

181 connectivity profile of effective DBS to the subthalamic nucleus (STN) and test its ability to predic

182 otor cortex and by a network composed of the subthalamic nucleus (STN) and the external segment of gl

183 The subthalamic nucleus (STN) and the zona incerta (ZI) are

184 rnal globus pallidus (GPe) and glutamatergic subthalamic nucleus (STN) are closely related to motor f

185 SMA) and inferior frontal gyrus (IFG) to the subthalamic nucleus (STN) are thought to support this fu

186 chronic deep brain stimulation (DBS) of the subthalamic nucleus (STN) at the commonly used frequency

187 l responses to electrical stimulation of the subthalamic nucleus (STN) at various frequencies between

188 The striatum and the subthalamic nucleus (STN) constitute the input stage of

189 changes in Parkinson disease (PD) induced by subthalamic nucleus (STN) DBS to determine whether these

190 function have been associated with bilateral subthalamic nucleus (STN) DBS.

191 Subthalamic nucleus (STN) deep brain stimulation (DBS) c

192 Subthalamic nucleus (STN) deep brain stimulation (DBS) h

193 Subthalamic nucleus (STN) deep brain stimulation (DBS) r

194 ty were compared with those occurring during subthalamic nucleus (STN) deep brain stimulation (DBS),

195 inson's disease (PD) patients with bilateral subthalamic nucleus (STN) deep brain stimulation underwe

196 The glutamatergic subthalamic nucleus (STN) exerts control over motor outp

197 local field potential (LFP) activity in the subthalamic nucleus (STN) from electrodes implanted in p

198 ders and deep brain stimulation (DBS) of the subthalamic nucleus (STN) has been reported to improve s

199 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) has no effect on the AER, but

200 The neuronal population of the subthalamic nucleus (STN) has the ability to prolong inc

201 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) improves symptoms of Parkinson

202 cephalography and direct recordings from the subthalamic nucleus (STN) in 17 PD patients.

203 regulated by the burst-firing pattern of the subthalamic nucleus (STN) in a feed-forward, or efferent

204 Recent studies have implicated the subthalamic nucleus (STN) in decisions that involve inhi

205 ans have demonstrated the involvement of the subthalamic nucleus (STN) in motivational and emotional

206 potential (LFP) activities recorded from the subthalamic nucleus (STN) in patients with deep brain st

207 ingers while recording LFP activity from the subthalamic nucleus (STN) in patients with Parkinson's d

208 ection highlighted the important role of the subthalamic nucleus (STN) in regulating behavior.

209 ng surgery for deep brain stimulation to the subthalamic nucleus (STN) indicate that spectral changes

210 ement in 12 PD patients receiving unilateral subthalamic nucleus (STN) infusion of an adenoassociated

211 The subthalamic nucleus (STN) is a critical excitatory signa

212 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is a highly effective symptoma

213 The subthalamic nucleus (STN) is a key area of the basal gan

214 The subthalamic nucleus (STN) is a key node in the network t

215 Evidence across species has shown that the subthalamic nucleus (STN) is activated by scenarios invo

216 Deep brain stimulation (DBS) in the subthalamic nucleus (STN) is an effective tool for the t

217 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is an effective treatment for

218 The subthalamic nucleus (STN) is an element of cortico-basal

219 The subthalamic nucleus (STN) is hypothesized to play a cent

220 The activity of the subthalamic nucleus (STN) is intimately related to movem

221 hown that beta-band desynchronization in the subthalamic nucleus (STN) is reduced just before and dur

222 ated in effortful control over behavior, the subthalamic nucleus (STN) is specifically thought to con

223 The subthalamic nucleus (STN) is the main target for neurosu

224 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is the most common neurosurgic

225 The subthalamic nucleus (STN) is thought to play a central r

226 Gamma activity in the subthalamic nucleus (STN) is widely viewed as a pro-kine

227 nt models of decision making assume that the subthalamic nucleus (STN) mediates this function by elev

228 to different patterns of synaptic input from subthalamic nucleus (STN) neuron.

229 In this study, subthalamic nucleus (STN) neuronal activity patterns in

230 In Parkinsonism, subthalamic nucleus (STN) neurons and two types of exter

231 The activity patterns of subthalamic nucleus (STN) neurons are intimately linked

232 ese models suggest that a network of GPe and subthalamic nucleus (STN) neurons computes the normaliza

233 Subthalamic nucleus (STN) neurons exhibited low-latency

234 cessive burst firing of action potentials in subthalamic nucleus (STN) neurons has been correlated wi

235 been reported as a pathological activity of subthalamic nucleus (STN) neurons in Parkinson's disease

236 Overactivity of subthalamic nucleus (STN) neurons is a consistent featur

237 ng methods to predict firing patterns of rat subthalamic nucleus (STN) neurons when their rhythmic fi

238 ypic GABAergic GPe neurons fire antiphase to subthalamic nucleus (STN) neurons, often express parvalb

239 neous rhythmic bursts of spikes generated by subthalamic nucleus (STN) neurons.

240 ior motor thalamus (Vim/Vop) neurons and 123 subthalamic nucleus (STN) neurons.

241 uency (13-30 Hz) oscillatory activity in the subthalamic nucleus (STN) of Parkinson's disease (PD) ha

242 ta and gamma ranges has been recorded in the subthalamic nucleus (STN) of Parkinson's disease (PD) pa

243 e applied glutamate receptor blockers to the subthalamic nucleus (STN) of parkinsonian rats and evalu

244 The subthalamic nucleus (STN) of the basal ganglia appears t

245 The subthalamic nucleus (STN) of the basal ganglia is an imp

246 uppresses motor activity, ostensibly via the subthalamic nucleus (STN) of the basal ganglia.

247 Parkinson's disease (PD) patients to either subthalamic nucleus (STN) or globus pallidus internus (G

248 whereby deep brain stimulation (DBS) of the subthalamic nucleus (STN) or internal globus pallidus (G

249 The subthalamic nucleus (STN) provides a second entry point

250 These models predict that the subthalamic nucleus (STN) provides feedback that normali

251 rget for deep brain stimulation (DBS) of the subthalamic nucleus (STN) remains controversial.

252 In the BG motor loop the subthalamic nucleus (STN) represents an important struct

253 ion and blockade of neuronal activity in the subthalamic nucleus (STN) results in a hyperkinetic move

254 ral electrocorticography in combination with subthalamic nucleus (STN) single-unit recording to study

255 We studied 14 PD patients with bilateral subthalamic nucleus (STN) stimulation and 16 age-matched

256 medical therapy (BMT); and (2) randomised to subthalamic nucleus (STN) versus globus pallidus interna

257 nded study the cognitive and mood effects of subthalamic nucleus (STN) vs. globus pallidus interna (G

258 gyrus, presupplementary motor area (preSMA), subthalamic nucleus (STN), and primary motor cortex duri

259 , globus pallidus interna and externa (GPe), subthalamic nucleus (STN), and substantia nigra pars com

260 tex provides strong excitatory inputs to the subthalamic nucleus (STN), and these cortico-STN inputs

261 d associated brainstem nuclei, including the subthalamic nucleus (STN), globus pallidus, striatum, an

262 projections to ventral tegmental area (VTA), subthalamic nucleus (STN), lateral hypothalamus, among o

263 the substantia nigra pars reticulata (SNr), subthalamic nucleus (STN), rostromedial lateral globus p

264 The subthalamic nucleus (STN), which receives excitatory inp

265 We hypothesize that repeated pairing of subthalamic nucleus (STN)-DBS and M1-TMS at specific tim

266 ated and coherent activity in the cortex and subthalamic nucleus (STN).

267 t and indirect pathways, interact within the subthalamic nucleus (STN).

268 were the major GPe cell type innervating the subthalamic nucleus (STN).

269 us (DR), pedunculopontine nucleus (PPN), and subthalamic nucleus (STN).

270 nflict: the medial prefrontal cortex and the subthalamic nucleus (STN).

271 ced by the glutamatergic projection from the subthalamic nucleus (STN).

272 e-supplementary motor area (preSMA), and the subthalamic nucleus (STN).

273 rnal globus pallidus (GPe) and glutamatergic subthalamic nucleus (STN).

274 right inferior frontal cortex (rIFC) and the subthalamic nucleus (STN).

275 ate activity in the globus pallidus (GP) and subthalamic nucleus (STN).

276 ngages a midbrain region consistent with the subthalamic nucleus (STN).

277 e-SMA), inferior frontal gyrus, caudate, and subthalamic nucleus (STN).

278 brain stimulation (DBS, n=164) at either the subthalamic nucleus (STN, n=84) or globus pallidus inter

279 Deep brain stimulation (DBS) of the subthalamic nucleus (STN-DBS) has largely replaced ablat

280 suggests that deep brain stimulation of the subthalamic nucleus (STN-DBS) may have a disease modifyi

281 lation (HFS) via implanted electrodes at the subthalamic nucleus (STN-HFS).

282 particular the subthalamic locomotor region (subthalamic nucleus, STN) and the periaqueductal grey (P

283 The subthalamic nucleus, substantia nigra and globus pallidu

284 s pallidus interna (pallidal stimulation) or subthalamic nucleus (subthalamic stimulation).

285 in the anterior frontal cortex, striatum and subthalamic nucleus suggests the striatal afferent conne

286 ety of bilateral infusion of AAV2-GAD in the subthalamic nucleus supports its further development for

287 uency band having much shorter net delays to subthalamic nucleus than those in the lower beta band.

288 ct evidence points to the involvement of the subthalamic nucleus-the most common target for deep brai

289 wever, suppression of the sensitivity of the subthalamic nucleus to its hyperdirect afferents by deep

290 pread of current to nonmotor portions of the subthalamic nucleus using Cicerone Deep Brain Stimulatio

291 Stimulation of the subthalamic nucleus was associated with dysarthria, fati

292 ociated suppression in their coupling to the subthalamic nucleus was not found to correlate with moto

293 ming asymmetric synapses in the dorsolateral subthalamic nucleus was reduced by 55.1% and 27.9%, resp

294 wledge of the anatomical organization of the subthalamic nucleus, we propose that the location of the

295 ral cortices, striatum, substantia nigra and subthalamic nucleus were assessed.

296 rmer from the somatosensory/motor cortex and subthalamic nucleus, which may explain their short-laten

297 and bilateral deep brain stimulation of the subthalamic nucleus with one contact in the dorsal and a

298 as associated with lower connectivity of the subthalamic nucleus with ventral striatum and subgenual

299 tical nodes and is posited to project to the subthalamic nucleus, with a putative global suppressive

300 ucleus predominantly involved driving of the subthalamic nucleus, with those drives in the higher bet