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1 cute hyperkalemia with the administration of succinylcholine.
2 nst the risk of side effects associated with succinylcholine.
3 ical evaluation as a possible alternative to succinylcholine.
4  changes were abolished after paralysis with succinylcholine.
5 sal muscle fibres, derived from testing with succinylcholine.
6 increased sensitivity to the muscle relaxant succinylcholine.
7  situation, laryngospasm can be treated with succinylcholine administration by intramuscular, intraos
8 a pharmacodynamic profile similar to that of succinylcholine) and AV002 (with an intermediate duratio
9 terase through genotyping patients with post-succinylcholine apnea.
10                                              Succinylcholine arguably remains the preferred neuromusc
11                  Upon injection of 820 pL of succinylcholine chloride in a 10-microm capillary, a con
12                 Two-microliter injections of succinylcholine chloride in a 5-cm x 1-mm C-18 column wi
13                       Static measurements of succinylcholine chloride in water have shown a detection
14 parations of citrate and nitrate, as well as succinylcholine chloride with sodium salicylate using ac
15 roduces a profound increase in activation by succinylcholine compared with either wild-type alpha7 or
16 tics and/or the depolarizing muscle relaxant succinylcholine in malignant hyperthermia-susceptible in
17 anophosphate toxicity, as drugs to alleviate succinylcholine-induced apnea, and as detoxification age
18 t was given 14 mg of etomidate and 100 mg of succinylcholine intravenously.
19  be added to the list of conditions in which succinylcholine is contraindicated.
20                          The side effects of succinylcholine occur in relatively predictable circumst
21 ensitivity to vibration and by the effect of succinylcholine on their response to ramp-and-hold muscl
22 to passive muscle stretch and the effects of succinylcholine (SCh) and by their sensitivity to vibrat
23                   The regimen of thiopental, succinylcholine (SCh) and unsupplemented nitrous oxide/o
24           Each afferent was characterised by succinylcholine testing with regard to its intrafusal fi
25  of muscle spindles to tendon stretch and to succinylcholine, whereas the high dose (5 microg kg(-1))
26 f fatal hyperkalemia after administration of succinylcholine, with a mechanism similar to that report

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