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1 opsis thaliana) also elongate in response to sucrose.
2 responding to the rate of change of cellular sucrose.
3  post-ingestive, nutritional consequences of sucrose.
4 t acquire their carbon essentially from leaf sucrose.
5 ylglyceride when mosquitoes were co-fed with sucrose.
6 ed for alcohol versus the nondrug reinforcer sucrose.
7 almost completely inhibited the detection of sucrose.
8  bioparticle analysis: isopropanol (IPA) and sucrose.
9 ns, 0.1g/100g reducing sugars and 0.12g/100g sucrose.
10 Hercules' synchronously to the appearance of sucrose.
11 at high rates after the tone associated with sucrose.
12 diated degradation of fructan, raffinose and sucrose.
13 - 8.6g/100g), maltose (41 +/- 15 g/100g) and sucrose (1.2 +/- 2.7 g/100g).
14 operties (set 1: lactone, diastase activity, sucrose - 100%) (set 2: free acidity, HMF, ash - 95%).
15  75 mM arginine, followed by the addition of sucrose (15 mM) after 0, 6, 20, or 48 h.
16  in fat (45% kcal), cholesterol (1% w/w) and sucrose (15% kcal).
17 ur types of biscuits were prepared: (1) with sucrose, (2) with glucose and fructose, (3) with fructos
18 ochlorite (0.2g.L(-1)), starch (5.0g.L(-1)), sucrose (5.4g.L(-1)) and water (150g.L(-1)).
19 severative and impulsive-like responding for sucrose, a disaccharide consisting of fructose and gluco
20 or ERK1, both at baseline and in response to sucrose, a signaling change associated with decreased st
21                                              Sucrose, a tuber-promoting factor in vitro, increases St
22 en 1175 and 950cm(-1) (glucose, fructose and sucrose absorption bands).
23 olved in stem growth, cell wall biology, and sucrose accumulation.
24          We propose a dynamic model in which sucrose acts via IR60b to activate a circuit that inhibi
25 p from 7 to 4.5, while biofilms treated with sucrose after 6, 20, or 48 h of preincubation with argin
26 2 times (Sf/Si ratio) in the presence of 10% sucrose after freeze drying.
27 ted that mixing the analog 1728 at 1 mM with sucrose almost completely inhibited the detection of suc
28                                         From sucrose alone, the corresponding recombinant protein, na
29 nlike previously with a diet high in fat and sucrose alone, the inclusion of cholesterol in the WD in
30 decreased social behavior and preference for sucrose, along with increased immobility in the tail sus
31 e rats learned to associate one context with sucrose and another context with less-desirable salt.
32  and med12 aux1 single and double mutants to sucrose and application of auxin transport inhibitor N-1
33 op (Glu-334-His-343) is essential in binding sucrose and beta(2-1)-linked oligosaccharides.
34 alities with opposing valence, such as sweet sucrose and bitter denatonium, reliant on different sens
35 a fatty rats fed a western diet high in fat, sucrose and cholesterol for 24 weeks developed a severe
36                     The relationship between sucrose and colour development during the roasting proce
37 ng the release of dopamine that is shared by sucrose and drugs of abuse, reinstated sucrose seeking d
38  neurons, after appetitive conditioning with sucrose and extinction learning.
39 es these are mixtures of aliphatic esters of sucrose and glucose of varying structures and quantities
40   Nonetheless, the Raman detection limits of sucrose and glucose were 5 x 10(-4) and 2.5 x 10(-3) mol
41                                        While sucrose and hexose levels were constitutively elevated i
42 ith highest changes in delta(18) O of starch/sucrose and lowest in delta(18) O of quercitol.
43    Following floral initiation, the level of sucrose and other non-structural carbohydrates increased
44  trained to press a right and left lever for sucrose and pellet outcomes, after which these contingen
45                    One compound also reduced sucrose and saccharin consumption, while the other was s
46 ut microbiota in the differential effects of sucrose and starch on blood lipids, as well as the influ
47 duced CO2 uptake, yet overaccumulate daytime sucrose and starch.
48  C for 60s results in the degradation of the sucrose and the formation of 5-(hydroxymethyl)-2-furfura
49 ently when one of them (CS+) was paired with sucrose and the other with quinine solution (CS-).
50 the presence/absence of non-reducing sugars (sucrose and trehalose) and polyols (glycerol and sorbito
51 which catalyzes the reversible conversion of sucrose and uridine diphosphate (UDP) into fructose and
52 olytic induced rupture of glucose, fructose, sucrose and vitamin C have been proposed.
53 ith the basic formulation (Jam 1) containing sucrose and without added pectin.
54 ed analysis indicates four pathways (starch, sucrose and xenobiotic metabolism; immune response and i
55 ux1 single and double mutants in response to sucrose and/or N-1-naphthylphthalamic acid (NPA) placed
56 foods high in calories, specifically fat and sucrose, and declining levels of daily physical activity
57 and BIO-samples, mainly decreasing lipid and sucrose, and increasing fructose, glucose and acetaldehy
58 N correlations were maintained with protein, sucrose, and major amino acids but not stored carbon sub
59 l-stimulated synaptic transmission; ethanol, sucrose, and quinine consumption; ethanol-induced loss o
60 1)) in viscous organic liquids (citric acid, sucrose, and shikimic acid) and inorganic gels (magnesiu
61                                        Using sucrose, and sucrose (donor) plus maltose (acceptor) as
62 hat modifying the carrier solution with PBS, sucrose, and/or IPA would enable characterization and se
63 evels from the 2 ALAT genes were analyzed in sucrose- and blood-fed A. aegypti tissues.
64                           Dicarboxylates and sucrose are the main carbon sources within the nodules;
65 diolytic decomposition of glucose, fructose, sucrose, ascorbic acid (H2A) and dehydroascorbic acid (D
66  and are associated with starch-at-maturity, sucrose-at-maturity and NSC-at-heading.
67 in, and exploiting a D-lactate- and mannitol/sucrose-based bioenergetic shunt that greatly minimizes
68 o not show activation of ERK1 in response to sucrose, but notably hemideletion females show elevated
69 centrations.Replacement of either glucose or sucrose by fructose resulted in significantly lowered pe
70 and glucose, released from wheat fructan and sucrose by invertase, compared to maltose is, however, n
71                         Tissues treated in a sucrose-calcium lactate-ascorbic acid solution did not s
72  phase states and phase state transitions of sucrose can be identified and ultimately propose that th
73 A could be used in ratios up to 60% and that sucrose can be used in concentrations up to 0.3 M.
74 to a carbon limitation via the disruption of sucrose cleavage by cell wall invertases in developing o
75  the growth was characterized by fluctuating sucrose concentration along with a constant increase in
76 s a consequence of prior pairing with a high sucrose concentration.
77 dings and raspberry beverages that varied in sucrose concentration.
78 he mean lick cluster size) across a range of sucrose concentrations.
79 mice showed normal satiety responses to high sucrose concentrations.
80 cantly enhanced in Esperase-derived peptides/sucrose conjugates (p<0.05).
81 tion, we identify a taste neuron that limits sucrose consumption in Drosophila.
82  those fed a high-starch diet suggested that sucrose consumption might be associated with elevated le
83 of Medicine that discounted evidence linking sucrose consumption to blood lipid levels and hence coro
84 oride infusions also significantly decreased sucrose consumption, but they did not affect consumption
85 nce, with no similar effects on saccharin or sucrose consumption.
86 ring liking ratings of 3 desserts of varying sucrose content (A: 26%, B: 39%, C: 60%).
87                           Diastase activity, sucrose content and hydroxymethylfurfural content were e
88 lustrate the importance of wheat fructan and sucrose content and their degradation for dough fermenta
89 dg1suc2 double mutant increases glucose plus sucrose content in leaves 80-fold relative to the wild t
90                                          The sucrose content was extremely low during the cycle.
91  an autoregulatory mechanism in perceiving a sucrose-controlled trans activity on one promoter and or
92                                Exposure to a sucrose cue elicited activation of neurons in both the N
93                         Here, we reveal that sucrose cue exposure recruited a more excitable ensemble
94 ges in host metabolism, including apoplastic sucrose degradation and consumption of carbohydrates and
95 resence of PrP(Sc) in fractions of different sucrose densities was indicative of the protein deposits
96 ve electrophoresis, immunoprecipitation, and sucrose density centrifugation.
97                            Enzymatic assays, sucrose density gradient centrifugation, immunoprecipita
98 Deriphat-polyacrylamide gel electrophoresis, sucrose density gradients, and isolated PSII particles,
99 ess hypocotyl elongation in a daylength- and sucrose-dependent manner.
100               Retinas of mice fed a high fat/sucrose diet also exhibited elevated levels of activated
101                          Mice fed a high fat/sucrose diet exhibited attenuated Akt phosphorylation in
102 a key molecular mechanism whereby a high fat/sucrose diet impairs insulin action in retina.
103 n nonhuman primates, a 2-year high-fat, high-sucrose diet increased hepatic mIndy expression.
104 as to evaluate the impact of a high fat/high sucrose diet on retinal insulin signaling and evaluate t
105 ion of excess cholesterol to a high-fat/high-sucrose diet produced greater steatosis in LCR and high
106 ptibility to acute and chronic high-fat/high-sucrose diet-induced steatosis, without observed increas
107  challenged with a lipogenic, high-fat, high-sucrose diet.
108 bolic endotoxemia in rats fed a high fat and sucrose diet.
109 pression of sugar transporters might explain sucrose differences among genotypes, but an unexpected d
110 signals to the circadian oscillator and that sucrose directly affects PIF binding to the promoters of
111                           Using sucrose, and sucrose (donor) plus maltose (acceptor) as substrates, t
112 lants with reduced expression of a tonoplast sucrose efflux transporter, PtaSUT4, exhibit reduced sho
113 tenoid bioaccessibility (17-42%) compared to sucrose ester emulsions (33-52% and 9-27%, respectively)
114 ion coating (CNFC: 0.3% CNF/1% oleic acid/1% sucrose ester fatty acid (w/w wet base)) had low contact
115 e oil (5%w/v) and stabilized with Tween80 or sucrose esters (0.5%w/v) with different hydrophilic-lipo
116                                              Sucrose esters induced an unstable system after gastric
117                                         Five sucrose esters isolated at 100 mug/mL (153.8, 138.8, 136
118                                              Sucrose esters prevented coalescence during 7days since
119               Glucose esters' and commercial sucrose esters' functionality as emulsifiers were compar
120 nation of extended nights, continuous light, sucrose feeding experiments, and photosynthesis inhibiti
121                                              Sucrose feeding, which is required to elicit fatty liver
122            Silencing of the neuron increases sucrose feeding; optogenetic activation decreases it.
123  heating the LMW peptides in the presence of sucrose for 2h at 90 degrees C, without or with US pre-t
124 ructure, water content, texture and Bloom of sucrose free white chocolate was investigated.
125 ontaining a range of carbohydrates (glucose, sucrose, fructose) and nitrogen sources (urea, NH4Cl) at
126             Changes in the concentrations of sucrose, fructose, glucose, amino acids, 3-deoxyglucoson
127   In addition, the signals of carbohydrates (sucrose, glucose and fructose) seemed to play the most i
128 factors, in sensing sucrose, potentially via sucrose/glucose/fructose/trehalose 6-phosphate signaling
129 rs co-sediment in the polysome fraction of a sucrose gradient and do not alter their behaviour with s
130                  Consistent with these data, sucrose gradient centrifugation and indirect immunofluor
131 s with other known virion proteins following sucrose gradient centrifugation.
132                                              Sucrose gradient density centrifugation revealed that Ea
133  the CPC in Xenopus laevis egg cytosol using sucrose gradient sedimentation and in HeLa cells using f
134 was measured by stabilization of caveolin-2, sucrose gradient, and electron microscopy.
135 tosynthetic membranes and fractionation on a sucrose gradient, but these preparations were only stabl
136                           Here, we integrate sucrose-gradient-assisted purification of nuclei with dr
137 d rRNA cofractionate with intact subunits in sucrose gradients and repopulate polysomes after a short
138 ing differential ultracentrifugation through sucrose gradients.
139  sulfate, sodium nitrate, succinic acid, and sucrose) had clearly identifiable features that correlat
140 er factors, such as overfeeding of bees with sucrose, harvesting prior to maturity, and adulteration
141 elta(18) OV into water and assimilates (e.g. sucrose, hexoses, quercitol and starch) in the leaf lami
142 e were fed a control (CD) or a high fat/high sucrose (HF/HS) diet for 4-10 weeks, and then mated.
143 ll expansion in response to a high-fat, high-sucrose (HFHS) diet was significantly impaired in betaTF
144           Here, we show that a high-fat high-sucrose (HFHS) diet, eliciting chronic hepatosteatosis r
145 n regulation of starch breakdown to maintain sucrose homeostasis.
146 inant protein, named BRS-B, mainly catalyzed sucrose hydrolysis and leucrose synthesis.
147                                              Sucrose, identified as the most effective elicitor by pr
148 ts that substituting fructose for glucose or sucrose in food or beverages lowers peak postprandial bl
149  the substitution of fructose for glucose or sucrose in food or beverages may be of benefit to indivi
150 onic, isoenergetic replacement of glucose or sucrose in foods or beverages by fructose.
151 soenergetic replacement of either glucose or sucrose in foods or beverages with fructose.We searched
152 s aim, rats were fed a high fat diet with 5% sucrose in the drinking water (HFS) for 7 months and the
153           We explore the possible origins of sucrose, including guard cell photosynthesis, and discus
154                      We employed data from a sucrose injection experiment in the Bengal Delta Plain t
155 servation that Drosophila flies double their sucrose intake at an early stage of long-term memory for
156 ) strengthened the case that the CHD risk of sucrose is greater than starch and (2) caused sucrose to
157                          Treatment with only sucrose led to a sustained pH drop from 7 to 4.5, while
158 ntiated by increased carbon photoassimilate (sucrose) levels.
159  hedonic orofacial reactions to the taste of sucrose ('liking' reactions).
160  a sucrose/proton symporter that facilitates sucrose loading from leaves into phloem.
161 ng starch and dicarboxylic acids, as well as sucrose, major amino acids, shikimate, and salicylic aci
162            The content of glucose, fructose, sucrose, maltose and water were determined for multiflor
163 ron spin relaxation rates in rigid trehalose/sucrose matrices reveal approximately temperature indepe
164 fructose and UDP-glucose, is a key enzyme in sucrose metabolism in higher plants.
165 ion, secondary metabolites biosynthesis, and sucrose metabolism pathway.
166 k are plant hormone transduction, starch and sucrose metabolism, RNA transport, protein processing in
167  contraction in gene families for starch and sucrose metabolism, the carnivore genomes showed evidenc
168 arge amount of CO2 was detected from glycine-sucrose model system under coffee roasting conditions, i
169  remodeling via complexes such as the switch-sucrose non-fermentable (SWI-SNF) chromatin remodeling c
170  transcription factor SS18:SSX alters SWItch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeling a
171  subunit of the chromatin-remodelling switch/sucrose non-fermentable (SWI/SNF) complex) are susceptib
172 y mechanistic links between KIN10, the major SUCROSE NON-FERMENTATION1-RELATED KINASE1 involved in su
173  through the target of rapamycin complex and sucrose non-fermentative-related kinase-based signaling
174                                              Sucrose non-fermenting 1-related protein kinases (SnRKs)
175 mponents of the chromatin remodelling switch/sucrose nonfermentable (SWI/SNF) complex are recurrently
176 in lymphoma pathogenesis (BCR, Notch, SWitch/sucrose nonfermentable (SWI/SNF), vacuolar ATPases) and
177 th BRG1, the catalytic subunit of the switch/sucrose nonfermentable chromatin-remodeling complex.
178                                              Sucrose nonfermenting related kinase1 (SnRK1) is a conse
179                           SWI/SNF (switching/sucrose nonfermenting)-dependent chromatin remodeling es
180 ned 240 individuals to treatment: 126 to the sucrose octasulfate dressing and 114 to the control dres
181          Three (2%) patients assigned to the sucrose octasulfate dressing and four (4%) assigned to t
182            These findings support the use of sucrose octasulfate dressing as a local treatment for ne
183 closure occurred in 60 patients (48%) in the sucrose octasulfate dressing group and 34 patients (30%)
184 nfections in 25 (20%) patients of 126 in the sucrose octasulfate dressing group and 36 in 32 (28%) pa
185 ere also reported in one (1%) patient in the sucrose octasulfate dressing group and two (2%) patients
186                            INTERPRETATION: A sucrose octasulfate dressing significantly improved woun
187           We aimed to assess the effect of a sucrose octasulfate dressing versus a control dressing o
188  and 5-30 cm(2)), to treatment with either a sucrose octasulfate wound dressing or a control dressing
189  control dressing (the same dressing without sucrose octasulfate) for 20 weeks.
190 energetic fructose replacement of glucose or sucrose on peak postprandial glucose, insulin, and trigl
191 cose administration significantly attenuated sucrose operant responding and D2R activation or blockad
192 ers transiently stopped the mice licking for sucrose or an empty sipper.
193            Acylsugars are composed of either sucrose or glucose esterified with varying numbers of ac
194 ceptor in a reaction catalyzed by SacB using sucrose or levan as fructosyl donor.
195 dium depletion, and in the absence of either sucrose or salt, we found that inhibiting the VP selecti
196 ials of isoenergetic replacement of glucose, sucrose, or both by fructose in adults or children with
197 a after isoenergetic replacement of glucose, sucrose, or both with fructose in healthy adults or chil
198  the time for complete ablation of 193 ng of sucrose particles was found to be approximately 2 s.
199 odologies for substrate deposited individual sucrose particles, Young's modulus and surface tension c
200 the first, rats learned to lever-press for a sucrose-pellet reward.
201 severative and impulsive-like responding for sucrose pellets and sustained reinforcement learning of
202 de reduced the seeking of chocolate-flavored sucrose pellets both pre- and post reward ingestion and
203 cue-controlled seeking of chocolate-flavored sucrose pellets under a second-order schedule of reinfor
204 ng) behavior motivated by chocolate-flavored sucrose pellets were evaluated in non-food-restricted ma
205 uced binge-like intake of chocolate-flavored sucrose pellets without affecting prior chow intake.
206 the first report that kinin analogs modulate sucrose perception in any insect.
207 des containing CpG-rich motifs linked to the sucrose polymer Ficoll, forming soluble 50-nm particles
208 y opposing transcription factors, in sensing sucrose, potentially via sucrose/glucose/fructose/trehal
209 erformed a task wherein competing shock- and sucrose-predictive cues were simultaneously presented.
210  identified anhedonia, manifest as decreased sucrose preference and social play, in adolescent male r
211 ffect observed in cacna1c HET mice using the sucrose preference test (SPT), forced swim test (FST), a
212                                              Sucrose preference was reduced in adolescent CES rats.
213                                              Sucrose preference was tested by measuring liking rating
214 ress and develop social avoidance, decreased sucrose preference, and decreased time in the open arms
215 nhedonia-like phenotype, observed as reduced sucrose preference, in weeks 2-3 of dim light at night,
216  mice were tested in the social interaction, sucrose preference, tail suspension, or elevated plus ma
217 ing control levels of social exploration and sucrose preference.
218 uantified by social interaction behavior and sucrose preference.
219 obesity, explaining 52% of their variance in sucrose preference.
220 th striatal D2R BPND and age correlated with sucrose preferences in subjects without obesity, explain
221                                 We evaluated sucrose preferences, perception of sweetness intensity,
222 t sugars were xylose, arabinose+fructose and sucrose, presenting dried samples with higher contents t
223                            Mosquitoes fed on sucrose primarily accumulate glycogen with increased bra
224  synthesis, and the suc2 mutation disables a sucrose/proton symporter that facilitates sucrose loadin
225                              Total RFOs, RFO/sucrose ratio, but not absolute individual RFO amounts,
226 henols, hydroxymethylfurfural content (HMF), sucrose, reducing and total sugars.
227  a striatal region that critically regulates sucrose reinforcement.
228 cocaine, as it did not impair responding for sucrose reinforcement.
229                                  Potentiated sucrose reinstatement by mGluR2/3 blockade was reversed
230 ively regulated by presynaptic mGluR2/3, and sucrose reinstatement was potentiated following mGluR2/3
231  the acylation of the R2 and R6 positions of sucrose, respectively, and no clear orthologs exist in t
232                                              Sucrose responses of IN1 interneurons in fed flies were
233 ototaxis, orientation to light, is linked to sucrose responsiveness and the transition of work from i
234 s received access to higher- and lower-value sucrose rewards over alternating 30 s periods.
235 atment alters self-administration of food or sucrose rewards.
236       Fucose, galactose, arabinose, glucose, sucrose, rhamnose, xylose, mannose, fructose, and ribose
237 aimed to determine the effect of varying the sucrose, RS, and whey protein content of cereal bars on
238 sequently funded animal research to evaluate sucrose's CHD risks.
239  memory performance in adulthood relative to sucrose SA controls, and performance was associated with
240 ed by sucrose and drugs of abuse, reinstated sucrose seeking does not induce glutamate spillover.
241 ely at mPFC-to-NAc terminals restored normal sucrose seeking in mice after SDe without affecting food
242 ersed by antagonizing mGluR5, but reinstated sucrose seeking in the absence of mGluR2/3 blockade was
243 g in drug seeking and potentiated reinstated sucrose seeking, but that downregulated glutamate transp
244 activating mGluR5 did not promote reinstated sucrose seeking, nor was potentiated reinstatement after
245 ffect locomotor activity or reinstatement to sucrose seeking.
246 sporters (GLT-1) had no effect on reinstated sucrose seeking.
247 f nNOS interneurons in the NAcore reinstated sucrose seeking.
248 cued reinstatement of cocaine seeking versus sucrose seeking.
249 tations and potentiate cued reinstatement of sucrose seeking.
250 striatum, selectively reduced cue-controlled sucrose seeking.
251  high-fat food, cue-induced reinstatement of sucrose-seeking, and motivation to work for sucrose were
252 ent manner to drive cocaine-seeking, but not sucrose-seeking, behavior.
253   It also reduced context-induced relapse to sucrose-seeking, but only at the 2.5 mg/kg dose.
254  acting as a dynamic homeostat responding to sucrose signals to maintain carbon homeostasis.
255  OD of kiwifruit was carried out in 61.5% of sucrose solution at 25 degrees C, for a contact period f
256 ded or not preceded by osmotic pretreatment (sucrose solution concentrations of 40% and 60%).
257                                              Sucrose solution visibly preserved the protoplast viabil
258 k GluA1 (Gria1 (-/-) mice) on consumption of sucrose solutions using a licking microstructure analysi
259 consumption across long periods of access to sucrose solutions were not affected by GluA1 deletion an
260  Gallic acid was used as a model compound in sucrose solutions.
261 wave frequencies for real-time monitoring of sucrose, sorbitol, d-glucose and d-fructose concentratio
262 g microwaves for the real-time monitoring of sucrose, sorbitol, d-glucose and d-fructose concentratio
263 r or better cryoprotection compared to an 8% sucrose-sorbitol blend, and a stabilizing effect of FPH
264 novelty suppressed feeding paradigm, and the sucrose splash test.
265 i, and is now being considered as a possible sucrose substitute due to its pleasant organoleptic prop
266 ed in mammalian cells, MtSWEET11 transported sucrose (Suc) but not glucose (Glc).
267                 We discovered that exogenous sucrose (Suc) in growth medium greatly enhances the redu
268                                              Sucrose (Suc) recovers partially between 14 and 24 h int
269 d at 136 degrees C presented lower amount of sucrose, suggesting the degradation of the carbohydrates
270                                              Sucrose supplementation reactivates both cell division a
271                                              Sucrose synthase (SuS), which catalyzes the reversible c
272 insight into the roles of this motif in rice sucrose synthase 3 (RSuS3), the two conserved glutamate
273 medial shell amplifies the hedonic impact of sucrose taste, but only within the same anatomically ros
274 n stimulations enhance the hedonic impact of sucrose taste.
275 higher rates during the tone associated with sucrose than during the tone associated with quinine del
276 l size, high amino acid contents and reduced sucrose) throughout development, along with a strong alt
277 ucrose is greater than starch and (2) caused sucrose to be scrutinized as a potential carcinogen.
278 nsucrase enzymes of lactic acid bacteria use sucrose to catalyze the synthesis of alpha-glucans with
279 ure and the time lead to a hydrolysis of the sucrose to glucose and fructose.
280 ial species Streptococcus mutans metabolizes sucrose to initiate biofilm formation on the tooth surfa
281                                 We presented sucrose to rats across a range of prices and modeled the
282  mutant phenotypes were rescued by supplying sucrose to the growth medium.
283 he temporary pH decrease associated with the sucrose transformation and mineralization caused pronoun
284 ression of the S gene OsSWEET11, a member of sucrose transporter gene family.
285 e show that GhSWEET10, encoding a functional sucrose transporter, is induced by Avrb6, a TAL effector
286                                          How sucrose transporters (SUTs) regulate phloem unloading in
287 ety of crystalline (sorbitol) and amorphous (sucrose, trehalose) structures, when investigated using
288 n saccharides (glucose, galactose, fructose, sucrose, trehalose, raffinose, and stachyose) at multipl
289 ctose, mannose, rhamnose), 11 disaccharides (sucrose, trehalose, turanose, maltose, maltulose, palati
290                               Similarly, the sucrose used in the growth media as a carbon source was
291  formulations of Jam 2 and 3, while in Jam 4 sucrose was completely replaced by fructose and steviosi
292     Progressive ratio operant responding for sucrose was examined in male and female rats, following
293                                              Sucrose was partially (50%) replaced by stevioside in fo
294 al product, isomelezitose (6(Fru)-alpha-Glcp-sucrose), was also produced when using lactose as an acc
295 ween low molecular weight (LMW) peptides and sucrose, was studied.
296  sucrose-seeking, and motivation to work for sucrose were employed following intra-PVT delivery of ei
297                        Fructose, glucose and sucrose were identified in all the petals and infusions.
298 e (nifH) nodules, particularly low levels of sucrose were observed, suggesting that plant sanctions a
299 uct of fructo-oligosaccharide synthesis from sucrose with fructosyltransferases.
300 ters to the R4, R3, R3', and R2 positions of sucrose, yielding a tetra-acylsucrose.

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