ライフサイエンスコーパス: sudden

1 s conventional care reduced the incidence of sudden and all-cause death.

2 Berry phase-induced spectroscopic feature: a sudden and large increase in the energy of angular-momen

3 c therapies to those at the greatest risk of sudden and nonsudden cardiac death.

4 il the 35(th) day of hospitalization, when a sudden and serious deterioration in the patient's condit

5 Acute biliary pancreatitis is a sudden and severe condition initiated by bile reflux int

6 implementation is feasible or if it reduces sudden and unexpected infant death rates remains to be s

7 n density at the landscape scale rather than sudden and widespread range retraction.

8 and reporting mortality outcomes (all-cause, sudden, any cardiac, or noncardiac) in the primary preve

9 ologists ever since Darwin referred to their sudden appearance in the fossil record as an abominable

10 as self-catalyzed pitting, visualized by the sudden appearance of circular pits with uniform diameter

11 ells need to express proteins to survive the sudden appearance of stressors.

12 apid presentation would reduce salience (the sudden appearance within the visual field) of a stimulus

13 Vinculin (VCL) was linked to sudden arrhythmia death in VCL knockout mice prior to th

14 g the insurability of families affected with Sudden Arrhythmia Death Syndromes (SADS) for the determi

15 the most common SCA-related conditions were sudden arrhythmic death syndrome (31%), coronary artery

16 Sudden arrhythmic death syndrome (SADS) describes a sudd

17 A sudden aversive event produces escape behaviors, an inna

18 denal and lymph nodes metastases developed a sudden bilateral visual acuity impairment and bilateral

19 an abrupt, dramatic shape change due to the sudden breakdown of the erythrocyte cytoskeleton, before

20 Cases were adjudicated as sudden cardiac arrest (i.e., having a cardiac cause) or

21 High-resolution stratification of risk of sudden cardiac arrest (SCA) in individual patients is a

22 Prevention of sudden cardiac arrest (SCA) in the young remains a large

23 Sudden cardiac arrest (SCA) ranks among the most common

24 In contrast, prehospital mortality from sudden cardiac arrest (SCA) remains high and difficult t

25 defibrillator for the primary prevention of sudden cardiac arrest after baseline clinical evaluation

26 ic marker, for example, for prostate cancer, sudden cardiac arrest and neurodegenerative disorders.

27 Sudden cardiac arrest and sudden cardiac death (SCD) are

28 ion, a cohort greatly contributing to global sudden cardiac arrest burden, this marker provides robus

29 The occurrence of sudden cardiac arrest due to structural heart disease wa

30 The incidence of sudden cardiac arrest during competitive sports was 0.76

31 of-hospital cardiac arrest, the incidence of sudden cardiac arrest during participation in competitiv

32 The incidence of sudden cardiac arrest during participation in sports act

33 ation screening programs aimed at preventing sudden cardiac arrest during sports activities are thoug

34 PA2 cause a mitochondrial disease leading to sudden cardiac arrest in infants.

35 CMR identified a likely pathogenesis for sudden cardiac arrest in nearly half of survivors in who

36 nce were followed for the primary outcome of sudden cardiac arrest or appropriate implantable cardiov

37 Determining the pathogenesis of sudden cardiac arrest or periarrest without significant

38 t to evaluate the role of CMR in determining sudden cardiac arrest pathogenesis and prognosis in surv

39 Three cases of sudden cardiac arrest that occurred during participation

40 Sudden cardiac arrest was correlated with a greater volu

41 s (15.6%) experienced the primary outcome (9 sudden cardiac arrest, 40 appropriate implantable cardio

42 rrences of sustained ventricular arrhythmia, sudden cardiac arrest, appropriate defibrillator shock,

43 een associated with increased survival after sudden cardiac arrest.

44 substantial portion of patients experiencing sudden cardiac arrest.

45 ion is a lifesaving technique for victims of sudden cardiac arrest.

46 During 4.1 years of follow-up, there were 33 sudden cardiac arrests (arrhythmic death or implantable

47 18.5 million person-years of observation, 74 sudden cardiac arrests occurred during participation in

48 We sought to identify all sudden cardiac arrests that occurred during participatio

49 strongest predictor for all-cause death and sudden cardiac death (10.09%, 95% CI 4.72-20.42% and 16.

50 Unexplained sudden cardiac death (40% of cases) was the predominant

51 ilure (aHR: 1.95; 95% CI: 1.11 to 3.41), and sudden cardiac death (aHR: 3.75; 95% CI: 1.26 to 11.2) o

52 l, 0.92-0.98; P=0.004) and family history of sudden cardiac death (odds ratio, 3.5; 95% confidence in

53 ibrillators (ICDs) for primary prevention of sudden cardiac death (primary prevention ICDs) in patien

54 Although cardio-vascular incidents and sudden cardiac death (SCD) are among the leading causes

55 Sudden cardiac arrest and sudden cardiac death (SCD) are terms often used intercha

56 The risk of sudden cardiac death (SCD) in patients with heart failur

57 cardiomyopathy for the primary prevention of sudden cardiac death (SCD) in those with a left ventricu

58 Sudden cardiac death (SCD) is a devastating event afflic

59 Sudden cardiac death (SCD) is a major cause of mortality

60 Sudden cardiac death (SCD) is the leading cause of morta

61 Sudden cardiac death (SCD) is the most devastating manif

62 l heart disease carries an elevated risk for sudden cardiac death (SCD), and implantable cardioverter

63 gation of the QT interval is associated with sudden cardiac death (SCD).

64 ament protein leads to an increased risk for sudden cardiac death (SCD).

65 al]) of cardiac death (1.45 [1.24 to 1.69]), sudden cardiac death [1.70 (1.34 to 2.15)], first cardio

66 in reducing risk of all-cause mortality and sudden cardiac death among patients with an EF </=35% at

67 muM) had a 4-fold higher risk of cardiac or sudden cardiac death and a 2-fold higher risk of any-cau

68 myectomy with long-term follow-up, (aborted) sudden cardiac death and mortality rates were found to b

69 Modes of death were divided into sudden cardiac death and nonsudden death and compared be

70 ons for prediction and prevention of SCA and sudden cardiac death and provides justification for a re

71 ations: For this review, we analyzed data on sudden cardiac death and SCA available from population s

72 ter defibrillators for primary prevention of sudden cardiac death are less likely to experience susta

73 nce of sustained ventricular arrhythmias and sudden cardiac death are lower in women than in men.

74 (AF), sustained ventricular arrhythmias, and sudden cardiac death are recognized.

75 s worse compared with other etiologies, with sudden cardiac death as an important mode of death.

76 ecutive families who experienced unexplained sudden cardiac death before 45 years of age were include

77 ular ejection fraction for the prediction of sudden cardiac death events.

78 id not receive defibrillators was related to sudden cardiac death in 8.0% among those with ICM but in

79 tive analysis examined data collected in the Sudden Cardiac Death in Heart Failure Trial (SCD-HeFT),

80 s generally and patients at risk for SCA and sudden cardiac death in particular is limited by the div

81 tor (ICD) is the standard therapy to prevent sudden cardiac death in patients with coronary artery di

82 bility is a well-established risk factor for sudden cardiac death in survivors of acute myocardial in

83 diomyopathy (ARVC) is a significant cause of sudden cardiac death in the young.

84 athy (AC) is a hereditary disease leading to sudden cardiac death or heart failure.

85 t failure (HR, 1.48; 95% CI, 1.43-1.53), and sudden cardiac death or ventricular arrhythmia (HR, 1.65

86 ars, whereas for patients >70 years old, the sudden cardiac death rate was 1.6 (95% CI, 0.8-3.2) and

87 For patients </=70 years old, the sudden cardiac death rate was 1.8 (95% CI, 1.3-2.5) and

88 l decision is whether a particular patient's sudden cardiac death risk is sufficient to justify place

89 Patients with an increased risk of sudden cardiac death undergo cardioverter-defibrillator

90 rillators (ICDs) for secondary prevention of sudden cardiac death were conducted nearly 2 decades ago

91 After sudden cardiac death with negative autopsy, clinical scr

92 g arrhythmic events (LAE) (cardiac arrest or sudden cardiac death) in SQTS patients.

93 fatal and nonfatal myocardial infarction and sudden cardiac death), and harms.

94 n 64 families (230 relatives, 80 unexplained sudden cardiac death).

95 romycin increases risk of cardiovascular and sudden cardiac death, although the underlying mechanisms

96 sease severity expression, high-incidence of sudden cardiac death, and absence of skeletal myopathy o

97 all-cause mortality, heart transplantation, sudden cardiac death, and appropriate implantable cardio

98 pically required due to an elevated risk for sudden cardiac death, and catheter ablation can be used

99 ar tachycardia, syncope, a family history of sudden cardiac death, and severe cardiac hypertrophy are

100 based on an estimate of a patient's risk of sudden cardiac death, as well as their preferences and v

101 recommended at autopsy in suspected cases of sudden cardiac death, but data on the role of nonselecti

102 hERG) ion channel, have been associated with sudden cardiac death, but only a subset of these variant

103 For sudden cardiac death, FHSD, nsVT, and obstruction showed

104 Some patients are at increased risk of sudden cardiac death, heart failure, and atrial fibrilla

105 tes conferred a higher risk of non-CV death, sudden cardiac death, hospitalization, CV hospitalizatio

106 for major arrhythmic events (sustained VAs, sudden cardiac death, or appropriate ICD therapy) in asy

107 d with a history of ventricular arrhythmias, sudden cardiac death, or implantable cardioverter-defibr

108 , sustained ventricular tachycardia, aborted sudden cardiac death, or sudden cardiac death.

109 However, HCM is also an important cause of sudden cardiac death, particularly in adolescents and yo

110 rited heart muscle disorder, predisposing to sudden cardiac death, particularly in young patients and

111 defined as stroke, myocardial infarction, or sudden cardiac death, were assessed prospectively over a

112 S2), a heart rhythm disorder associated with sudden cardiac death.

113 ias, leading to ventricular fibrillation and sudden cardiac death.

114 riggered syncope, aborted cardiac arrest, or sudden cardiac death.

115 ricular tachycardia (VT) is a major cause of sudden cardiac death.

116 table cardioverter-defibrillator shocks, and sudden cardiac death.

117 n HFpEF, which in turn predisposes to VA and sudden cardiac death.

118 All-cause mortality and sudden cardiac death.

119 ricular arrhythmias and an increased risk of sudden cardiac death.

120 e been linked to ventricular arrhythmias and sudden cardiac death.

121 achycardia, aborted sudden cardiac death, or sudden cardiac death.

122 ical interventions with potential to prevent sudden cardiac death.

123 osis (CS) may manifest as arrhythmia or even sudden cardiac death.

124 -myocyte junction and are a leading cause of sudden cardiac death.

125 ading to impaired mechanical contraction and sudden cardiac death.

126 tor (ICD) in the treatment and prevention of sudden cardiac death.

127 rdiac hypertrophy are major risk factors for sudden cardiac death.

128 n associated with delayed repolarization and sudden cardiac death.

129 rs are indicated for prevention of secondary sudden cardiac death.

130 of lesions lead to heart attack, stroke, or sudden cardiac death.

131 main goal of treatment is the prevention of sudden cardiac death.

132 cturally normal heart carries a low risk for sudden cardiac death; accordingly, there is typically no

133 observe a dynamical phase transition after a sudden change of the Hamiltonian, in a regime in which c

134 ameters, including allergen release time and sudden change rate, to evaluate the potential and streng

135 on these "epiphanies" has shown evidence of sudden changes in behavior, but it remains unclear how s

136 ted the effect on soil CO2 efflux (FCO2 ) of sudden changes in photosynthetic rates by altering CO2 c

137 Although spiral scanning avoids the sudden changes in the beam location (fly-back distortion

138 y to self-heal to perturbations and adapt to sudden changes in the drive.

139 ge, NYHA functional class, family history of sudden death (FHSD), syncope, atrial fibrillation, non-s

140 e interval, 1.6-9.7) and a family history of sudden death (odds ratio, 3.2; 95% confidence interval,

141 with CTCAE terms: disease progression [n=3], sudden death [n=1], and not specified [n=1]).

142 Although sudden death accounted for approximately 25% to 30% of d

143 There was a 44% decline in the rate of sudden death across the trials (P=0.03).

144 was driven by reductions in out-of-hospital sudden death and hospitalized ST-segment-elevation myoca

145 ticipatory planning for therapies to prevent sudden death and manage HF, is warranted.

146 nterpreting mtDNA variants in the context of sudden death and rare diseases.

147 al aneurysms are at high risk for arrhythmic sudden death and thromboembolic events.

148 One death was cardiac sudden death and two were due to sepsis.

149 Prior syncope and family history of sudden death are predictors of a positive genetic test.

150 The cumulative incidence of sudden death at 90 days after randomization was 2.4% in

151 </=5.7%, those with a SPRM-predicted risk of sudden death below the median had no reduction in mortal

152 Rates of sudden death declined substantially over time among ambu

153 d worse cardiac function, than those in whom sudden death did not occur.

154 The sudden death event rate was 4.7%/year.

155 The rate of sudden death fell from 12.15% with conventional care to

156 EG suppression, cardiorespiratory arrest and sudden death following a seizure.

157 ttributed to coronary heart disease, stroke, sudden death from an unknown cause, or a combination of

158 The risk of sudden death has changed over time among patients with s

159 gical connection between epilepsy itself and sudden death have fuelled increased attention to this ph

160 etes mellitus, and (2) all end points except sudden death in a model including all baseline character

161 cular tachycardia (CPVT), a leading cause of sudden death in apparently healthy individuals exposed t

162 uch sources as the U.S. National Registry of Sudden Death in Athletes (which uses news media, Interne

163 ROS contribute to sudden death in diabetics after myocardial infarction by

164 Adjusted hazard ratios for sudden death in each trial group were calculated with th

165 ibrillators (ICDs) for primary prevention of sudden death in patients with an ejection fraction (EF)

166 ting the increased risk for exertion-related sudden death in SCT carriers is unlikely related to fitn

167 represents the latest molecular autopsy for sudden death in the young (SDY).

168 is of clinical importance given the risk of sudden death in these patients, but so far contradictory

169 common cardiomyopathies and a major cause of sudden death in young athletes.

170 en without autopsy, familial screening after sudden death in young patients is effective.

171 ects the fact that ARVC is a common cause of sudden death in young people and that sudden death may b

172 Methods to predict a higher or lower risk of sudden death include the detection of myocardial fibrosi

173 Sudden death is rare.

174 As measurement of LVWT impacts diagnosis and sudden death management, CMR should be considered as par

175 use of sudden death in young people and that sudden death may be the first manifestation of the disea

176 teral prefrontal cortex (Brodmann Area 9) of sudden death medication-free individuals post mortem.

177 ssion was used to examine trends in rates of sudden death over time.

178 ed a longer mitral annulus disjunction in 50 sudden death patients with MVP and LV fibrosis than in 2

179 orphology index has the potential to improve sudden death risk stratification and patient selection f

180 liforme is a soil borne pathogen that causes sudden death syndrome (SDS) in soybean plants.

181 ulation in Fusarium virguliforme that causes sudden death syndrome in soybean.

182 to F. virguliforme, the pathogen that causes sudden death syndrome.

183 nd 39 females) with >/=1 HCM risk factor for sudden death underwent S-ICD ECG screening at rest and o

184 The rate of sudden death was not higher among patients with a recent

185 Sudden death was reported in 3583 patients.

186 The cumulative incidence rates of sudden death were assessed at different time points afte

187 autopsy studies report an associated risk of sudden death with interarterial anomalous left coronary

188 arrhythmic death syndrome (SADS) describes a sudden death with negative autopsy and toxicological ana

189 neous disorders with the common phenotype of sudden death without explanation upon postmortem investi

190 th from stroke, 2.1 (95% CI, 1.5 to 2.9) for sudden death, and 3.5 (95% CI, 2.9 to 4.1) for death fro

191 at baseline and (2) the incidence of death, sudden death, and other cardiac adverse events.

192 gnificantly associated with (1) total death, sudden death, and pacemaker implantation in a model, inc

193 The leading cause of mortality in HFpEF is sudden death, but little is known about the underlying m

194 alth-care expenditure as a result of stroke, sudden death, heart failure, unplanned hospital admissio

195 l Risk Model (SPRM) for proportional risk of sudden death, including death from ventricular arrhythmi

196 Cardiomyopathy patients are at risk of sudden death, typically from scar-related abnormalities

197 function of hERG causes long QT syndrome and sudden death, which occur in patients with cardiac ische

198 etion of Slc8b1 in adult mouse hearts causes sudden death, with less than 13% of affected mice surviv

199 d by variants found in genes associated with sudden death-predisposing catecholaminergic polymorphic

200 Overall, 17 sudden death-susceptibility gene variants were identifie

201 epth of 10x across 90% of nucleotides within sudden death-susceptibility genes in 100% of parental ex

202 s of ultrarare, nonsynonymous variants in 99 sudden death-susceptibility genes.

203 ed susceptibility to cardiac arrhythmias and sudden death.

204 that SCT is associated with exertion-related sudden death.

205 entricular arrhythmias and can predispose to sudden death.

206 odels to identify patients at higher risk of sudden death.

207 f internal cardiac defibrillators to prevent sudden death.

208 enotypes, including spontaneous seizures and sudden death.

209 nges modeling bipolar disorder, epilepsy and sudden death.

210 CI) increases the risk of cardiomyopathy and sudden death.

211 t failure morbidity or a cause of arrhythmic sudden death; when treated, it is associated with low di

212 recommended programming accounted for 56% of sudden deaths and 11% of all deaths during the study per

213 Most sudden deaths and cardiac arrests occurred in the swim s

214 Most sudden deaths in triathletes happened during the swim se

215 Sudden deaths that occurred out of hospital accounted fo

216 type 1 patients is associated with total and sudden deaths, conduction defects, left ventricular dysf

217 A total of 135 sudden deaths, resuscitated cardiac arrests, and trauma-

218 ynamics of exponential growth interrupted by sudden declines.

219 neous group of conditions characterized by a sudden decrease in glomerular filtration rate, manifeste

220 stributions, ranging from gradual buildup to sudden delivery, based only on the channel dimensions.

221 l support for the immunization programs, and sudden discontinuation of their employment would potenti

222 ults indicate that exploration is related to sudden disruptions in prefrontal sensorimotor control an

223 n the waist of the balloon released and by a sudden drop in inflation pressure, often accompanied by

224 ies interactions to drive or protect against sudden ecosystem shifts.

225 han the entire A1/D1 East cluster caused the sudden emergence of Verticillium stem striping in the UK

226 researchers invoke such external factors as sudden environmental change, rapid cognitive or morpholo

227 y may tune the cell's capacity to respond to sudden environmental perturbations.

228 in England and Wales to identify periods of sudden escalation of scarlet fever.

229 at P1 to 401.4 +/- 80.6 hours at P43, with a sudden extension between P15 and P22.

230 ss response proteins given environments with sudden fluctuations between low and high stress.

231 re a more direct mechanism that may describe sudden forest degradation in the south-eastern Amazon.

232 An appropriately timed sequence of sudden frequency changes reduces the quantum noise fluct

233 but follow each other rapidly, separated by sudden gaze shifts.

234 to permit long-term survival in response to sudden glucose depletion, yeast cells activate lipid-dro

235 y starving cells to quickly meet demand upon sudden improvement in growth conditions.

236 Here we show a sudden increase in summer temperature at 6,000 cal.

237 quilibration phase of the system, leading to sudden increase of fluid pressure in the process zone.

238 emperature, the sharp drop in V LSSE and the sudden increases in H K and H KS at 175 K are associate

239 shapes the WSS changes rapidly resulting in sudden increases in leukocyte TEM suggesting lumen volum

240 Many sudden infant death syndrome (SIDS) cases exhibit a part

241 Sudden infant death syndrome (SIDS), the leading cause o

242 nicotinic exposure (PNE), is responsible for sudden infant death syndrome (SIDS).

243 ervations: While its exact cause is unknown, sudden infant death syndrome is believed to be multifact

244 Hazard ratios for total, respiratory, and sudden infant death syndrome mortality per-interquartile

245 endent covariates to total, respiratory, and sudden infant death syndrome mortality.

246 Sudden infant death syndrome remains the leading cause o

247 ty present in a variety of disorders such as sudden infant death syndrome, depression, and anxiety.

248 ing of the physiologic mechanisms that cause sudden infant death, the mainstay of risk reduction cont

249 consequences in newborn babies and result in sudden infant death.

250 y occurs gradually with time with occasional sudden jumps in frequency.

251 ainly corresponds to force ramps followed by sudden jumps.

252 w do astrocyte-derived neurons deal with the sudden loss of their glial identity?

253 r that this decision appears to be made in a sudden moment of "insight."

254 cterized by chronic sleepiness and cataplexy-sudden muscle paralysis triggered by strong, positive em

255 INTERPRETATION: For most sudden natural adult deaths investigated by HM Coroners,

256 94I was found in a SUNDS victim who suffered sudden nocturnal tachypnea and lacked pathogenic variant

257 ients who develop constitutional symptoms or sudden onset chest pain that start days or weeks after a

258 CASE PRESENTATION: A 34-year-old man had sudden onset of central scotoma and photopsia in the lef

259 se of premature infants characterized by the sudden onset of intestinal necrosis.

260 All patients presented with a sudden onset of multiple lesions on sun-exposed areas of

261 benign fasciculation syndrome, even when of sudden onset.

262 revious stent insertion who presented with a sudden - onset severe abdominal pain.

263 ey process of living organisms to counteract sudden osmotic changes.

264 venth post-operative-day, she presented with sudden painful, visual loss.

265 He complained of sudden painless profound visual loss in the left eye (LE

266 nonpolar molecule upon photoexcitation (the sudden polarization effect) earlier discussed for 90 deg

267 Hemisphere, with many changes ascribed to a sudden poleward shift in the Southern Hemisphere westerl

268 The cavitation domain nucleates due to sudden pressure drop upon flow past a cylindrical obstac

269 ermination of spot machines as a result of a sudden price increase.

270 ng point" at which complex systems undergo a sudden qualitative shift.

271 echanism, known as deformation twinning (the sudden re-orientation of the crystal lattice), takes ove

272 of majority spins at the interface causes a sudden, reversible transition of the spin alignment of i

273 e variety of hearing-loss disorders, such as sudden sensorineural hearing loss and Meniere's disease

274 sitivity in lipopolysaccharide (LPS)-induced sudden sensorineural hearing loss in guinea pigs.

275 avioral and autonomic hyperresponsiveness to sudden sounds in PTSD is associated with locus coeruleus

276 in-screw extruder that had been brought to a sudden stop and molecular weights were determined by int

277 accurate, reflex-like pathway are fitter in sudden stress, whereas cells with only the slow, more ac

278 ge amounts of ubiquitin needed to respond to sudden stress.

279 gent of Chagas cardiomyopathy, we observed a sudden switch from the classical M1 macrophage (microbic

280 as it provides an efficient solution to the sudden termination of spot machines as a result of a sud

281 r a rapid quantum quench that results from a sudden transition across the phase boundary.

282 On the other hand, sudden transition from coarse-to-fine grain sizes promot

283 hallmark of narcolepsy characterized by the sudden uncontrollable onset of muscle weakness or paraly

284 the risk of social instability, conflict and sudden, uncontrolled population migrations triggered by

285 or a significant portion of autopsy-negative sudden unexpected death (SUD) cases, but molecular autop

286 We also discovered spontaneous sudden unexpected death in epilepsy (SUDEP) in two of ou

287 Sudden unexpected death in epilepsy (SUDEP) is the most

288 evelopmental delays and are at high risk for sudden unexpected death in epilepsy (SUDEP).

289 ed probability of premature mortality due to sudden unexpected death in epilepsy (SUDEP).

290 , cognitive disability, and elevated risk of sudden unexpected death in epilepsy (SUDEP).

291 intractable seizures, and the potential for sudden unexpected death in epilepsy (SUDEP).

292 ive impairment and are at increased risk for sudden unexpected death in epilepsy (SUDEP).

293 Sudden unexpected death in epilepsy is a very rare outco

294 recommendations known to reduce the risk of sudden unexpected infant death has contributed to a slow

295 hy genes in a large, demographically diverse sudden unexplained death cohort that underwent thorough

296 ld was similar for family members of UCA and sudden unexplained death victims (31% versus 27%; P=0.59

297 es to testing yield differences reported for sudden unexplained death.

298 evere neurodevelopmental disorder, including sudden unexplained deaths and audiogenic seizures.

299 Sudden unexplained nocturnal death syndrome (SUNDS) rema

300 ack typically regain it by auditory scanning-sudden, wide deviations in their heading-and females ini