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1 resent a new, potentially treatable cause of sudden infant death.
2 der can have serious consequences, including sudden infant death.
3  failure to thrive, behavioral deficits, and sudden infant death.
4 consequences in newborn babies and result in sudden infant death.
5                                          The sudden infant death/ALTE families had a greater frequenc
6            Recurrent late-term fetal loss or sudden infant death can result from unsuspected parental
7 entified a lethal phenotype characterized by sudden infant death (from cardiac and respiratory arrest
8 ychological effects on the parents following sudden infant death is discussed and reveals maternal an
9                            After an event of sudden infant death, many parents moved without a forwar
10 mine the most recent published literature on Sudden Infant Death, Sudden Unexplained Infant Death, in
11 ation-based cohort of 221 anonymous cases of sudden infant death syndrome (84 females, 137 males; ave
12  death certificates/autopsy reports included sudden infant death syndrome (n = 544 [44%]), asphyxia (
13 the birth of an infant that had succumbed to sudden infant death syndrome (SIDS) (and no other cause
14                      The association between sudden infant death syndrome (SIDS) and maternal smoking
15 Abnormalities in HMs have been implicated in sudden infant death syndrome (SIDS) and obstructive slee
16 ernal smoking is the highest risk factor for sudden infant death syndrome (SIDS) and prenatal nicotin
17  There is growth in our understanding of how sudden infant death syndrome (SIDS) and the symptom comp
18                                         Many sudden infant death syndrome (SIDS) cases exhibit a part
19                   An estimated 10% to 15% of sudden infant death syndrome (SIDS) cases may stem from
20                                              Sudden infant death syndrome (SIDS) cases often have abn
21 s in prevalence during the past two decades, sudden infant death syndrome (SIDS) continues to be the
22 isk reduction campaigns have been conducted, sudden infant death syndrome (SIDS) has become increasin
23 ast 5 years suggest that the epidemiology of sudden infant death syndrome (SIDS) has changed since th
24 between infant sleeping position and risk of sudden infant death syndrome (SIDS) in an ethnically div
25                                              Sudden infant death syndrome (SIDS) is a leading cause o
26              The likelihood of recurrence of sudden infant death syndrome (SIDS) is an issue of biolo
27                                              Sudden infant death syndrome (SIDS) is postulated to res
28                                          The sudden infant death syndrome (SIDS) is the sudden death
29                     The recent US decline in sudden infant death syndrome (SIDS) rates may be explain
30 reported here support a recent proposal that sudden infant death syndrome (SIDS) results from a devel
31               Unexplained stillbirth and the sudden infant death syndrome (SIDS) share some features.
32       To examine recent research relevant to sudden infant death syndrome (SIDS) to determine whether
33 inding in the medullae of infants dying from sudden infant death syndrome (SIDS) were identified, sug
34 tion is associated with an increased risk of sudden infant death syndrome (SIDS), but few studies hav
35 uently used in infants at increased risk for sudden infant death syndrome (SIDS), but the efficacy of
36 levant to understanding the aetiology of the sudden infant death syndrome (SIDS), in which there is m
37            This has special implications for sudden infant death syndrome (SIDS), insofar as seemingl
38 erozygous state in two infants who died from sudden infant death syndrome (SIDS), one with documented
39 ancy predisposes an infant to a high risk of sudden infant death syndrome (SIDS), the authors conduct
40                                              Sudden infant death syndrome (SIDS), the leading cause o
41 he prone sleep position (on the stomach) and sudden infant death syndrome (SIDS).
42 eritis, necrotizing enterocolitis (NEC), and sudden infant death syndrome (SIDS).
43 cans is associated with an increased risk of sudden infant death syndrome (SIDS).
44 nicotinic exposure (PNE), is responsible for sudden infant death syndrome (SIDS).
45                 Thousands die each year from sudden infant death syndrome (SIDS).
46 lateralis (PGCL), in many infants who die of sudden infant death syndrome (SIDS).
47 ne exposure are the highest risk factors for sudden infant death syndrome (SIDS).
48 linically associated with cardiomyopathy and sudden infant death syndrome (SIDS).
49 yndrome may be responsible for some cases of sudden infant death syndrome (SIDS).
50 normalities are present in many cases of the sudden infant death syndrome (SIDS).Mice with a targeted
51 targets for disorders such as sleep apnea or sudden infant death syndrome and for regulating uterine
52 t there is often a fine line differentiating sudden infant death syndrome and infanticide.
53 characterized SCN5A variants associated with sudden infant death syndrome and provide further biophys
54 sproportionately affect males, including the sudden infant death syndrome and sleep apnea.
55 s recommend against it to reduce the risk of sudden infant death syndrome and suffocation deaths.
56 rom congenital abnormalities, accidents, and sudden infant death syndrome are predicted to continue i
57                                We classified sudden infant death syndrome as a separate cause of deat
58 en aged 12 months or younger; most (52%) had sudden infant death syndrome as the reported cause of de
59 ut the evaluation and prevention of possible Sudden Infant Death Syndrome cases were published this y
60 estions and an apparent relationship to some sudden infant death syndrome cases.
61  near drowning, respiratory arrest, and near sudden infant death syndrome cause significant mortality
62 CN5A variants were identified in a Norwegian sudden infant death syndrome cohort (n=201).
63                                              Sudden infant death syndrome contributed to 5% of deaths
64 demy of Pediatrics in 1992, the incidence of Sudden infant death syndrome has decreased by almost 50%
65 esting in a large population-based cohort of sudden infant death syndrome has elucidated mutations in
66 nsistent with the observed 50% male bias for Sudden Infant Death Syndrome in humans.
67 igns, there has been a dramatic reduction in sudden infant death syndrome in this country.
68 4 sources were used to locate 230 parents of sudden infant death syndrome infants who died in Souther
69 or a standardized and systematic approach to sudden infant death syndrome is also reviewed.
70 ervations: While its exact cause is unknown, sudden infant death syndrome is believed to be multifact
71                    The current literature on sudden infant death syndrome is reviewed in this section
72 inding strongly supports the hypothesis that sudden infant death syndrome is the result of dysregulat
73    Hazard ratios for total, respiratory, and sudden infant death syndrome mortality per-interquartile
74 endent covariates to total, respiratory, and sudden infant death syndrome mortality.
75 g association between infant bed sharing and sudden infant death syndrome or unintentional sleep-rela
76        Controversy regarding the etiology of sudden infant death syndrome persists, and risk factors
77 Academy of Pediatrics new recommendations on sudden infant death syndrome prevention.
78  national "Back to Sleep" campaign to reduce sudden infant death syndrome provides an opportunity to
79                            Given the risk of sudden infant death syndrome related to bedsharing, mult
80                                              Sudden infant death syndrome remains the leading cause o
81         Despite a putative diagnostic shift, sudden infant death syndrome remains the most common cau
82 between apparent life-threatening events and sudden infant death syndrome remains to be explored furt
83                                              Sudden infant death syndrome reports after IPV were cons
84  as a pathogenic cause for a small subset of sudden infant death syndrome via a secondary loss-of-fun
85 ciation between meteorologic temperature and sudden infant death syndrome was investigated in the 198
86 between Apgar score at 5 min and the risk of sudden infant death syndrome was noted at any gestationa
87 quiries identified 18 families with two SIDS(sudden infant death syndrome) deaths and two families wi
88 p position continues to be a risk factor for sudden infant death syndrome, although immunizations may
89 ematurity, apparent life-threatening events, sudden infant death syndrome, and central hypoventilatio
90 bute to sudden unexpected death in epilepsy, sudden infant death syndrome, and sleep apnea.
91 negative sudden unexplained death, including sudden infant death syndrome, can be caused by cardiac c
92 ty present in a variety of disorders such as sudden infant death syndrome, depression, and anxiety.
93 omyopathy, and now encompasses more cases of sudden infant death syndrome, fulminant hepatic failure,
94 factors thought to underlie the aetiology of sudden infant death syndrome, including: (1) a vulnerabl
95 and preventable-cause mortality in children (sudden infant death syndrome, unintentional injury, and
96 jor risk factor for late fetal death and the sudden infant death syndrome, we investigated cardioresp
97 dings provide insight into the mechanisms of sudden infant death syndrome, which has been associated
98  prolonged QT electrocardiogram interval and sudden infant death syndrome.
99 n the greatest influence on the reduction of sudden infant death syndrome.
100 rts to find other modifiable risk factors of sudden infant death syndrome.
101 explain the relationship between smoking and sudden infant death syndrome.
102  prolonged QT electrocardiogram interval and sudden infant death syndrome.
103 low heart rates and cardiac function such as sudden infant death syndrome.
104 e patients who are labeled as having died of sudden infant death syndrome.
105 as been associated with an increased risk of sudden infant death syndrome.
106 onin abnormalities thought to be involved in sudden infant death syndrome.
107 with histo-pathological findings reported in sudden infant death syndrome.
108 s, apneas of prematurity, Rett syndrome, and sudden infant death syndrome.
109 nd most recently long-QT syndrome (LQTS) and sudden infant death syndrome.
110 failure phenotype in mice, with relevance to Sudden Infant Death Syndrome.
111 defective AR capacity has been implicated in Sudden Infant Death Syndrome.
112 hood manifestations include fetal hydrops or sudden infant death syndrome.
113 SCN5A, have been identified in some cases of sudden infant death syndrome.
114 e for some cases of sudden unexplained death/sudden infant death syndrome.
115  syndrome, familial atrial fibrillation, and sudden infant death syndrome.
116 erlying pathophysiology and risk factors for sudden infant death syndrome.
117 eping is one of the biggest risk factors for sudden infant death syndrome.
118 r factors that may contribute to the risk of Sudden Infant Death Syndrome.
119 ediatric 5-HT brainstem disorders, including sudden infant death syndrome.
120 between KCNH2 gain of function mutations and sudden infant death syndrome.
121 moke is associated with an increased risk of sudden infant death syndrome.
122 , in disturbances culminating in events like Sudden Infant Death Syndrome.
123 tory symptoms in children and be a factor in sudden infant death syndrome.
124 effectiveness of these campaigns in reducing sudden infant death syndrome.
125 ing of the physiologic mechanisms that cause sudden infant death, the mainstay of risk reduction cont
126 ptibility loci for a rare monogenic disease (sudden infant death with dysgenesis of the testes syndro
127                  The possibility of a second sudden infant death within a family is discussed.

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