ライフサイエンスコーパス: sudden infant death syndrome

1 onin abnormalities thought to be involved in sudden infant death syndrome.

2 low heart rates and cardiac function such as sudden infant death syndrome.

3 e patients who are labeled as having died of sudden infant death syndrome.

4 as been associated with an increased risk of sudden infant death syndrome.

5 with histo-pathological findings reported in sudden infant death syndrome.

6 s, apneas of prematurity, Rett syndrome, and sudden infant death syndrome.

7 nd most recently long-QT syndrome (LQTS) and sudden infant death syndrome.

8 failure phenotype in mice, with relevance to Sudden Infant Death Syndrome.

9 defective AR capacity has been implicated in Sudden Infant Death Syndrome.

10 hood manifestations include fetal hydrops or sudden infant death syndrome.

11 SCN5A, have been identified in some cases of sudden infant death syndrome.

12 e for some cases of sudden unexplained death/sudden infant death syndrome.

13 syndrome, familial atrial fibrillation, and sudden infant death syndrome.

14 erlying pathophysiology and risk factors for sudden infant death syndrome.

15 eping is one of the biggest risk factors for sudden infant death syndrome.

16 r factors that may contribute to the risk of Sudden Infant Death Syndrome.

17 ediatric 5-HT brainstem disorders, including sudden infant death syndrome.

18 between KCNH2 gain of function mutations and sudden infant death syndrome.

19 moke is associated with an increased risk of sudden infant death syndrome.

20 , in disturbances culminating in events like Sudden Infant Death Syndrome.

21 tory symptoms in children and be a factor in sudden infant death syndrome.

22 effectiveness of these campaigns in reducing sudden infant death syndrome.

23 prolonged QT electrocardiogram interval and sudden infant death syndrome.

24 n the greatest influence on the reduction of sudden infant death syndrome.

25 rts to find other modifiable risk factors of sudden infant death syndrome.

26 explain the relationship between smoking and sudden infant death syndrome.

27 prolonged QT electrocardiogram interval and sudden infant death syndrome.

28 ation-based cohort of 221 anonymous cases of sudden infant death syndrome (84 females, 137 males; ave

29 p position continues to be a risk factor for sudden infant death syndrome, although immunizations may

30 targets for disorders such as sleep apnea or sudden infant death syndrome and for regulating uterine

31 t there is often a fine line differentiating sudden infant death syndrome and infanticide.

32 characterized SCN5A variants associated with sudden infant death syndrome and provide further biophys

33 sproportionately affect males, including the sudden infant death syndrome and sleep apnea.

34 s recommend against it to reduce the risk of sudden infant death syndrome and suffocation deaths.

35 ematurity, apparent life-threatening events, sudden infant death syndrome, and central hypoventilatio

36 bute to sudden unexpected death in epilepsy, sudden infant death syndrome, and sleep apnea.

37 rom congenital abnormalities, accidents, and sudden infant death syndrome are predicted to continue i

38 We classified sudden infant death syndrome as a separate cause of deat

39 en aged 12 months or younger; most (52%) had sudden infant death syndrome as the reported cause of de

40 negative sudden unexplained death, including sudden infant death syndrome, can be caused by cardiac c

41 ut the evaluation and prevention of possible Sudden Infant Death Syndrome cases were published this y

42 estions and an apparent relationship to some sudden infant death syndrome cases.

43 near drowning, respiratory arrest, and near sudden infant death syndrome cause significant mortality

44 CN5A variants were identified in a Norwegian sudden infant death syndrome cohort (n=201).

45 Sudden infant death syndrome contributed to 5% of deaths

46 quiries identified 18 families with two SIDS(sudden infant death syndrome) deaths and two families wi

47 ty present in a variety of disorders such as sudden infant death syndrome, depression, and anxiety.

48 omyopathy, and now encompasses more cases of sudden infant death syndrome, fulminant hepatic failure,

49 demy of Pediatrics in 1992, the incidence of Sudden infant death syndrome has decreased by almost 50%

50 esting in a large population-based cohort of sudden infant death syndrome has elucidated mutations in

51 nsistent with the observed 50% male bias for Sudden Infant Death Syndrome in humans.

52 igns, there has been a dramatic reduction in sudden infant death syndrome in this country.

53 factors thought to underlie the aetiology of sudden infant death syndrome, including: (1) a vulnerabl

54 4 sources were used to locate 230 parents of sudden infant death syndrome infants who died in Souther

55 or a standardized and systematic approach to sudden infant death syndrome is also reviewed.

56 ervations: While its exact cause is unknown, sudden infant death syndrome is believed to be multifact

57 The current literature on sudden infant death syndrome is reviewed in this section

58 inding strongly supports the hypothesis that sudden infant death syndrome is the result of dysregulat

59 Hazard ratios for total, respiratory, and sudden infant death syndrome mortality per-interquartile

60 endent covariates to total, respiratory, and sudden infant death syndrome mortality.

61 death certificates/autopsy reports included sudden infant death syndrome (n = 544 [44%]), asphyxia (

62 g association between infant bed sharing and sudden infant death syndrome or unintentional sleep-rela

63 Controversy regarding the etiology of sudden infant death syndrome persists, and risk factors

64 Academy of Pediatrics new recommendations on sudden infant death syndrome prevention.

65 national "Back to Sleep" campaign to reduce sudden infant death syndrome provides an opportunity to

66 Given the risk of sudden infant death syndrome related to bedsharing, mult

67 Sudden infant death syndrome remains the leading cause o

68 Despite a putative diagnostic shift, sudden infant death syndrome remains the most common cau

69 between apparent life-threatening events and sudden infant death syndrome remains to be explored furt

70 Sudden infant death syndrome reports after IPV were cons

71 the birth of an infant that had succumbed to sudden infant death syndrome (SIDS) (and no other cause

72 The association between sudden infant death syndrome (SIDS) and maternal smoking

73 Abnormalities in HMs have been implicated in sudden infant death syndrome (SIDS) and obstructive slee

74 ernal smoking is the highest risk factor for sudden infant death syndrome (SIDS) and prenatal nicotin

75 There is growth in our understanding of how sudden infant death syndrome (SIDS) and the symptom comp

76 Many sudden infant death syndrome (SIDS) cases exhibit a part

77 An estimated 10% to 15% of sudden infant death syndrome (SIDS) cases may stem from

78 Sudden infant death syndrome (SIDS) cases often have abn

79 s in prevalence during the past two decades, sudden infant death syndrome (SIDS) continues to be the

80 isk reduction campaigns have been conducted, sudden infant death syndrome (SIDS) has become increasin

81 ast 5 years suggest that the epidemiology of sudden infant death syndrome (SIDS) has changed since th

82 between infant sleeping position and risk of sudden infant death syndrome (SIDS) in an ethnically div

83 Sudden infant death syndrome (SIDS) is a leading cause o

84 The likelihood of recurrence of sudden infant death syndrome (SIDS) is an issue of biolo

85 Sudden infant death syndrome (SIDS) is postulated to res

86 The sudden infant death syndrome (SIDS) is the sudden death

87 The recent US decline in sudden infant death syndrome (SIDS) rates may be explain

88 reported here support a recent proposal that sudden infant death syndrome (SIDS) results from a devel

89 Unexplained stillbirth and the sudden infant death syndrome (SIDS) share some features.

90 To examine recent research relevant to sudden infant death syndrome (SIDS) to determine whether

91 inding in the medullae of infants dying from sudden infant death syndrome (SIDS) were identified, sug

92 tion is associated with an increased risk of sudden infant death syndrome (SIDS), but few studies hav

93 uently used in infants at increased risk for sudden infant death syndrome (SIDS), but the efficacy of

94 levant to understanding the aetiology of the sudden infant death syndrome (SIDS), in which there is m

95 This has special implications for sudden infant death syndrome (SIDS), insofar as seemingl

96 erozygous state in two infants who died from sudden infant death syndrome (SIDS), one with documented

97 ancy predisposes an infant to a high risk of sudden infant death syndrome (SIDS), the authors conduct

98 Sudden infant death syndrome (SIDS), the leading cause o

99 he prone sleep position (on the stomach) and sudden infant death syndrome (SIDS).

100 eritis, necrotizing enterocolitis (NEC), and sudden infant death syndrome (SIDS).

101 nicotinic exposure (PNE), is responsible for sudden infant death syndrome (SIDS).

102 cans is associated with an increased risk of sudden infant death syndrome (SIDS).

103 Thousands die each year from sudden infant death syndrome (SIDS).

104 lateralis (PGCL), in many infants who die of sudden infant death syndrome (SIDS).

105 ne exposure are the highest risk factors for sudden infant death syndrome (SIDS).

106 linically associated with cardiomyopathy and sudden infant death syndrome (SIDS).

107 yndrome may be responsible for some cases of sudden infant death syndrome (SIDS).

108 normalities are present in many cases of the sudden infant death syndrome (SIDS).Mice with a targeted

109 and preventable-cause mortality in children (sudden infant death syndrome, unintentional injury, and

110 as a pathogenic cause for a small subset of sudden infant death syndrome via a secondary loss-of-fun

111 ciation between meteorologic temperature and sudden infant death syndrome was investigated in the 198

112 between Apgar score at 5 min and the risk of sudden infant death syndrome was noted at any gestationa

113 jor risk factor for late fetal death and the sudden infant death syndrome, we investigated cardioresp

114 dings provide insight into the mechanisms of sudden infant death syndrome, which has been associated