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1 dentified only CPS and PG sugars in the appropriate ratios, suggesting the absence of an oligosaccharide linker.
2 f mRNAs in the control group but not in the exposed groups, suggesting the disruption of miRNA activity due to chemical e
3 collapse of the density of states at low transition energy, suggesting the emergence of the MDF linear dispersion in the
4 econdary structural features in both enzymes are perturbed, suggesting the existence of a common mechanism for substrate
5 ly increase or decrease the longer cells are in quiescence, suggesting the existence of a continuum of quiescence depths.
6 es activated when pre-synaptic proteins are over-expressed, suggesting the existence of a feedback circuit to match synap
7 bit sporadic expression of many of these same marker genes, suggesting the existence of a general program in which expres
8 ed response gene and a repressor of IFN-gene transcription, suggesting the existence of a negative-feedback regulatory lo
9 les become refractory to remating within seconds [1, 3, 4], suggesting the existence of a rapid mechanism to prevent fema
10 ure to vismodegib is necessary to prevent tumor recurrence, suggesting the existence of a vismodegib-resistant reservoir
11 l motile in the mutants where MglA localizes symmetrically, suggesting the existence of additional regulators that contro
12 ra of spontaneous mutations similar to MMR-bearing species, suggesting the existence of an alternative to the canonical M
13 ans-eQTL hotspots, affecting from ten to hundreds of genes, suggesting the existence of master genetic regulators.
14 ns, cytokinetic abscission is delayed but can be completed, suggesting the existence of parallel membrane-reorganizing pa
15 neral are exempt from Rtt109-dependent dosage compensation, suggesting the existence of pathways excluding specific loci
16 detected or detected with very low frequency in the blood, suggesting the existence of separate compartments of T cells
17 o exhibit decreased resistance to a hypo-osmotic challenge, suggesting the function of caveolae as membrane reservoir is
18 verall conformation than those formed along the on-pathway, suggesting the generation of distinct conformeric strains of
19 gE, and cell-specific signatures are regulated in patients, suggesting the imbalance of immune and inflammatory cells in
20 yrinogen III level and reduced protoporphyrinogen IX level, suggesting the impairment of tetrapyrrole biosynthesis.
21 gain relative to control in a diet-induced obese dog model, suggesting the importance of systemic inhibition of DGAT1 for
22 r CD40L in combination with IL-21, but not BCR stimulation, suggesting the importance of the immune-stimulatory context f
23 tered gene expression in CRC, this was not the case in ACF, suggesting the insufficiency of methylation changes to modula
24 G, mobilizes Ca(2+) and activates protein kinase C and ERK, suggesting the involvement of a G protein-coupled receptor (G
25 eration of mitochondrial replication and import components, suggesting the involvement of additional H. pylori activities
26 cytes in the presence of detectable inflammatory responses, suggesting the involvement of complex viral mechanisms of imm
27 ma membrane was differentially sensitive to BFA treatments, suggesting the involvement of different protein kinases or tr
28 Like CD, RCDII is strongly associated with HLA-DQ2, suggesting the involvement of HLA-DQ2-restricted gluten-speci
30 associated with more unbalanced tumour phylogenetic trees, suggesting the need of denser sampling of tumours at relapse.
31 nvolved in chemokine-induced RhoA and Rac1 activation, thus suggesting the occurrence of a SOS1 specificity shift in the
32 induced ABA-responsive genes CsABI5 and CsDREB2A similarly, suggesting the occurrence of ABA signaling in roots of floode
33 y partly explain litter decomposition under dry conditions, suggesting the operation of an additional mechanism.
34 e preferential mu-opioid receptor inverse agonist naloxone, suggesting the participation of mu-opioid receptors in the re
35 thesize reactive oxygen species (ROS) in response to light, suggesting the possibility of an alternate signaling mechanis
36 of the tissue that diverges at the liquid-solid transition, suggesting the possibility of extracting rheological informat
37 These changes were detected 30 days after blast exposure, suggesting the possibility of long-lasting retinal injury and
38 the ion current displays high sensitivity to redox species, suggesting the possibility of trace-level analysis.
39 Our results reveal new modes of action for TAL effectors, suggesting the possibility of yet unrecognized targets import
40 educed therapeutic response, and shorter remission periods, suggesting the presence of a persistent and potentially progr
41 gand positioned trans to the photolabile CH3CN ligand in 2, suggesting the presence of a trans-type influence in the exci
42 2 repeat expansion does not fully account for this finding, suggesting the presence of additional pleiotropic genes assoc
43 ssing CEACAM6 was significantly increased in severe asthma, suggesting the presence of an altered neutrophil phenotype.
44 of viruses in the panel, and 53% neutralized more than 50%, suggesting the presence of broadly NAbs in these individuals.
45 de are located to different mitochondrial sub-compartments, suggesting the presence of multiple cAMP/PKA signalling domai
46 f the DNA can protect against vaginal challenge with HPV16, suggesting the promise of this approach to both prevent and t
48 ocular surface homeostasis in unmanipulated NK1R(-/-) mice, suggesting the role of SP-NK1R signaling in ocular surface ho
49 Coulombic efficiencies near 100 % for every cycle, suggesting the suppression of polysulfide shuttle through the
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