ライフサイエンスコーパス: sulcal

1 e criteria of previous authors, although the sulcal anatomy of the human temporal pole has great vari

2 tism and 14 healthy comparison subjects, and sulcal and gyral thickness were measured for the total b

3 Increases in total cerebral sulcal and gyral thickness were observed in children wit

4 al cortical gray matter decline and cortical sulcal and lateral ventricular expansion.

5 Regionally specific increases in sulcal and subarachnoid CSF occur during adulthood and a

6 d white matter as well as greater volumes of sulcal and ventricular CSF than nonalcoholic women.

7 iations, except for a lack of association of sulcal and ventricular size with race.

8 Total, sulcal, and smooth-surface molar caries in the GBP59- an

9 on could be further subdivided into an outer sulcal anterior auditory field (sAAF) and an inner field

10 an posterior cingulate gyrus without marking sulcal areas.

11 espread as well as spatially coherent within sulcal boundaries and decreases in amplitude across the

12 y had statistically significant decreases in sulcal caries severity.

13 of smooth-surface caries and the severity of sulcal caries, relative to controls.

14 We found accelerated enlargement in cortical sulcal cerebrospinal fluid spaces early in the course of

15 n clusters were associated with formation of sulcal clots (P < 0.01), a high likelihood of adjacent c

16 al morphology of the cingulate/paracingulate sulcal complex and feedback-related activity.

17 Tracer injection into the sulcal cortex demonstrated that at least some axonal fib

18 he anteroventral field and the pseudosylvian sulcal cortex.

19 d with larger left (9.66%) and right (9.61%) sulcal CSF relative volumes were found in women with SPD

20 y matter volume and significant increases in sulcal CSF volume compared with controls.

21 ng groups, gray matter volume reductions and sulcal CSF volume increases were significantly more pron

22 er neocortical gray matter volume and larger sulcal CSF volume provide evidence of the brain basis of

23 as segmented into gray matter, white matter, sulcal CSF, and ventricular CSF from a T(1)-weighted mag

24 al lobe gray matter, lateral ventricles, and sulcal CSF.

25 Maps of sulcal depth (distance from the cerebral hull) were comb

26 Regional analysis of sulcal depth and cortical shape showed the greatest impa

27 We performed a detailed analysis of sulcal depth based on geometric cortical surface represe

28 riability was also related to variability in sulcal depth but not cortical thickness, positively corr

29 ch significant sulcal widening and decreased sulcal depth co-occurred.

30 ior frontal gyrus that was associated with a sulcal depth difference in the anterior insula and front

31 Sulcal depth findings in the PS corresponded closely to

32 ross individuals to generate maps of average sulcal depth for WS and control subjects, along with dep

33 We found bilateral reductions in sulcal depth in the intraparietal/occipitoparietal sulcu

34 We generated sulcal depth maps from structural magnetic resonance ima

35 on two measures (verbal memory and cortical sulcal depth).

36 (measured by gray- and white-matter volume, sulcal depth, and gyrification index) that localize to t

37 ersubject variability in cortical curvature, sulcal depth, cortical thickness, and cortical surface a

38 Population average maps of sulcal depth, depth variability, three-dimensional posit

39 rmalities suggestive of vasogenic oedema and sulcal effusions (ARIA-E) and microhaemorrhages and haem

40 lume increases in the lateral ventricles and sulcal (especially frontal and temporal) CSF than Val ho

41 Prefrontal gray matter decline and sulcal expansion were associated with greater BPRS negat

42 Faster rates of frontal sulcal expansion were related to greater BPRS total and

43 cortical gray matter volume loss, as well as sulcal expansion.

44 ution atlas (including labeling of gyral and sulcal features).

45 al human brains covering a period of primary sulcal formation (20-28 gestational weeks).

46 generated flat maps in relation to gyral and sulcal geography, thereby facilitating the analysis of c

47 hat the gray matter volume loss and abnormal sulcal geometry may be related.

48 Statistical analysis of sulcal geometry will facilitate multisubject atlasing, n

49 sion were used to characterize variations in sulcal geometry.

50 h increasing age, and higher ventricular and sulcal grades in men and in nonblack individuals.

51 By using models of the cortical surface and sulcal landmarks and a statistical model for gray matter

52 cortical pattern-matching technique used 72 sulcal landmarks traced on each brain as anchors to alig

53 olded cortical surface relative to gyral and sulcal landmarks.

54 ess cortical gray and white matter, and less sulcal, lateral, and third ventricular CSF.

55 lation-based interhemispheric asymmetries of sulcal length existed that could be considered markers o

56 prefrontal cortex, with a 1 cm reduction in sulcal length increasing the likelihood of hallucination

57 s, photographs, morphological recordings and sulcal length metrics, our data illustrate the interindi

58 outer cortical surface (hull surface area), sulcal length, depth, and width.

59 nduced by head impact loading is greatest in sulcal locations, where pathology in cases of chronic tr

60 Sulcal outlines were reparameterized for surface compari

61 nches, and connections) and for a particular sulcal pattern, namely, the interposition of a precentra

62 y and white matter, macrogyria, and abnormal sulcal pattern.

63 Although gyral and sulcal patterns are highly heritable, and emerge in a ti

64 Most of Einstein's sulci are identified, and sulcal patterns in various parts of the brain are compar

65 Two of the photographs reveal sulcal patterns of the medial surfaces of the hemisphere

66 Heritability for gyral-sulcal patterns, as reflected in the cross-correlation d

67 ral poles, and parietal cortex; and lower in sulcal, perirolandic, and medial occipital cortex.

68 allel to the cortical surface, most often in sulcal regions but also in many gyral crowns.

69 The volume fraction of sulcal regions exceeding brain injury thresholds were si

70 Sulcal regions exhibited higher contrast compared to gyr

71 ickness, with positive relationships seen in sulcal regions in prefrontal and temporal cortices, and

72 Positive relationships were seen in sulcal regions in prefrontal and temporal cortices, whil

73 owed converging imaging abnormalities within sulcal regions with a significant decrease in fractional

74 ntal and orbitofrontal and superior temporal sulcal regions with areas of relative preservation in vi

75 measured in 20 tissue and 5 ventricular and sulcal regions.

76 n, namely, the interposition of a precentral sulcal segment between the central sulcus and one of its

77 anifested in global alterations of gyral and sulcal shape.

78 Ventricular size, sulcal size, and white matter signal intensity changes w

79 The results show that gyral and sulcal structures are aligned with high but variable acc

80 the basilar cisterns (P <.001) and cerebral sulcal subarachnoid space (P <.001) after Fio(2) was red

81 tans and a cariogenic diet, total smooth and sulcal surface lesions are more than 2- and 1.6-fold hig

82 ts by using elastic transformations to align sulcal topography.

83 the relationship between thickness and local sulcal topology arises in development is still not well

84 roup had smaller thalamic and larger frontal sulcal volumes than control subjects.

85 Slopes relating cortical gray matter and sulcal volumes to age were steeper in alcoholic than in

86 x interactions for cortical white matter and sulcal volumes were due to abnormalities in alcoholic me

87 and occipital cortices, in which significant sulcal widening and decreased sulcal depth co-occurred.

88 ences were consistent with cortical thinning/sulcal widening and ventricular enlargement.

89 Globally, sulcal widening was associated with cortical thinning an

90 Sulcal width, ventricular size, and white matter signal