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1 , pendrin has been proposed to function as a sulfate transporter.
2 tion of sulfate uptake and expression of the sulfate transporters.
3 ane glycoprotein and a member of a family of sulfate transporters.
5 panning alpha-helices and a C-terminal STAS (sulfate transporters and anti-sigma-factor) domain homol
6 cal for the activity of Arabidopsis thaliana sulfate transporters, and specific lesions in this domai
7 impaired, but deletion of the adjacent STAS (sulfate transporter anti-sigma factor antagonist) domain
8 gulator (CFTR), (R)CFTR, via the Slc26-STAS (sulfate transporter anti-sigma) domain, resulting in mut
9 s of SULTR1;2, which encodes a high-affinity sulfate transporter, are defective in sulfate transport
10 conserved miRNA that targets a low-affinity sulfate transporter (AST68) and three ATP sulfurylases (
11 th treatments also caused up-regulation of a sulfate transporter, but only in the case of sulfur defi
13 e exchange of STAS domains between different sulfate transporters, can severely impair transport acti
14 ed sulfate/anion transporters that contain a sulfate transporter domain and are found in a widely dis
15 ral residue substitutions near the conserved sulfate transporter domain of prestin either greatly red
16 nsporter (encoded by DTD; 32%) and the human sulfate transporter 'downregulated in adenoma' (encoded
20 e identity), the human diastrophic dysplasia sulfate transporter (encoded by DTD; 32%) and the human
21 e; however, root proliferation and increased sulfate transporter expression occurred as in S-deficien
27 he sequence analysis classified the Brassica sulfate transporter genes into four different groups.
32 SLC26A2 is the primary sodium-independent sulfate transporter in cartilage and bone and is importa
36 reased expression of genes encoding specific sulfate transporters in roots and other plant parts.
37 ing or modifying the STAS domain of dominant sulfate transporters in roots of Arabidopsis thaliana.
38 ons 260 and 288, are conserved in all of the sulfate transporters in the family whereas the NaDC cont
40 teractions between residues surrounding the "sulfate transporter motif" is essential for normal prest
41 ing invasive pulmonary aspergillosis since a sulfate transporter mutant strain and a sulfite reductas
43 pecifically, uptake of selenate (probably by sulfate transporters) occurred at a much higher rate tha
46 logy and the presence of a slightly modified sulfate-transporter signature sequence comprising its pu
47 y analyzing the expression of genes encoding sulfate transporters (STs) of groups 1, 2, and 4 (SlST1.
50 d constitutive upregulation in S. pinnata of sulfate transporters SULTR1;2 (root influx) and SULTR2;1
51 icroarray studies identified a high-affinity sulfate transporter (SULTR1;2) among the most robust and
52 ion to its known function as a high-affinity sulfate transporter, SULTR1;2 may have a regulatory role
53 fate assimilation, as well as a low-affinity sulfate transporter, SULTR2;1, is strongly induced by su
54 show that SLC26a2/7 is a ventrally expressed sulfate transporter that promotes a ventral accumulation
55 in has a role in controlling the activity of sulfate transporters, their stability, or their localiza
57 ux pumps employed to remove cadmium, while a sulfate transporter was down-regulated to reduce nonspec
58 rivation, although the expression of Group 3 sulfate transporters was not affected by the sulfate sta
59 pecies, abundant gene expression of putative sulfate transporters was observed for both Se-hyperaccum
60 d the sphX gene, and transcripts of multiple sulfate transporter were also significantly more abundan
61 quisition genes encoding for a high-affinity sulfate transporter were significantly induced, while de
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