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1 e methylene group with a nitrogenous base or sulfhydryl compound.
2 he ortho-hydroxylated derivatives with model sulfhydryl compounds.
3 tionally, replacing a single hydroxyl with a sulfhydryl (compound 23, IC50 = 5.8 microM) results in o
6 with an acid pH optimum that is activated by sulfhydryl compounds and preferentially hydrolyzes the m
7 t sensitive to reduction by externally added sulfhydryl compounds, but apparently reacted with intrac
8 s S-methylation of aromatic and heterocyclic sulfhydryl compounds, including anticancer and immunosup
9 e S-methylation of aromatic and heterocyclic sulfhydryl compounds, including medications such as merc
10 um was activated by thiocyanate, cyanate, or sulfhydryl compounds; inhibited by sulfite, bisulfite, o
11 les and could be suppressed by the synthetic sulfhydryl compounds, N-acetylcysteine and bucillamine.
12 such as aniline and veratrylamine as well as sulfhydryl compounds such as l-cysteine and beta-mercapt
13 do react readily with biologically important sulfhydryl compounds to give products derived from eithe
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