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1 ancement increased with the concentration of sulfite.
2 s showed sensitivity and overaccumulation of sulfite.
3 mproved by 29 times in the presence of 20 mM sulfite.
4 kely metabolite of thiosulfate assimilation, sulfite.
5 reflecting a decrease in the ability to bind sulfite.
6 f ammonium with o-phthaldialdehyde (OPA) and sulfite.
7 ains that function together to reduce APS to sulfite.
8 ty to form complexes with glycine betaine or sulfite.
9 donor for the reduction of APS to 5'-AMP and sulfite.
10 for methanogenesis and a possible target for sulfite.
11 nversion of taurine to aminoacetaldehyde and sulfite.
12 rapidly oxidised sulfide to thiosulfate and sulfite.
13 ts dibenzothiophene to 2-hydroxybiphenyl and sulfite.
14 ) during the free radical chain oxidation of sulfite.
15 fide and unexpectedly, when it is exposed to sulfite.
16 t rate by reaction with sulfide but not with sulfite.
17 which is then partitioned to thiosulfate or sulfite.
19 reacts with OPA and forms in the presence of sulfite a product, which can be detected by spectrophoto
25 ic red wines produced without sulfur dioxide/sulfites addition are comparable to conventional red win
26 ic red wines produced without sulfur dioxide/sulfites addition in comparison to those of eight conven
27 udies (stabilizations of the covalent FAD-N5-sulfite adduct and p-quinonoid form of 8-mercapto-FAD),
28 model wine was present in free and hydrogen sulfite adduct forms and the measured total, free and pe
31 nd Mn(II) caused damage to DNA while neither sulfite alone nor metal ions alone did have the same eff
32 lly, the effect of the reducing agent sodium sulfite also was evaluated to characterize the nature of
36 her reactive sulfur species (RSS), including sulfite and bisulfite, as well as sulfane sulfur species
37 vasculature system, where it is reduced into sulfite and finally sulfide within the subcellular organ
38 transfer in the direction of thiosulfate to sulfite and glutathione persulfide; sulfur transfer in t
39 everely impaired both in the ability to bind sulfite and in catalysis, with a second-order rate const
40 ctivity of human rhodanese to cyanide versus sulfite and might be important in differences in suscept
43 suggests an efficient oxidation pathway via sulfite and sulfate radical anions on droplets possibly
45 ection for simultaneous analysis of sulfate, sulfite, and chloride in human urine, plasma, and sweat
46 ple thiophilic acceptors, including sulfide, sulfite, and glutathione, to form as products, hydrodisu
48 gradation rate of MO was greatly enhanced by sulfite, and the enhancement increased with the concentr
50 spontaneously desulfinated to succinate and sulfite; and (iii) whereas succinate enters the central
51 icals generated by the reactions of sulfite (sulfite anions or bisulfite anions) with holes or hydrox
52 ts in maintaining sulfite homeostasis, where sulfite appears to act as an orchestrating signal molecu
53 and activity levels are likewise promoted by sulfite application as compared with water injection con
56 -stressed wild-type plants were resistant to sulfite applications, but SO RNA interference plants sho
57 containing compounds, augmented by exogenous sulfite applications, underlines the role of SO and othe
58 r of their ability to induce DNA damage with sulfite as follows: Fe(III) > Co(II) > Cu(II) > Cr(VI) >
59 hat it is essential for growth on sulfate or sulfite as the sole sulfur source and, further, that the
63 linear in the range 0.032-0.320 mg L(-1) of sulfite (as SO2), with a correlation coefficient of 0.99
67 another low pH form, whereas reduction with sulfite at higher pH values gives a mixture of Species 1
69 NA damage induced by radicals generated from sulfite autoxidation using cyclic voltammetry (CV) and e
70 R is consistent with glutathione rather than sulfite being the predominant acceptor at physiologicall
73 idation and insight into the impact of these sulfite bound carbonyls on antimicrobial and antioxidant
76 iderable photocurrent for photo-oxidation of sulfite, but generated significantly reduced photocurren
77 d many bacteria, this compound is reduced to sulfite by APS reductase (APR); in fungi and some cyanob
79 The 1,2-eliminations in cyclic carbonate and sulfite by regioselective abstraction of methine protons
80 ytoplasmic membrane for further oxidation to sulfite by the dissimilatory reductase DsrAB is incomple
82 of this species prepared with (33)S-labeled sulfite clearly show the presence of coordinated sulfate
86 However, under pathological conditions when sulfite concentrations are high, sulfite could compete w
88 itions when sulfite concentrations are high, sulfite could compete with sulfide for addition to the a
92 AB is a protein-based trisulfide, in which a sulfite-derived sulfur is bridging two conserved cystein
93 ant SO (PSO) also plays an important role in sulfite detoxification and in addition serves as an inte
98 gh sulfite application is manifested by fast sulfite disappearance and an increase in glutathione lev
103 sulfur from glutathione persulfide (GSSH) to sulfite generating thiosulfate and from thiosulfate to c
106 on being: H2S --> glutathione persulfide --> sulfite --> sulfate, than with a more convoluted route t
107 er sulfite network components in maintaining sulfite homeostasis, where sulfite appears to act as an
108 ssible to observe and sometimes quantify the sulfite, hydrate, and acetal forms of the carbonyl compo
109 s PRF is poised to metabolize thiosulfate to sulfite in a sulfur assimilation pathway rather than in
111 was evaluated with 96.46% recovery of added sulfite in red wine and 1.7% and 3.3% within and between
112 ults in a covalent adduct between NAD(+) and sulfite in the active site of the enzyme that binds very
113 action method (HS-SDME) for determination of sulfite in the form of sulfur dioxide was developed.
116 in the overall reaction process whereby the sulfite, in the presence of transition metals, may cause
117 imum concentration of a transition metal for sulfite induced DNA damage revealed that electrochemical
118 is also formed when sulfide is added to the sulfite-induced CT intermediate, representing a new mech
119 SiR transcript and activity within 30 min of sulfite injection into Arabidopsis and tomato leaves.
120 rn about food preservation, the reduction of sulfite input plays a major role in the wine industry.
121 te oxidation to sulfate and incorporation of sulfite into sulfoquinovosyl diacylglycerols were not su
122 stance to ectopically applied sulfur dioxide/sulfite is a function of SiR expression levels and that
124 ts for the removal of water pollutants since sulfite is a waste from flue gas desulfurization process
125 In biological systems, the detoxification of sulfite is catalyzed by the sulfite-oxidizing enzymes (S
126 eas succinate enters the central metabolism, sulfite is detoxified by the previously identified putat
130 s a role in the protection of plants against sulfite is supported by the rapid up-regulation of SiR t
132 1A mutation causing a 5-fold increase in the sulfite K(m) value, perhaps reflecting a decrease in the
133 sis thaliana has been reduced at pH = 6 with sulfite labeled with 33S (nuclear spin I = 3/2), followe
136 Little is known about the homeostasis of sulfite levels, a cytotoxic by-product of plant sulfur t
137 ls were not sufficient to maintain low basal sulfite levels, resulting in accumulative leaf damage in
139 at it has excellent electrocatalysis towards sulfite, lower detection limit, higher storage stability
141 re found for specific Kraft, mechanical, and sulfite mills, suggesting yet unidentified causative age
143 However, under nonstressed conditions, the sulfite network can control sulfite levels in the absenc
144 cations, underlines the role of SO and other sulfite network components in maintaining sulfite homeos
146 ic pathways, we followed key elements of the sulfite network enzymes that include adenosine-5'-phosph
152 nter of the pathogenic R160Q mutant of human sulfite oxidase (hSO) confirms the presence of three dis
153 dies on the pathogenic R160Q mutant of human sulfite oxidase (HSO) have shown that Mo-heme intramolec
157 8D were identified in patients with isolated sulfite oxidase (SO) deficiency, and the equivalent amin
158 Dimethylsulfoxide reductase (DMSOR) and sulfite oxidase (SO) families were the most widespread m
162 sulfate reductase and the sulfite scavengers sulfite oxidase (SO), sulfite reductase, UDP-sulfoquinov
165 o either the xanthine dehydrogenase (XDH) or sulfite oxidase (SUOX) families, and these have pyranopt
166 ing partial misfolding and monomerization of sulfite oxidase and attenuating both substrate binding a
167 persulfide dioxygenase (PDO), rhodanese, and sulfite oxidase and converts H2S to thiosulfate and sulf
169 ed the characteristic absorption spectrum of sulfite oxidase and exhibited steady state and rapid kin
170 The physiological implications of plant sulfite oxidase as a copious generator of superoxide are
171 onance Raman spectra of oxidized A. thaliana sulfite oxidase catalytically cycled in both H2(16)O and
175 tations identified in patients with isolated sulfite oxidase deficiency, the G473D variant is of part
176 for confirmatory testing of cystic fibrosis, sulfite oxidase deficiency, urolithiasis, and other diso
177 crystal structures of the wild type and the sulfite oxidase deficiency-causing R138Q (R160Q in human
184 0Q in humans) variant of recombinant chicken sulfite oxidase in the resting and sulfate-bound forms.
188 te forms of the molybdenum-containing enzyme sulfite oxidase possess a b-type cytochrome prosthetic g
190 sulfite oxidase, Arabidopsis thaliana plant sulfite oxidase, and the bacterial sulfite dehydrogenase
191 remarkably similar to that found in chicken sulfite oxidase, Arabidopsis thaliana plant sulfite oxid
192 f this residue in the catalytic mechanism of sulfite oxidase, serine and alanine variants at position
193 subsequent reconstitution of MoCo-free human sulfite oxidase-molybdenum domain yielding a fully activ
197 s in crystallizing recombinant human and rat sulfite oxidases and the failure to clone the chicken su
207 etoxification of sulfite is catalyzed by the sulfite-oxidizing enzymes (SOEs), which interact with an
208 similar results were observed for samples of sulfite-oxidizing enzymes from other organisms that were
210 sis of the possible mechanistic pathways for sulfite-oxidizing enzymes is presented and related to av
212 ons were measured by HPLC using a stable OPA/sulfite precolumn derivatization and an electrochemical
213 ed wild-type plants, while expression of the sulfite producer, adenosine-5'-phosphosulfate reductase,
215 n rapidly oxidise sulfide to thiosulfate and sulfite, providing the foundation for using heterotrophi
216 Studies using hole scavengers suggest that sulfite radicals generated by the reactions of sulfite (
219 f infected oat tissue homogenate with sodium sulfite reduces transmission of the purified virus by ap
221 lic processes, and profiles of dissimilatory sulfite reductase (dsr) transcripts are consistent with
222 CR targeting the 16S rRNA, dissimilatory (bi)sulfite reductase (dsrAB), and dissimilatory arsenate re
231 oad similarity to the hemoprotein subunit of sulfite reductase but has many significant differences i
233 flavin oxidoreductase component of the CysJI sulfite reductase complex (CysJ(8)I(4)), we show that th
235 The initial rate parameters for the purified sulfite reductase from M. tuberculosis were determined u
236 t may interact with HdrABC and dissimilatory sulfite reductase gamma subunit (DsrC) to perform novel
237 hic patterns of the functional dissimilatory sulfite reductase gene (dsrA) and the 16S rRNA gene in s
238 ce a sulfate transporter mutant strain and a sulfite reductase mutant strain are fully virulent.
239 ences or could arise from alterations of the sulfite reductase structure that arise from the isolatio
240 dentification of virus-encoded dissimilatory sulfite reductase suggests SUP05 viruses reprogram their
244 -terminal half a dissimilatory-type siroheme sulfite reductase, and Fsr catalyzes the corresponding p
245 s coding for DsrAB, the enzyme dissimilatory sulfite reductase, inevitably also contain the gene codi
247 the sulfite scavengers sulfite oxidase (SO), sulfite reductase, UDP-sulfoquinovose synthase, and beta
251 Like their nitrite reductase counterparts, sulfite reductases require a siroheme cofactor for catal
253 anaerobic S(0) reduction, anaerobic sulfate/sulfite reduction and anaerobic respiration of organic s
254 acterial system, we show that the product of sulfite reduction by DsrAB is a protein-based trisulfide
255 C biosynthesis and that pseudomonads utilize sulfite reduction enzymology distinct from that of E. co
261 enzyme with NAD(+) and low concentrations of sulfite results in a covalent adduct between NAD(+) and
262 ite application induced up-regulation of the sulfite scavenger activities in dark-stressed or unstres
263 denosine-5'-phosphosulfate reductase and the sulfite scavengers sulfite oxidase (SO), sulfite reducta
264 od for measuring APR activity by using novel sulfite-selective colorimetric or "off-on" fluorescent l
266 tion, sulfide serves as the sulfur donor and sulfite serves as the acceptor, forming thiosulfate.
267 produce GSSH; PDO oxidises GSSH to sulfite; sulfite spontaneously reacts with polysulfides to genera
268 as a result of these mutations; however, the sulfite-stimulated activity decreased by more than 60%.
269 consequence of SiR impairment, the levels of sulfite, sulfate, and thiosulfate were higher and glutat
270 ase is capable of being regenerated from its sulfite/sulfate heat stable salt, which enables the simu
271 lfite radicals generated by the reactions of sulfite (sulfite anions or bisulfite anions) with holes
272 thione to produce GSSH; PDO oxidises GSSH to sulfite; sulfite spontaneously reacts with polysulfides
273 that in the C185S variant, in the absence of sulfite, the active site residue Tyr322 became disordere
275 185S variant crystallized in the presence of sulfite, the Tyr322 residue relocalized to the active si
277 animals, SO catalyzes the oxidation of toxic sulfite to sulfate as the final step in the catabolism o
279 sociated with the AMP-dependent oxidation of sulfite to sulfate), some of which occur in multiple (up
282 mmission 1.8.7.1) catalyzes the reduction of sulfite to sulfide in the reductive sulfate assimilation
287 The method is based on the conversion of sulfite to volatile sulfur dioxide by acidification of t
290 are not mutually exclusive: (a) that sodium sulfite treatment disrupts critical virion-host protein
293 good correlation (r=0.99) between red wines sulfite value by standard DTNB (5,5'-dithio-bis-(2-nitro
295 at values, and the large increases in the Km(sulfite) values, rationalize the fatal impact of these m
296 in the sulfur transfer reaction from GSSH to sulfite were 1.6- (Asp-102) and 4-fold (Ala-285) lower t
297 a more convoluted route that would result if sulfite were the primary acceptor of sulfane sulfur.
299 ted the effects of both conventional and low sulfite wines on ex vivo human erythrocytes under oxidat
300 r artifact-free determination of sulfate and sulfite with consistent results for chloride when compar
301 te (APS) to adenosine 5'-phosphate (AMP) and sulfite with reducing equivalents from the protein cofac
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