ライフサイエンスコーパス: sulfonic acid

1 (iodoacetyl)amino]ethyl}amino)naphthalene-1- sulfonic acid).

2 tracolonic administration of trinitrobenzene sulfonic acid.

3 intraductal injection of 2% trinitrobenzene sulfonic acid.

4 allows the binding of 8-anilino-1-naphtalene sulfonic acid.

5 nflammation was induced with trinitrobenzene sulfonic acid.

6 flammatory pain induced with trinitrobenzene sulfonic acid.

7 eated 6 days previously with trinitrobenzene sulfonic acid.

8 nnatural amino acids homoserine and cysteine sulfonic acid.

9 ag2(-/-) recipients and (ii) trinitrobenzene sulfonic acid.

10 y doses of intrarectal 2,4,6-trinitrobenzene sulfonic acid.

11 by intraductal injection of trinitrobenzene sulfonic acid.

12 n with supplementation with 2-mercaptoethane sulfonic acid.

13 to an unreactive form such as a sulfinic or sulfonic acid.

14 with the metal chelator 8-hydroxyquinoline-5-sulfonic acid.

15 ROS due to its overoxidation to sulfinic or sulfonic acid.

16 e fluorescent probe, 8-anilino-1-naphthalene-sulfonic acid.

17 -piperidine; GSH was primarily oxidized into sulfonic acid.

18 rosis and Cys674 was found to be oxidized to sulfonic acid.

19 emical modification by 2,4,6-trinitrobenzene sulfonic acid.

20 l, and 2,2'-azinobis3-ethylbenzothiazoline-6-sulfonic acid.

21 of dextran sodium sulfate or trinitrobenzene sulfonic acid.

22 ntracolonic administration of dinitrobenzene sulfonic acid.

23 enamide, or overoxidation to the sulfinic or sulfonic acids.

24 or by cysteine overoxidation to sulfinic and sulfonic acids.

25 hereas Cys46 and Cys53 are fully oxidized to sulfonic acids.

26 s the first study of internal elimination in sulfonic acids.

27 2-iodoacetyl)amino)ethyl)amino)naphthalene-1-sulfonic acid (1,5-IAEDANS) without loss of the ability

28 minopurine (2-AP-6-SCH3) and 2-aminopurine-6-sulfonic acid (2-AP-6-SO3H) upon reaction with S-adenosy

29 NMR study of three (17)O-labeled crystalline sulfonic acids: 2-aminoethane-1-sulfonic acid (taurine,

30 -ylmethylene]-2-ox o-2,3-dihydro-1H-indole-5-sulfonic acid (3-chlorophenyl)methylamide, termed [11C]S

31 hydro-[1,2,4]triazol -4-ylmethyl]-biphenyl-2-sulfonic acid (3-methyl-thiophene-2-carbonyl)-amide].

32 Radiolysis of cysteine leads to cysteine sulfonic acid (+48 Da mass shift) and cystine as the maj

33 Dinonylnaphthalene sulfonic acid (a cation exchanger) and chromoionophore I

34 acid (homotaurine, HT), and 4-aminobutane-1-sulfonic acid (ABSA).

35 n of 2,2'-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS(*+)).

36 nic 2,2'-azino-bis-(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS(+)) radical, as well as the stable

37 n of 2,2'-Azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS) and Laccase on Vulcan XC-72, which

38 ing 2, 2-Azino-bis-(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS) as a mediator, while no significan

39 sing 2,2'-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS) as an electron mediator].

40 on of 2,2'-azino-bis(3-ethylbenzthiazoline-6-sulfonic acid) (ABTS) by H(2)O(2) and iNOS(heme) was use

41 and 2,2'-azinobis-(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS) radical scavenging activity even a

42 and 2,2'-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS) reactions.

43 the 2,2'-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS)-H2O2 reaction catalyzed by G-quadr

44 and 2,2'-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS).

45 ) and 2,2'-azinobis(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS).

46 reased in colon tissues after dinitrobenzene sulfonic acid administration.

47 nor 5-(iodoacetamidoethyl)aminonaphthalene-1-sulfonic acid (AEDANS) attached to the residues 131, 151

48 3 with 2-(4'-iodoacetamidoanilo)napthalene-6-sulfonic acid allowed us to accurately measure the rate

49 t using catalytic amorphous milled cellulose sulfonic acid (AMCell-SO3H), substituted anilines, prope

50 onylbis(azanediyl)bis(4-hydroxynaphthalene-2-sulfonic acid (AMI-1) was a potent antagonist of Nox-der

51 l alcohol (PVA)-2-Acrylamido-2-methylpropane sulfonic acid (AMPS) hydrogel beads (DC Bead) that are c

52 3O4-VTMS) using 2-acrylamido-2-methylpropane sulfonic acid (AMPS), N,N'-methylenebisacrylamide (MBA)

53 Sulfonic acid analogues of 4-[1-(3,5,5,8,8-pentamethyl-5

54 the formation of the active species in both sulfonic acid and acetate-containing buffer systems.

55 Using both the trinitrobenzene sulfonic acid and dextran sodium sulfate models of colit

56 ormed at 130 degrees C was the corresponding sulfonic acid and not the phenol itself.

57 The proportions of sulfonic acid and phosphate groups in both the self-supp

58 he disulfide bond and generation of cysteine sulfonic acid and sulfinic acid; however, the rate of ox

59 rfaces functionalized with discrete pairs of sulfonic acid and thiol groups are reported.

60 sets of fragments, namely, the naphthylamide sulfonic acids and the naphthalene-benzamides and -nicot

61 [(iodoacetyl)amino]ethyl]amino]naphthalene-1-sulfonic acid) and acceptor (i.e., fluorescein 5-isothio

62 BTS (2,2'-azinobis-(3-ethylbenzothiazoline-6-sulfonic acid) and DPPH (2,2-diphenyl-1-picryl-hydrazyl-

63 d gels were "doped" with sodium poly(styrene sulfonic acid) and poly(diallyl dimethylammonium chlorid

64 ,4-tetrazolium-bis(4-methoxy-6-nitro)benzene sulfonic acid) and TUNEL (terminal deoxynucleotidyl tran

65 ABTS (2,2-azino-bis(3-ethylbenzthiazoline-6-sulfonic acid)) and FRAP (ferric-reducing antioxidant po

66 escence properties of 1-anilinonaphthalene-8-sulfonic acid, and (iv) glycolipid intermembrane transfe

67 ication with a partial agonist, piperidine-4-sulfonic acid, and a gating-impaired mutation, alpha1(L2

68 glutathione, s-hexylglutathione, glutathione sulfonic acid, and glutathione-s-dinitrobenzene.

69 g HCl, KCl, KNO 3, Ni(NO 3) 2, LiCl, camphor sulfonic acid, and K 3Fe(CN) 6 ionic solutions.

70 ile perfluoroheptanoic acid, perfluorobutane sulfonic acid, and perfluorohexane sulfonic acid were de

71 bicity as detected by 1-anilinonaphthalene-8-sulfonic acid (ANS) fluorescence.

72 plate impregnated with 8-amino-1-naphthol-5-sulfonic acid (ANS) is used as the collection/sensor ele

73 d in the presence of 8-anilino-1-naphthalene-sulfonic acid (ANS) or Congo Red (CR), perturbants that

74 r signal compared to 8-anilino-1-naphthalene sulfonic acid (ANS), a commonly used hydrophobic probe;

75 y in the presence of 8-anilino-1-naphthalene sulfonic acid (ANS), we show that RBP populates a state

76 lefins is reported that uses hydroxylamine-O-sulfonic acids as inexpensive, readily available, and ni

77 using 2,2'-azino-bis(3-ethylbenzthiazoline-6-sulfonic acid) as an electron transfer mediator.

78 TS (2,2'-azino-bis-3-ethyl-benzothiazoline-6-sulfonic acid) assays.

79 B) and 2,2-azinobis-(3-ethylbenzothizoline-6-sulfonic acid) (AzBTS), and an assay was designed for sc

80 tic polyelectrolytes composed of an aromatic sulfonic acid backbone exhibit specific affinity for cav

81 otoacid generators (PAGs) for carboxylic and sulfonic acids based on N-hydroxyanthracene-1,9-dicarbox

82 8-Anilino-1-naphthalene sulfonic acid binding data showed solvation of the myris

83 trinsic fluorescence, 8-anilinonaphthalene-1-sulfonic acid binding, visible absorption, copper conten

84 ed by gel filtration, 1-anilinonaphthalene-8-sulfonic acid-binding fluorescence, and phosphocholine-b

85 olyphenylenes that incorporate a novel tetra-sulfonic acid bistetracyclone monomer.

86 The pH-activity profile measured in a sulfonic acid buffer exhibited a maximum at pH 5.8 and b

87 oxidizing azinobis(3-ethylbenzothiazoline-6-sulfonic acid) but not L-Trp.

88 ) to the acetylation of 4-aminoazobenzene-4'-sulfonic acid by arylamine N-acetyltransferase.

89 [(iodoacetyl)amino]ethyl}amino)naphthalene-1-sulfonic acid) can be used as a versatile fluorescence-b

90 is quickly converted into either a sulfinic/sulfonic acid (catalytically dead and irreversible inact

91 onanesulfonic acid (PFNS)], and four unknown sulfonic acids (Cl-PFOS, ketone-PFOS, ether-PFHxS, and C

92 ibited a reduced severity of trinitrobenzene sulfonic acid colitis in association with decreased inte

93 s show that in chronic 2,4,6-trinitrobenzene sulfonic acid colitis, fibrosis is dependent on the deve

94 s of IBD: colitis induced by trinitrobenzene sulfonic acid, colitis induced by microflora (in gnotobi

95 tion after exposure to 2,4,6-trinitrobenzene sulfonic acid, compared with control mice.

96 We focus in particular on hydrated perfluoro-sulfonic acid compounds employed as electrolytes in fuel

97 lecular furan/iminium ion cyclization, and a sulfonic acid-controlled Z-selective macrocyclic ring-cl

98 traluminal administration of trinitrobenzene sulfonic acid; controls received saline.

99 of Cys(34) sulfinic acid (Cys-SO2H), Cys(34)-sulfonic acid (Cys-SO3H), and Met(329) sulfoxide were gr

100 ever, the dicarboxylic acid 2 and carboxylic/sulfonic acid derivative 3 showed excellent water solubi

101 ed both Cys residues in the CT-25 peptide to sulfonic acid derivatives, without altering the secondar

102 of 2,2'-azino-bis(3-ethylbenzothiazoline)-6-sulfonic acid diammonium salt (ABTS).

103 nd 2,2'-azino-bis-(3-ethylbenzo-thiazoline-6-sulfonic acid) diammonium salt (ABTS(+)), Fe(III) reduci

104 ndol-2-ylmethylene)-2,3-dihydro-1H-indole- 5-sulfonic acid dimethylamide (SU6656)] markedly inhibited

105 xtran sulfate sodium (DSS) or dinitrobenzene sulfonic acid (DNBS) in mice treated with selective 5-HT

106 antly reduced the severity of dinitrobenzene sulfonic acid (DNBS)-, oxazolone-, and dextran-sodium su

107 2,4-Dinitrobenzene sulfonic acid (DNBS)-induced colitis is an experimental

108 Adoptive transfer of 2,4-dinitrobenzene sulfonic acid (DNBS)-pulsed DCs directly implicated the

109 2-iodoacetyl-amino-ethyl-amino)naphthalene-1-sulfonic acid donor at either Cys-36 of the beta-chain o

110 MB), 2,2'-azino-bis (3-ethylbenzthiazoline-6-sulfonic acid), dopamine, and methylene blue in the pres

111 The fluorophore, dapoxyl sulfonic acid (DSA), is incorporated into two different

112 Six weekly 2,4,6-trinitrobenzene sulfonic acid enemas were given to establish colitis and

113 Lewis rats underwent weekly trinitrobenzene sulfonic acid enemas yielding models of acute inflammato

114 tocatalytic kinetics to generate fluorinated sulfonic acids, essential for the simultaneous improveme

115 al selective imprinted polyaniline-ferrocene-sulfonic acid film has been electrochemically fabricated

116 (2''-iodoacetamido)phenyl)aminonaphthalene-6-sulfonic acid fluorescence in isolated cTnC or the cTn c

117 hroism, aromatic and 8-anilino-naphthalene-1-sulfonic acid fluorescence, visible absorption, activity

118 ing changes in Trp or 1-anilinonaphthalene-8-sulfonic acid fluorescence.

119 extrinsic (binding of 1-anilinonaphthalene-8-sulfonic acid) fluorescence, near- and far-UV CD, and 1D

120 lls (PBMCs)-DCs) treated with dinitrobenzene sulfonic acid for predicting skin-sensitizing chemicals.

121 ysis of Cdc25B by mass spectrometry revealed sulfonic acid formation on the catalytic cysteine of Cdc

122 tion of the sulfenic acid to the sulfinic or sulfonic acid forms.

123 acing the fluorophore 1-anilinonaphthalene 8-sulfonic acid from the lipid binding cavity.

124 ethanols (FOSA/FOSEs), and one fluorotelomer sulfonic acid (FTSA)) were compared in household dust sa

125 n mechanism was followed by the formation of sulfonic acid functions, thus indicating a second degrad

126 Furthermore, sulfonic acids gave polyurethanes with higher molecular

127 g interactions with the polypeptide, such as sulfonic acid, gives rise to the formation of a long-liv

128 Furthermore, the sulfonic acid group can be partially substituted with ot

129 mer containing either a phosphate group or a sulfonic acid group in terms of (i) higher Pu(IV) sorpti

130 2',1'-f]pyrimidine-3,5-dione) nucleus, and a sulfonic acid group is then introduced at C(8) by reacti

131 ena: (1) dissociation of the proton from the sulfonic acid groups even in the presence of small amoun

132 eracts with drugs using ion-exchange between sulfonic acid groups on the polymer and the positively c

133 The sulfonic acid groups on the surface of the SWNT acted as

134 e presence of a macromolecular template with sulfonic acid groups that provide a unique environment f

135 ansformed to nanoporous carbon (NPC) and the sulfonic acid groups were cleaved from the SWNT.

136 olymer and the attraction between it and the sulfonic acid groups were confirmed by IR spectra.

137 pyrolysis pathways by selecting n-heptane-4-sulfonic acid (H4S) as a model compound.

138 , while inputs of the legacy perfluorooctane sulfonic acid had decreased.

139 sulfonic acid (taurine, T), 3-aminopropane-1-sulfonic acid (homotaurine, HT), and 4-aminobutane-1-sul

140 ith the chelating agent 8-hydroxyquinoline-5-sulfonic acid (HQS) to form Al(HQS)3, followed by oxidat

141 2-iodoacetyl)amino)ethyl)amino)naphthalene-1-sulfonic acid (IAEDANS (K(d) approximately 13 nM)), as w

142 2-iodoacetyl)amino)ethyl)amino)naphthalene-1-sulfonic acid (IAEDANS) and the DNA substrate with Dabcy

143 es, reduced serum anti-2,4,6-trinitrobenzene sulfonic acid IgG, decreased leukocyte infiltration, and

144 3-Methyl-1-sulfonic acid imidazolium nitrate ([Msim]NO(3)) as a new

145 tracolonic administration of trinitrobenzene sulfonic acid in mice causes inflammation in the colon t

146 by intraductal injection of trinitrobenzene sulfonic acid in rats.

147 leading to the generation of carboxylic and sulfonic acids in good quantum and chemical yields.

148 12 novel perfluoroalkyl ether carboxylic and sulfonic acids in surface water in North Carolina, USA u

149 oselectivity toward 16 chiral phosphoric and sulfonic acids in the polar organic mode (e.g., methanol

150 diction of the biotransformation of aromatic sulfonic acids in WWTPs.

151 lfide) and indirectly irreversible (sulfinic/sulfonic acids)] in liver from young and old C57BL/6 mic

152 and treated a model of acute trinitrobenzene sulfonic acid-induced (TNBS-induced) colitis, as assesse

153 vivo reduced the severity of trinitrobenzene sulfonic acid-induced colitis and associated bacterial t

154 Ab treatment also suppressed trinitrobenzene sulfonic acid-induced colitis in SJL/J mice.

155 this phenomenon in a chronic trinitrobenzene sulfonic acid-induced colitis model, an experimental col

156 organ inflammation (i.e., 2,4-dinitrobenzene sulfonic acid-induced colitis).

157 ific PPAR-gamma null mice to trinitrobenzene sulfonic acid-induced colitis.

158 nst the development of 2,4,6-trinitrobenzene sulfonic acid-induced colitis.

159 tenuated both DSS- and 2,4,6-trinitrobenzene sulfonic acid-induced colonic inflammation by improving

160 inflammation and fibrosis in trinitrobenzene sulfonic acid-induced murine colitis.

161 perfluorooctanesulfonate, or perfluorooctane sulfonic acid) is an anthropogenic fluorosurfactant wide

162 rmeable reagents, like 2,4,6-trinitrobenzene sulfonic acid, is also reduced severalfold.

163 confirmed cysteinyl sulfinic acid (m/z 435), sulfonic acid (m/z 443), and disulfide (m/z 836) bond fo

164 peroxidatic Cys is over-oxidized to cysteine sulfonic acid, making this the first Prx structure seen

165 , but also the presumably stronger acid aryl sulfonic acid material 11.SBA.

166 reactions to higher oxidation states (e.g., sulfonic acid) may be irreversible under biological cond

167 e second route is characterized by a camphor sulfonic acid mediated isomerization of a beta-hydroxyep

168 rease in 2-(4-maleimidoanilino)naphthalene-6-sulfonic acid (MIANS) fluorescence upon addition of fess

169 m, a molecule of Mes [2-(N-morpholino)ethane sulfonic acid] mimics the target uridine of an RNA subst

170 In the murine trinitrobenzene sulfonic acid model of colitis, inhibition of this enzym

171 The alkyl sulfonic acid modified material 10.SBA derived from the

172 with hydrogen peroxide to produce the alkyl sulfonic acid modified materials 9.SBA and 10.SBA, respe

173 vatization of the grafted species, thiol and sulfonic acid modified silicas are obtained.

174 Poly(4-amino-3-hydroxynaphthalene sulfonic acid)-modified glassy carbon electrode (poly(AH

175 phosphate buffer with 3-(N-morpolino)propane sulfonic acid (MOPS) led to increased interparticle inte

176 f sulfur dioxide and increase that of methyl sulfonic acid (MSA), which is needed to close the gap be

177 hydrazine, hydroxyl, phenydicarboxy, phenyl, sulfonic acid, n-butyl, diphenylcyclopropyl, and hydrazi

178 (6-sulfonaphthalen-2-yl)diazenyl-quinoline-5-sulfonic acid (NSC-87877) as a potent Shp2 PTP inhibitor

179 dation of the thiol group at position 315 to sulfonic acid occurring via a sulfenic acid intermediate

180 e-3, benzophenone-4, 2-phenylbenzimidazole-5-sulfonic acid, octocrylene, octylmethoxycinnamate, butyl

181 on of tyrosines 151 and 472 and oxidation to sulfonic acid of cysteines 126 and 385.

182 rmined as sulfinic acid on alpha-Cys-104 and sulfonic acid on alpha-Cys-104, beta-Cys-93, and beta-Cy

183 Colitis was induced with trinitrobenzene sulfonic acid or dextran sulfate sodium, and the intensi

184 e impermeant quencher, 2,4,6-trinitrobenzene sulfonic acid, or lipid-based quenchers that are restric

185 f ion transport in mice with trinitrobenzene sulfonic acid- or dextran sodium sulfate-induced colitis

186 ures of Prxs in Cys-sulfenic, -sulfinic and -sulfonic acid oxidation states known, the structural ste

187 ring 2,2'-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) oxidation.

188 d onto the poly-(4-amino-3-hydroxynapthalene sulfonic acid) (p-AHNSA) modified edge plane pyrolytic g

189 ds, and the agonist activity of piperidine-4-sulfonic acid (P4S) were determined.

190 ly(3-aminobenzoic acid-co-2-methoxyaniline-5-sulfonic acid), PABMSA]-modified electrodes.

191 leneglycol (PEG-SWNTs) or m-polyaminobenzene sulfonic acid (PABS-SWNTs).

192 Poly(aminobenzene sulfonic acid) (PABS) and polyethylene glycol (PEG) were

193 Here, a new route to thiol/sulfonic acid paired catalysts is reported.

194 ferentially decreased the GABA, piperidine-4-sulfonic acid (partial agonist), and SR-95531 sensitivit

195 The thiol agent p-chloromercuribenzene sulfonic acid (pCMBS) and the active metabolites from an

196 ven PFCAs (C8-C14), PFOS and perfluorodecane sulfonic acid (PFDS) showed significantly increasing tre

197 uorodecanoic acid (PFDA) and perfluorodecane sulfonic acid (PFDS).

198 tal exposures to PFOS, PFOA, perfluorohexane sulfonic acid (PFHxS), and perfluoronanoic acid (PFNA) i

199 octane sulfonic acid (PFOS), perfluorohexane sulfonic acid (PFHxS), and perfluorononanoic acid (PFNA)

200 luoroheptanoic acid (PFHpA), perfluorohexane sulfonic acid (PFHxS), perfluorodecanoic acid (PFDA) and

201 Perfluorohexane sulfonic acid (PFHxS), perfluorooctane sulfonic acid (PF

202 orononanoic acid (PFNA), and perfluorohexane sulfonic acid (PFHxS), with self-reported lifetime asthm

203 st serum concentrations were perfluorooctane sulfonic acid (PFOS) and perfluorooctanoic acid (PFOA).

204 shown that concentrations of perfluorooctane sulfonic acid (PFOS) in men are higher than in women.

205 Exposure to perfluorooctane sulfonic acid (PFOS) or to perfluorooctanoic acid (PFOA)

206 FCAs; C6-15) along with six perfluoro-octane sulfonic acid (PFOS) precursors and 11 polyfluoroalkyl p

207 Perfluorooctane sulfonic acid (PFOS) was the dominant compound accountin

208 uorooctanoic acid (PFOA) and perfluorooctane sulfonic acid (PFOS) were previously detected (>/= 4 ng/

209 rfluorooctanoic acid (PFOA), perfluorooctane sulfonic acid (PFOS), perfluorohexane sulfonic acid (PFH

210 rfluorooctanoic acid (PFOA), perfluorooctane sulfonic acid (PFOS), perfluorononanoic acid (PFNA), and

211 exane sulfonic acid (PFHxS), perfluorooctane sulfonic acid (PFOS), perfluorooctanoic acid (PFOA), and

212 route for human exposure to perfluorooctane sulfonic acid (PFOS).

213 Two target sulfonic acids [PFOS and perfluorohexanesulfonic acid (P

214 progress and developments on perfluorinated sulfonic-acid (PFSA) membranes have been summarized on m

215 c acids (PFCAs), C4 and C6-C8 perfluoroalkyl sulfonic acids (PFSAs) and two neutral precursors perflu

216 xylic acids (PFCAs) and three perfluorinated sulfonic acids (PFSAs) were analyzed by HPLC-MS/MS.

217 yl carboxylic acids (PFCAs), perfluoroalkane sulfonic acids (PFSAs), fluorotelomer alcohols (FTOHs),

218 7 PFAS classes [i.e., PFCAs, perfluoroalkane sulfonic acids (PFSAs), fluorotelomer alcohols (FTOHs),

219 lemot eggs were analyzed for perfluoroalkane sulfonic acids (PFSAs; C4,6,8,10) and perfluoroalkyl car

220 lic acids (PFCAs; C6-C15), 4 perfluoroalkane sulfonic acids (PFSAs; C4,C6,C8,C10) and perfluorooctane

221 quercetin pentaphosphate (QPP) and quercetin sulfonic acid (QSA).

222 ) and 2,2'-azinobis(3-ethylbenzothiazoline-6-sulfonic acid) radical cation (ABTS(+)).

223 high 2,2'-azino-bis (3-ethylbenzthiazoline-6-sulfonic acid) radical scavenging activity (EC50 17.52+/

224 ABTS (2,2'-azinobis(3-ethylbenzothiazoline-6-sulfonic acid)) radical medium.

225 by an indirect colorimetric (trinitrobenzene sulfonic acid) reaction, which detects loss of free amin

226 Hydrogen chloride and trifluoromethane sulfonic acid readily add to the symmetrically substitut

227 ((((2-acetyl)amino)ethyl)amino)naphthalene-1-sulfonic acid reporter groups attached to L76C and Y124C

228 g to TE is by introducing negatively charged sulfonic acid residues near the positively charged clust

229 oordination of Lewis acids to carboxylic and sulfonic acids results in a significant increase in the

230 2-[(4'-iodoacetamide)-aniline]naphthalene-6-sulfonic acid showed a decrease in myofilament Ca(2+) se

231 hREG3A and exposed to 2,4,6-trinitrobenzene sulfonic acid showed less colon damage and inflammation

232 -((iodoacetyl)amino)ethyl)aminonaphthalene-1-sulfonic acid showed that PLB decreased the amplitude of

233 ran sulfate sodium (DSS) and trinitrobenzene sulfonic acid showed that TG FVB/6 mice were more suscep

234 with fluorescent marker (anilinonaphthalene sulfonic acid) shows partitioning of hydrophobic protein

235 gs with shorter, but not longer, alkyl amino sulfonic acid side chains.

236 oxidized by UVA light to render guanine-S(6)-sulfonic acid ((SO3H)G).

237 organic linkers with two functional groups, sulfonic acid (-SO3H, S) and ammonium (-NH3(+), N), are

238 hydrochloride/hydroxy-2,5-dioxopyrrolidine-3-sulfonic acid sodium salt).

239 charged small molecule (9,10-anthraquinone-2-sulfonic acid sodium salt, AQ), through tuning of the ch

240 TG mice by administration of trinitrobenzene sulfonic acid; some mice were given intrarectal injectio

241 irreversible inactivation to the sulfinic or sulfonic acid species.

242 esolution, despite continued trinitrobenzene sulfonic acid stimulation.

243 thiol modifications other than oxidation to sulfonic acid, such as S-glutathionylation, also decreas

244 is was induced in mice using trinitrobenzene sulfonic acid; symptoms were monitored as a function of

245 crystalline sulfonic acids: 2-aminoethane-1-sulfonic acid (taurine, T), 3-aminopropane-1-sulfonic ac

246 After hydrolysis with trifluoroacetic and sulfonic acids, the desired optically pure l-[5-(11)C]-g

247 ulfur atom is described, including seven 1-C-sulfonic acids, three sulfonate esters, three sulfinate

248 Trinitrobenzene sulfonic acid (TNBS) administration was used to induce c

249 n NCM460-NK-1R cells and the trinitrobenzene sulfonic acid (TNBS) and dextran sodium sulfate (DSS) mo

250 tran sulfate sodium (DSS) or trinitrobenzene sulfonic acid (TNBS) as well as nonsteroidal anti-inflam

251 (i) loss of available 2,4,6-trinitrobenzene sulfonic acid (TNBS) binding to primary amines, (ii) ele

252 ly resistant to induction of trinitrobenzene sulfonic acid (TNBS) colitis and (2) mice administered a

253 ran sodium sulfate (DSS) and trinitrobenzene sulfonic acid (TNBS) colitis models were used to evaluat

254 -alpha (TNFalpha)] in murine trinitrobenzene sulfonic acid (TNBS) colitis, most likely because of the

255 Chronic trinitrobenzene sulfonic acid (TNBS) colitis-associated intestinal fibro

256 ministered intrarectal 2,4,6-trinitrobenzene sulfonic acid (TNBS) develop inflammation and fibrosis t

257 litis was induced via a 2.5% trinitrobenzene sulfonic acid (TNBS) enema.

258 eived 20 mg/rat intracolonic trinitrobenzene sulfonic acid (TNBS) in 50% ethanol or saline.

259 Colitis, induced by trinitrobenzene sulfonic acid (TNBS) in guinea pig, leads to decreased p

260 cell helper 1 (Th1)-mediated trinitrobenzene sulfonic acid (TNBS) model of colitis.

261 given intrarectal saline or trinitrobenzene sulfonic acid (TNBS) on day 10 postinfection, and were s

262 itis was induced using 2,4,6-trinitrobenzene sulfonic acid (TNBS) or dextran sodium sulfate (DSS) and

263 tran sulfate sodium (DSS) or trinitrobenzene sulfonic acid (TNBS) to induce intestinal inflammation.

264 -null mice were subjected to trinitrobenzene sulfonic acid (TNBS) treatment.

265 k-old mice administered with trinitrobenzene sulfonic acid (TNBS) with or without neutralizing anti-T

266 sodium sulfate (DSS), 2,4,6-trinitrobenzene sulfonic acid (TNBS), and Salmonella typhimurium were us

267 nsplantation by injection of trinitrobenzene sulfonic acid (TNBS), and tissues were analyzed 1-14 day

268 B/c mice with oxazolone- and trinitrobenzene sulfonic acid (TNBS)-induced colitides by nicotine.

269 effect of baicalein on 2,4,6-trinitrobenzene sulfonic acid (TNBS)-induced colitis, which shares patho

270 nd health-compromised, 2,4,6-trinitrobenzene sulfonic acid (TNBS)-treated mice.

271 olonic instillation of 2,4,6-trinitrobenzene sulfonic acid (TNBS).

272 e sodium salt (DSS) or 2,4,6-trinitrobenzene sulfonic acid (TNBS).

273 ng dextran sodium sulfate or trinitrobenzene sulfonic acid (TNBS).

274 onic administration of 2,4,6 trinitrobenzene sulfonic acid (TNBS).

275 ntrarectal administration of trinitrobenzene sulfonic acid (TNBS).

276 ileitis by means of 2, 4, 6-trinitrobenzene sulfonic acid (TNBS)/ethanol installation, early inflamm

277 ntrarectal administration of trinitrobenzene sulfonic acid (TNBS-colitis), as well as colitis and ski

278 tion of colitis with 2, 4, 6-trinitrobenzene-sulfonic-acid (TNBS), and the course of colitis in recom

279 stration of ethanol; 2, 4, 6-trinitrobenzene sulfonic-acid (TNBS); or infection with Citrobacter rode

280 ore colitis was induced with trinitrobenzene sulfonic acid to monitor the effects of MSCs.

281 addition of HCl, HF and also phosphoric and sulfonic acids to alkenes, with complete regioselectivit

282 ed from commercially obtained [(35)S]methane sulfonic acid) to generate [(35)S]Sch225336.

283 more susceptible to DSS and trinitrobenzene sulfonic acid treatment than wild-type FVB/6 mice, as de

284 d others not so reversed, e.g., formation of sulfonic acids, Trp oxides, and Met sulfoxides.

285 An anilinonaphthalene-8-sulfonic acid uptake assay reveals two distinct modes of

286 The 8-anilinonaphthalene-1-sulfonic acid uptake experiment using Escherichia coli c

287 with 2,2'-azino-bis-(3-ethylbenzthiazoline-6-sulfonic acid) used as a substrate was 2047 U mg(-1), 20

288 The sulfonic acid was observed anchored to the zinc-coordina

289 from distal colons of rats; trinitrobenzene sulfonic acid was used to induce inflammation.

290 scent probe TNS (2-p-toluidinylnaphthalene-6-sulfonic acid) was characterized as an active site fluor

291 soxazolo[5,4-c]pyridin-3-ol and piperidine-4-sulfonic acid, we examined BDZ modulation using a simple

292 orobutane sulfonic acid, and perfluorohexane sulfonic acid were detected less frequently.

293 Among organic acids evaluated, sulfonic acids were found the most effective for urethan

294 Perfluoroalkane sulfonic acids were not volatilized, suggesting that the

295 kyl carboxylic acids and four perfluoroalkyl sulfonic acids were quantified for different pHs, contac

296 xH-containing species (sulfenic, sulfinic or sulfonic acid), which destabilizes the toxin.

297 n experiments between 8-anilino-1-naphtalene sulfonic acid, which has fluorescent properties, and var

298 oxidatic cysteine Cys52 was over-oxidized to sulfonic acid, which in turn resulted in MKP-1 oxidation

299 e chiral sulfur ylides (derived from camphor sulfonic acid) with different aryl groups were reacted w

300 1-deoxynojirimycin, and 1-deoxynojirimycin-1-sulfonic acid, with K0.5 values of 0.5 to 9 microM.