ライフサイエンスコーパス: sulprostone

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1 yryl cyclic AMP, but not by the EP3R agonist sulprostone.

2 kolin or dibutyrl-cAMP altered the action of sulprostone.

3 vators 16,16-dimethyl-prostaglandin E(2) and sulprostone, also increased mineralization.

4 trinor prostaglandin E2 (an EP1 agonist) and sulprostone (an EP3 agonist) had little effect on membra

5 The effects were mimicked by sulprostone, an agonist to PGE(2) EP(3) receptors.

6 gonist, increased MUC5AC production, whereas sulprostone, an EP1 and EP3 agonist, did not.

7 In addition, treatment of HepG2 cells with sulprostone, an EP1-selective agonist, up-regulated the

8 e pressor response to EP1-selective agonists sulprostone and 17-phenyltrinor PGE2 were blunted by SC5

9 Y10598 and not at all by the EP3/EP1 agonist sulprostone and confirmed by protein measurements of cyc

10 our prostanoid receptors, direct addition of sulprostone but not butaprost mimicked PGE2, suggesting

11 t 17-trinor-PGE(2), and the EP1/EP3 agonist, sulprostone, but not the EP2 receptor agonist, butaprost

12 E2 were mimicked by the EP3-selective agent, sulprostone, by cAMP reduction, and lost upon inhibition

13 ncentration of calcium ([Ca2+]i) by PGE2 and sulprostone demonstrated the respective involvement of E

14 Sulprostone (EP(3)>EP(1) agonist), GR63799, and ONO-AE-2

15 The EP3/EP1 R-selective agonist sulprostone, EP3/EP2/EP4 R-selective agonists M&B 28767

16 t PGE2 and misoprostol, but not butaprost or sulprostone, evoked increases in the intracellular conce

17 specific EP2 and EP3 agonists (butaprost and sulprostone) in cultured trigeminal ganglia neurons.

18 on of HSB.2 cells with 10(-8)-10(-6) M PGE2, sulprostone, M&B 28767, and misoprostol but not with 10(

19 stanoid receptors EP2 (butaprost) and EP1/3 (sulprostone) mimicked PGE2, while misoprostol (E1-4) act

20 t butaprost, but neither the EP1/EP3 agonist sulprostone nor the EP4-selective agonist ONO-AE1-329, m

21 However, sulprostone resulted in increased staining intensity and

22 Stimulation of the EP3 receptor with sulprostone results in a robust increase in JNK activati

23 Sulprostone severely compromised ChAT activity, dendrite

24 st butaprost, not the E1/E3 receptor agonist sulprostone, stimulated the expression of Cx43 and gap j

25 ter, misoprostol-free acid, misoprostol, and sulprostone suggested that a negative charge at the C1 p

26 of [3H]PGE2 binding by the EP3/EP1R agonist sulprostone, the EP3/EP2/EP4R agonist M&B 28767, and the

27 Northern analysis indicated that PGE2 and sulprostone time-dependently increased expression of MMP

28 The application of butaprost and sulprostone triggered neuropeptide exocytosis, and butap

29 The effects of sulprostone were not blocked by SC-19220, an EP(1) recep

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