ライフサイエンスコーパス: superficial

1 out higher level processing remains largely superficial.

2 eyes compared with control eyes both in the superficial (0.427 mm(2) vs. 0.275 mm(2); P < 0.001) and

3 eyes compared with control eyes in both the superficial (17.68 mm(-1) vs. 21.55 mm(-1); P < 0.001) a

4 Models A and B had solitary, superficial, 2 cm and 1 cm soft masses, respectively.

5 of the GABAA receptor is higher in layers 2/superficial 3 of the prefrontal cortex.

6 ly between earlyROA and riskROA knees in the superficial (48.0 +/- 3.5 ms vs. 48.1 +/- 3.1 ms) or the

7 RVO eyes compared to fellow eyes in both the superficial (48.07% vs 52.60%, respectively; P < .001) a

8 eyes compared with control eyes in both the superficial (49.44% vs. 55.09%; P < 0.001) and deep (56.

9 ageal mucosa of patients with NERD have more superficial afferent nerves compared with controls or pa

10 26, and 0.56 +/- 0.118, respectively, at the superficial and 0.34 +/- 0.116, 2.50 +/- 0.462, 0.69 +/-

11 hat a large number of pretectal cells in the superficial and central areas extend dendrites into the

12 these areas were compared with the areas of superficial and deep capillary flow loss on OCT angiogra

13 ion (FD), and foveal avascular zone (FAZ) of superficial and deep capillary plexus in healthy volunte

14 eable regarding VD, FD, and FAZ for both the superficial and deep capillary plexus.

15 aphy generated OCT angiograms of the retinal superficial and deep capillary plexuses.

16 udied baseline values and one-year change in superficial and deep cartilage T2 layers in 60 subjects

17 n PRC, LEC, and MEC, including those in both superficial and deep cortical areas and in grid, border,

18 0 healthy subjects underwent measurements of superficial and deep foveal and parafoveal vessel densit

19 ant differences in the binarized flow index (superficial and deep layers), both including and excludi

20 etween diabetic and normal eyes in FAZ area (superficial and deep layers), perimeter (superficial lay

21 h (superficial layer), and minor axis layer (superficial and deep layers).

22 h the pallial component further divided into superficial and deep nuclei.

23 area and parafoveal vascular density in the superficial and deep plexi.

24 greater in gigantopyramidal neurons than in superficial and deep pyramidal neurons.

25 The study found that OCTA reveals subnormal superficial and deep retinal capillary density in the ma

26 ing of the macula and flow voids in both the superficial and deep retinal capillary plexus were found

27 eal subclinical macular ischemia at both the superficial and deep retinal capillary plexuses in diabe

28 Comparison of measurements made in the superficial and deep retinal capillary plexuses of diabe

29 Reduced superficial and deep retinal capillary vessel density on

30 show significant vascular impairment at both superficial and deep retinal plexuses, correlating with

31 tically generated en face OCTA images of the superficial and deep retinal vasculatures using vessel-b

32 Culture-proven superficial and deep SSIs occurred in 3% and 15% of pati

33 ations across multiple brain regions in both superficial and deep structures.

34 on the macula and en face angiograms of the superficial and deep vascular networks were acquired.

35 nfirm decreased vascular density in both the superficial and deep vascular networks.

36 nts in the central macula, an area where the superficial and deep vascular plexuses terminate.

37 erent errors in segmentation, studies of the superficial and deep vascular plexuses using manufacture

38 nsal bacterium that has the ability to cause superficial and deep-seated infections.

39 based on somatodendritic morphology for both superficial and gigantopyramidal neurons.

40 Superficial and input layer neurons exhibit attention-de

41 utons form glomerulus-like structures in the superficial and intermediate tectal layers, establishing

42 When this protection breaks down, superficial and invasive fungal infections cause disease

43 pses from long-range projections, within the superficial and middle laminar zones of the human DLPFC.

44 PV-containing thalamic nuclei extends across superficial and middle layers of the human DLPFC.

45 To stratify superficial and organ-space SSIs after pancreatectomy an

46 ral juxtafoveal region and involved both the superficial and the deep capillary plexuses.

47 Here, we describe the superficial appearance of the brain of the rarely studie

48 vely correlated with total root length, root superficial area, and root volume.

49 on of deep short-axon cell interneurons with superficial axonal projections to the sensory input laye

50 randomized to imiquimod 5% cream once daily (superficial basal cell carcinoma, 6 weeks; nodular basal

51 e used genomic analyses to determine whether superficial behavioral similarities in humans and the hi

52 is, is widely used as adjunctive therapy for superficial bladder cancer.

53 in vivo calcium imaging has been limited to superficial brain structures during head-fixed behaviora

54 n NAA and causes "spongiform" vacuolation of superficial brain white matter and neighboring gray matt

55 These findings demonstrate that superficial CA1 PCs provide a more stable map of an envi

56 Activity of superficial CA2 cells preceded the activity surge in CA3

57 Plaques responsible for MACE had larger superficial calcium inclusions, which acted to increase

58 TLR agonists are effective at treating superficial cancerous lesions, but their use internally

59 el densities, and perfusion densities of the superficial capillary plexus (SCP) and deep capillary pl

60 ith those on SDOCT, we superimposed the OCTA superficial capillary plexus (SCP) vascular landmarks on

61 lities were observed in all patients in both superficial capillary plexus and deep capillary plexus o

62 The comparative analysis between the superficial capillary plexus and the deep capillary plex

63 Registration was first performed using superficial capillary plexus images, and this transforma

64 The mean vessel density of the superficial capillary plexus in normal fellow eyes was 1

65 ctive error, the mean (SD) difference in the superficial capillary plexus in the 6 x 6-mm scan was st

66 The macular vessel density of the superficial capillary plexus was lower in the amblyopic

67 and Eed/Ezh2 double mutants exhibit delayed superficial cell differentiation.

68 Superficial cells are self-renewing chondrocyte progenit

69 Thus, superficial cells are self-renewing progenitors that are

70 fluorescent protein retention revealed that superficial cells divide more slowly than underlying art

71 t cartilage renewal occurs as the progeny of superficial cells fully replace fetal chondrocytes durin

72 anced micro-computed tomography, showed that superficial cells generate chondrocytes and contribute t

73 oot hairs are filamentous protuberances from superficial cells of plant roots that are critical for n

74 ined with immunohistochemistry revealed that superficial cells renew their number by symmetric divisi

75 Superficial cells showed wider integration along the ton

76 t line the ureter fail to differentiate into superficial cells, which are responsible for producing u

77 ent of PRC2, causes upregulation of Upk3a(+) superficial cells.

78 mposed of layers of basal, intermediate, and superficial cells.

79 te cells differentiated into basal cells and superficial cells.

80 on cellular location and projection pattern: superficial, central, and periventricular.

81 In this study, we found no superficial changes in Rbfox2 expression in the SCN, but

82 nce to support delaying manual dermabrasion, superficial chemical peels, cutaneous surgery, laser hai

83 , metamorphosed or degraded, displaying some superficial chemical similarities to abiotic meteoritic

84 ariate analysis, combined hemangiomas with a superficial component and a step border were associated

85 angiomas with a step or abrupt border of the superficial component left more severe sequelae than tho

86 anetodermal skin, redundant skin, persistent superficial component, and the degree of sequelae rangin

87 that may be confused with infections of the superficial cornea, such as herpes simplex virus keratit

88 Nevertheless, these neurons localize at more superficial cortical layers than their control counterpa

89 latory activity is predominantly observed in superficial cortical layers, whereas alpha- and beta-ban

90 We achieved implantation into a superficial cortical vein overlying the motor cortex via

91 mcinolone, alone or in combination with open superficial cryosurgery.

92 riamcinolone; 2 patients also underwent open superficial cryosurgery.

93 ean [SEM] expression, 5.06 [1.27]; P = .05), superficial cSCC (mean [SEM] expression, 3.58 [1.50]; P

94 gymnotiform fish, Apteronotus leptorhynchus: superficial (DDs), intermediate (DDi) and magnocellular

95 Superficial, deep-incisional, and organ-space SSIs, as d

96 Their presence in large numbers in the superficial dermis, where Leishmania is encountered, sug

97 naptic length of R-profiles within the Vc/C2 superficial dorsal horn (lamina I) 3 weeks post-CCI-ION.

98 se in the excitability of neurons within the superficial dorsal horn (SDH) of the spinal cord is thou

99 ly understood, as nothing is known about how superficial dorsal horn neurons process sensory input fr

100 tracellular signal-regulated kinase (ERK) in superficial dorsal horn neurons.

101 old and heat are initially integrated in the superficial dorsal horn of the spinal cord.

102 Each type was counted within the superficial dorsal horn of the Vc/C2 and the means from

103 in the number of excitatory synapses in the superficial dorsal horn of Vc/C2 could lead to enhanced

104 pressed by primary afferent terminals in the superficial dorsal horn that co-expressed the neuropepti

105 2 (stargazin) is present in lamina II of the superficial dorsal horn, an area involved in nociception

106 ies of glutamatergic transmission within the superficial dorsal horn.

107 nfGNPs-VLPs is not mediated by the opposing superficial electrostatic charges, suggesting that non-e

108 ee of mixed selectivity and heterogeneity in superficial entorhinal neurons.

109 reversed when saline was reinfused into the superficial epigastric artery (21 +/- 2 mmHg; P < 0.01 v

110 used a superoxide scavenger, tiron, into the superficial epigastric artery of decerebrated rats.

111 ulticolor system, skinbow, that barcodes the superficial epithelial cell (SEC) population of zebrafis

112 hogens whose primary ecological niche is the superficial epithelial layers of the throat and/or skin.

113 RATIONALE: Superficial erosion currently causes up to a third of ac

114 2 signaling as mechanisms that contribute to superficial erosion, a cause of acute coronary syndrome

115 cial intimal injury, a process implicated in superficial erosion.

116 Interobserver agreement was high for all superficial FAZ measurements (ICC >/=0.90) but did not m

117 light by tissue limits the imaging depth to superficial features.

118 symptomatic peripheral artery disease of the superficial femoral and popliteal arteries.

119 N) has shown to improve vessel patency after superficial femoral angioplasty.

120 ve provided new options for the treatment of superficial femoral artery disease; however, the compara

121 Atherosclerosis in the superficial femoral artery is common in patients sufferi

122 Treatment of de novo superficial femoral artery lesions with PEB angioplasty

123 r Study to Evaluate Treatment of Obstructive Superficial Femoral Artery or Popliteal Lesions With A N

124 tic peripheral artery disease due to de novo superficial femoral artery stenotic or occlusive lesions

125 am group or for carotid arterial compliance, superficial femoral intima media thickness or endotheliu

126 ere performed on seven lower limb (including superficial femoral, deep femoral and popliteal) artery

127 iver catchments with contrasting bedrock and superficial geologies, N2O and nitrate (NO3(-)) concentr

128 ed differences in the activation of deep and superficial granule cells.

129 hould select nonpharmacologic treatment with superficial heat (moderate-quality evidence), massage, a

130 he developed sensor can be attributed to its superficial highly matched imprinted cavities on the exc

131 , with 3 crack patterns identified: Type I - superficial horizontal cracks; Type II - vertically (occ

132 CGRP-positive nerves were significantly more superficial in mucosa from patients with NERD-both dista

133 ginating prions was ephemeral and adaptation superficial in TgEq and TgD.

134 The primary outcome was SSI, defined as any superficial incisional, deep incisional, or organ/space

135 an asymptomatic lifestyle or as an agent of superficial infections.

136 ivity in retinal ganglion cell terminals and superficial inhibitory interneurons in the tectum during

137 ntiation primary response gene 88), or after superficial injury by blotting with tissue paper.

138 After superficial injury without thrombin generation, no multi

139 segmentation algorithm divided PR-OCTA into superficial, intermediate, and deep retinal plexuses.

140 al cardinal, spinal, superficial lateral and superficial intersegmental lymphatics.

141 scopy, CLIO-CyAm7 primarily deposited in the superficial intima within plaque macrophages, endothelia

142 Thrombi because of superficial intimal erosion characteristically complicat

143 uronan, and Toll-like receptor 2 ligation in superficial intimal injury, a process implicated in supe

144 ral keratitis, the linear form of Thygeson's superficial keratitis, epithelial regeneration line, Aca

145 Cells in superficial L2 show more extensive columnar integration

146 I PV-IR synapses were not identified in the superficial laminar zone.

147 ater-developing collateral cardinal, spinal, superficial lateral and superficial intersegmental lymph

148 SLEs in the piriform cortex initiate in the superficial layer 1 lacking principal neurons with an ac

149 ature PFC, both ChCs and BCs are abundant in superficial layer 2, but only BCs are present in deeper

150 lasticity impairment is first observed in EC superficial layer and further affected with time.

151 15.5 subventricular zone, during the peak of superficial layer CPN birth, with a progressive postmito

152 itors to both broadly regulate generation of superficial layer CPN throughout the neocortex, and to r

153 in obese adolescents characterized by a thin superficial layer of abdominal subcutaneous adipose tiss

154 tina and visual cortex and converge onto the superficial layer of the SC (sSC).

155 ound a large percentage of neurons in the V1 superficial layer responded more strongly to complex pat

156 r ventral sensory center (AVSC), occupying a superficial layer within the ventromedial tritocerebrum.

157 eter (superficial layer), major axis length (superficial layer), and minor axis layer (superficial an

158 ea (superficial and deep layers), perimeter (superficial layer), major axis length (superficial layer

159 receives visual input from the retina in its superficial layers (sSC) and induces eye/head-orientatin

160 nd earlier decision-related responses in the superficial layers and more action execution-related sig

161 ity with brain areas that target its deep vs superficial layers and thereby contribute to the behavio

162 depression of layer V synapses arising from superficial layers consistent with enhanced neurotransmi

163 Broad-spiking neurons in the superficial layers exhibit attention-mediated increases

164 ayer 13 in the avian tectum and in the lower superficial layers in the mammalian colliculus.

165 "Increased" neurons were more prevalent in superficial layers of dorsal premotor cortex; deeper lay

166 ence for somatosensory and premotor input in superficial layers of M1 and for cortico-spinal motor ou

167 evealed in a new analysis of recordings from superficial layers of macaque primary visual cortex.

168 In the superficial layers of the superior colliculus of the mid

169 enhanced recruitment of inputs arising from superficial layers to layer V.

170 in 15 layers where visual input targets the superficial layers while auditory input terminates in de

171 ng and increased power at gamma frequency in superficial layers.

172 e investigated the structural development of superficial-layers of medial entorhinal cortex and paras

173 s of tissue penetration that restrict PDT to superficial lesions, inability to treat hypoxic tumours,

174 positive neurons in the medial aspect of the superficial medullary and spinal dorsal horn from the tr

175 of a 50muL bolus appears to create multiple superficial microdisruptions in the papillary dermis and

176 the vein of Labbe, sphenoparietal sinus, and superficial middle cerebral vein was graded by one neuro

177 More superficial models of language, based solely on sequenti

178 Here, we show that a specific subset of superficial mPFC projections to a subfield of nucleus ac

179 e activity dynamics of both single cells and superficial neuropil distributed across the majority of

180 nstrated no hyperfluorescence, and eyes with superficial ODD showed nodular staining.

181 ), and 3 children (6 eyes) with PPE owing to superficial ODD were included.

182 s the presence or absence of SSI, defined as superficial or deep SSI, within 30 days postdischarge.

183 topical imiquimod compared with surgery for superficial or nodular basal cell carcinoma at low-risk

184 olonisation was substantially greater in the superficial organic layer compared with the mineral soil

185 Incompleteness of the superficial palmar arch (SPA) was present in 46%, the de

186 The "superficial parasubiculum" (layers 2 and 1) divides into

187 detected in the livers, kidneys, spleens and superficial parotid lymph nodes of the mice.

188 io-Emotion (ASE) model reduces sentiments to superficial patterns of emotional responding that emerge

189 le activity chronically and to stimulate the superficial peroneal nerve electrically to evoke cutaneo

190 RCs of human sympathetic nerve fibres of the superficial peroneal nerve innervating the dorsum of the

191 Specifically, the chorda tympani and greater superficial petrosal nerve terminal fields were 1.4x and

192 through the chorda tympani (27%) and greater superficial petrosal nerves (15%) expressed Phox2b durin

193 lossopharyngeal, chorda tympani, and greater superficial petrosal nerves were labeled to examine thei

194 to rely on the peculiar organization of the superficial piriform cortex layers, which are characteri

195 ptical coherence tomography demonstrate that superficial plaque erosion is more common than previousl

196 Major superficial plexiform neurofibromas and symptomatic spin

197 of uniform density and caliber, whereas the superficial plexus revealed vessels in the familiar cent

198 FVD and PFVD of the superficial plexus were not significantly lower in CRVO

199 electively in pyramidal cells located in the superficial portion of the CA1 pyramidal cell layer, whe

200 (primarily layer V) pyramidal (n = 203) and superficial (primarily layer III) pyramidal (n = 233) ne

201 made up of distinct deep (subplate-like) and superficial (principal) parts.

202 presented with corneal findings ranging from superficial punctate epitheliopathy to bilateral corneal

203 eniscus height, Schirmer test I, presence of superficial punctate keratopathy (SPK), LG volume, and m

204 divided into 2 subtypes: semilunar (SL) and superficial pyramidal (SP) cells.

205 e leukocyte infiltration was observed in the superficial retina.

206 OCTA cube scans per eye were obtained and 9 superficial retinal layer (SRL) and deep retinal layer (

207 Density maps based on superficial retinal layer (SRL) and deeper retinal layer

208 Retinal vasculature was assessed in the superficial retinal layer (SRL), deep retinal layer (DRL

209 t of the deep retinal plexus compared to the superficial retinal plexus due to ischemia that did not

210 ogMAR visual acuity and FAZ area in both the superficial (rho = 0.29; P < 0.01) and deep (rho = 0.48;

211 The superficial SC layers (sSC) contain cells that respond t

212 To evaluate vascular abnormalities at superficial (SCP) and deep (DCP) capillary plexuses and

213 rcles around telangiectasias, projected over superficial (SCP) and deep capillary plexuses (DCP).

214 alize areas of capillary nonperfusion at the superficial (SCP) or DCP, we used the spectral-domain op

215 roplastic contamination is ubiquitous within superficial sediments and bottom water along the western

216 se without, especially disseminated cortical superficial siderosis (49% vs 19%; p<0.0001).

217 bar cerebral microbleeds (CMBs) and cortical superficial siderosis (CSS) are the characteristic marke

218 for probable CAA were analysed for cortical superficial siderosis (focal, </=3 sulci; disseminated,

219 Central nervous system infratentorial superficial siderosis (iSS) is increasingly detected by

220 Disseminated superficial siderosis (subhazard ratio [SHR] 7.45, 95% C

221 multivariable logistic regression, cortical superficial siderosis burden (OR 5.53; 95% CI 2.82 to 10

222 Cortical superficial siderosis prevalence (but no other CAA sever

223 We compared clinical, imaging and cortical superficial siderosis topographical mapping data between

224 are medical center, the presence of cortical superficial siderosis was an independent variable associ

225 The presence of cortical superficial siderosis was an independent variable associ

226 Cerebral microbleeds, cortical superficial siderosis, and white matter hyperintensity v

227 trictly lobar cerebral microbleeds, cortical superficial siderosis, centrum semiovale perivascular sp

228 ted for lobar cerebral microbleeds, cortical superficial siderosis, centrum semiovale perivascular sp

229 ividual focal lesions (microbleeds, cortical superficial siderosis, microinfarcts) and large-scale al

230 the overlap between acute cSAH and cortical superficial siderosis-a new CAA haemorrhagic imaging sig

231 neurological episodes compared with cortical superficial siderosis-positive, but cSAH-negative subjec

232 ermined by the presence of cSAH and cortical superficial siderosis.

233 cterial biofilm in vivo in a murine model of superficial skin infection.

234 < 0.01) and 1.3% (P < 0.01) for SSI rates in superficial space, deep space, and organ space, respecti

235 Transcriptional changes in superficial spinal dorsal horn neurons (SSDHN) are essen

236 d in central processes of these cells in the superficial spinal dorsal horn.

237 , were significantly less likely to have the superficial spreading histologic subtype (P = 0.01), occ

238 were less likely to have BRAF mutations or a superficial spreading histologic subtype (P = 0.05) comp

239 e features can share some of the criteria of superficial spreading melanoma and some of facial lentig

240 d texture was an independent risk factor for superficial SSI (OR, 1.45; 95% CI, 1.14-1.85; P = .002)

241 confirmed as an independent risk factor for superficial SSI (OR, 2.07; 95% CI, 1.58-2.71; P < .001).

242 Organ/space but not superficial SSI was associated with an increased rate of

243 Independent risk factors for superficial SSI were preoperative biliary stenting (odds

244 eived MBP plus OABP had a lower incidence of superficial SSI, deep SSI, organ space SSI, any SSI, ana

245 ry stenting and coriticosteroid use increase superficial SSI, even in patients receiving perioperativ

246 ative, 2057 (19.8%) had any SSI; 719 (6.9%), superficial SSI; 207 (2%), deep-incisional SSI; and 1287

247 years]), 58 had any SSI (28.9%); 28 (13.9%), superficial SSI; 8 (4%), deep-incisional SSI; and 24 (11

248 Recipients with superficial SSIs and those without SSIs were similar in

249 ikewise express Nr4a2 and occupy a corticoid superficial stratum of the mesopallium, which is clearly

250 Histologically, there was a variable superficial stromal lymphocytic infiltrate, involving th

251 from anterior to posterior, and from deep to superficial subcortical white matter.

252 -0.30, -0.03 cm), lower volumes of abdominal superficial subcutaneous adipose tissue (-4.53 mL; 95% C

253 ith improved insulin sensitivity, and losing superficial subcutaneous adipose tissue remained neutral

254 e dynamics of different fat depots (deep and superficial subcutaneous, liver, pericardial, muscle, pa

255 gnostics for the routine detection of HSV in superficial swab specimens.

256 g within slow-cycling cell niches at the (i) superficial taste/tooth junction (T/TJ), and (ii) deep s

257 most common anatomical site of isolation was superficial tissue (66.5%), followed by the respiratory

258 alysis demonstrated that lesion location was superficial to areas of abnormally high microsphere depo

259 From superficial to deep, there is the subcutaneous layer, wh

260 gi are becoming more frequent and range from superficial to systemic dissemination.

261 tended arrhythmic substrate, whether deep or superficial, to be visualized immediately and ablated ir

262 olution of oxide formation has only received superficial treatment.

263 iO2 NPs effectively suppressed the growth of superficial tumours after ultrasound treatments.

264 ase, invasive squamous cell carcinoma, and a superficial type of basal cell carcinoma (BCC).

265 acuity and the vascular density in both the superficial (VAD, rho = -0.52; VLD, rho = -0.54; P < 0.0

266 uitment in human venous stasis, we show that superficial varicose veins preferentially contain activa

267 Four en face OCTA slabs were analyzed: the superficial vascular complex (SVC), intermediate capilla

268 in the intermediate capillary plexus and the superficial vascular complex, but these changes were sma

269 but not the intermediate capillary plexus or superficial vascular complex.

270 d reliable method for quantifying FAZ at the superficial vascular network.

271 ndex (CDI) and fractal dimension (FD) at the superficial vascular plexus (SVP) and deep retinal vascu

272 the laminin gamma3-null (Lamc3(-/-)) retinal superficial vascular plexus and consequently the vascula

273 he segmentation slab designed to isolate the superficial vascular plexus included the deep vascular p

274 onary embolism, progression or recurrence of superficial vein-thrombosis, and all-cause mortality at

275 onary embolism, progression or recurrence of superficial vein-thrombosis, and all-cause mortality, an

276 nderlying pathology is thrombophlebitis of a superficial vein.

277 c thrombosis (at least 5 cm in a supragenual superficial-vein segment) and at least one additional ri

278 oxaban could offer patients with symptomatic superficial-vein thrombosis a less burdensome and less e

279 compared efficacy outcomes in patients with superficial-vein thrombosis and additional risk factors

280 Superficial-vein thrombosis can lead to deep-vein thromb

281 ents aged 18 years or older with symptomatic superficial-vein thrombosis from 27 sites (academic, com

282 on-inferior to fondaparinux for treatment of superficial-vein thrombosis in terms of symptomatic deep

283 ajectories between ChCs and BCs, and between superficial versus deep-layer BCs.

284 vascular depth discrimination is limited by superficial vessels projecting flow signal artifact onto

285 ies, many of which have been associated with superficial, visceral, or systemic infections in humans,

286 We find that the activity of SC superficial visual-layer neurons (SCs), specifically, is

287 lier for V1 neurons than superior colliculus superficial visual-layer neurons (SCs), the saliency rep

288 Non-recovered patients showed widespread superficial white matter damage in comparison to recover

289 AR encephalitis is associated with extensive superficial white matter damage in patients with incompl

290 significantly correlated with damage of the superficial white matter in patients.

291 performed between cognitive performance and superficial white matter integrity.

292 RI data were collected from all subjects and superficial white matter mean diffusivity derived from d

293 In contrast, the superficial white matter was intact in recovered patient

294 able brain compartment, the late myelinating superficial white matter.

295 Superficial wound complication was the most common wound

296 Serious adverse events included a superficial wound infection in one patient that resolved

297 ling and enabling ingress of bacteria from a superficial wound to deep tissue.

298 y, AQP3(-/-) mice showed impaired healing of superficial wounds in the colon and impaired mucosal inn

299 stry detected high expression of PHD2 in the superficial zone (SZ), while PHD3 and HIF-1alpha (target

300 entire sample and specific cartilage zones (superficial zone [SZ], transitional zone [TZ], and deep