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1 lls were then superinfected with VSV or mock superinfected.
2 ith a complex ACD recombinant strain, became superinfected 6 to 9 months later with an AC recombinant
4 logous B. burgdorferi clones to successfully superinfect a mouse host, primary- and secondary-infecti
7 DNA in the serum samples collected from all superinfected animals during weeks one through six after
8 aive hosts by ticks previously fed either on superinfected animals or on animals singly infected with
10 rease in primary structural diversity in the superinfected animals, and (iii) the increase in primary
11 observed in sera and lung lavage fluid from superinfected animals, suggesting that G-CSF is a major
15 ld-type B31-A3 clone in order to distinguish superinfecting B. burgdorferi from primary-infecting spi
17 acid Nun protein of prophage HK022 excludes superinfecting bacteriophage lambda by blocking transcri
18 ic complementation assay in which tumors are superinfected before dox withdrawal with other RCAS viru
19 examined NAb responses in 6 women who became superinfected between approximately 1 to 5 years followi
21 of 6 SIV239Deltanef-immunized monkeys became superinfected by challenge virus mismatched in its Env s
22 nd phylogenetic analyses suggest CT02 became superinfected by CT01 with subsequent production of a re
24 in chronically infected patients who become superinfected by other hepatotropic viruses; they sugges
25 p and two in the Delta4 group clearly became superinfected by the challenge virus, but these animals
26 s, growth of L. pneumophila was as robust in superinfected cell cultures as in those singly infected.
27 mathematical model that dynamic stability of superinfected cell growth is crucial in determining the
28 apoptosis is correlated with its ability to superinfect cells and that this occurs as an early step
30 ), proliferative virus principally occurs as superinfected cells (wild type with defective deletion m
31 ective deletion mutants can be replicated in superinfected cells by parasitism of the intact virus' r
32 nt revealed that in both transfected and VZV-superinfected cells it is a fusion of two unidirectional
38 ift mutation could be isolated readily after superinfecting EBV-positive cell lines, but not if recom
44 nfection, individuals who would later become superinfected had significantly weaker NAb activity agai
45 t that the replication space occupied by the superinfecting hepadnavirus in chronically infected live
46 an inability of homologous B. burgdorferi to superinfect immunocompetent mice as opposed to heterolog
47 cell types that vMA-1c infected but did not superinfect, indicating that the entry pathway used by v
54 oma (BL) cells that maintain latency I, when superinfected, initially supported transcription from th
55 n which relatively high titers of virus were superinfected into the eyes of latently infected rabbits
59 ular inflammatory infiltrate in the lungs of superinfected mice compared to singly infected animals.
66 e structures, recent studies have shown that superinfecting Mycobacterium marinum traffic rapidly to
69 CD4 levels, virus burden, and the ability to superinfect peripheral blood mononuclear cells in vitro
71 cipient strain which represses expression of superinfecting phage genomes and minimizes the contribut
72 otein expressed from prophage HK022 excludes superinfecting phage lambda by arresting transcription o
73 a virulence factor but functions to exclude superinfecting phage, possibly by blocking the injection
78 These data suggest that the ability of a superinfecting strain of HIV to overcome preexisting imm
79 In conclusion, the rapid overgrowth of a superinfecting strain of HIV-1 of the same subtype raise
82 train-specific nested PCR confirmed that the superinfecting strain was not present until the 9 month
84 intercompartment dynamics of the initial and superinfecting strains and recombinants derived from the
87 dogene repertoire would be distinct in these superinfecting strains, consistent with encoding differe
89 At various times after primary infection, we superinfected T cells in vitro by exposure to a genetica
92 ally distinct A. marginale strain capable of superinfecting the mammalian host can subsequently be co
94 n the generation of a virus that was able to superinfect these cells, presumably by the use of a nove
95 sidue CDR H3, and neutralized the virus that superinfected this individual 15 weeks after initial inf
96 eficiency virus (HIV) depends on whether the superinfecting transmission resembles primary infection,
102 uch as ED cells that EIAV(vMA-1c) is able to superinfect, viral entry is pH independent and appears t
103 cellular machinery, induce the repulsion of superinfecting virions away toward uninfected cells.
106 mine the specificity of the NAbs against the superinfecting virus, these variants were cloned from fi
108 atency III program of transcription from the superinfecting-virus genomes, failing to transition to l
111 cted with a plasmid encoding procaspase 3 or superinfected with a proapoptotic mutant virus lacking t
114 otein fused to green fluorescent protein and superinfected with an HSV-1 mutant lacking the U(S)8-12
117 Procaspase-3 levels remained unaltered if superinfected with Bac-U(S)3 at 3 h after d120 mutant in
118 nt amounts of procaspase-3 remained in cells superinfected with Bac-Us3 at 9 h postinfection with d12
120 transmission settings, individuals are often superinfected with complex mixtures of genetically disti
121 occurring after seroconversion: 2 IDUs were superinfected with different HCV genotypes, and 3 were s
123 ns show that HCV-infected individuals can be superinfected with different strains, and this event may
125 ted with different HCV genotypes, and 3 were superinfected with divergent strains of the same genotyp
126 ted with woodchuck hepatitis virus (WHV) are superinfected with HDV, they produce HDV with a WHV enve
127 viruses expressing U(L)47 or U(S)11 and then superinfected with HSV-1 under conditions that blocked D
128 mature C. burnetii replication vacuoles were superinfected with L. pneumophila, and then the acidity,
129 ding of a chronically infected host becoming superinfected with MDR HIV-1 with subsequent formation o
131 ES individuals remained healthy and were not superinfected with other HIV-1 strains despite continued
134 LB/cJ mice infected with influenza virus and superinfected with S. pneumoniae were treated with eithe
137 macentor andersoni ticks were fed on animals superinfected with the Anaplasma marginale subsp. centra
138 8+ T cell clearance of CD4+ T cells that are superinfected with the HIV-1 strain JR-CSF or infected w
144 of the livers and HCCs collected from three superinfected woodchucks, and (iii) finding WHVNY DNA re
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