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1 lls were then superinfected with VSV or mock superinfected.
2 ith a complex ACD recombinant strain, became superinfected 6 to 9 months later with an AC recombinant
3 ility of homologous B. burgdorferi clones to superinfect a host has not been studied in detail.
4 logous B. burgdorferi clones to successfully superinfect a mouse host, primary- and secondary-infecti
5  results in the production of Pf4 phage that superinfect and kill cells or inhibit their growth.
6                                              Superinfected animals could serve as a reservoir for onw
7  DNA in the serum samples collected from all superinfected animals during weeks one through six after
8 aive hosts by ticks previously fed either on superinfected animals or on animals singly infected with
9      This phenomenon in mice was observed in superinfected animals undergoing an active viral infecti
10 rease in primary structural diversity in the superinfected animals, and (iii) the increase in primary
11  observed in sera and lung lavage fluid from superinfected animals, suggesting that G-CSF is a major
12 pressed in the spleens, lungs, and brains of superinfected animals.
13 oires between the strains that were found in superinfected animals.
14 tative of the strains that were found in the superinfected animals.
15 ld-type B31-A3 clone in order to distinguish superinfecting B. burgdorferi from primary-infecting spi
16                                     Lysis of superinfecting bacteria by ampicillin caused an extensiv
17  acid Nun protein of prophage HK022 excludes superinfecting bacteriophage lambda by blocking transcri
18 ic complementation assay in which tumors are superinfected before dox withdrawal with other RCAS viru
19 examined NAb responses in 6 women who became superinfected between approximately 1 to 5 years followi
20                                              Superinfecting BVDV failed to deliver a translatable gen
21 of 6 SIV239Deltanef-immunized monkeys became superinfected by challenge virus mismatched in its Env s
22 nd phylogenetic analyses suggest CT02 became superinfected by CT01 with subsequent production of a re
23  sexually transmitted infections, and may be superinfected by HIV.
24  in chronically infected patients who become superinfected by other hepatotropic viruses; they sugges
25 p and two in the Delta4 group clearly became superinfected by the challenge virus, but these animals
26 s, growth of L. pneumophila was as robust in superinfected cell cultures as in those singly infected.
27 mathematical model that dynamic stability of superinfected cell growth is crucial in determining the
28  apoptosis is correlated with its ability to superinfect cells and that this occurs as an early step
29                       It was not possible to superinfect cells that failed to express HLA-DR unless e
30 ), proliferative virus principally occurs as superinfected cells (wild type with defective deletion m
31 ective deletion mutants can be replicated in superinfected cells by parasitism of the intact virus' r
32 nt revealed that in both transfected and VZV-superinfected cells it is a fusion of two unidirectional
33 , nucleolus, and cytoplasm in transfected or superinfected cells.
34 sm of the wild-type replication machinery in superinfected cells.
35  of expression of luciferase activity in the superinfected cells.
36 etions attenuated CPE in FeT-cells caused by superinfecting cytopathic virus.
37                                  Attempts to superinfect different types of immunodeficient mice with
38 ift mutation could be isolated readily after superinfecting EBV-positive cell lines, but not if recom
39 with an additional, unidentified receptor to superinfect ED cells.
40 f vMA-1c suggested that this strain might be superinfecting equine fibroblasts.
41 e was observed in comparisons of single- and superinfected females.
42 ophilus strains, and other bacteria known to superinfect genital ulcers.
43  density-gradient centrifugation and used to superinfect Giardia trophozoites.
44 nfection, individuals who would later become superinfected had significantly weaker NAb activity agai
45 t that the replication space occupied by the superinfecting hepadnavirus in chronically infected live
46 an inability of homologous B. burgdorferi to superinfect immunocompetent mice as opposed to heterolog
47  cell types that vMA-1c infected but did not superinfect, indicating that the entry pathway used by v
48 monoclonal antibodies (mAbs) isolated from a superinfected individual.
49 e, we characterize the genetic bottleneck in superinfected individuals for the first time.
50                                     Although superinfected individuals had less NAb breadth than matc
51                            In addition, most superinfected individuals had NAbs that could neutralize
52                                Moreover, the superinfected individuals were able to mount autologous
53 e response strengthens significantly in some superinfected individuals.
54 oma (BL) cells that maintain latency I, when superinfected, initially supported transcription from th
55 n which relatively high titers of virus were superinfected into the eyes of latently infected rabbits
56  The Nun protein of coliphage HK022 excludes superinfecting lambda phage.
57 RNA in the mfd mutant titrates Nun, allowing superinfecting lambda to form plaques.
58 d and supported the efficient replication of superinfecting LDV-P.
59 ular inflammatory infiltrate in the lungs of superinfected mice compared to singly infected animals.
60                                      Because superinfected mice had little PrP-res just before the on
61                                       All 18 superinfected mice showed incubation times identical to
62 rtical pathology of FU was not detectable in superinfected mice.
63 n, which indicated that MCF13 MLV is able to superinfect mink cells.
64 quence of either a low initial MOI or a high superinfecting MOI.
65                            We show here that superinfecting Mycobacterium marinum traffic rapidly int
66 e structures, recent studies have shown that superinfecting Mycobacterium marinum traffic rapidly to
67                 The present study shows that superinfecting Mycobacterium tuberculosis and Mycobacter
68  HIV-1 intersubtype recombination in dual or superinfected patients.
69 CD4 levels, virus burden, and the ability to superinfect peripheral blood mononuclear cells in vitro
70                    Cytomegalovirus (CMV) can superinfect persistently infected hosts despite CMV-spec
71 cipient strain which represses expression of superinfecting phage genomes and minimizes the contribut
72 otein expressed from prophage HK022 excludes superinfecting phage lambda by arresting transcription o
73  a virulence factor but functions to exclude superinfecting phage, possibly by blocking the injection
74                                         When superinfecting poliovirus resistant to the antiviral com
75  or replicons, were encapsidated in trans by superinfecting polioviruses.
76                                          The superinfecting strain can replace the initial strain, be
77                               In contrast, a superinfecting strain of EIAV, EIAV(vMA-1c), utilizes tw
78     These data suggest that the ability of a superinfecting strain of HIV to overcome preexisting imm
79     In conclusion, the rapid overgrowth of a superinfecting strain of HIV-1 of the same subtype raise
80                        In 4 individuals, the superinfecting strain replaced the original strain.
81                           In both cases, the superinfecting strain was detected by molecular and sero
82 train-specific nested PCR confirmed that the superinfecting strain was not present until the 9 month
83 in association with reduced containment of a superinfecting strain.
84 intercompartment dynamics of the initial and superinfecting strains and recombinants derived from the
85           In 2 individuals, both initial and superinfecting strains continued to cocirculate.
86                                          The superinfecting strains were not detected as minority var
87 dogene repertoire would be distinct in these superinfecting strains, consistent with encoding differe
88 ive recombination in env between initial and superinfecting strains.
89 At various times after primary infection, we superinfected T cells in vitro by exposure to a genetica
90 ned if spirochetes from the two groups could superinfect the tick vector Ornithodoros hermsi.
91 infection can be extended and/or enhanced by superinfecting the cultures.
92 ally distinct A. marginale strain capable of superinfecting the mammalian host can subsequently be co
93                       In animals that became superinfected, there was a reduction in peak replication
94 n the generation of a virus that was able to superinfect these cells, presumably by the use of a nove
95 sidue CDR H3, and neutralized the virus that superinfected this individual 15 weeks after initial inf
96 eficiency virus (HIV) depends on whether the superinfecting transmission resembles primary infection,
97                                     Although superinfecting vaccinia virus bound to cells, infection
98 more infectious virions excluded hundreds of superinfecting vaccinia virus particles.
99                                            A superinfecting variant strain of EIAV, vMA-1c, did not r
100                                          The superinfecting variants did not appear to be inherently
101         In the present study, the ability to superinfect various mouse models by homologous wild-type
102 uch as ED cells that EIAV(vMA-1c) is able to superinfect, viral entry is pH independent and appears t
103  cellular machinery, induce the repulsion of superinfecting virions away toward uninfected cells.
104 table on-going viral replication of distinct superinfecting virus in the env coding region.
105 ene expression nor genome replication of the superinfecting virus occurred.
106 mine the specificity of the NAbs against the superinfecting virus, these variants were cloned from fi
107 virus but did confer relative control of the superinfecting virus.
108 atency III program of transcription from the superinfecting-virus genomes, failing to transition to l
109                                              Superinfecting viruses were typically not susceptible to
110 iviral activity that inhibits replication of superinfecting VSV.
111 cted with a plasmid encoding procaspase 3 or superinfected with a proapoptotic mutant virus lacking t
112                  When the infected mice were superinfected with a VlsE-deficient clone (DeltaVlsE) at
113 d subsequent rescue from the chromosome when superinfected with Ad and wild-type AAV.
114 otein fused to green fluorescent protein and superinfected with an HSV-1 mutant lacking the U(S)8-12
115     There were 47 examples of patients being superinfected with an unrelated strain.
116 duced hepatocellular carcinomas (HCCs), were superinfected with another WHV strain, WHVNY.
117    Procaspase-3 levels remained unaltered if superinfected with Bac-U(S)3 at 3 h after d120 mutant in
118 nt amounts of procaspase-3 remained in cells superinfected with Bac-Us3 at 9 h postinfection with d12
119 ptimal infectivity if the producer cells are superinfected with certain gammaretroviruses.
120 transmission settings, individuals are often superinfected with complex mixtures of genetically disti
121  occurring after seroconversion: 2 IDUs were superinfected with different HCV genotypes, and 3 were s
122        However, we recently detected animals superinfected with different strains of A. marginale and
123 ns show that HCV-infected individuals can be superinfected with different strains, and this event may
124 ency virus (HIV)-positive individuals can be superinfected with different virus strains.
125 ted with different HCV genotypes, and 3 were superinfected with divergent strains of the same genotyp
126 ted with woodchuck hepatitis virus (WHV) are superinfected with HDV, they produce HDV with a WHV enve
127 viruses expressing U(L)47 or U(S)11 and then superinfected with HSV-1 under conditions that blocked D
128 mature C. burnetii replication vacuoles were superinfected with L. pneumophila, and then the acidity,
129 ding of a chronically infected host becoming superinfected with MDR HIV-1 with subsequent formation o
130                                    (v) Cells superinfected with mutants lacking both methionine codon
131 ES individuals remained healthy and were not superinfected with other HIV-1 strains despite continued
132 re passaged onto fresh cells which were then superinfected with RSV.
133 ing and was also effective in healing wounds superinfected with S. aureus.
134 LB/cJ mice infected with influenza virus and superinfected with S. pneumoniae were treated with eithe
135 with sublethal inocula of Pseudomonas become superinfected with secondary bacterial strains.
136  nucleus and bound DNA in MHV-infected cells superinfected with SeV.
137 macentor andersoni ticks were fed on animals superinfected with the Anaplasma marginale subsp. centra
138 8+ T cell clearance of CD4+ T cells that are superinfected with the HIV-1 strain JR-CSF or infected w
139  first methionine codon of the U(L)3 ORF and superinfected with the U(L)3(-) mutant.
140  (M12) methionine codon of the U(L)3 ORF and superinfected with the U(L)3(-) mutant.
141                     Tsetse were successfully superinfected with their mutualistic facultative symbion
142                              Cells were then superinfected with VSV or mock superinfected.
143 fter surgery the WHV carrier woodchucks were superinfected with WHV-enveloped HDV (wHDV).
144  of the livers and HCCs collected from three superinfected woodchucks, and (iii) finding WHVNY DNA re

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