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1 l stimulation of the trigeminal ganglion and superior sagittal sinus.
2 ring and firing evoked by stimulation of the superior sagittal sinus.
3 ning along the borders of the transverse and superior sagittal sinuses.
4 el of craniovascular pain by stimulating the superior sagittal sinus and monitoring trigeminal neuron
5 dural venous sinus thrombosis involving the superior sagittal sinus and right transverse-sigmoid sin
6 ualization with color Doppler imaging of the superior sagittal sinus and transverse sinuses through t
7 ted cell firing evoked by stimulation of the superior sagittal sinus as well as the background activi
9 ts with a JCH except 1 had thrombosis of the superior sagittal sinus, compared to 49% of patients wit
10 il, resulted in a dose-dependent recovery of superior sagittal sinus evoked trigeminocervical nucleus
11 , resulted in a dose-dependent inhibition of superior sagittal sinus evoked trigeminocervical nucleus
12 cat after (i) sham, (ii) stimulation of the superior sagittal sinus, (iii) stimulation of the superi
15 e PO2 and on venous oxygen saturation of the superior sagittal sinus (n = 5 rats per group); and 3) t
16 cell firing, elicited by stimulation of the superior sagittal sinus or by microiontophoretic applica
17 s that respond to noxious stimulation of the superior sagittal sinus or to ventrolateral PAG stimulat
20 hypothesized that trigeminal input from the superior sagittal sinus (SSS) can excite C1-C2 spinothal
27 , among the skull fractures extending to the superior sagittal sinus, those of the occipital bone had
28 5 95% CI=1.58 to 84.46, p=0.01) and isolated superior sagittal sinus thrombosis (HR=0.39, 95% CI=0.14
29 sue PO2, and venous oxygen saturation of the superior sagittal sinus values concomitant with diffuse
34 ior sagittal sinus, (iii) stimulation of the superior sagittal sinus with PAG stimulation, or (iv) st
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