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2 dea of using the Schottky contact to achieve supersensitive and fast response nanowire-based nanosens
3 ng the NR1 subunit of the NMDA receptor show supersensitive behavioral responses to kainic acid and t
4 triatal monoamine levels and no evidence for supersensitive D1, D3, or D4 dopamine receptors in the D
5 vation of the nigrostriatal pathway and that supersensitive dopamine D(1) receptors may mediate the e
7 signal transduction mechanisms to produce a supersensitive form of D1- mediated neuronal plasticity.
8 cetylcholine receptor subunits or expressing supersensitive forms of these subunits have begun to tie
11 amine (DA) D1 receptors is the hallmark of a supersensitive molecular response associated with dyskin
13 ine system, direct pathway neurons display a supersensitive response to D1 dopamine receptor agonists
14 nisms underlying the irreversibility of this supersensitive response with long-term dopamine agonist
17 iral therapy and followed intensively with a supersensitive reverse transcriptase PCR assay with a lo
19 tal cortex of PKCepsilon null mice were also supersensitive to allosteric activation by ethanol and f
21 as serine (S)563, phosphorylation at S603 is supersensitive to calcineurin-mediated dephosphorylation
22 negative alleles of cdr1, which render cells supersensitive to Chk1 levels, and suppress the checkpoi
24 ies have suggested that bipolar patients are supersensitive to light suppression of melatonin and tha
27 ggest that the juvenile prefrontal cortex is supersensitive to methylphenidate, and the accepted ther
31 ing protein kinase Cepsilon (PKCepsilon) are supersensitive to positive allosteric modulators of gamm
34 data suggest that neurons in the CPu become supersensitive to the effects of DA agonists after 6-OHD
35 chronic morphine-treated cells which become supersensitive to the excitatory effects of mu-, delta-
36 Here we show that GRK6-deficient mice are supersensitive to the locomotor-stimulating effect of ps
37 n ribonucleotide reductase; rnr4 mutants are supersensitive to the ribonucleotide reductase inhibitor
38 wth of the strain with the pst1-1 allele was supersensitive to the specific histone deacetylase inhib
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