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1          Further, D1-dopamine receptors were supersensitive; adenylate cyclase activity, locomotor ac
2 dea of using the Schottky contact to achieve supersensitive and fast response nanowire-based nanosens
3 ng the NR1 subunit of the NMDA receptor show supersensitive behavioral responses to kainic acid and t
4 triatal monoamine levels and no evidence for supersensitive D1, D3, or D4 dopamine receptors in the D
5 vation of the nigrostriatal pathway and that supersensitive dopamine D(1) receptors may mediate the e
6  phenotypic anomalies seen in the interferon supersensitive Down Syndrome patient.
7  signal transduction mechanisms to produce a supersensitive form of D1- mediated neuronal plasticity.
8 cetylcholine receptor subunits or expressing supersensitive forms of these subunits have begun to tie
9                                 In step 2, a supersensitive fraction of the cancer cells, including s
10 e dopamine-depleted striatum, as well as the supersensitive induction of IEGs.
11 amine (DA) D1 receptors is the hallmark of a supersensitive molecular response associated with dyskin
12  suffered from inefficient, delayed and EGTA-supersensitive release.
13 ine system, direct pathway neurons display a supersensitive response to D1 dopamine receptor agonists
14 nisms underlying the irreversibility of this supersensitive response with long-term dopamine agonist
15 nd depletion of tissue norepinephrine and by supersensitive responses to norepinephrine.
16     However, following dopamine depletion, a supersensitive responsiveness of D1 striatal neurons to
17 iral therapy and followed intensively with a supersensitive reverse transcriptase PCR assay with a lo
18                        The Arabidopsis sad1 (supersensitive to ABA and drought) mutation increases pl
19 tal cortex of PKCepsilon null mice were also supersensitive to allosteric activation by ethanol and f
20                           Yet, they are also supersensitive to antidepressants and show enhanced sens
21 as serine (S)563, phosphorylation at S603 is supersensitive to calcineurin-mediated dephosphorylation
22 negative alleles of cdr1, which render cells supersensitive to Chk1 levels, and suppress the checkpoi
23 5- to 7-fold, suggesting that DA-/- mice are supersensitive to DA.
24 ies have suggested that bipolar patients are supersensitive to light suppression of melatonin and tha
25            Furthermore, DKO mice are acutely supersensitive to low doses of cocaine and fail to displ
26      Second, matings to an MAT alpha partner supersensitive to mating pheromone (sst2 delta) suppress
27 ggest that the juvenile prefrontal cortex is supersensitive to methylphenidate, and the accepted ther
28                      These neurons were also supersensitive to neurotoxin.
29  of PKA, whereas the glutamic acid mutant is supersensitive to overexpression of PKA.
30 sidues is not phosphorylated, the protein is supersensitive to phosphatase action.
31 ing protein kinase Cepsilon (PKCepsilon) are supersensitive to positive allosteric modulators of gamm
32             The A281T and T282A mutants were supersensitive to S. cerevisiae alpha-factor, but were d
33                        cdc1 mutant cells are supersensitive to the DNA synthesis inhibitor hydroxyure
34  data suggest that neurons in the CPu become supersensitive to the effects of DA agonists after 6-OHD
35  chronic morphine-treated cells which become supersensitive to the excitatory effects of mu-, delta-
36    Here we show that GRK6-deficient mice are supersensitive to the locomotor-stimulating effect of ps
37 n ribonucleotide reductase; rnr4 mutants are supersensitive to the ribonucleotide reductase inhibitor
38 wth of the strain with the pst1-1 allele was supersensitive to the specific histone deacetylase inhib

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