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1 Con A hepatitis, suggesting inhibition of a suppressor cell.
2 d granulocytic and monocytic myeloid-derived suppressor cells.
3 on of T regulatory cells and myeloid-derived suppressor cells.
4 ne suppressive activities of myeloid-derived suppressor cells.
5 lls, regulatory T cells, and myeloid-derived suppressor cells.
6 severely depressed numbers of lung CD8(+) T suppressor cells.
7 aematopoietic progenitor and myeloid-derived suppressor cells.
8 to induce the generation of CD8(+)CD28(-) T suppressor cells.
9 r-associated macrophages and myeloid-derived suppressor cells.
10 y exhibit characteristics of myeloid-derived suppressor cells.
11 velopment, survival, and function of myeloid suppressor cells.
12 ncer: regulatory T cells and myeloid-derived suppressor cells.
13 -DC, DC-10, and PGE2-induced myeloid-derived suppressor cells.
14 ressed on tumor infiltrating myeloid-derived suppressor cells.
15 (+) macrophages and Ly-6C(+) myeloid-derived suppressor cells.
16 gest that these cells may be myeloid derived suppressor cells.
17 effects by the elimination of CD38(+) immune-suppressor cells.
18 ored more M2 macrophages and myeloid-derived suppressor cells.
19 associated with expansion of myeloid-derived suppressor cells.
21 owever, in vivo depletion of myeloid-derived suppressor cells abrogated both Treg expansion and prote
22 ith cancer and in animal tumor models, these suppressor cells accumulate in the tumor microenvironmen
24 enograft assays to establish that the tumour suppressor Cell adhesion molecule 1 (CADM1) inhibits SqC
25 Depletion of neutrophils/myeloid-derived suppressor cells also suppressed metastasis suggesting a
26 ng the presence of monocytic myeloid-derived suppressor cells and increasing the presence of activate
27 acterized by an abundance of myeloid-derived suppressor cells and inhibition of cytotoxic T-cell infi
28 ecruitment of both monocytic myeloid-derived suppressor cells and macrophages, thereby promoting meta
29 by cell populations such as myeloid-derived suppressor cells and regulatory T cells, as well as immu
30 nt, including development of myeloid-derived suppressor cells and regulatory T cells; therefore, thes
34 withstanding the presence of myeloid-derived suppressor cells, and CD8(+) dendritic cells cross-prese
36 +) T cells and CD11(+)Gr1(+) myeloid-derived suppressor cells, and changes in the chemokine/cytokine
39 cells, type II macrophages, myeloid-derived suppressor cells, and other immunosuppressive cells that
40 macrophages and granulocytic myeloid-derived suppressor cells, and protumor T-regulatory/Th17 cell re
41 es in regulatory T cells and myeloid-derived suppressor cells, and the leukemic cells themselves were
42 matopoietic stem cell niche, myeloid-derived suppressor cells, and the sympathetic nervous system.
44 honuclear cells/granulocytic myeloid-derived suppressor cells are due to the loss of CD44 upon enzyma
46 e deficits through compensatory trans-acting suppressors, cells are penalized in the long term by cha
47 r-associated macrophages and myeloid-derived suppressor cells, are abundant in the HCC microenvironme
48 xpansion of Gr-1(+) CD11b(+) myeloid-derived suppressor cells, as well as elevated levels of immune a
49 tion of CD11b+Gr1intF4/80int myeloid-derived suppressor cells at the resection margin and increased t
51 xpands a heterogeneous population of myeloid suppressor cells capable of inhibiting T cell function.
52 in cargo of exosomes shed by myeloid-derived suppressor cells collected under high and low conditions
53 ted increased recruitment of myeloid-derived suppressor cells, consistent with protection of the epit
54 uding regulatory T cells and myeloid-derived suppressor cells, correlating with an increased therapeu
56 ater numbers of macrophages, myeloid-derived suppressor cells, dendritic cells, and granulocytes than
57 indicate that denileukin diftitox and other suppressor cell-depleting therapies may be useful adjunc
58 f phenformin on granulocytic myeloid-derived suppressor cell-driven immune suppression and support th
60 s document the generation of myeloid-derived suppressor cells during TB, suggesting their role in TB
61 cell (DC) differentiation to myeloid-derived suppressor cell expansion during tumour progression.
62 d to the finding that alloantigen-specific T suppressor cells express IL-2 receptor (CD25) and that I
63 rogenic dendritic cells, and myeloid-derived suppressor cells, for the induction of transplantation t
64 lly and functionally, to the myeloid-derived suppressor cells found in cancer because they exerted a
65 henotypically similar to the myeloid-derived suppressor cells found in patients with cancer, have rec
67 suppressor macrophages, and myeloid-derived suppressor cells, generated in vitro from the same mouse
68 reg), Th17, and granulocytic myeloid-derived suppressor cells (gMDSC) were increased significantly in
71 s, the phenotype typical for myeloid-derived suppressor cells in cancer or immature myeloid cells (IM
72 und they differentiated into myeloid-derived suppressor cells in early metastatic sites of tumor-bear
74 tively inhibits granulocytic myeloid-derived suppressor cells in spleens of tumor-bearing mice and ex
76 only reduced tumor-promoting myeloid-derived suppressor cells in the lung, but also down-regulated th
77 ficiency, which also lessens myeloid-derived suppressor cells in the premetastatic niche, synergized
80 ines, including TGF-beta, by myeloid-derived suppressor cells in tumor-draining lymph nodes, leading
81 nulocytic (gMDSC) subsets of myeloid-derived suppressor cells infiltrate in the primary tumour and di
82 ell killing while decreasing myeloid-derived suppressor cell infiltration and IL10 production in the
83 splenic proliferation of bone marrow-derived suppressor cells, inhibiting the adaptive immune respons
84 the suppressive function of myeloid-derived suppressor cells, inhibits the release of IL-12 p70 by m
85 ta drives differentiation of myeloid-derived suppressor cells into protumorigenic terminally differen
86 aling mediates maturation of myeloid-derived suppressor cells into TDMMCs with high levels of cell su
87 erodimer ligands can recruit myeloid-derived suppressor cells into the skin, countering rather than p
88 othelial, haematopoietic and myeloid-derived suppressor cells is sufficient to cause regression of tu
89 y a balanced distribution of myeloid-derived suppressor cell-like and APC-like myeloid phenotypes and
92 e monocytic and granulocytic myeloid-derived suppressor cells (M- and G-MDSCs) defined by their abili
94 trophils, T cells, monocytic myeloid-derived suppressor cells (M-MDSCs), and group 2 innate lymphoid
95 myeloid cells expressing the myeloid-derived suppressor cell marker S100A9 only in a TN breast cancer
97 ociated macrophage (TAM) and myeloid-derived suppressor cell (MDSC) infiltration in tumors via chemok
98 tomach, where they exhibited myeloid-derived suppressor cell (MDSC) markers and acquired the ability
99 s) and (ii) CD45(+) CD34(-) [myeloid-derived suppressor cell (MDSC)-like fibrocytes] cells in stable
100 c cells were driven toward a myeloid-derived suppressor cell (MDSC)-like phenotype by the GRO chemoki
101 d medium differentiated into myeloid derived suppressor cells (MDSC) (CD33(+)CD11b(+)HLA-DR(-/low)).
102 umors exhibit an increase in myeloid-derived suppressor cells (MDSC) and a decrease in T cells, indic
103 ve an increased frequency of myeloid derived suppressor cells (MDSC) and are at increased risk for ca
104 umulation of lung-associated myeloid-derived suppressor cells (MDSC) and MDSC-derived production of T
117 cells allowed generation of myeloid-derived suppressor cells (MDSC) from precursors in mouse bone ma
120 (CD11b(+)Ly6G(-)Ly6C(high)) myeloid-derived suppressor cells (MDSC) in chronically infected mice, wh
121 nction of tumor-infiltrating myeloid-derived suppressor cells (MDSC) in ganglioside-deficient tumors,
123 st, it reduced the levels of myeloid-derived suppressor cells (MDSC) in mice bearing Lewis lung carci
124 induction of CD11b(+)Gr-1(+) myeloid-derived suppressor cells (MDSC) in the peritoneum, which express
125 sociated with restoration of myeloid-derived suppressor cells (MDSC) in the tumor microenvironment, i
126 y, we show the importance of myeloid-derived suppressor cells (MDSC) in this process at bone metastat
127 cy may be the recruitment of myeloid-derived suppressor cells (MDSC) into the tumor microenvironment.
130 sue recruited CXCR2-positive myeloid-derived suppressor cells (MDSC) to form a premetastatic niche th
131 d CD4(+) helper T cells, and myeloid-derived suppressor cells (MDSC) to gain concurrent views on the
132 mobilization of granulocytic myeloid-derived suppressor cells (MDSC) to the breast cancer lung metast
133 gulatory T cells (Treg), and myeloid-derived suppressor cells (MDSC) were profiled by flow cytometry.
134 clitaxel to tumor-associated myeloid-derived suppressor cells (MDSC), (ii) MPO-regulated release, and
135 etic progenitor cells (HPC), myeloid-derived suppressor cells (MDSC), and dendritic cells is directly
136 T regulatory cells (Tregs), myeloid-derived suppressor cells (MDSC), and exhausted T effector cells
137 mune suppression mediated by myeloid-derived suppressor cells (MDSC), as derived from cytokine induct
138 increases in CD11b(+)Gr-1(+) myeloid-derived suppressor cells (MDSC), FoxP3(+) regulatory T cells, an
139 ory T lymphocytes (Treg) and myeloid-derived suppressor cells (MDSC), major components of these inhib
140 M), and CD11b(+)Gr-1 (LY6G)+ myeloid-derived suppressor cells (MDSC), respond to cancer-related stres
141 ated with an accumulation of myeloid-derived suppressor cells (MDSC), the numbers of which are reduce
142 ave previously reported that myeloid-derived suppressor cells (MDSC), which are a heterogeneous popul
145 ry T cells (Treg) and innate myeloid-derived suppressor cells (MDSC), which facilitate immune escape
158 d to induce the expansion of myeloid-derived suppressor cells (MDSC); however, little is known regard
160 ncreased in myeloid cells as myeloid-derived suppressor cells (MDSCs) accumulated, which was associat
161 ingly, a subset of monocytic myeloid-derived suppressor cells (MDSCs) also expressed CD2 and CREMalph
162 Recruitment of monocytic myeloid-derived suppressor cells (MDSCs) and differentiation of tumor-as
165 latory mechanism mediated by myeloid-derived suppressor cells (MDSCs) and showed that MDSCs expanded
166 as phenotypically similar to myeloid-derived suppressor cells (MDSCs) and that suppressed T cell resp
167 ion of myeloid cells such as myeloid-derived suppressor cells (MDSCs) and tumor-associated macrophage
169 rating myeloid cells such as myeloid-derived suppressor cells (MDSCs) and tumor-associated macrophage
175 cancer metastasis, in which myeloid-derived suppressor cells (MDSCs) are an important participant.
187 uction, with CD11b(+)Gr-1(+) myeloid-derived suppressor cells (MDSCs) as the most expanded population
188 ntiation and accumulation of myeloid-derived suppressor cells (MDSCs) both in vitro and in vivo.
193 The systemic accumulation of myeloid-derived suppressor cells (MDSCs) decreased significantly in flox
202 understanding of the role of myeloid-derived suppressor cells (MDSCs) in cancer is becoming increasin
203 n immune-suppressive role of myeloid-derived suppressor cells (MDSCs) in melanoma has long been specu
204 iency causes infiltration of myeloid-derived suppressor cells (MDSCs) in multiple organs and subseque
205 ed iNKT cells cooperate with myeloid-derived suppressor cells (MDSCs) in protecting mice against the
206 entified a critical role for myeloid-derived suppressor cells (MDSCs) in S. aureus biofilm persistenc
208 t al show the involvement of myeloid-derived suppressor cells (MDSCs) in the pathogenesis of immune t
209 ion of the immunosuppressive myeloid-derived suppressor cells (MDSCs) in the spleen by alpha-GalCer t
210 es to understand the role of myeloid-derived suppressor cells (MDSCs) in the tumor microenvironment a
211 greater absolute numbers of myeloid-derived suppressor cells (MDSCs) in tm24KO mice and showed that
212 dentify increased numbers of myeloid-derived suppressor cells (MDSCs) in untreated patients with chro
215 immune-suppressive activity, myeloid-derived suppressor cells (MDSCs) influence tumor progression in
217 h inhibiting infiltration of myeloid-derived suppressor cells (MDSCs) into colonic mucosa and tumors
219 cells, accumulation of donor myeloid-derived suppressor cells (MDSCs) mediated by ILC2 production of
220 xpansion of Gr-1(+) CD11b(+) myeloid-derived suppressor cells (MDSCs) occurring intraprostatically im
225 ations produced by patients' myeloid-derived suppressor cells (MDSCs) support the development of RORg
226 lymphocyte antigen 6G (Ly6G) myeloid-derived suppressor cells (MDSCs) that caused myeloproliferative
227 of cells with similarity to myeloid-derived suppressor cells (MDSCs) that may have suppressed vaccin
228 of immunosuppressive CD33(+) myeloid-derived suppressor cells (MDSCs) that negatively correlated with
229 vestigated the potential for myeloid-derived suppressor cells (MDSCs) to suppress T cell-mediated imm
230 1(high)Ly-6C(+) granulocytic myeloid-derived suppressor cells (MDSCs) to the liver of HLA-DR4 transge
231 DCs and Gr-1(high)CD11c(neg) myeloid-derived suppressor cells (MDSCs) were enriched in GVHD target or
233 AR-deficient mice, monocytic myeloid-derived suppressor cells (MDSCs) were recruited to the liver.
234 restingly, immunosuppressive myeloid-derived suppressor cells (MDSCs) were recruited to tumor tissue
235 al by cotransplantation with myeloid-derived suppressor cells (MDSCs) without requirement of immunosu
236 were increased, notably the myeloid-derived suppressor cells (MDSCs), a CD11b(+) subset characterize
237 e paclitaxel on functions of myeloid-derived suppressor cells (MDSCs), chronic inflammatory mediators
238 G-F4/80(low)) with monocytic myeloid-derived suppressor cells (MDSCs), had similar morphology, and su
239 ovel subset of innate cells, myeloid-derived suppressor cells (MDSCs), has been described in cancer,
240 nally resemble tumor-induced myeloid-derived suppressor cells (MDSCs), indicating an essential role o
242 Two major populations of myeloid-derived suppressor cells (MDSCs), monocytic MDSCs (M-MDSCs) and
243 immune-inhibitory responses (myeloid-derived suppressor cells (MDSCs), regulatory T cells (Tregs), an
244 and massive accumulation of myeloid-derived suppressor cells (MDSCs), which actively suppressed anti
245 tinct subset of neutrophilic myeloid-derived suppressor cells (MDSCs), which functionally suppress T
246 lation of tumor-infiltrating myeloid-derived suppressor cells (MDSCs), which suppress T cells and alt
261 trated from the circulation (myeloid-derived suppressor cells [MDSCs], monocyte-derived macrophages [
262 8.35; P = .01) and monocytic myeloid derived suppressor cells (mMDSC) (95% CI, 3.62-12.74; P = .001)
264 riven expansion of monocytic myeloid-derived suppressor cells (mMDSC) from human or mouse myeloid pro
265 creased numbers of monocytic myeloid-derived suppressor cells (Mo-MDSC) in psoriasis patients and exa
266 4(+) HLA-DR(-/low) monocytic myeloid-derived suppressor cells (Mo-MDSCs) have been shown to suppress
269 The increase in NK cell and myeloid-derived suppressor cell numbers was associated with increased bo
270 their murine counterparts, are highly potent suppressor cells of both CD4(+) and CD8(+) T cell respon
271 to generate ex vivo a subpopulation of human suppressor cells of monocytic origin, referred to as hum
273 umor-associated macrophages, myeloid-derived suppressor cells, or inflammatory monocytes, functions o
274 ng combined approaches to deplete endogenous suppressor cell populations that can also expand effecto
275 secreted tissue signals and myeloid-derived suppressor cell populations were altered in surgically s
278 ammaR-dependent mechanism regulates CD8(+) T suppressor cell recruitment, limits immunopathogenesis,
279 high levels of granulocytic myeloid-derived suppressor cells resulted in loss of immunotherapeutic p
280 s, we identified a CD14/CD24 myeloid-derived suppressor cell subset with the capability of killing ac
281 cell death pathways, we found the monocytic suppressor-cell subset, but not the granulocytic subset,
285 ise to highly activated short-lived Klrg1(+) suppressor cells to optimize immune regulation and maint
286 ctional plasticity and can be converted from suppressor cells to pathogenic effector cells, enhancing
287 ulatory dendritic cells, and myeloid-derived suppressor cells to regulate alloimmunity, their potenti
289 unosuppressive cells such as myeloid-derived suppressor cells, tolerogenic monocytes, and T regulator
290 er, significant expansion of myeloid-derived suppressor cells was detected in the recipients of CD47-
293 ns of T-regulatory cells and myeloid-derived suppressor cells were observed in both rejection and tol
294 ygen species in granulocytic myeloid-derived suppressor cells, whereas the antioxidant N-acetylcystei
295 oration and were enriched in myeloid-derived suppressor cells which could be responsible for immunosu
297 r-associated macrophages and myeloid-derived suppressor cells, which not only mediate immune suppress
298 aling responses in monocytic myeloid-derived suppressor cells, which was paired with their pronounced
299 effector T cells, Tregs, and myeloid-derived suppressor cells, while effector T cells expressed more
300 revealed a subpopulation of myeloid-derived suppressor cells within the CD11b(+) Gr-1(high) cell fra
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