ライフサイエンスコーパス: supragingival

1 oral cavity and a significant constituent of supragingival and subgingival dental plaque in children

2 Here, saliva, supragingival and subgingival plaque samples from period

3 , supports the viewpoint of dysbiosis of the supragingival biofilms.

4 bjects with > or =10% versus <10% sites with supragingival calculus (OR = 3.6).

5 P <0.0001), gingival bleeding (P <0.05), and supragingival calculus (P <0.0001) than normal subjects.

6 ental prophylaxis, which includes removal of supragingival calculus and plaque, has been shown to arr

7 There is evidence that supragingival calculus contains unmineralized channels a

8 stain, were found within cavities/lacunae in supragingival calculus cryosections.

9 n probing demonstrated more LCAL and PD, and supragingival calculus had an apparently protective effe

10 In contrast to the outcomes LCAL/PD, supragingival calculus had no significant protective eff

11 Supragingival calculus harvested from patients with mode

12 ay be important, since incomplete removal of supragingival calculus may expose these reservoirs of po

13 rcentage of sites with gingival bleeding and supragingival calculus only and subgingival calculus wit

14 The percentage of sites with supragingival calculus was not different between the gro

15 ble model, cigarette smoking and presence of supragingival calculus were the factors most significant

16 , bleeding on probing (BOP), visible plaque, supragingival calculus, and mean tooth loss.

17 and anaerobic bacteria may be present within supragingival calculus, specifically within the internal

18 g and root planing (SRP) in patients free of supragingival calculus, the chip was placed in target si

19 and the lowest percentage of sites with only supragingival calculus.

20 nly and subgingival calculus with or without supragingival calculus.

21 caling and root planing, whereas CG received supragingival cleaning at baseline and scaling and root

22 clinically at 6-month intervals followed by supragingival cleaning.

23 gienist-delivered full mouth subgingival and supragingival debridement with a host-modulating agent,

24 ignificantly associated with a high level of supragingival dental calculus and cigarette smoking.

25 plaque index (VPI), marginal bleeding index, supragingival dental calculus, probing depth (PD), clini

26 The microbiomes of supragingival dental plaque differ substantially among t

27 Supragingival dental plaque was collected from tooth sur

28 , multigenus consortium in the microbiome of supragingival dental plaque.

29 nt of the multispecies oral biofilm known as supragingival dental plaque; they grow by fermentation o

30 to analyze the microbiomes of site-specific supragingival dental plaques from children with differen

31 rmine whether there was any adverse shift in supragingival flora.

32 ce: 25.6%) was significantly associated with supragingival plaque (OR = 1.74; 95% CI: 1.22 to 2.50) a

33 CCBs and the widespread presence of abundant supragingival plaque (PI > or =2 on >40% of tooth surfac

34 , presence of bleeding on probing (BOP), and supragingival plaque (PL) were assessed at six sites aro

35 ing on probing (BOP), suppuration (SUP), and supragingival plaque (PL).

36 nt level, dichotomous presence or absence of supragingival plaque accumulation, and bleeding on probi

37 Supragingival plaque and calculus indices, salivary flow

38 The presence or absence of supragingival plaque and clinical attachment loss (CAL)

39 , probing depth (PD), gingival bleeding, and supragingival plaque and measures to define MetS using N

40 On real supragingival plaque and stool MG datasets that were gen

41 An in vitro model of supragingival plaque associated with gingivitis was char

42 gingival bleeding at >/=50% of sites; and 4) supragingival plaque at >/=50% of sites.

43 ed primarily by disruption of the contiguous supragingival plaque by the mouthrinse.

44 ies have shown that the nature and amount of supragingival plaque can influence the composition of th

45 Because studies have shown that control of supragingival plaque can influence the onset and/or prog

46 epth and attachment loss) in both saliva and supragingival plaque habitats.

47 Supragingival plaque had no significant effect on cLCAL/

48 This study evaluates the role of supragingival plaque level on the relationship between s

49 croorganisms with CAL changes in relation to supragingival plaque levels in older adult women.

50 At low supragingival plaque levels, only the presence of Pg was

51 At high supragingival plaque levels, the presence of Tf (OR: 2.4

52 ly associated with CAL at either low or high supragingival plaque levels.

53 er mercaptoalkylguanidine appeared to affect supragingival plaque levels.

54 ition and caries phenotypes, we profiled the supragingival plaque microbiome of 485 dizygotic and mon

55 ween baseline and other time points for both supragingival plaque microbiota structure and salivary m

56 ers attenuated observed associations, though supragingival plaque remained significant (OR = 1.47; 95

57 An association between MetS and supragingival plaque requires further investigation.

58 Supragingival plaque samples were taken from 55 dentate

59 etected in 6 of 20, 1 of 20, and 11 of 20 of supragingival plaque samples, respectively, and 4 of 20,

60 ility, we employed a computational model for supragingival plaque to systematically sample combinatio

61 ingival inflammation, bleeding tendency, and supragingival plaque were clinically measured at baselin

62 odontitis subjects had a higher frequency of supragingival plaque without increasing gingival inflamm

63 itis patients, including explorer-detectable supragingival plaque, bleeding on probing (BOP) and rela

64 iodontal status included probing depth (PD), supragingival plaque, gingival bleeding on probing, and

65 Clinical assessment of supragingival plaque, gingival bleeding, subgingival cal

66 hifts in sugar transporter types between the supragingival plaque, other oral surfaces, and stool; hy

67 oral cavity by interacting with organisms in supragingival plaque, such as the oralis group of oral s

68 ffect of calcium channel blockers (CCBs) and supragingival plaque.

69 requency in subgingival plaque as opposed to supragingival plaque.

70 gens that may be harbored in subgingival and supragingival plaque.

71 aque reflected contributions from saliva and supragingival plaque.

72 ion, cigarette smoking, body mass index, and supragingival plaque.

73 Neither target species was detected in supragingival plaque; A. actinomycetemcomitans was detec

74 g and root planing and control subjects with supragingival prophylaxis.

75 Thus, in the supragingival regime, SPO and MPO work in unison for the

76 on 24 subjects for saliva, subgingival, and supragingival samples.

77 d as an adjunct to a maintenance schedule of supragingival scaling and dental prophylaxis.

78 ) that received plaque control instructions, supragingival scaling, and two placebos.

79 lysis of plaque samples from subgingival and supragingival sites in all diseases categories for react

80 ion of the oral cavity is likely to occur on supragingival surfaces that already support robust biofi

81 After initial supragingival therapy, patients were treated by OSFMUD.

82 e formation of the salivary pellicle coating supragingival tooth surfaces.